Search results for "interleukin"

showing 10 items of 1856 documents

Effects of metformin on mitochondrial function of leukocytes from polycystic ovary syndrome patients with insulin resistance.

2015

ObjectiveOxidative stress and mitochondrial dysfunction are implicated in polycystic ovary syndrome (PCOS). The present study assesses the effect of metformin treatment on mitochondrial function in polymorphonuclear cells from PCOS subjects. Additionally, we evaluate endocrine parameters and levels of interleukin 6 (IL6) and tumour necrosis factor alpha (TNFα).Design and methodsOur study population was comprised of 35 women of reproductive age diagnosed with PCOS and treated with metformin for 12 weeks, and their corresponding controls (n=41), adjusted by age and BMI. We evaluated the alteration of endocrinological and anthropometrical parameters and androgen levels. Mitochondrial O2 consum…

Adultmedicine.medical_specialtyendocrine system diseasesNeutrophilsEndocrinology Diabetes and MetabolismMitochondrionBiologymedicine.disease_causechemistry.chemical_compoundYoung AdultEndocrinologyDehydroepiandrosterone sulfateInsulin resistanceInternal medicinemedicineHumansHypoglycemic Agentschemistry.chemical_classificationReactive oxygen speciesInterleukin-6Tumor Necrosis Factor-alphanutritional and metabolic diseasesGeneral MedicineGlutathionemedicine.diseasePolycystic ovaryMetforminMetforminMitochondriaEndocrinologyTreatment OutcomechemistryFemaleInsulin ResistanceOxidative stressmedicine.drugPolycystic Ovary Syndrome
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The role of regulatory T cell (Treg) subsets in gestational diabetes mellitus.

2014

Physiological changes during normal pregnancy are characterized by an inflammatory immune response and insulin resistance. Therefore, we hypothesize that gestational diabetes mellitus (GDM) may be caused by an inappropriate adaption of the maternal immune system to pregnancy. In this study we examined the role of regulatory T cell (Treg) differentiation for the development of GDM during pregnancy. We used six-colour flow cytometric analysis to demonstrate that the total CD4(+) CD127(low+/-) CD25(+) forkhead box protein 3 (FoxP3(+)) T(reg) pool consists of four different T(reg) subsets: naive CD45RA(+) T(regs), HLA-DR(-) CD45RA(-) memory T(regs) (DR(-) T(regs)) and the highly differentiated …

Adultmedicine.medical_specialtyendocrine system diseasesRegulatory T cellImmunologychemical and pharmacologic phenomenaCell SeparationLymphocyte ActivationT-Lymphocytes RegulatoryImmunophenotypingYoung AdultImmune systemInsulin resistanceimmune system diseasesPregnancyT-Lymphocyte Subsetshemic and lymphatic diseasesInternal medicinemedicineImmune ToleranceImmunology and AllergyHomeostasisHumansIL-2 receptorInterleukin-7 receptorbusiness.industryFOXP3hemic and immune systemsCell DifferentiationForkhead Transcription FactorsT helper cellOriginal Articlesmedicine.diseaseFlow CytometryGestational diabetesDiabetes GestationalEndocrinologymedicine.anatomical_structureImmunologyCD4 AntigensFemalebusinessImmunologic MemoryClinical and experimental immunology
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Role of interleukin-4 in pathogenesis of oral lichen planus : a systematic review

2020

Background Oral lichen planus (OLP) is a premalignant mucocutaneous disease that affects 1-2% of the adult population. Immunological factor may act as etiological factor. The cellular immune cells such as T cells are important in pathogenesis of OLP. Interleukin-4 (IL-4) is secreted by T-helper 2 (Th2). Several studies have been carried out on the role of IL-4 in OLP. The aim of this study was to review the level of IL-4 in OLP, effective factors in the production of IL-4 and its role in the development of OLP. Material and Methods A search in PubMed was performed on the literature published from 2000 until august 2019 using the following keywords: “oral lichen planus” or “OLP” and “interle…

Adultmedicine.medical_specialtymedicine.medical_treatmentMucocutaneous zoneDiseaseReviewPathogenesisstomatognathic systemmedicineHumansGeneral DentistryInterleukin 4Oral Medicine and Pathologybusiness.industryCase-control studymedicine.disease:CIENCIAS MÉDICAS [UNESCO]Dermatologystomatognathic diseasesCytokineOtorhinolaryngologyCase-Control StudiesUNESCO::CIENCIAS MÉDICASEtiologyCytokinesSurgeryOral lichen planusInterleukin-4businessPrecancerous ConditionsLichen Planus Oral
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IL-15 in human visceral leishmaniasis caused byLeishmania infantum

