Search results for "kinetics"

showing 10 items of 2224 documents

Kinetic characterization of mitochondrial complex I inhibitors using annonaceous acetogenins

1999

The NADH:ubiquinone oxidoreductase (complex I) of the mitochondrial respiratory chain is by far the largest and most complicated of the proton-translocating enzymes involved in the oxidative phosphorylation. Many clues regarding the electron pathways from matrix NADH to membrane ubiquinone and the links of this process with the translocation of protons are highly controversial. Different types of inhibitors become valuable tools to dissect the electron and proton pathways of this complex enzyme. Therefore, further knowledge of the mode of action of complex I inhibitors is needed to understand the underlying mechanism of energy conservation. This study presents for the first time a detailed …

UbiquinoneSubmitochondrial ParticlesBiophysicsOxidative phosphorylationBiologyBiochemistryMitochondria HeartLactonesOxidoreductaseRotenoneNAD(P)H Dehydrogenase (Quinone)Mammalian enzymeAnimalsFuransMolecular Biologychemistry.chemical_classificationNADH-Ubiquinone OxidoreductasePlant ExtractsNADKineticsMitochondrial respiratory chainEnzymechemistryBiochemistryCattleAnnonaceous AcetogeninsMitochondrial Complex I
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Heparin-binding protein targeted to mitochondrial compartments protects endothelial cells from apoptosis.

1999

Neutrophil-borne heparin-binding protein (HBP) is a multifunctional protein involved in the progression of inflammation. HBP is stored in neutrophil granules and released upon stimulation of the cells in proximity to endothelial cells. HBP affects endothelial cells in multiple ways; however, the molecular and cellular mechanisms underlying the interaction of HBP with these cells are unknown. Affinity isolation and enzymatic degradation demonstrated that HBP released from human neutrophils binds to endothelial cell-surface proteoglycans, such as syndecans and glypican. Flow cytometry indicated that a significant fraction of proteoglycan-bound HBP is taken up by the endothelial cells, and we …

Umbilical VeinsEndotheliumCell SurvivalNeutrophilsmedia_common.quotation_subjectmedicine.medical_treatmentInflammationApoptosisBiologyFibroblast growth factorLeukotriene B4ArticleChromatography AffinityFlow cytometryParacrine CommunicationLeukocytesmedicineAnimalsHumansInternalizationCells Culturedmedia_commonInflammationmedicine.diagnostic_testHeparinMonocyteGrowth factorBiological TransportGeneral MedicineBlood ProteinsMolecular biologyRecombinant ProteinsMitochondriaN-Formylmethionine Leucyl-PhenylalanineKineticsmedicine.anatomical_structureApoptosisCommentaryTetradecanoylphorbol AcetateProteoglycansEndothelium Vascularmedicine.symptomCarrier ProteinsAntimicrobial Cationic PeptidesThe Journal of clinical investigation
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Enzymatically modified, nonoxidized LDL induces selective adhesion and transmigration of monocytes and T-lymphocytes through human endothelial cell m…

1999

Abstract —Circulating monocytes and T lymphocytes extravasate through the endothelium at sites of developing atheromatous lesions, where they tend to accumulate and mediate the progression of the disease. We have previously demonstrated the presence of an enzymatically degraded, nonoxidized form of LDL (E-LDL) in early human fatty streaks, which possesses major biological properties of an atherogenic lipoprotein. The effects of E-LDL on human endothelial cells have now been studied with respect to adhesion and transmigration of monocytes and T lymphocytes. E-LDL induced a rapid and dose-dependent selective adhesion of monocytes and T lymphocytes to endothelial cell monolayers within 30 min…

Umbilical VeinsP-selectinArteriosclerosisT-LymphocytesIntercellular Adhesion Molecule-1HL-60 CellsBiologyMonocytesMuscle Smooth VascularCell MovementE-selectinmedicineCell AdhesionHumansLymphocyte homing receptorCell adhesionDose-Response Relationship DrugMonocyteT lymphocyteCholesterol LDLIntercellular Adhesion Molecule-1Molecular biologyEndothelial stem cellLipoproteins LDLPlatelet Endothelial Cell Adhesion Molecule-1KineticsP-Selectinmedicine.anatomical_structureImmunologybiology.proteinlipids (amino acids peptides and proteins)Endothelium VascularCardiology and Cardiovascular MedicineE-SelectinArteriosclerosis, thrombosis, and vascular biology
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Association of AUUUA-binding Protein with A + U-rich mRNA during nucleo-cytoplasmic transport

