Search results for "lcsh:Pathology"

showing 10 items of 44 documents

Cellular Senescence and Inflammaging in Age-Related Diseases

2018

0301 basic medicineAgingArticle SubjectImmunologyCellular senescenceCell BiologyBiologymedicine.disease_causeBioinformatics03 medical and health sciencesOxidative Stress030104 developmental biologyEditorialAge relatedmedicinelcsh:PathologyAnimalsHumansSettore MED/05 - Patologia ClinicaCellular Senescence Inflammaging Age-Related DiseasesOxidative stressCellular Senescencelcsh:RB1-214Mediators of Inflammation
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Rabphilin involvement in filtration and molecular uptake in Drosophila nephrocytes suggests a similar role in human podocytes

2020

ABSTRACT Drosophila nephrocytes share functional, structural and molecular similarities with human podocytes. It is known that podocytes express the rabphilin 3A (RPH3A)-RAB3A complex, and its expression is altered in mouse and human proteinuric disease. Furthermore, we previously identified a polymorphism that suggested a role for RPH3A protein in the development of urinary albumin excretion. As endocytosis and vesicle trafficking are fundamental pathways for nephrocytes, the objective of this study was to assess the role of the RPH3A orthologue in Drosophila, Rabphilin (Rph), in the structure and function of nephrocytes. We confirmed that Rph is required for the correct function of the en…

0301 basic medicineEndocytic cycle030232 urology & nephrologyRetinoic acidlcsh:MedicineMedicine (miscellaneous)Labyrinthine channelschemistry.chemical_compound0302 clinical medicineImmunology and Microbiology (miscellaneous)Chronic kidney diseaseDrosophila ProteinsSlit diaphragmGene knockdownPodocytesIntracellular Signaling Peptides and ProteinsDrosophila nephrocyteEndocytosisCell biologyProtein TransportDrosophila melanogasterLarvaSlit diaphragmFemaleRNA InterferenceEndocytic pathwaylcsh:RB1-214Research ArticleEndosomeNeuroscience (miscellaneous)Nerve Tissue ProteinsTretinoinCell fate determinationBiologyEndocytosisGeneral Biochemistry Genetics and Molecular Biology03 medical and health scienceslcsh:PathologyRabphilinAnimalsHumansCell Lineagelcsh:RCytoplasmic VesiclesDrosCubilinSurvival Analysis030104 developmental biologychemistrySilver NitrateDisease Models & Mechanisms
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Anti-Inflamm-Ageing and/or Anti-Age-Related Disease Emerging Treatments: A Historical Alchemy or Revolutionary Effective Procedures?

2018

The “long-life elixir” has long represented for humans a dream, a vanity’s sin for remaining young and to long survive. Today, because of ageing population phenomenon, the research of antiageing interventions appears to be more important than ever, for preserving health in old age and retarding/or delaying the onset of age-related diseases. A hope is given by experimental data, which evidence the possibility of retarding ageing in animal models. In addition, it has been also demonstrated in animal life-extending studies not only the possibility of increasing longevity but also the ability to retard the onset of age-related diseases. Interestingly, this recent evidence is leading to promise …

0301 basic medicineGerontologyPopulation ageingAgingmedia_common.quotation_subjectImmunologyReview ArticleRegenerative medicineAlchemy03 medical and health sciencesImmunology; Cell Biologylcsh:PathologySettore MED/05 - Patologia ClinicaMedicineAnimalsHumansmedia_commonInflammationbusiness.industryLongevityCell Biology030104 developmental biologyAgeingModels AnimalbusinessAge related diseaselcsh:RB1-214Mediators of inflammation
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The Drosophila junctophilin gene is functionally equivalent to its four mammalian counterparts and is a modifier of a Huntingtin poly-Q expansion and…

2018

[EN] Members of the Junctophilin (JPH) protein family have emerged as key actors in all excitable cells, with crucial implications for human pathophysiology. In mammals, this family consists of four members (JPH1-JPH4) that are differentially expressed throughout excitable cells. The analysis of knockout mice lacking JPH subtypes has demonstrated their essential contribution to physiological functions in skeletal and cardiac muscles and in neurons. Moreover, mutations in the human JPH2 gene are associated with hypertrophic and dilated cardiomyopathies; mutations in JPH3 are responsible for the neurodegenerative Huntington's disease-like-2 (HDL2), whereas JPH1 acts as a genetic modifier in C…

0301 basic medicineHuntingtinNotchProtein familyCardiomyopathyNeuroscience (miscellaneous)Notch signaling pathwayMedicine (miscellaneous)lcsh:Medicinemedicine.disease_causeGeneral Biochemistry Genetics and Molecular Biology03 medical and health sciencesImmunology and Microbiology (miscellaneous)JPH2BIOQUIMICA Y BIOLOGIA MOLECULARHuntingtin Proteinmedicinelcsh:PathologyGeneticsMutationbiologylcsh:RHuntington's diseasebiology.organism_classification030104 developmental biologyJunctophilinDrosophilaDrosophila melanogasterDrosophila Proteinlcsh:RB1-214Disease Models & Mechanisms
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IL-33/IL-31 Axis: A Potential Inflammatory Pathway

