Search results for "interleukin-1"

showing 10 items of 660 documents

Human CD4+CD25+ T cells derived from the majority of atopic donors are able to suppress TH1 and TH2 cytokine production

2003

Abstract Background: Recently, it has been established that CD4 + CD25 + T cells with regulatory capacity are present in human peripheral blood, inhibiting allogeneic proliferation and cytokine production of preactivated CD4 + CD25 − respond-er T cells. Objective: The aim of this study was to analyze in an allergen-specific setting whether such regulatory CD4 + CD25 + T cells also exist and function normally in atopic individuals, especially concerning the inhibition of T H 2 cytokines. Methods: For this purpose, CD4 + CD25 − or CD4 + CD25 + T cells from donors allergic to grass or birch pollen (mainly with rhinitis) or from healthy nonatopic donors were stimulated in the presence of autolo…

Immunoconjugatesmedicine.medical_treatmentImmunologychemical and pharmacologic phenomenaBiologyLymphocyte ActivationImmunophenotypingAbataceptInterleukin 21Th2 CellsAntigenAntigens CDTransforming Growth Factor betaHypersensitivitymedicineHumansImmunology and AllergyCytotoxic T cellCTLA-4 AntigenIL-2 receptorGrowth factorReceptors Interleukin-2hemic and immune systemsT lymphocyteDendritic cellTh1 CellsAntigens DifferentiationInterleukin-10CytokineCD4 AntigensImmunologyCytokinesJournal of Allergy and Clinical Immunology
researchProduct

Cytoskeletal stabilization of inhibitory interactions in immunologic synapses of mature human dendritic cells with natural killer cells

2011

Abstract Human mature dendritic cells (DCs) can efficiently stimulate natural killer (NK)–cell responses without being targeted by their cytotoxicity. To understand this important regulatory crosstalk, we characterized the development of the immunologic synapse between mature DCs and resting NK cells. Conjugates between these 2 innate leukocyte populations formed rapidly, persisted for prolonged time periods and matured with DC-derived f-actin polymerization at the synapse. Polarization of IL-12 and IL-12R to the synapse coincided with f-actin polymerization, while other activating and inhibitory molecules were enriched at the interface between DCs and NK cells earlier. Functional assays re…

Immunological SynapsesImmunologyCell Communicationmacromolecular substancesBiochemistryImmunological synapseNatural killer cell03 medical and health sciences0302 clinical medicineInterleukin-15 Receptor alpha SubunitMicroscopy Electron TransmissionReceptors KIRMHC class ImedicineHumansAntigen-presenting cellCells CulturedCytoskeleton030304 developmental biology0303 health sciencesMicroscopy ConfocalbiologyReceptors Interleukin-12Dendritic CellsCell BiologyHematologyDendritic cellFlow CytometryInterleukin-12Immunological SynapsesActinsCoculture Techniques3. Good healthCell biologyKiller Cells Naturalmedicine.anatomical_structureMicroscopy Fluorescencebiology.proteinInterleukin 12RNA InterferenceK562 CellsMicrotubule-Organizing CenterWiskott-Aldrich Syndrome Protein030215 immunologyK562 cellsBlood
researchProduct

Humoral immune response in IL-12 and IFN-gamma deficient mice after infection with Cryptosporidium parvum.

2008

Infection with Cryptosporidium spp. causes diarrhoeal disease and has become an important medical and veterinary problem especially in the immunocompromised host. The importance of the adaptive immune response, with CD4+ T-lymphocytes being the major players, has been clearly demonstrated. The requirement of IL-12 and IFN-gamma identifies this response as a Th1-dominated reaction. IFN-gamma is also important in the early phase of the host-parasite interaction. We analysed the outcome of infection in IL-12p40 (IL-12KO) and IFN-gamma (GKO) deficient C57BL/6 mice after primary and secondary challenge with the parasite and, for the first time, we demonstrate the resulting Ig response in sera an…

ImmunologyAntibodies ProtozoanCryptosporidiosisMicrobiologyFecesInterferon-gammaMiceImmune systemIleumParasite Egg CountParasite hostingAnimalsParasite Egg CountCryptosporidium parvumMice KnockoutbiologyCryptosporidiumAcquired immune systembiology.organism_classificationInterleukin-12Immunoglobulin AMice Inbred C57BLCryptosporidium parvumImmunoglobulin GImmunologyVaginaInterleukin 12biology.proteinVaginal DouchingParasitologyFemaleAntibodyParasite immunology
researchProduct