2002

SummaryInterleukin (IL)-15 is a recently discovered cytokine with the ability to stimulate the proliferation activity of Th1 and/or Th2 lymphocytes. Here, we investigated the involvement of IL-15 in the immune response to Leishmania infantum infection by studying patients with visceral leishmaniasis (VL). We found that IL-15 is produced by leishmanial antigen (LAg)-stimulated peripheral blood mononuclear cells (PBMC) from active VL patients at a significantly higher level than those produced by cells from healed VL subjects or healthy controls. A significant increase in IL-15 serum blood levels was also observed in acute VL patients compared with healed ones. Furthermore, recombinant IL-15 …

Adultmedicine.medical_treatmentImmunologyAntigens ProtozoanLymphocyte ActivationInterferon-gammaTh2 CellsAntigenmedicineAnimalsHumansImmunology and AllergyLeishmania infantumInterleukin-15biologyAntibodies MonoclonalInterleukinOriginal ArticlesTh1 Cellsbiology.organism_classificationmedicine.diseaseInterleukin-12Recombinant ProteinsCytokineVisceral leishmaniasisInterleukin 15ImmunologyLeukocytes MononuclearInterleukin 12Leishmaniasis VisceralInterleukin-4Leishmania infantumCell activationClinical and Experimental Immunology
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In the presence of IL-21 human cord blood T cells differentiate to IL-10-producing Th1 but not Th17 or Th2 cells.

2012

Abstract IL-21, a member of the IL-2 cytokine family, is mainly produced by activated CD4+ T cells and controls the activity of immune and also non-immune cells. As a pleiotropic cytokine, IL-21 acts on both innate and adaptive immune responses, suggesting that IL-21 may be a master regulator of the T-cell-dependent adaptive immune response. Although IL-21 is described as mostly promoting inflammation, evidence also suggests inhibitory effects of IL-21. However, its role, particularly in the human neonatal immune system, has not been detailed so far. Here, we assessed the effect of IL-21 in the specific context of the neonatal immune response and delineated differences between the human new…

Adultmedicine.medical_treatmentImmunologyCell Culture TechniquesBiologyInterferon-gammaImmune systemTh2 CellsT-Lymphocyte SubsetsmedicineImmunology and AllergyHumansIL-2 receptorTh1-Th2 BalanceCells CulturedInnate immune systemGene Expression ProfilingInterleukinsCCL18LymphokineInfant NewbornCell DifferentiationGeneral MedicineTh1 CellsAcquired immune systemFetal BloodInterleukin-10Interleukin 10CytokineImmunologyTh17 CellsInternational immunology
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Effect of interleukin-6 polymorphisms on human longevity: a systematic review and meta-analysis.

2008

Udgivelsesdato: 2009-Jan Several studies have assessed changes in frequency of -174 interleukin (IL)-6 single nucleotide polymorphism (SNP) with age. If IL-6 tracks with disability and age-related diseases, then there should be reduction, in the oldest old, of the frequency of homozygous GG subjects, who produce higher IL-6 levels. However, discordant results have been obtained. To explore the relationship between this polymorphism and longevity, we analyzed individual data on long-living subjects and controls from eight case-control studies conducted in Europeans, using meta-analysis. There was no significant difference in the IL-6 genotype between the oldest old and controls (Odds Ratio […

AgingGenotypemedia_common.quotation_subjectLongevityPhysiologySingle-nucleotide polymorphismBiologyEnvironmentBiochemistryPolymorphism Single NucleotideGene FrequencyPolymorphism (computer science)GenotypeOdds RatioSNPHumansMolecular BiologyAllele frequencymedia_commonGeneticsAged 80 and overInterleukin-6LongevityOdds ratioNeurologyMeta-analysismeta-analysis interleukin-6 polymorphismsBiotechnologyAgeing research reviews
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Phosphatidylserine counteracts physiological and pharmacological suppression of humoral immune response

1990

Phosphatidylserine (PS) is a necessary cofactor for protein kinase C (PKC) activation, and changes in the synthesis of PS have been shown to participate in the mechanism(s) involved in the transmembrane signaling of interleukin 1 (IL-1). In view of the age-associated defects in T-cell functions, in the present study we have addressed the question of whether an in vivo treatment with PS might interfere with such processes. Furthermore, the effect of an in vitro treatment with PS in human peripheral blood monocytes (PBMC) or splenocytes activated with a lectin mitogen, on the expression of IL-2 receptor, was assessed. While the process of ageing was accompanied by a marked decline of humoral …

