Search results for "lcsh:Immunologic diseases. Allergy"

showing 10 items of 238 documents

Resveratrol-induced xenophagy promotes intracellular bacteria clearance in intestinal epithelial cells and macrophages

2019

International audience; Autophagy is a lysosomal degradation process that contributes to host immunity by eliminating invasive pathogens and the modulating inflammatory response. Several infectious and immune disorders are associated with autophagy defects, suggesting that stimulation of autophagy in these diseases should be bene ficial. Here, we show that resveratrol is able to boost xenophagy, a selective form of autophagy that target invasive bacteria. We demonstrated that resveratrol promotes in vitro autophagy-dependent clearance of intracellular bacteria in intestinal epithelial cells and macrophages. These results were validated in vivo using infection in a transgenic GFP-LC3 zebra f…

Salmonella typhimuriumrestrictionResveratrolresveratrolMicechemistry.chemical_compound0302 clinical medicine[SDV.IDA]Life Sciences [q-bio]/Food engineeringImmunologieXenophagyImmunology and AllergyIntestinal MucosaZebrafishOriginal Research0303 health sciencessalmonella infectionbiologyChemistrycrohns-disease[SDV.IDA] Life Sciences [q-bio]/Food engineering3. Good healthCell biologyrégime alimentaire030220 oncology & carcinogenesisHost-Pathogen InteractionsAIEClcsh:Immunologic diseases. AllergyautophagysalmonelleTransgenesalmonellaImmunologyautophagieCell Line03 medical and health sciencesImmune systemxenophagyEscherichia coliAnimalsHumans030304 developmental biologyselective autophagyhealthy-volunteersmodelEnterocolitisMacrophagesIntracellular parasiteAutophagylife-span extensionautophagy;resveratrol;xenophagy;salmonella;AIECagent resveratrolEpithelial Cellsbiology.organism_classification[SDV.MP.BAC]Life Sciences [q-bio]/Microbiology and Parasitology/BacteriologyCell cultureactivation[SDV.MP.BAC] Life Sciences [q-bio]/Microbiology and Parasitology/Bacteriologyproteinlcsh:RC581-607Bacteria
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Smoking, health and ageing.

2008

Abstract On March 19, 2008 a Symposium on Pathophysiology of Ageing and Age-Related diseases was held in Palermo, Italy. Here, the lecture of V. Nicita-Mauro on Smoking, health and ageing is summarized. Smoking represents an important ageing accelerator, both directly by triggering ana inflammatory responses, and indirectly by favoring the occurrence of several diseases where smoking is a recognized risk factor. Hence, non-smokers can delay the appearance of diseases and of ageing process, so attaining longevity.

Settore MED/04 - Patologia GeneraleGerontologylcsh:Immunologic diseases. AllergyAgingmedicine.medical_specialtyGeriatrics gerontologybusiness.industrymedia_common.quotation_subjectNicotine patchmedicine.medical_treatmentPublic healthImmunologyLongevityShort Reportlcsh:GeriatricsNicotine replacement therapyAgeinglcsh:RC952-954.6AgeingMedicineSmoking health ageingRisk factorbusinesslcsh:RC581-607media_commonImmunityageing : IA
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Beliefs and preferences regarding biological treatments for severe asthma

2020

Background: Severe asthma is a serious condition with a significant burden on patients' morbidity, mortality, and quality of life. Some biological therapies targeting the IgE and interleukin-5 (IL5) mediated pathways are now available. Due to the lack of direct comparison studies, the choice of which medication to use varies. We aimed to explore the beliefs and practices in the use of biological therapies in severe asthma, hypothesizing that differences will occur depending on the prescribers’ specialty and experience. Methods: We conducted an online survey composed of 35 questions in English. The survey was circulated via the INterasma Scientific Network (INESNET) platform as well as throu…

