Search results for "kinetics"

showing 10 items of 2224 documents

Pharmacokinetics of methylmethacrylate monomer during total hip replacement in man.

1988

The concentration of methylmethacrylate monomer (MMA) in the blood stream after implantation of the components of 15 total hip prostheses using bone cement was determined in the pulmonary artery, the radial artery, and the superior vena cava after cement application, and correlated with the observed drop in blood pressure and the increase in the pulmonary arterial pressure. In all samples MMA was found. The values ranged from 0.02 micrograms/ml to 59 micrograms/ml. The mean maximum value after implantation of the stem was measured to be 7.8 micrograms/ml in the pulmonary artery, 4.6 micrograms/ml in the radial artery, and 1.75 micrograms/ml in the superior vena cava. After implantation of t…

MaleMean arterial pressureChromatography GasVena Cava SuperiorBlood PressurePulmonary ArteryPharmacokineticsSuperior vena cavamedicine.arterymedicineHumansMethylmethacrylatesOrthopedics and Sports MedicineRadial arteryAgedAged 80 and overbusiness.industryGeneral MedicineMiddle AgedBone cementBlood pressureAnesthesiaCirculatory systemPulmonary arterySurgeryFemaleHip ProsthesisNuclear medicinebusinessArchives of orthopaedic and traumatic surgery. Archiv fur orthopadische und Unfall-Chirurgie
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Acceleration of glutathione efflux and inhibition of gamma-glutamyltranspeptidase sensitize metastatic B16 melanoma cells to endothelium-induced cyto…

2005

Highly metastatic B16 melanoma (B16M)-F10 cells, as compared with the low metastatic B16M-F1 line, have higher GSH content and preferentially overexpress BCL-2. In addition to its anti-apoptotic properties, BCL-2 inhibits efflux of GSH from B16M-F10 cells and thereby may facilitate metastatic cell resistance against endothelium-induced oxidative/nitrosative stress. Thus, we investigated in B16M-F10 cells which molecular mechanisms channel GSH release and whether their modulation may influence metastatic activity. GSH efflux was abolished in multidrug resistance protein 1 knock-out (MRP-/-1) B16M-F10 transfected with the Bcl-2 gene or in MRP-/-1 B16M-F10 cells incubated with l-methionine, wh…

MaleMelanoma ExperimentalCystic Fibrosis Transmembrane Conductance RegulatorApoptosisBiochemistryOligodeoxyribonucleotides Antisensechemistry.chemical_compoundMiceCell AdhesionAnimalsEndotheliumNeoplasm MetastasisCytotoxicityCell adhesionMolecular BiologybiologyActivator (genetics)Cell BiologyGlutathioneTransfectiongamma-GlutamyltransferaseMolecular biologyGlutathioneCystic fibrosis transmembrane conductance regulatorMice Inbred C57BLKineticsOxidative StresschemistryProto-Oncogene Proteins c-bcl-2VerapamilApoptosisbiology.proteinEffluxMultidrug Resistance-Associated ProteinsThe Journal of biological chemistry
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Controlled transdermal iontophoresis for poly-pharmacotherapy: Simultaneous delivery of granisetron, metoclopramide and dexamethasone sodium phosphat…

2015

Iontophoresis has been used to deliver small molecules, peptides and proteins into and across the skin. In principle, it provides a controlled, non-invasive method for poly-pharmacotherapy since it is possible to formulate and to deliver multiple therapeutic agents simultaneously from the anodal and cathodal compartments. The objective of this proof-of-principle study was to investigate the simultaneous anodal iontophoretic delivery of granisetron (GST) and metoclopramide (MCL) and cathodal iontophoresis of dexamethasone sodium phosphate (DEX-P). In addition to validating the hypothesis, these are medications that are routinely used in combination to treat chemotherapy-induced emesis. Two p…

MaleMetoclopramideSwinePharmaceutical Science02 engineering and technologyPharmacologyGranisetronAdministration Cutaneous030226 pharmacology & pharmacyDexamethasoneGranisetron03 medical and health sciences0302 clinical medicineDexamethasone Sodium PhosphatePharmacokineticsIn vivomedicineAnimalsRats WistarDexamethasoneActive metaboliteTransdermalSkinIontophoresisChemistryHydrolysisIontophoresis021001 nanoscience & nanotechnologyRatsPolypharmacy0210 nano-technologymedicine.drugEuropean journal of pharmaceutical sciences : official journal of the European Federation for Pharmaceutical Sciences
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Stimulatory and inhibitory effects of ethanol on hippocampal acetylcholine release

