Search results for "interleukin-1"

showing 10 items of 660 documents

Interleukin-10-treated dendritic cells modulate immune responses of naive and sensitized T cells in vivo.

2002

Interleukin-10 is a pleiotropic cytokine known to have inhibitory effects on the accessory functions of dendritic cells. In vitro, interleukin-10 converts immature dendritic cells into tolerizing antigen- presenting cells. To assess whether interleukin-10-treated dendritic cells exert tolerizing effects in vivo, CD4+ T cells from DO11.10 ovalbumin-T cell receptor transgenic mice were transferred to syngeneic BALB/c recipients. Recipient animals were treated with ovalbumin-pulsed/unpulsed, interleukin-10-treated/untreated CD11c+ dendritic cells thereafter and ovalbumin-specific proliferation of lymph node cells was assessed by restimulation with the peptide in vitro. In prophylactic experime…

OvalbuminT cellT-LymphocytesReceptors Antigen T-CellDermatologyBiochemistryMicemedicineCytotoxic T cellAnimalsHypersensitivity DelayedAntigen-presenting cellMolecular BiologyMice Inbred BALB CCD40biologyFollicular dendritic cellsDendritic cellCell BiologyDendritic CellsNatural killer T cellInterleukin-10medicine.anatomical_structureImmunologybiology.proteinInterleukin 12The Journal of investigative dermatology
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Signals involved in the early TH1/TH2 polarization of an immune response depending on the type of antigen.

1999

Abstract Background: The early production of distinct cytokines by epidermal cells (ECs) in response to antigen exposure may govern the development of T H1 -like immune responses, such as contact sensitivity, or T H2 -like immune responses, such as IgE-dependent allergies of the immediate type, depending on the type of antigen. Objective: The aim of this study was to compare the signals induced by protein allergens with those induced by haptens in ECs and subsequently in local draining lymph node cells (LNCs) or splenocytes. Methods: BALB/c mice were primed in vivo with the protein allergens ovalbumin or birch pollen or the haptens 2,4-dinitrofluorobenzene or trinitrochlorbenzene, respectiv…

Ovalbuminmedicine.medical_treatmentImmunologyImmunoglobulinsEnzyme-Linked Immunosorbent AssayPicryl ChlorideBiologyMiceImmune systemTh2 CellsAntigenmedicineDinitrochlorobenzeneImmunology and AllergyAnimalsRNA MessengerCells CulturedMice Inbred BALB CReverse Transcriptase Polymerase Chain ReactionCell PolarityEpithelial CellsT lymphocyteAllergensTh1 CellsInterleukin-10Interleukin 10OvalbuminBlotting SouthernKineticsCytokineImmunologybiology.proteinCytokinesPollenFemaleLymph NodesAntibodyHaptenHaptensSpleenSignal TransductionThe Journal of allergy and clinical immunology
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Neuroimmunomodulatory Alterations in Non-Lesional Peritoneum Close to Peritoneal Endometriosis

2012

<b><i>Objectives:</i></b> An imbalance in the ratio of sensory to sympathetic nerve fibre (NF) density in peritoneal endometriotic lesions (pEL) has recently been demonstrated and leads to the assumption that this preponderance of the sensory pro-inflammatory milieu is a major cause of pain in endometriosis. Therefore, the density of sensory and sympathetic NFs was determined in distal unaffected peritoneum of endometriosis patients to be able to detect possible alterations in unaffected peritoneum. <b><i>Methods:</i></b> In serial pEL sections (n = 40), lesional and matching unaffected peritoneum as well as healthy peritoneum (HP) from patien…

PathologyEndometriosisSympathetic nerveSubstance PSensory nerve fibreSubstance PPeritoneal Diseaseschemistry.chemical_compoundNerve growth factorPelvic painEndocrinologyhemic and lymphatic diseasesEndometriosiintegumentary systemvirus diseasesMiddle AgedInterleukin-1βImmunohistochemistrymedicine.anatomical_structureNeurologyImmunohistochemistryFemalemedicine.symptomPeritoneal diseasesPeritoneumHumanAdultmedicine.medical_specialtyAdolescentImmunologyEndometriosisEndocrine and Autonomic SystemYoung AdultPeritoneumAdrenergic FiberInternal medicinemedicineHumansPathogenesis endometriosiEndocrine and Autonomic Systemsbusiness.industryPelvic painmedicine.diseaseNerve growth factorEndocrinologychemistrySympathetic nerve fibrePeritoneal DiseasebusinessAdrenergic Fibers
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IL-22 is produced by innate lymphoid cells and limits inflammation in allergic airway disease

2011

Interleukin (IL)-22 is an effector cytokine, which acts primarily on epithelial cells in the skin, gut, liver and lung. Both pro- and anti-inflammatory properties have been reported for IL-22 depending on the tissue and disease model. In a murine model of allergic airway inflammation, we found that IL-22 is predominantly produced by innate lymphoid cells in the inflamed lungs, rather than TH cells. To determine the impact of IL-22 on airway inflammation, we used allergen-sensitized IL-22-deficient mice and found that they suffer from significantly higher airway hyperreactivity upon airway challenge. IL-22-deficiency led to increased eosinophil infiltration lymphocyte invasion and production…