Severe asthmaBehavior; Belief; Biological drug; Severe asthmainterleukine 5 IgEMULTICENTERDiseaseOmalizumabADD-ON THERAPYBehavior Belief Biological drug Severe asthmaDOUBLE-BLIND0302 clinical medicineMedicine and Health SciencesImmunology and AllergyEosinophil IL5 [Eos]030223 otorhinolaryngologyPulmonologistssevere asthma biological drug belief behavior3. Good healthBeliefSAFETYmedicine.druglcsh:Immunologic diseases. AllergyPulmonary and Respiratory Medicinemedicine.medical_specialtyEos: Eosinophil IL5ImmunologyBiological drugSpecialtylong-acting beta2-agonist OMASocio-culturaleSettore MED/10 - Malattie Dell'Apparato RespiratorioArticle03 medical and health sciencesQuality of life (healthcare)Omalizumab. OMA/IL5 Omalizumab plus anti-IL5 moleculeOMALIZUMABmedicineIntensive care medicineAsthmaBehaviorINterasma Scientific Network LABAbusiness.industryMEPOLIZUMABmedicine.diseaseEFFICACYICS inhaled corticosteroidsLIFE030228 respiratory systemImmunoglobulin E INESNETAllergistslcsh:RC581-607businessMepolizumab
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Paenibacillus larvae Chitin-Degrading Protein PlCBP49 Is a Key Virulence Factor in American Foulbrood of Honey Bees

2014

Paenibacillus larvae, the etiological agent of the globally occurring epizootic American Foulbrood (AFB) of honey bees, causes intestinal infections in honey bee larvae which develop into systemic infections inevitably leading to larval death. Massive brood mortality might eventually lead to collapse of the entire colony. Molecular mechanisms of host-microbe interactions in this system and of differences in virulence between P. larvae genotypes are poorly understood. Recently, it was demonstrated that the degradation of the peritrophic matrix lining the midgut epithelium is a key step in pathogenesis of P. larvae infections. Here, we present the isolation and identification of PlCBP49, a mo…

Veterinary MicrobiologyChitinPathogenesisPathology and Laboratory MedicineVirulence factorchemistry.chemical_compoundMedicine and Health SciencesPeritrophic matrixlcsh:QH301-705.5biologyVirulenceGram Positive BacteriaBeesVeterinary BacteriologyBacterial PathogensVeterinary DiseasesMedical MicrobiologyLarvaHost-Pathogen InteractionsPaenibacillusResearch Articlelcsh:Immunologic diseases. Allergy570American foulbroodVirulence FactorsImmunologyMolecular Sequence DataVirulenceMicrobiologyMicrobiologyChitinBacterial ProteinsVirologyGeneticsAnimalsAmino Acid SequenceMolecular BiologyMicrobial PathogensGram-Positive Bacterial InfectionsSequence Homology Amino AcidfungiBiology and Life SciencesMidgutBacteriologyHoney beebiology.organism_classificationlcsh:Biology (General)chemistryProteolysisParasitologyVeterinary Sciencelcsh:RC581-607BacteriaPLoS Pathogens
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Effects of a structured educational intervention in moderate-to-severe elderly asthmatic subjects.

2019

Background: Adherence to inhaled drugs is linked to patients’ satisfaction with their device, and an incorrect use can negatively affect the outcomes of asthma treatment. We speculated that this is particularly true in elderly asthmatic subjects. Aim: We performed a national pre-post interventional multicentre study, enrolling moderate-to-severe asthmatic subjects aged ≥65 ​years treated with fixed inhaled combination drugs by dry powder inhaler (DPI) or pressurized metered dose inhaler (pMDI). Adherence and critical errors were evaluated by means of validated questionnaires at first visit (V1) and after 3–6 months (V2). At V1, subjects underwent intensive training on the correct use of the…

Vital capacityACT asthma control testExacerbationSAE severe asthma exacerbationSF12chlorofluorocarbonsmental health composite scoreEDUCA elderly and device use in chronic asthmaPCS physical health composite scoreACT asthma control test; AHDS hospital anxiety depression scale; Asthma; CFC chlorofluorocarbons; Device misuse; EDUCA elderly and device use in chronic asthma; Education; Elderly; FEV1 forced expiratory volume 1s second; FVC forced vital capacity; ICS inhaled corticosteroids; LABA long-acting β2 agonist; LAMA long-acting muscarinic antagonists; MCS mental health composite score; PCS physical health composite score; PROs patient-reported outcomes; SAE severe asthma exacerbation; SF12 short form health survey; mMRC modified medical research councilEDUCAFEV10302 clinical medicineElderlyImmunology and AllergyMedicine030223 otorhinolaryngologybiologymMRC modified medical research councilsevere asthma exacerbationmodified medical research councilLAMASAELamaelderly and device use in chronic asthmaMetered-dose inhalerDry-powder inhalerDevice misusePROs patient-reported outcomesCFCPCSpatient-reported outcomesmMRClcsh:Immunologic diseases. AllergyPulmonary and Respiratory Medicinelong-acting β2 agonistmedicine.medical_specialtyImmunologyLABAphysical health composite scoreArticleLAMA long-acting muscarinic antagonistsAHDSEducation03 medical and health sciencesFEV1/FVC ratiolong-acting muscarinic antagonistsforced vital capacitySF12 short form health surveyAsthma; Device misuse; Education; ElderlyInternal medicineMCS mental health composite scoreforced expiratory volume 1s secondAsthmabusiness.industryFEV1 forced expiratory volume 1s secondInhalerasthma control testbiology.organism_classificationmedicine.diseaseCFC chlorofluorocarbonsACTICS inhaled corticosteroidsFVCAsthmaMCShospital anxiety depression scale030228 respiratory systemICSAHDS hospital anxiety depression scaleFVC forced vital capacityPROsshort form health surveyinhaled corticosteroidslcsh:RC581-607businessLABA long-acting β2 agonistThe World Allergy Organization journal
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IL-17 and TNF-α Are Key Mediators of Moraxella catarrhalis Triggered Exacerbation of Allergic Airway Inflammation