1998

Using the microdialysis technique and sensitive HPLC procedures for the determination of acetylcholine (ACh) and ethanol, we investigated the release of ACh in rat hippocampus after acute ethanol administration. Systemic administration of ethanol (0.8 and 2.4 g/kg i.p.) led to peak ethanol concentrations of 21 and 42 mM in the hippocampus, respectively. The high dose caused a long-lasting inhibition of basal ACh release by up to 33%. Local infusion of scopolamine (1 microM) enhanced hippocampal ACh release up to eightfold in the presence of neostigmine (10 microM), and this stimulated release was also inhibited after systemic ethanol administration (by up to 45%). The low dose of ethanol (0…

MaleMicrodialysisMicrodialysisScopolamineHippocampusStimulationMuscarinic AntagonistsHippocampal formationPharmacologyHippocampuschemistry.chemical_compoundmedicineAnimalsRats WistarChromatography High Pressure LiquidPharmacologyEthanolEthanolCentral Nervous System DepressantsGeneral MedicineAcetylcholineRatsKineticschemistrySystemic administrationCholinergicExtracellular SpaceAcetylcholinemedicine.drugNaunyn-Schmiedeberg's Archives of Pharmacology
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Mitochondrial reactive oxygen species are obligatory signals for glucose-induced insulin secretion.

2009

OBJECTIVE—Insulin secretion involves complex events in which the mitochondria play a pivotal role in the generation of signals that couple glucose detection to insulin secretion. Studies on the mitochondrial generation of reactive oxygen species (ROS) generally focus on chronic nutrient exposure. Here, we investigate whether transient mitochondrial ROS production linked to glucose-induced increased respiration might act as a signal for monitoring insulin secretion. RESEARCH DESIGN AND METHODS—ROS production in response to glucose was investigated in freshly isolated rat islets. ROS effects were studied using a pharmacological approach and calcium imaging. RESULTS—Transient glucose increase …

MaleMitochondrial ROSmedicine.medical_specialtyEndocrinology Diabetes and Metabolismmedicine.medical_treatment[ SDV.AEN ] Life Sciences [q-bio]/Food and Nutritionchemistry.chemical_elementCalciumMitochondrionBiologySuperoxide dismutaseIslets of Langerhans03 medical and health scienceschemistry.chemical_compoundAdenosine Triphosphate0302 clinical medicineSuperoxidesInternal medicineInsulin SecretionInternal MedicinemedicineAnimalsInsulinSecretionChromansRats Wistar030304 developmental biologychemistry.chemical_classification0303 health sciencesReactive oxygen speciesSuperoxide DismutaseSuperoxideInsulinNADMitochondriaRatsKinetics[SDV.AEN] Life Sciences [q-bio]/Food and NutritionGlucoseEndocrinologyIslet Studieschemistrybiology.proteinThapsigarginCalciumReactive Oxygen Species[SDV.AEN]Life Sciences [q-bio]/Food and Nutrition030217 neurology & neurosurgerySignal Transduction
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A pharmacokinetic approach to model-guided design of infliximab schedules in ulcerative colitis patients

2015

Background: Infliximab, an anti-tumour necrosis factor approved for treatment of Crohn's disease and ulcerative colitis, is administered at predefined interdose-intervals. On insufficient response or loss of response, treatment can be intensified. The lack or loss of response is likely related to complex pharmacokinetics of infliximab. Aims: To explore optimal dosing strategies of infliximab in treatment-naïve patients with ulcerative colitis through predictive Monte Carlo simulations based on a validated population PK model. Methods: A population of 2,000 treatment-naïve patients was generated by Montecarlo simulation. Six dosing strategies for maintenance therapy were simulated on this po…

MaleModels StatisticalDose-Response Relationship DrugUlcerativeColitisInfliximabTreatment OutcomeGastrointestinal AgentsHumanslcsh:Diseases of the digestive system. GastroenterologyColitis UlcerativeComputer SimulationFemalePharmacokineticslcsh:RC799-869Monte Carlo Method
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A nuclear juvenile hormone-binding protein from larvae of Manduca sexta: a putative receptor for the metamorphic action of juvenile hormone

1994

0027-8424 (Print) Journal Article Research Support, Non-U.S. Gov't Research Support, U.S. Gov't, Non-P.H.S. Research Support, U.S. Gov't, P.H.S.; A 29-kDa nuclear juvenile hormone (JH)-binding protein from the epidermis of Manduca sexta larvae was purified by using the photoaffinity analog for JH II ([3H]epoxyhomofarnesyldiazoacetate) and partially sequenced. A 1.1-kb cDNA was isolated by using degenerate oligonucleotide primers for PCR based on these sequences. The cDNA encoded a 262-amino acid protein that showed no similarity with other known proteins, except for short stretches of the interphotoreceptor retinoid-binding protein, rhodopsin, and human nuclear protein p68. Recombinant bacu…