Costimulatory signalling potential of murine MHC class II‐positive T‐clone cells

1996

Activated human and rat T cells as well as mouse T-cell clones have been reported to synthesize and express major histocompatibility complex (MHC) class II molecules. However, the capacity of class II+ antigen (Ag) presenting T cells to induce proliferation of Ag-specific cloned T cells has been controversial. We analysed whether the failure of some T-cell clones to proliferate in response to Ag presented by class II+ T cells is because of a lack of costimulatory cytokine production by the antigen-presenting cells (APC). As a model system the mouse class II+ cloned BI/O4.1 T cells were used as APC in order to activate the T cell clone KIII5. This T-helper 1 (Th1) type, GAT (synthetic copoly…

ImmunologyAntigen presentationCD1Antigen-Presenting CellsPolymerase Chain ReactionCell LineMiceInterleukin 21T-Lymphocyte SubsetsAnimalsImmunology and AllergyCytotoxic T cellIL-2 receptorAntigen-presenting cellMice Inbred C3HMHC class IICD40biologyHistocompatibility Antigens Class IIReceptors Interleukin-2Th1 CellsInterleukin-12Molecular biologyMice Inbred C57BLbiology.proteinInterleukin-2Cell DivisionSpleenSignal TransductionResearch ArticleImmunology
researchProduct

Cyclic adenosine monophosphate and IL-10 coordinately contribute to nTreg cell-mediated suppression of dendritic cell activation

2010

In humans and mice naturally occurring regulatory T cells (nTregs) are crucial for the maintenance of peripheral tolerance by controlling not only potentially autoreactive T cells but virtually all cells of the adaptive and innate immune system. Here we show that co-culture of murine dendritic cells (DC) and nTregs results in an immediate increase of cAMP in DC, responsible for a rapid down-regulation of co-stimulatory molecules (CD80, CD86). In addition, the inhibitory surface molecule B7-H3 on DC is up-regulated. Subsequently, nTreg-derived IL-10 inhibits the cytokine production (IL-6, IL-12) of suppressed DC therewith preserving their silent phenotype. Hence, our data indicate that nTreg…

ImmunologyDown-RegulationCell CommunicationBiologyT-Lymphocytes RegulatoryImmune toleranceMiceImmune systemCyclic AMPImmune ToleranceAnimalsCD86Innate immune systemInterleukin-6Peripheral toleranceDendritic CellsDendritic cellInterleukin-12Coculture TechniquesInterleukin-10Cell biologyInterleukin 10B7-1 AntigenB7-2 AntigenCD80Signal TransductionCellular Immunology
researchProduct

Specific and Redundant Roles for NFAT Transcription Factors in the Expression of Mast Cell-Derived Cytokines

2006

Abstract By virtue of their ability to express a plethora of biologically highly active mediators, mast cells (MC) are involved in both adaptive and innate immune responses. MC-derived Th2-type cytokines are thought to act as local amplifiers of Th2 reactions, including chronic inflammatory disorders such as allergic asthma, whereas MC-derived TNF-α is a critical initiator of antimicrobial defense. In this study, we demonstrate that the transcription factors NFATc1 and NFATc2 are part of a MC-specific signaling network that regulates the expression of TNF-α and IL-13, whereas NFATc3 is dispensable. Primary murine bone marrow-derived MC from NFATc2−/− mice, activated by either ionomycin or I…

ImmunologyDown-RegulationImmunoglobulin EMicechemistry.chemical_compoundTh2 CellsCell Line TumormedicineAnimalsImmunology and AllergyMast CellsTranscription factorCells CulturedMice KnockoutMice Inbred BALB CGene knockdownInterleukin-13Innate immune systemNFATC Transcription FactorsbiologyTumor Necrosis Factor-alphaDegranulationNFATMast cellUp-RegulationCell biologymedicine.anatomical_structurechemistryIonomycinImmunologybiology.proteinCytokinesThe Journal of Immunology
researchProduct

Inhibition of human allergic T-helper type 2 immune responses by induced regulatory T cells requires the combination of interleukin-10-treated dendri…

2006

Summary Background In grass pollen-allergic individuals, T cell anergy can be induced by IL-10-treated dendritic cells (IL-10-DC) resulting in the suppression of T helper type 1 (Th1) as well as Th2 cells. This study was performed to analyse whether such IL-10-DC-treated T cells are able to act as regulatory T cells (Treg) suppressing the function of other T cells in the periphery. As transforming growth factor (TGF)-β is also a potential inducer of Treg, we additionally analysed the inhibitory capacity of TGF-β-treated T cells in this system. Materials and Methods Freshly isolated CD4+ or CD4+CD25− T cells from grass pollen-allergic donors were stimulated with autologous mature monocyte-de…