2017

Alterations of the airway microbiome is often associated with pulmonary diseases. For example, detection of the bacterial pathogen Moraxella catarrhalis in the upper airways is linked with an increased risk to develop or exacerbate asthma. However, the mechanisms by which M. cattarhalis augments allergic airway inflammation (AAI) remains unclear. We here characterized the cellular and soluble mediators of M. catarrhalis triggered excacerbation of AAI in wt and IL-17 deficient as well as in animals treated with TNF-alpha and IL-6 neutralizing antibodies. We compared the type of inflammatory response in M. catarrhalis infected, HDM-allergic and animals infected with M. catarrhalis at differen…

exacerbation of allergic reactionslcsh:Immunologic diseases. Allergy0301 basic medicineExacerbationT cellImmunologyexacerbation of pulmonary inflammationpulmonary inflammationMoraxella catarrhalisAllergic sensitization03 medical and health sciences0302 clinical medicineMoraxellaceae infectionsinfection and allergyImmunology and AllergyMedicineSensitizationOriginal Researchbiologybusiness.industrymicrobial exacerbation of pulmonary inflammationbiology.organism_classificationIL-17030104 developmental biologymedicine.anatomical_structureTNF-αImmunologyTumor necrosis factor alphaInterleukin 17lcsh:RC581-607businessAirway030215 immunologyFrontiers in Immunology
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Anti-inflammatory effects of pre-seasonal Th1-adjuvant vaccine to Parietaria judaica in asthmatics

2011

Nicola Scichilone, Chiara Minaldi, Roberta Santagata, Salvatore Battaglia, Gaetana Camarda, Vincenzo Bellia Dipartimento Biomedico di Medicina Interna e Specialistica (Di.Bi.M.I.S.), Sezione di Pneumologia, University of Palermo, Palermo, ItalyBackground: The ultra-short course pre-seasonal allergy vaccine, containing appropriate allergoids with the adjuvant monophosphoryl lipid A (MPL), may be effective in treating allergic symptoms. Objective: To explore the timing of the immunological responses to the pre-seasonal allergy vaccine.Methods: Four subcutaneous injections of the active product (Pollinex Quattro) were administered to 20 Parietaria-sensitive intermittent asthmatics (M/F: 12/8; …

exhaled breath condensatelcsh:Immunologic diseases. AllergyPulmonary and Respiratory MedicinePediatricsmedicine.medical_specialtymedicine.drug_classmedicine.medical_treatmentSettore MED/10 - Malattie Dell'Apparato Respiratoriomedicine.disease_causeAnti-inflammatoryAllergenJournal of Asthma and AllergymedicineImmunology and AllergyExhaled breath condensateOriginal ResearchAsthmabiologybusiness.industryasthmabiology.organism_classificationmedicine.diseaseinflammationpollenallergen asthma immunotherapy inflammation pollen exhaled breath condensateImmunologyParietaria judaicaimmunotherapylcsh:RC581-607businessAdjuvantallergenJournal of Asthma and Allergy
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Transfer of Immunity from Mother to Offspring Is Mediated via Egg-Yolk Protein Vitellogenin.

