Search results for "isoproterenol"

showing 10 items of 56 documents

Induction of Apoptosis in Rat Cardiocytes by A3 Adenosine Receptor Activation and Its Suppression by Isoproterenol

2000

The purpose of the present study was to investigate the mechanisms involved in the induction of apoptosis in newborn cultured cardiomyocytes by activation of adenosine (ADO) A3 receptors and to examine the protective effects of beta-adrenoceptors. The selective agonist for A3 ADO receptors Cl-IB-MECA (2-chloro-N6-iodobenzyl-5-N-methylcarboxamidoadenosine) and the antagonist MRS1523 (5-propyl-2-ethyl-4-propyl-3-(ethylsulfanylcarbonyl)-6-phenylpy rid ine-5-carboxylate) were used. High concentrations of the Cl-IB-MECA (or = 10 microM) agonist induced morphological modifications of myogenic cells, such as rounding and retraction of cell body and dissolution of contractile filaments, followed by…

Agonistmedicine.medical_specialtyProgrammed cell deathAdenosineCardiotonic Agentsmedicine.drug_classApoptosisStimulationBiologyInternal medicinePurinergic P1 Receptor AgonistsmedicineAnimalsProtein kinase AReceptorCells CulturedMyocardiumReceptor Adenosine A3IsoproterenolReceptors Purinergic P1HeartCell BiologyAdenosineAdenosine receptorRatsCell biologyEndocrinologyApoptosisSignal Transductionmedicine.drugExperimental Cell Research
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Spontaneous Cardiomyocyte Differentiation From Adipose Tissue Stroma Cells

2004

Cardiomyocyte regeneration is limited in adult life. Thus, the identification of a putative source of cardiomyocyte progenitors is of great interest to provide a usable model in vitro and new perspective in regenerative therapy. As adipose tissues were recently demonstrated to contain pluripotent stem cells, the emergence of cardiomyocyte phenotype from adipose-derived cells was investigated. We demonstrated that rare beating cells with cardiomyocyte features could be identified after culture of adipose stroma cells without addition of 5-azacytidine. The cardiomyocyte phenotype was first identified by morphological observation, confirmed with expression of specific cardiac markers, immunocy…

AtropineMalemedicine.medical_specialtyStromal cellPhysiologyCellular differentiationHeart VentriclesCholinergic AgentsAdipose tissueAdipose tissueCardiomyocytes ; Adipose tissue ; Differentiation ; Stem cells ; Cell therapyStem cellsBiologyCell therapyCell therapyMiceAdrenergic Agents:CIENCIAS MÉDICAS ::Medicina interna [UNESCO]Internal medicinemedicineAnimalsMyocytes CardiacHeart AtriaProgenitor cellInduced pluripotent stem cellCells CulturedUNESCO::CIENCIAS MÉDICAS ::Medicina internaCardiomyocytesRegeneration (biology)Multipotent Stem CellsIsoproterenolCell Differentiation:CIENCIAS MÉDICAS [UNESCO]Myocardial ContractionPropranololCell biologyClone CellsMice Inbred C57BLEndocrinologyPhenotypeAdipose TissueDifferentiationUNESCO::CIENCIAS MÉDICASRNACarbacholStem cellStromal CellsCardiology and Cardiovascular MedicineMyoblasts Cardiac
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Simultaneous stimulation of GABA and beta adrenergic receptors stabilizes isotypes of activated adenylyl cyclase heterocomplex

2004

Abstract Background We investigated how the synthesis of cAMP, stimulated by isoproterenol acting through β-adrenoreceptors and Gs, is strongly amplified by simultaneous incubation with baclofen. Baclofen is an agonist of δ-aminobutyric acid type B receptors [GABAB], known to inhibit adenylyl cyclase via Gi. Because these agents have opposite effects on cAMP levels, the unexpected increase in cAMP synthesis when they are applied simultaneously has been intensively investigated. From previous reports, it appears that cyclase type II contributes most significantly to this phenomenon. Results We found that simultaneous application of isoproterenol and baclofen specifically influences the assoc…

Baclofenlcsh:CytologyGTP-Binding Protein beta SubunitsIsoproterenolAdrenergic beta-AgonistsRatsIsoenzymesRats Sprague-DawleyReceptors GABAGTP-Binding Protein gamma SubunitsMultiprotein ComplexesReceptors Adrenergic betaSynapsesCyclic AMPGTP-Binding Protein alpha Subunits GsAnimalslcsh:QH573-671GABA AgonistsResearch ArticleAdenylyl CyclasesSignal TransductionBMC Cell Biology
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Paradoxical effect of increased diastolic Ca(2+) release and decreased sinoatrial node activity in a mouse model of catecholaminergic polymorphic ven…