1992

Resealed nuclear envelope (NE) vesicles from rat liver containing entrapped exogenous RNA were used to study the effect of adenosine+uridine binding factor (AUBF), present in cytosolic cell extracts, on ATP-dependent transport of A+U-rich RNA (AU+RNA) and A+U-free RNA (AU-RNA) across the NE. This factor specifically binds to A+U-rich sequences present in the 3' untranslated regions of lymphokine and cytokine mRNAs, containing overlapping AUUUA boxes (granulocyte-macrophage colony stimulating factor, interleukin-3). Addition of AUBF to the extravesicular compartment markedly increased the efflux of the in vitro transcribed, capped and polyadenylated AU+ RNAs. Export of entrapped AU- control …

Untranslated regionCytoplasmAdenosineTranscription GeneticPolyadenylationNuclear EnvelopeMolecular Sequence DataRNA-binding proteinBiologyCell LineStructural BiologyTranscription (biology)EndoribonucleasesAnimalsHumansNuclear MatrixRNA MessengerBinding siteNuclear export signalUridineMolecular BiologyCell NucleusMessenger RNABinding SitesBase SequenceGranulocyte-Macrophage Colony-Stimulating FactorInterferon-alphaRNA-Binding ProteinsRNAMolecular biologyRatsKineticsLiverRibonucleoproteinsInterleukin-3Carrier ProteinsPlasmidsPolyribonucleotidesProtein BindingJournal of Molecular Biology
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Interaction of valproic acid and some analogues with microsomal epoxide hydrolase.

1992

Abstract Valproic acid (VPA) and its analogues valpromide (VPM), valproyl-Coenzyme A (VP-CoA) and valproyl-ethylester (VPE) were examined as potential inhibitors of microsomal epoxide hydrolase (mEHb) using styrene-7,8-oxide (STO) and benzo(a)pyrene-4,5-oxide (BPO) as enzyme substrates. The effect of each potential inhibitor was examined using mEHb from rat liver, human livers (from a child, woman and man) and from human placenta. Of the compounds tested, only VPM (2 mM) expressed significant inhibition of mEHb activity with a maximum inhibition of 49%, 48%, 35% and 33% for liver microsomes from the child, woman, man and rat, respectively, using STO (2 mM) as substrate. Human placenta mEHb …

ValpromideAdultMalePharmacologyBiochemistrymedicineAnimalsHumansEpoxide hydrolasePharmacologychemistry.chemical_classificationEpoxide HydrolasesBinding SitesbiologyDose-Response Relationship DrugValproic AcidRats Inbred StrainsMiddle Agedbiology.organism_classificationRatsKineticsEnzymeBiochemistryMicrosomachemistryMechanism of actionEnzyme inhibitorMicrosomal epoxide hydrolaseChild PreschoolMicrosomebiology.proteinMicrosomes LiverFemalemedicine.symptommedicine.drugBiochemical pharmacology
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Hypoxia-stimulated expression of angiogenic growth factors in cervical cancer cells and cervical cancer-derived fibroblasts

2001

It is generally accepted that local growth of solid tumors and their ability to establish distant metastases are dependent on the formation of new blood vessels arising from preexisting ones (angiogenesis). The angiogenic response of the host is mediated by angiogenic molecules that are released from cancer and normal stroma cells, especially fibroblasts. The goal of the present study was to quantitatively compare the expression of the two most important angiogenic growth factors (VEGF, angiogenin) of cervical cancer cells (HeLa and Me-180) with that of cervical cancer-derived fibroblasts (from one tumor/patient) under defined normoxic and hypoxic conditions in vitro. The growth kinetics of…

Vascular Endothelial Growth Factor APathologymedicine.medical_specialtyStromal cellAngiogeninAngiogenesismedicine.medical_treatmentCellUterine Cervical NeoplasmsEnzyme-Linked Immunosorbent AssayEndothelial Growth FactorsHeLamedicineHumansHypoxiaLymphokinesNeovascularization PathologicbiologyVascular Endothelial Growth FactorsGrowth factorObstetrics and GynecologyRibonuclease PancreaticFibroblastsbiology.organism_classificationIn vitroGene Expression Regulation NeoplasticKineticsmedicine.anatomical_structureOncologyCell cultureCancer researchFemaleCell DivisionHeLa CellsInternational Journal of Gynecological Cancer
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Interaction of endothelial cells and neutrophilsin vitro: kinetics of thrombomodulin, intercellular adhesion molecule-1 (ICAM-1), E-selectin, and vas…

1999

SUMMARYRecently markers of endothelial cell activation or injury gained increasing interest as serological parameters of disease activation in vasculitides. Among these, soluble serum thrombomodulin, ICAM-1, VCAM-1 and E-selectin are of particular interest. However, only thrombomodulin showed the expected close correlation. The objective of this study was to investigate in vitro the kinetics of these endothelial cell receptors after interaction of unstimulated or cytokine-activated polymorphonuclear neutrophils (PMN) and endothelial cells in order to find evidence explaining these different clinical findings. Over the time period of up to 48 h of incubation the kinetics of thrombomodulin, I…