2018

Cytokines play an important role in the regulation of the immune system (adaptive and innate). Given their importance in proinflammatory processes, cytokines have been used for understanding the pathogenesis and as biomarkers in many diseases. IL-31 and IL-33 are still considered novel cytokines. IL-31 controls signalling and regulates a huge amount of biological functions: it induces proinflammatory cytokines, regulates cell proliferation, and is involved also in tissue remodelling. On the other hand, IL-33 has been identified as an “alarmin” released from the epithelial cells and from different human tissues and organs after a damage following, that is, an inflammatory process. The aim of…

0301 basic medicineIL-33/IL-31 AxiImmunologyInflammationReview ArticleAdaptive ImmunityBiologyProinflammatory cytokinePathogenesis03 medical and health sciences0302 clinical medicineImmune systemSettore MED/43 - Medicina Legalelcsh:PathologymedicineAnimalsHumansIL-33/IL-31 Axis; Inflammatory PathwayInflammationAdaptive Immunity; Animals; Biomarkers; Humans; Inflammation; Interleukin-33; Interleukins; Immunology; Cell BiologyCell growthInterleukinsInterleukinCell BiologyInterleukin-33Acquired immune systemInflammatory PathwayInterleukin 33030104 developmental biologyImmunologymedicine.symptomBiomarkerslcsh:RB1-214030215 immunology
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Psammomatoid Calcification-Rich Oncocytic Thyroid Adenoma With Clear-Cell Change

2018

Abundant calcifications and clear-cell change may occur in oncocytic thyroid adenoma (OTA). We aimed to report the features of a case of OTA peculiar by the presence of extensive calcifications and of a clear-cell component. The OTA was detected on a right thyroid lobectomy specimen. Clear-cell change was observed in 15%-20% of the cells. Numerous, focally confluent psammomatoid calcifications, resulting in macrocalcifications, were identified. The nodule cells, uni- or multinucleated oncocytes, including those with clear cytoplasm, expressed heterogeneously TTF1, S100 protein, Bcl2, CD10, as well as CD138 and CD56. Lipid-droplet markers adipophilin and TIP47 were also expressed. In conclus…

0301 basic medicineMicrobiology (medical)oncocytePathologymedicine.medical_specialtyHistologyAdenomanoduleOncocytePathology and Forensic Medicineclear cellcalcification03 medical and health sciences0302 clinical medicinehemic and lymphatic diseasesmedicinelcsh:PathologyThyroidbusiness.industryThyroid adenomaThyroidNodule (medicine)medicine.diseasepsammomatoid/psammoma-like/pseudo-psammomamitochondria030104 developmental biologymedicine.anatomical_structure030220 oncology & carcinogenesisimmunohistochemistryCommentaryImmunohistochemistryadenomamedicine.symptombusinessClear cellCalcificationlcsh:RB1-214Clinical Pathology
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Two different pathogenic mechanisms, dying-back axonal neuropathy and pancreatic senescence, are present in the YG8R mouse model of Friedreich ataxia

2016

Frataxin (FXN) deficiency causes Friedreich's ataxia (FRDA), a multisystem disorder with neurological and non-neurological symptoms. FRDA pathophysiology combines developmental and degenerative processes of dorsal root ganglia (DRG), sensory nerves, dorsal columns and other central nervous structures. A dying-back mechanism has been proposed to explain the peripheral neuropathy and neuropathology. In addition, affected individuals have non-neuronal symptoms such as diabetes mellitus or glucose intolerance. To go further in the understanding of the pathogenic mechanisms of neuropathy and diabetes associated with the disease, we have investigated the humanized mouse YG8R model of FRDA. By bio…

0301 basic medicineNervous systemAgingPathologylcsh:MedicineMedicine (miscellaneous)Mice0302 clinical medicineImmunology and Microbiology (miscellaneous)Ganglia SpinalInsulin-Secreting CellsInsulin SecretionInsulinMuscle spindleDorsal root gangliaCellular SenescenceDiabetisbiologyMusclesDiabetesAnatomyMitochondria3. Good healthmedicine.anatomical_structureSistema nerviós simpàticDying-back neuropathyPeripheral nervous systemCell senescencemedicine.symptomOxidation-Reductionlcsh:RB1-214Research ArticleSenescencemedicine.medical_specialtyAtaxiaNeuroscience (miscellaneous)Friedreich’s ataxiaNeuropathologyGeneral Biochemistry Genetics and Molecular BiologyPàncreesMalalties del sistema nerviós03 medical and health sciencesPeripheral Nervous Systemlcsh:PathologymedicineAnimalsHumansPancreasIslet of Langerhanslcsh:R302Friedreich's ataxiaNervous system Diseasesmedicine.diseaseAxonsMice Inbred C57BLDisease Models Animal030104 developmental biologyPeripheral neuropathyFriedreich AtaxiaSympathetic nervous systemMutationHumanized mouseFrataxinbiology.proteinEnergy Metabolism030217 neurology & neurosurgeryDisease Models & Mechanisms
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Amyotrophic Lateral Sclerosis, a Multisystem Pathology: Insights into the Role of TNFα