2012

Background— Catecholaminergic polymorphic ventricular tachycardia is characterized by stress-triggered syncope and sudden death. Patients with catecholaminergic polymorphic ventricular tachycardia manifest sinoatrial node (SAN) dysfunction, the mechanisms of which remain unexplored. Methods and Results— We investigated SAN [Ca 2+ ] i handling in mice carrying the catecholaminergic polymorphic ventricular tachycardia–linked mutation of ryanodine receptor (RyR2 R4496C ) and their wild-type (WT) littermates. In vivo telemetric recordings showed impaired SAN automaticity in RyR2 R4496C mice after isoproterenol injection, analogous to what was observed in catecholaminergic polymorphic ventricul…

ChronotropicTachycardiaMalePatch-Clamp TechniquesAction Potentials030204 cardiovascular system & hematologyVentricular tachycardiaMice0302 clinical medicineSinoatrial NodeCatecholaminergic0303 health sciencesRyanodine receptorAdrenergic beta-AgonistsMiddle AgedSarcoplasmic Reticulummedicine.anatomical_structurecardiovascular systemCardiologyFemalemedicine.symptomCardiology and Cardiovascular MedicineAdultmedicine.medical_specialtyIn Vitro TechniquesCatecholaminergic polymorphic ventricular tachycardiaSudden deathArticle03 medical and health sciencesPhysiology (medical)Internal medicinemedicineAnimalsHumansCalcium SignalingExercise030304 developmental biologyAgedbusiness.industrySinoatrial nodeIsoproterenolRyanodine Receptor Calcium Release Channelmedicine.diseaseMice Mutant StrainsMice Inbred C57BLDisease Models AnimalEndocrinologyMutationTachycardia VentricularCalciumbusinessCirculation
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Polyunsaturated fatty acids in cultured cardiomyocytes : effect on physiology and beta-adrenoceptor function

1992

This study was carried out to investigate the influence of the membrane fatty acid composition on the basal electrical and contractile activities and the response to beta-adrenergic stimulation of rat cardiac muscle cells in culture. Cells were grown for 3 days in a conventional serum culture medium and then incubated for 24 h in synthetic media containing either n-6 or n-3 as the sole source of polyunsaturated fatty acids (PUFA). The n-6/n-3 ratio in the phospholipids was 0.9 in the n-3 cells and 13.1 in the n-6 cells compared with 6.3 in controls cells. Such modifications did not alter action potentials and the main parameters related to contraction, although shortening was slightly acce…

Chronotropicmedicine.medical_specialtyContraction (grammar)PhysiologyPhospholipidStimulation030204 cardiovascular system & hematologyBiology03 medical and health scienceschemistry.chemical_compound0302 clinical medicineHeart RatePhysiology (medical)Internal medicineReceptors Adrenergic betamedicineMyocyteAnimalsCells CulturedComputingMilieux_MISCELLANEOUS030304 developmental biologychemistry.chemical_classification0303 health sciences[SDV.MHEP] Life Sciences [q-bio]/Human health and pathologyPOTENTIEL ELECTRIQUEMyocardiumFatty AcidsCardiac muscleIsoproterenolHeartMyocardial ContractionElectrophysiologyEndocrinologymedicine.anatomical_structureACIDE GRAS POLYINSATURE N-6chemistryCell cultureFatty Acids UnsaturatedACIDE GRAS POLYINSATURE N-3RATCardiology and Cardiovascular Medicine[SDV.MHEP]Life Sciences [q-bio]/Human health and pathologyPolyunsaturated fatty acid
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The relaxant effects of cromakalim (BRL 34915) on human isolated airway smooth muscle

1992

Cromakalim (BRL 34915) is a potassium channel opener with therapeutic potential as a bronchodilator in asthma. Cromakalim (0.1–30 μmol/l) inhibited the spontaneous tone of human isolated bronchi n a concentration-related manner being nearly as effective as isoprenaline or theophylline. The order of relaxant potencies (expressed as -log10 IC50 mol/l; mean ±SEM) was isoprenaline (7.29 ± 0.27; n = 8) > cromakalim (5.89 ± 0.12; n = 7) > theophylline (4.07 ±0.13; n = 10). In human bronchi where tone had been raised by addition of histamine (0.1 mmol/l), acetylcholine (0.1 mmol/l) or leukotriene D4 (LTD4, 0.1 μmol/l), the relaxant effect of cromakalim was substantially reduced. Cromakalim suppres…

CromakalimMuscle RelaxationBronchiPharmacologyGlibenclamidechemistry.chemical_compoundTheophyllineIsoprenalinemedicineHumansBenzopyransDrug InteractionsPyrrolesPharmacologyTetraethylammoniumIsoproterenolMuscle SmoothGeneral MedicineAcetylcholineBronchodilator AgentschemistryAnesthesiaSRS-APotassium channel openermedicine.symptomCromakalimHistamineAcetylcholineHistamineMuscle contractionmedicine.drugNaunyn-Schmiedeberg's Archives of Pharmacology
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Lipolysis in isolated epididymal adipocytes from genetically obese Zucker rat treated with 3,5,3'-L-triiodothyronine.