VasculitisNeutrophilsThrombomodulinImmunologyIntercellular Adhesion Molecule-1Vascular Cell Adhesion Molecule-1BiologyThrombomodulinAutoimmune Diseaseschemistry.chemical_compoundCell–cell interactionE-selectinHumansImmunology and AllergyVascular DiseasesVCAM-1ICAM-1Cell adhesion moleculeIntercellular Adhesion Molecule-1Coculture TechniquesEndothelial stem cellKineticsSolubilitychemistryImmunologycardiovascular systembiology.proteinEndothelium VascularE-SelectinBiomarkersClinical and Experimental Immunology
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Effects of tetraethylammonium ions on frequency-dependent vasopressin release from the rat neurohypophysis.

1988

1. Isolated rat neurohypophyses were fixed by their stalks to a platinum wire electrode and superfused with oxygenated Krebs-HEPES solution. Vasopressin release into the medium was determined by radioimmunoassay. Vasopressin secretion was increased by electrical stimulation at different frequencies (3-30 Hz) and different train lengths (75-900 pulses). The effects of tetraethylammonium (TEA) ions and of enhanced calcium were tested. 2. Electrical stimulation at 7.5 or 15 Hz evoked a markedly larger release of vasopressin than stimulation at 3 Hz. During continuous stimulation at 7.5 and 15 Hz the evoked vasopressin release per pulse declined rapidly, but with similar time constants for both…

Vasopressinmedicine.medical_specialtyTime FactorsPhysiologychemistry.chemical_elementStimulationStimulus (physiology)CalciumIn Vitro Techniqueschemistry.chemical_compoundPituitary Gland PosteriorInternal medicinemedicineExtracellularAnimalsTetraethylammoniumChemistryTetraethylammoniumRadioimmunoassayRats Inbred StrainsTetraethylammonium CompoundsElectric StimulationRatsArginine VasopressinKineticsEndocrinologyVasopressin secretionCalciumFemaleResearch ArticleThe Journal of physiology
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Sprint Acceleration Mechanics in Fatigue Conditions: Compensatory Role of Gluteal Muscles in Horizontal Force Production and Potential Protection of …

2018

Aim: Hamstring muscle injury is the main injury related to sports requiring sprint acceleration. In addition, hamstring muscles have been reported to play a role in horizontal force production during sprint acceleration performance. The aim of the present study was to analyze (i) the determinants of horizontal force production and (ii) the role of hip extensors, and hamstring muscles in particular, for horizontal force production during repeated sprint-induced fatigue conditions. Method: In this experimental laboratory setting study including 14 sprint-trained male athletes, we analyzed (i) the changes in sprint mechanics, peak torque of the knee and hip extensors and flexors, muscle activi…

Vastus lateralis musclePhysiologymuscle[SDV]Life Sciences [q-bio]sprint kineticsConcentricBicepslcsh:Physiology03 medical and health sciences0302 clinical medicinehamstringPhysiology (medical)medicinerisk factorsTreadmillGluteal musclesComputingMilieux_MISCELLANEOUSOriginal Research030222 orthopedicslcsh:QP1-981business.industrysports injury prevention030229 sport sciencesMechanicsmusculoskeletal systemSagittal planegluteus maximusmedicine.anatomical_structureSprintbusinesshuman activitiesHamstringperformanceFrontiers in physiology
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Phosphatidylinositol phosphate kinase type I gamma regulates dynamics of large dense-core vesicle fusion.

2005

Phosphatidylinositol-4,5-bisphosphate was proposed to be an important regulator of large dense-core vesicle exocytosis from neuroendocrine tissues. Here, we have examined the kinetics of secretion in chromaffin cells from mice lacking phosphatidylinositol phosphate kinase type Iγ, the major neuronal phosphatidylinositol-4-phosphate 5-kinase. Absence of this enzyme caused a reduction of the readily releasable vesicle pool and its refilling rate, with a small increase in morphologically docked vesicles, indicating a defect in vesicle priming. Furthermore, amperometry revealed a delay in fusion pore expansion. These results provide direct genetic evidence for a key role of phosphatidylinositol…

Vesicle fusionChromaffin CellsBiologyIn Vitro TechniquesMembrane FusionExocytosisExocytosischemistry.chemical_compoundMiceAnimalsPhosphatidylinositolCells CulturedMultidisciplinaryVesicleSecretory VesiclesSNAP25Munc-18Kiss-and-run fusionBiological SciencesSecretory VesicleCell biologyKineticsMicroscopy ElectronPhosphotransferases (Alcohol Group Acceptor)chemistryCalciumProceedings of the National Academy of Sciences of the United States of America
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