2017

Amyotrophic lateral sclerosis (ALS) is considered a multifactorial, multisystem disease in which inflammation and the immune system play important roles in development and progression. The pleiotropic cytokine TNFαis one of the major players governing the inflammation in the central nervous system and peripheral districts such as the neuromuscular and immune system. Changes in TNFαlevels are reported in blood, cerebrospinal fluid, and nerve tissues of ALS patients and animal models. However, whether they play a detrimental or protective role on the disease progression is still not clear. Our group and others have recently reported opposite involvements of TNFR1 and TNFR2 in motor neuron dea…

0301 basic medicinePathologymedicine.medical_specialtyAmyotrophic Lateral Sclerosis; Animals; Humans; Motor Neurons; T-Lymphocytes Regulatory; Tumor Necrosis Factor-alphamedicine.medical_treatmentT-LymphocytesCentral nervous systemImmunologyAmyotrophic lateral sclerosis inflammation TNFαInflammationReview ArticleT-Lymphocytes Regulatory03 medical and health sciences0302 clinical medicineImmune systemCerebrospinal fluidImmunology; Cell BiologyTNFαlcsh:PathologyMedicineAnimalsHumansAmyotrophic lateral sclerosisMotor Neuronsbusiness.industryTumor Necrosis Factor-alphaAmyotrophic Lateral SclerosisCell BiologyMotor neuronmedicine.diseaseRegulatory3. Good health030104 developmental biologymedicine.anatomical_structureCytokineinflammationImmunologyTumor necrosis factor alphamedicine.symptombusiness030217 neurology & neurosurgerylcsh:RB1-214
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Unusual presentation of blastic plasmacytoid dendritic cell neoplasm: Pitfalls in other hematolymphoid neoplasms

2020

Abstract Blastic plasmacytoid dendritic cell neoplasm (BPDCN) is a rare CD4+/CD56+ hematological malignancy with aggressive clinical course and poor prognosis. Histologically, BPDCN is characterized by a diffuse, monomorphous infiltration of cutaneous, subcutaneous, and sometimes other tissues such as lymph nodes and bone marrow, by medium-sized neoplastic cells with blastoid morphology. Typically, there is absence of lymphocytic infiltrate. Diagnosis relies on immunophenotypic expression of CD4, CD56, and the more specific markers of plasmacytoid dendritic cells CD123, CD303/BDCA2, and TCL1. We report a case of a 57-year-old man who presented a 4 cm-long solitary, erythemateous lesion on t…

0301 basic medicinePathologymedicine.medical_specialtyMyeloidMYCASXL1BlastoidPathology and Forensic MedicineLymphocytic Infiltrate03 medical and health sciences0302 clinical medicineBlastic plasmacytoid dendritic cell neoplasmT-cells infiltrationlcsh:PathologyMedicineTET2biologybusiness.industryETV6medicine.diseasebiology.organism_classificationLymphomaETV6030104 developmental biologymedicine.anatomical_structure030220 oncology & carcinogenesisInterleukin-3 receptorBone marrowbusinessInfiltration (medical)lcsh:RB1-214Human Pathology: Case Reports
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Gastroblastoma in Adulthood—A Rarity among Rare Cancers—A Case Report and Review of the Literature

2019

Gastroblastoma (GB) is a rare gastric epithelial-mesenchymal neoplasm, first described by Miettinen et al. So far, all reported cases described the tumor in children or young adults, and similarities with other childhood blastomas have been postulated. We report a case of GB in a 43-year-old patient with long follow up and no recurrence up to 100 months after surgery. So far, this is the second case of GB occurring in the adult age >40-year-old. Hence, GB should be considered in the differential diagnosis of microscopically comparable conditions in adults carrying a worse prognosis and different clinical approach.

0301 basic medicineepithelial-mesenchymal neoplasmPediatricsmedicine.medical_specialtyPathologyGastroblastomabusiness.industryrare biphasic neoplasmMEDLINECase ReportGeneral MedicinegastroblastomaSettore MED/08 - Anatomia PatologicaAdult age03 medical and health sciences030104 developmental biology0302 clinical medicine030220 oncology & carcinogenesislcsh:PathologyMedicineDifferential diagnosisYoung adultbusinesslcsh:RB1-214Case Reports in Pathology
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