1990

Abstract. Glycerol released by isolated epididymal adipocytes was measured in 6-8 weeks old genetically obese (fa/fa) and lean (C.c.a/-) male Zucker rats, ip treated or not treated with T3 (0.46, 2.3 and 4.6 nmol/100 g in a single injection or 4.6 nmol · (100 g)−1 · day−1 for 5 days). In the non-treated rats, lipolysis was less stimulated by adrenalin or isoproterenol in obese than in lean rats, whereas it was stimulated by theophylline up to the same level in both kinds of rats. A single injection of the T3 (highest dose) increased isoproterenol-induced lipolysis in the lean but not in the obese rats. The 5-day T3 treatment caused a stimulation of the adrenalin- or isoproterenol-induced li…

GlycerolMalemedicine.medical_specialtyEpinephrineEndocrinology Diabetes and MetabolismLipolysisAdipose tissueStimulationBiologychemistry.chemical_compoundEndocrinologyTheophyllineInternal medicineAdipocytemedicineLipolysisAnimalsTheophyllineObesityEpididymisTriiodothyronineIsoproterenolGeneral MedicineEpididymisPathophysiologyRatsRats ZuckerEndocrinologymedicine.anatomical_structurechemistryAdipose TissueTriiodothyroninemedicine.drugActa endocrinologica
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Isoproterenol inhibits fluid-phase endocytosis from early to late endosomes

1999

We have shown recently that isoproterenol affects both the cellular location and the morphology of late endosomes in a pH-dependent manner [Marjomäki et al., Eur. J. Cell Biol. 65, 1-13 (1994)]. In this study, using fluorescence and quantitative electron microscopy, we wanted to examine further what is the fate of internalized markers during their translocation from early to late endosomes under isoproterenol treatment. Fluorescein dextran internalized for 30 min (10-min pulse followed by a 20-min chase) showed accumulation in the cellular periphery during isoproterenol treatment in contrast to the control cells, which accumulated dextran in the perinuclear region. Quantitative electron mic…

HistologyEndosomeCell CountEndosomesBiologyEndocytosisPathology and Forensic Medicinechemistry.chemical_compoundCyclic AMPAnimalsFragmentation (cell biology)Cells CulturedVesicleIsoproterenolCell BiologyGeneral MedicineHydrogen-Ion ConcentrationMembrane transportEndocytosisIn vitroRatsCell biologyDextranchemistryBiochemistryCalciumRabbitsPercollBiomarkers
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Electrophysiologic and inotropic effects of alpha-adrenoceptor stimulation in human isolated atrial heart muscle.

1992

The effects of α-adrenoceptor stimulation on force of contraction were investigated in human atrial heart muscle and compared with those of β-adrenoceptor stimulation. The maximal positive inotropic effect produced by stimulation of α-adrenoceptors with phenylephrine (in the presence of atenolol 10 μmol/l) was significantly smaller than that seen in response to β-adrenoceptor stimulation with isoprenaline. The maximal effect of phenylephrine (25% of the maximal effect of isoprenaline) required far higher concentrations (1 mmol/l) than isoprenaline (100 nmol/l); the EC50 values amounted to 33.1 μmol/l and 3.3 nmol/l, respectively. In the presence of the α-adrenoceptor blocking agent phentola…

InotropeAdultMalemedicine.medical_specialtyAdolescentAdrenergicAction PotentialsStimulationIn Vitro TechniquesNorepinephrinePhenylephrinePhentolamineInternal medicineIsoprenalinePrazosinmedicineHumansHeart AtriaChildPhenylephrineAgedPharmacologyDose-Response Relationship Drugbusiness.industryIsoproterenolInfantGeneral MedicineMiddle AgedReceptors Adrenergic alphaAtenololMyocardial ContractionStimulation ChemicalElectrophysiologyEndocrinologyChild PreschoolFemalebusinessmedicine.drugNaunyn-Schmiedeberg's archives of pharmacology
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Adrenoceptor-mediated changes of action potential and force of contraction in human isolated ventricular heart muscle.

1988

Abstract 1. The effects of alpha-adrenoceptor stimulation on the action potential and force of contraction were investigated in human isolated ventricular heart muscle and compared with those of beta-adrenoceptor stimulation. 2. The maximal stimulation by isoprenaline of beta-adrenoceptors produced large changes in the force of contraction, which were accompanied by moderate increases in the height of the action potential. The maximal inotropic effect produced by stimulation of alpha-adrenoceptors with phenylephrine, in the presence of propranolol (1 mumol 1(-1)) was much smaller (about 10% of that seen in response to beta-adrenoceptor stimulation), and no significant changes of the action …

InotropeAdultMalemedicine.medical_specialtyContraction (grammar)EpinephrineAction PotentialsStimulationPropranololContractilityNorepinephrineInternal medicineIsoprenalineReceptors Adrenergic betamedicinePrazosinHumansPhenylephrineAgedPharmacologyChemistryIsoproterenolMiddle AgedPapillary MusclesReceptors Adrenergic alphaMyocardial ContractionPropranololEndocrinologyFemalemedicine.drugResearch ArticleBritish journal of pharmacology
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