Agingmedicine.medical_specialtyReceptor expressionPhosphatidylserinesIn Vitro Techniqueschemistry.chemical_compoundImmune systemInternal medicineImmune TolerancemedicineAnimalsHumansReceptorProtein Kinase CProtein kinase CPharmacologybiologyInterleukinRats Inbred StrainsReceptors Interleukin-2PhosphatidylserineRatsEndocrinologychemistryHumoral immunityLeukocytes Mononuclearbiology.proteinFemaleAntibodySpleenImmunopharmacology
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Anthropometric Variables as Mediators of the Association of Changes in Diet and Physical Activity With Inflammatory Profile

2020

AbstractBackgroundMechanisms underlying the associations of high levels of physical activity (PA) and adherence to the Mediterranean diet (MedDiet) with a better inflammatory profile remain unclear. Our objective was to assess the mediating role of changes in body mass index (BMI) and waist circumference (WC), as markers of body fat in the association of changes in PA and adherence to the MedDiet, with changes in the inflammatory profile.MethodThis study included 489 adults, aged 55–75 years, from the PREDIMED-Plus multicenter lifestyle intervention trial. An inflammatory score was calculated, based on 8 blood biomarkers: high-sensitivity C-reactive protein, interleukin 6, interleukin 8, in…

Agingmedicine.medical_specialtyWaistMediterranean diet030204 cardiovascular system & hematologyDiet Mediterraneanlaw.invention03 medical and health sciences0302 clinical medicineRandomized controlled triallawMediterranean dietInternal medicinemedicineHumans030212 general & internal medicineskin and connective tissue diseasesExerciseLife StyleBody mass indexInflammation2. Zero hungerbiologybusiness.industryLeptinC-reactive proteinAnthropometrybiology.proteinMediation analysisWaist circumferenceInterleukin 18sense organsGeriatrics and GerontologybusinessBody mass indexBiomarkersThe Journals of Gerontology: Series A
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Early activation of γδ T lymphocytes in the elderly

2001

T cell function is altered in vivo and in vitro in elderly compared with young subjects, and this alteration is believed to contribute to morbidity and mortality in man due to the greater incidence of infection, as well as autoimmunity and cancer in elderly. The majority of T cells express TCRalphabeta whereas TCRgammadelta is expressed on a minority of T cells. Moreover, it is known that gammadelta T lymphocytes display major histocompatibility complex (MHC)- unrestricted cytotoxicity that is reminiscent of natural killer (NK) activity. In view of earlier findings on both T cells and NK cells in the elderly, we hypothesised a different behaviour of gammadelta T lymphocytes from old subject…

Agingmedicine.medical_specialtybiologyT cellchemical and pharmacologic phenomenaT lymphocyteMajor histocompatibility complexNatural killer T cellInterleukin 21medicine.anatomical_structureEndocrinologyImmune systemAntigenInternal medicineImmunologymedicinebiology.proteinCytotoxic T cellDevelopmental BiologyMechanisms of Ageing and Development
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Inflammation, genes and zinc in ageing and age-related diseases.

2006

Lifelong antigenic burden determines a condition of chronic inflammation, with increased lymphocyte activation and pro-inflammatory cytokine production. A large number of studies have documented changes in Zn metabolism in experimental animal models of acute and chronic inflammation and in human chronic inflammatory diseases. In particular, modification of zinc plasma concentration as well as intracellular disturbance of antioxidant intracellular pathways have been found associated to age-related inflammatory diseases, like atherosclerosis. Zinc deficiency is extremely diffused in aged people that are educated to avoid meat and other high Zn-content foods due to fear of cholesterol. Rather,…

Agingmedicine.medical_treatmentLongevityGene ExpressionInflammationBiologychemistry.chemical_compoundmedicinecytokine interleukin 6 metallothionein tumor necrosis factor alpha zincAnimalsHumansGeneTranscription factorCellular SenescenceInflammationPolymorphism GeneticCholesterolInterleukin-6Tumor Necrosis Factor-alphamedicine.diseaseAtherosclerosisImmunity InnateZincCytokinechemistryAgeingImmunologyZinc deficiencyCytokinesMetallothioneinGeriatrics and Gerontologymedicine.symptomGerontologyIntracellular
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