PathologyPulmonologymedicine.medical_treatmentT-LymphocytesIntracellular Spacelcsh:Medicine10263 Institute of Experimental ImmunologyInterleukin 22Mice0302 clinical medicineLymphocytesPhosphorylationlcsh:ScienceLung0303 health sciencesMultidisciplinaryInterleukin-13T CellsAllergy and HypersensitivityInnate lymphoid cellInterleukinrespiratory systemInnate ImmunityRecombinant Proteins3. Good healthCytokinemedicine.anatomical_structureInterleukin 13CytokinesMedicineTumor necrosis factor alphaBiological Markersmedicine.symptomResearch ArticleSTAT3 Transcription Factormedicine.medical_specialtyImmune CellsImmunologyAntigen-Presenting CellsImmunoglobulinsInflammation610 Medicine & health1100 General Agricultural and Biological SciencesBiology03 medical and health sciences1300 General Biochemistry Genetics and Molecular BiologymedicineRespiratory HypersensitivityAnimalsBiology030304 developmental biologyInflammation1000 MultidisciplinaryTumor Necrosis Factor-alphaInterleukinslcsh:RImmunityEpithelial CellsEosinophilAllergensAsthmaImmunity Innaterespiratory tract diseasesImmune SystemImmunology570 Life sciences; biologylcsh:QImmunizationBiomarkers030215 immunology
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IL-6 Regulates Neutrophil Microabscess Formation in IL-17A-Driven Psoriasiform Lesions

2014

The lack of a generally accepted animal model for human psoriasis has hindered progress with respect to understanding the pathogenesis of the disease. Here we present a model in which transgenic IL-17A expression is targeted to the skin in mice, achievable after crossing our IL-17A(ind) allele to the K14-Cre strain. K14-IL-17A(ind/+) mice invariably develop an overt skin inflammation bearing many hallmark characteristics of human psoriasis including dermal infiltration of effector T cells, formation of neutrophil microabscesses, and hyperkeratosis. IL-17A expression in the skin results in upregulated granulopoiesis and migration of IL-6R-expressing neutrophils into the skin. Neutralization …

Pathologymedicine.medical_specialty1303 BiochemistryNeutrophilsT-LymphocytesHyperkeratosisGene Expression610 Medicine & healthInflammationDermatology10263 Institute of Experimental ImmunologyBiochemistryGranulopoiesis2708 Dermatology1307 Cell BiologyPathogenesisMicePsoriasis1312 Molecular BiologymedicineAnimalsPsoriasisMicroabscessMolecular BiologyMice Knockoutintegumentary systemInterleukin-6business.industryMacrophagesInterleukin-17Cell Biologymedicine.diseaseReceptors Interleukin-6AbscessDisease Models AnimalImmunology570 Life sciences; biologyEpidermismedicine.symptombusinessInfiltration (medical)GranulocytesSignal TransductionEpidermal thickeningJournal of Investigative Dermatology
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Interleukin 6 (IL-6) deficiency delays lupus nephritis in MRL-Faslpr mice: the IL-6 pathway as a new therapeutic target in treatment of autoimmune ki…

2009

Objective.To investigate the pathophysiological effect of interleukin 6 (IL-6) on lupus nephritis in MRL-Faslprmice.Methods.We generated IL-6-deficient MRL-Faslprmice using a backcross/intercross breeding scheme. Renal pathology was evaluated using immunohistochemistry detection for macrophages, lymphocytes, vascular cell adhesion molecule-1 (VCAM-1), and TUNEL (terminal deoxynucleotide transferase-mediated dUTP nick end-labeling) for apoptotic cells, and renal IgG and C3 deposition by immunofluorescence staining. Expression of inflammatory markers in the spleen was analyzed by quantitative real-time reverse transcription-polymerase chain reaction. Serum cytokine concentrations were detecte…

Pathologymedicine.medical_specialtyMice Inbred MRL lprImmunologyLupus nephritisVascular Cell Adhesion Molecule-1SpleenKidneyInterferon-gammaMiceLife ExpectancyRheumatologyImmunology and AllergyMedicineAnimalsHumansLupus Erythematosus Systemicfas ReceptorInterleukin 6InflammationMice KnockoutKidneyMice Inbred BALB CLupus erythematosusbiologybusiness.industryInterleukin-6Kidney metabolismmedicine.diseaseLupus NephritisInterleukin-10Proteinuriamedicine.anatomical_structureRenal pathologyImmunologybiology.proteinFemalebusinessBiomarkersSpleenKidney diseaseSignal TransductionThe Journal of rheumatology
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Pro-inflammatory, Pleiotropic, and Anti-inflammatory TNF-α, IL-6, and IL-10 in Experimental Porcine Intervertebral Disk Degeneration