MaleMoths/growth & development/*metabolism/physiologyBase SequenceMetamorphosisPolymerase Chain Reaction/methodsSesquiterpenes/metabolismMolecular Sequence DataDNABiological/*physiologyTritiumJuvenile Hormones/metabolismMolecular WeightKineticsIsomerismOligodeoxyribonucleotidesLarvaAnimalsComplementary/isolation & purificationInsect ProteinsAmino Acid SequenceCarrier Proteins/genetics/isolation & purification/*metabolism
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The NK Cell Response to Mouse Cytomegalovirus Infection Affects the Level and Kinetics of the Early CD8+ T-Cell Response

2012

ABSTRACT Natural killer (NK) cells and CD8 + T cells play a prominent role in the clearance of mouse cytomegalovirus (MCMV) infection. The role of NK cells in modulating the CD8 + T-cell response to MCMV infection is still the subject of intensive research. For analyzing the impact of NK cells on mounting of a CD8 + T-cell response and the contribution of these cells to virus control during the first days postinfection (p.i.), we used C57BL/6 mice in which NK cells are specifically activated through the Ly49H receptor engaged by the MCMV-encoded ligand m157. Our results indicate that the requirement for CD8 + T cells in early MCMV control inversely correlates with the engagement of Ly49H. W…

MaleMuromegalovirusImmunologyNK cellsCD8-Positive T-LymphocytesBiologym157MicrobiologyRodent DiseasesMice03 medical and health sciencesInterleukin 210302 clinical medicineVirologyAnimalsCytotoxic T cellmouse cytomegalovirus; NK cells; T-cell response; Ly49H; m157IL-2 receptor030304 developmental biologyMice Inbred BALB C0303 health sciencesJanus kinase 3ZAP70BIOMEDICINE AND HEALTHCARE. Basic Medical Sciences.Herpesviridae InfectionsNatural killer T cellMouse cytomegalovirus3. Good healthKiller Cells NaturalMice Inbred C57BLKineticsT-cell responseInsect ScienceImmunologyInterleukin 12CytokinesPathogenesis and ImmunityFemaleLy49HBIOMEDICINA I ZDRAVSTVO. Temeljne medicinske znanosti.CD8030215 immunology
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Glutathione and the rate of cellular proliferation determine tumour cell sensitivity to tumour necrosis factor in vivo.

1997

Low rates of cellular proliferation are associated with low GSH content and enhanced sensitivity of Ehrlich ascites-tumour (EAT) cells to the cytotoxic effects of recombinant human tumour necrosis factor (rhTNF-alpha). Buthionine sulphoximine, a selective inhibitor of GSH synthesis, inhibited tumour growth and increased rhTNF-alpha cytoxicity in vitro. Administration of sublethal doses (10(6)units/kg per day) of rhTNF-alpha to EAT-bearing mice promoted oxidative stress (as measured by increases in intracellular peroxide levels, O2(-); generation and mitochondrial GSSG) and resulted in a slight reduction (19%) in tumour cell number when controls showed the highest rate of cellular proliferat…

MaleNecrosisCell SurvivalMice Inbred StrainsBiologyPharmacologymedicine.disease_causeBiochemistrychemistry.chemical_compoundMiceIn vivomedicineTumor Cells CulturedCytotoxic T cellAnimalsHumansCytotoxicityCarcinoma Ehrlich TumorMolecular BiologyButhionine SulfoximineTumor Necrosis Factor-alphaDrug SynergismCell BiologyGlutathioneGlutathioneRecombinant ProteinsKineticschemistryBiochemistryCancer cellmedicine.symptomOxidative stressIntracellularCell DivisionResearch ArticleThe Biochemical journal
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Pharmacokinetic Interaction between Nevirapine and Nortriptyline in Rats: Inhibition of Nevirapine Metabolism by Nortriptyline

2014

ABSTRACTOne of the most frequent comorbidities of HIV infection is depression, with a lifetime prevalence of 22 to 45%. Therefore, it was decided to study a potential pharmacokinetic interaction between the nonnucleoside reverse transcriptase inhibitor nevirapine (NVP) and the tricyclic antidepressant nortriptyline (NT). NVP and NT were administered to rats either orally, intraduodenally, or intravenously, and the changes in plasma levels and pharmacokinetic parameters were analyzed. Experiments with rat and human hepatic microsomes were carried out to evaluate the inhibitory effects of NT on NVP metabolism. NVP plasma concentrations were significantly higher when this drug was coadminister…

MaleNevirapineAnti-HIV AgentsAdministration OralNortriptylineAntidepressive Agents TricyclicPharmacologyPharmacokineticsimmune system diseasesIn vivomedicineAnimalsHumansPharmacology (medical)NevirapineRats WistarBiotransformationPharmacologyDose-Response Relationship DrugReverse-transcriptase inhibitorbusiness.industryvirus diseasesRatsDose–response relationshipInfectious DiseasesArea Under CurveInjections IntravenousMicrosomes LiverMicrosomeReverse Transcriptase InhibitorsNortriptylinebusinessDrug AntagonismDrug metabolismmedicine.drugAntimicrobial Agents and Chemotherapy
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