ImmunologyEnzyme-Linked Immunosorbent AssayCell CommunicationBiologyPoaceaeT-Lymphocytes RegulatoryInterleukin 21Interferon-gammaTh2 CellsAntigens CDTransforming Growth Factor betaHypersensitivityImmunology and AllergyCytotoxic T cellHumansCTLA-4 AntigenIL-2 receptorAntigen-presenting cellCells CulturedInterleukin 3Cell ProliferationDendritic cellDendritic CellsAllergensNatural killer T cellFlow CytometryAntigens DifferentiationCell biologyInterleukin-10ImmunologyInterleukin 12PollenImmunizationInterleukin-4Interleukin-5Clinical and experimental allergy : journal of the British Society for Allergy and Clinical Immunology
researchProduct

Modulation of C1q mRNA Expression and Secretion by Interleukin-1,Interleukin-6, and Interferon-g in Resident and Stimulated Murine Peritoneal Macroph…

2002

The complement system plays an important role in the humoral immune response. Activation of the classical complement pathway is mediated by its subcomponent, C1q. Among the main C1q-synthesising tissues, macrophages have been attributed as a source of particular importance. We investigated the effects of cytokines (IL-1, IL-6 and Interferon-gamma) on local C1q mRNA expression and C1q secretion in resident and in thioglycollate-stimulated murine peritoneal macrophages in vitro. The macrophages were isolated from murine peritoneal lavage fluid, maintained in culture and incubated with the cytokines. Among the cytokines, only IL-6 had a stimulatory effect on C1q production (25% increase vs. co…

ImmunologyGene Expressionchemical and pharmacologic phenomenaIn Vitro TechniquesProinflammatory cytokineInterferon-gammaMiceClassical complement pathwayImmune systemmedicineAnimalsImmunology and AllergyMacrophageInterferon gammaRNA MessengerInterleukin 6Macrophage inflammatory proteinMice Inbred BALB CbiologyInterleukin-6ChemistryComplement C1qInterleukinHematologyMacrophage ActivationRecombinant ProteinsCell biologyThioglycolatesMacrophages Peritonealbiology.proteinInterleukin-1medicine.drugImmunobiology
researchProduct

Predominant role of NF-kappa B p65 in the pathogenesis of chronic intestinal inflammation.

1998

NF-kappa B is a key transcription factor of lymphocytes and macrophages with important regulatory functions in the immune system and inflammatory processes. These functions are at least partially based on its ability to regulate the promoters of a variety of genes whose products, such as cytokines, adhesion molecules and acute phase proteins, are critical for inflammatory processes. In the present study, we describe a method to treat established intestinal inflammation by local or systemic application of antisense phosphorothioate oligonucleotides targeting the translation start site of the p65 subunit of NF-kappa B. Specific downregulation of p65 by administration of antisense phosphorothi…

ImmunologyInflammationBiologyPathogenesisMiceImmune systemDownregulation and upregulationCrohn DiseasemedicineImmunology and AllergyAnimalsHumansCells CulturedInflammationPhosphorothioate OligonucleotidesOligonucleotideInterleukin-6Tumor Necrosis Factor-alphaMacrophagesAcute-phase proteinNF-kappa BTranscription Factor RelAHematologyOligonucleotides AntisenseNFKB1ColitisIntestinesDisease Models AnimalImmunologyChronic DiseaseCancer researchFemalemedicine.symptomInterleukin-1Immunobiology
researchProduct

Mast cells are crucial for early inflammation, migration of Langerhans cells, and CTL responses following topical application of TLR7 ligand in mice.

2007

Abstract Until recently, IgE-activated mast cells have been regarded merely as effector cells of adaptive immune responses, involved in allergic reactions and mucosal immunity to parasites. Herein, we report that murine dermal mast cells, activated by local administration of a cream containing the synthetic TLR7 ligand imiquimod, are essential to initiate an early inflammatory reaction. The mast-cell–derived cytokines TNF-α and IL-1β play an important role in this process. Furthermore, TLR7-activated mast cells are also able to promote the emigration of Langerhans cells, which partly depends on the expression of mast-cell–derived IL-1β. We have previously shown that TLR7 ligation enhances t…

ImmunologyInterleukin-1betaInflammationImmunoglobulin ELigandsBiochemistryMiceImmune systemAdjuvants ImmunologicCell MovementmedicineCytotoxic T cellAnimalsMast CellsAntigensSkinInflammationImmunity CellularMice Inbred BALB CVaccinesImiquimodMembrane GlycoproteinsbiologyTumor Necrosis Factor-alphaDegranulationCell BiologyHematologyTLR7Immunoglobulin EAcquired immune systemImmunity InnateInterleukin 33Toll-Like Receptor 7Langerhans CellsImmunologybiology.proteinAminoquinolinesImmunizationmedicine.symptomAgranulocytosisT-Lymphocytes CytotoxicBlood
researchProduct