2015

Insect immune systems can recognize specific pathogens and prime offspring immunity. High specificity of immune priming can be achieved when insect females transfer immune elicitors into developing oocytes. The molecular mechanism behind this transfer has been a mystery. Here, we establish that the egg-yolk protein vitellogenin is the carrier of immune elicitors. Using the honey bee, Apis mellifera, model system, we demonstrate with microscopy and western blotting that vitellogenin binds to bacteria, both Paenibacillus larvae – the gram-positive bacterium causing American foulbrood disease – and to Escherichia coli that represents gram-negative bacteria. Next, we verify that vitellogenin bi…

honey beestrans-generational immunityEgg proteinmedicine.disease_causebakteeritchemistry.chemical_compoundVitellogeninsbacterial pathogensimmuniteettibacterialcsh:QH301-705.5biologyfood and beveragesBees3. Good healthCell biologyFemaleVitellogeninsResearch Articlelcsh:Immunologic diseases. Allergyfood.ingredientanimal structuresImmunologyBlotting WesternMicrobiologyVitellogeninfoodImmune systemImmunityVirologyYolkGeneticsmedicineAnimalsMolecular BiologyEscherichia coliOvumfungiEgg Proteinsta1182Surface Plasmon Resonanceimmunitylcsh:Biology (General)chemistryImmunologybiology.proteinta1181bacteriaParasitologyPeptidoglycanlcsh:RC581-607vitellogeninPLoS pathogens
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NPM-ALK-reactive T-cell responses in children and adolescents with NPM-ALK positive anaplastic large cell lymphoma

2019

ABSTRACT The oncoantigen nucleophosmin-anaplastic lymphoma kinase (NPM-ALK) induces cellular and humoral immune responses in patients with NPM-ALK-positive anaplastic large cell lymphoma (ALCL). We characterize the NPM-ALK-specific T-cell responses in a cohort of pediatric and adolescent ALCL-patients in remission without Human Leucocyte Antigen (HLA)-preselection. First, we assessed NPM-ALK-reactive T-cell responses and their HLA-class I restriction in patients by using dendritic cells (DCs) transfected with in vitro transcribed (IVT) NPM-ALK-RNA for CD8 (n = 20) or CD3 (n = 9) T-cell stimulation. NPM-ALK-specific T-cells were detected in twelve of 29 patients (nine of 20 with CD8-selected…

lcsh:Immunologic diseases. Allergy0301 basic medicineALK-Positive Anaplastic Large Cell LymphomaT cellImmunologylcsh:RC254-282IFN-γ ELISPOT03 medical and health sciences0302 clinical medicineImmune systemimmune system diseaseshemic and lymphatic diseasesmedicineImmunology and AllergyIn patientNPM-ALKImmune responseAnaplastic large-cell lymphomaOriginal Researchintegumentary systemKinasebusiness.industryT-cellslcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogensmedicine.diseaseALCL3. Good healthLymphomaOncoantigen030104 developmental biologymedicine.anatomical_structureOncology030220 oncology & carcinogenesisCancer researchlcsh:RC581-607businessOncoImmunology
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Human Memory Th17 Cell Populations Change Into Anti-inflammatory Cells With Regulatory Capacity Upon Exposure to Active Vitamin D

2019

Autoimmune diseases are characterized by an aberrantly activated immune system, resulting in tissue damage and functional disability in patients. An important therapeutic goal is to restore the deregulated immunological balance between pro- A nd anti-inflammatory T cells. This imbalance is illustrated by elevated levels and activity of memory Th17 cell populations, such as Th17, Th1/Th17, and Th17.1 cells, in various autoimmune diseases. These cells are characterized by the chemokine receptor CCR6, RORC expression and production of IL-17A, IFNγ, and TNFα. Using rheumatoid arthritis (RA) as a model of autoimmune disease, we here demonstrate that pro-inflammatory memory CCR6+ Th cells can swi…

lcsh:Immunologic diseases. Allergy0301 basic medicineAdultMaleReceptors CCR6rheumatoid arthritisCD3 ComplexCD3CellImmunologyAnti-Inflammatory Agentschemical and pharmacologic phenomenavitamin DC-C chemokine receptor type 6Autoimmune DiseasesArthritis Rheumatoid03 medical and health sciencesChemokine receptor0302 clinical medicineImmune systemsynovial fluidRAR-related orphan receptor gammamedicineImmunology and AllergyHumansCells CulturedOriginal ResearchAutoimmune diseasebiologyChemistryTumor Necrosis Factor-alphaInterleukinsMiddle Agedmedicine.diseaseTreg030104 developmental biologymedicine.anatomical_structureImmunologybiology.proteinTh17 CellsTumor necrosis factor alphaFemaleTh17lcsh:RC581-607Immunologic Memory030215 immunologyFrontiers in Immunology
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