2009

The aim of this study was to check the balance between tumor necrosis factor-α (TNF-α), interleukin-6 (IL-6), and interleukin-10 (IL-10) in well-developed end-stage disk disease in the disk itself as well as in paradiskal spine. In 6 domestic pigs the cranial bony end plate of the L4 vertebra was perforated to the nucleus pulposus. At 3 months the degenerated experimental and contiguous control disks, together with the adjoining bony and cartilaginous vertebral end plates, bone marrow, and spinal ligaments, were excised and used for immunohistochemical analysis. In general, there were more TNF-α and in particular IL-10 positive cells in the degenerated disks than in the control disks, where…

Pathologymedicine.medical_specialtyNecrosisSwineIntervertebral Disc DegenerationLesionmedicineAnimalsIntervertebral DiscSwine DiseasesGeneral VeterinaryInterleukin-6Tumor Necrosis Factor-alphabusiness.industryCartilageIntervertebral discAnatomyImmunohistochemistryInterleukin-10VertebraIntervertebral diskmedicine.anatomical_structureTumor necrosis factor alphaBone marrowmedicine.symptombusinessVeterinary Pathology
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Increased TNF-alfa, IL-6 and decreased IL-1beta immunohistochemical expression by the stromal spindle-shaped cells in the central giant cell granulom…

2010

Objectives: the exp ress ion of the osteoclastogenic cytokines TNF-α, IL-6 and IL-1β were immunohistochemically evaluated in periph eral (PGCG) and central (CGCG) giant cell granulomas of the jaws in order to determine diff erences between these two lesions and between the two distinct tumor cell populations (multinucleated giant cells, MGCs and stromal sp indle-sh aped cells). Study Design: Paraffin-embedd ed tiss ue sections from 40 PGCG and 40 CGCG were immunohistochemically stained using antibodies against TNF-α, IL-6 and IL-1β. The percentage of positively stained cells and the staining intensity were ass ess ed to provide a combined immunoreactivity score value. Results: TNF-α, IL-6 a…

Pathologymedicine.medical_specialtyStromal cellInterleukin-1betaProinflammatory cytokineGranuloma Giant CellOsteoclastmedicineHumansProgenitor cellGeneral DentistryOral Medicine and PathologyInterleukin-6Tumor Necrosis Factor-alphaChemistry:CIENCIAS MÉDICAS [UNESCO]medicine.diseaseImmunohistochemistrymedicine.anatomical_structureOtorhinolaryngologyGiant cellUNESCO::CIENCIAS MÉDICASImmunohistochemistryResearch-ArticleSurgeryTumor necrosis factor alphaStromal CellsCentral giant-cell granulomaMedicina Oral Patología Oral y Cirugia Bucal
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Interleukins IL-6, IL-8, IL-10, IL-12 and periimplant disease. An update

2010

Introduction: A study is made of the usefulness of cytokines (such as interleukin-6 (IL-6), interleukin-8 (IL-8), interleukin-10 (IL-10) and interleukin-12 (IL-12)) as markers of periimplant disease (mucositis and periimplantitis). An increase in the levels of these cytokines in dental implant crevicular fluid may give rise to a lack of osteointegration, bbone loss or implant failure. Obbjective: To review the literature relating IL-6, IL-8, IL-10 and IL-12 levels to dental implant surgery and periimplantitis. Material and Method: A PubbMed literature search was made of articles in English and Spanish, using the key words "cytokine and dental implants", cytokine and periimplantitis", "IL-6,…

Peri-implantitismedicine.medical_treatmentDentistryOsseointegrationMucositisHumansMedicineInterleukin 6Dental implantGeneral DentistrybiologyInterleukin-6business.industryInterleukin-8Implant failureInterleukin:CIENCIAS MÉDICAS [UNESCO]medicine.diseaseInterleukin-12Peri-ImplantitisInterleukin-10stomatognathic diseasesCytokineOtorhinolaryngologyUNESCO::CIENCIAS MÉDICASbiology.proteinSurgerybusinessBiomarkersMedicina Oral Patología Oral y Cirugia Bucal
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Expression of the human complement C8 subunits is independently regulated by interleukin 1β, interleukin 6, and interferon γ

1998

The eighth component of human complement (C8) is composed of two subunits which are products from three separate genes. The alpha-gamma- and the beta-subunit of C8 are expressed independently, and are part of the membrane attack complex. C8 is primarily synthesized in the liver. It has been shown in previous studies that the human hepatoma cell line HepG2 constitutively expresses C8, and thus is a suitable model system for studying C8 biosynthesis in vitro. Expression is modulated by the cytokines IL-1 beta, IL-6 and IFN-gamma. The effect of the different cytokines on the expression of these subunits was examined using biosynthetical labelling and immunoprecipitation methods. C8 alpha-gamma…

PharmacologybiologyInterleukin-6ImmunoprecipitationAntineoplastic AgentsSulfur RadioisotopesComplement C8Precipitin TestsMolecular biologyIn vitroComplement systemInterferon-gammaGene expressionTumor Cells Culturedbiology.proteinHumansSecretionInterleukin 6Beta (finance)Complement membrane attack complexInterleukin-1Immunopharmacology
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