Search results for "lcsh:Pathology"

showing 10 items of 44 documents

Glial expression of Swiss cheese (SWS), the Drosophila orthologue of neuropathy target esterase (NTE), is required for neuronal ensheathment and func…

2016

ABSTRACT Mutations in Drosophila Swiss cheese (SWS) or its vertebrate orthologue neuropathy target esterase (NTE), respectively, cause progressive neuronal degeneration in Drosophila and mice and a complex syndrome in humans that includes mental retardation, spastic paraplegia and blindness. SWS and NTE are widely expressed in neurons but can also be found in glia; however, their function in glia has, until now, remained unknown. We have used a knockdown approach to specifically address SWS function in glia and to probe for resulting neuronal dysfunctions. This revealed that loss of SWS in pseudocartridge glia causes the formation of multi-layered glial whorls in the lamina cortex, the firs…

Medicine (miscellaneous)lcsh:MedicineAxonal degenerationSynaptic Transmission0302 clinical medicineImmunology and Microbiology (miscellaneous)Drosophila ProteinsNeurons0303 health sciencesGene knockdownCell Deathmusculoskeletal neural and ocular physiologyPhototaxisAnatomyCell biologymedicine.anatomical_structureDrosophila melanogasterPhospholipasesGene Knockdown TechniquesNeurogliaNeurogliaDrosophila Proteinpsychological phenomena and processesResearch Articlelcsh:RB1-214Programmed cell deathNeuriteNeuroscience (miscellaneous)Nerve Tissue ProteinsNeuropathy target esteraseNeurotransmissionBiologyMotor ActivityGeneral Biochemistry Genetics and Molecular Biology03 medical and health sciencesPNPLA6mental disordersNeuropilmedicineNeuriteslcsh:PathologyAnimalsPhospholipaseCell Shape030304 developmental biologySequence Homology Amino AcidSpastic paraplegialcsh:R302Reproducibility of ResultsEnsheathing gliabody regionsnervous systemVacuolesbiology.proteinCarboxylic Ester Hydrolases030217 neurology & neurosurgeryDisease Models & Mechanisms
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Application of fnbA gene as new target for the species-specific and quantitative detection of Staphylococcus aureus directly from lower respiratory t…

2013

Staphylococcus aureus is a significant cause of hospital-acquired pneumonia (HAP), particularly in mechanically ventilated patients. We used the fibronectin-binding protein A gene (fnbA) for the species-specific and quantitative detection of S. aureus directly from lower respiratory tract (LRT) specimens by a Taq Man real time PCR. For this reason, a total of 269 lower respiratory tract (LRT) specimens collected from patients with hospital-acquired pneumonia were assayed. Amplification of fnbA in serial dilutions ranged from 10(9) CFU/ ml to 10(2) CFU/ml. Standard curve of triplicate every dilution had slope 3.34±0.1 and R2>0.99 with SD 0.1. Based on these data, the sensitivity and specif…

Microbiology (medical)fnbA Gene real time PCR respiratory infection Staphylococcus aureusSettore MED/07 - Microbiologia E Microbiologia ClinicaStaphylococcus aureusSerial dilutionRespiratory Systemlcsh:QR1-502medicine.disease_causeReal-Time Polymerase Chain ReactionSensitivity and SpecificityfnbA Genelcsh:MicrobiologyPathology and Forensic MedicineMicrobiologyrespiratory infectionPneumonia StaphylococcalmedicineTaqManlcsh:PathologyHumansAdhesins BacterialCross InfectionbiologyStaphylococcus. aureusRespiratory infectionGeneral Medicinemedicine.diseasePneumoniareal time PCRmedicine.anatomical_structureReal-time polymerase chain reactionMolecular Diagnostic TechniquesStaphylococcus aureusbiology.proteinProtein ARespiratory tractlcsh:RB1-214Indian journal of pathologymicrobiology
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Development of aDrosophila melanogasterspliceosensor system forin vivohigh-throughput screening in myotonic dystrophy type 1

2014

AbstractAlternative splicing of pre-mRNAs is an important mechanism that regulates cellular function in higher eukaryotes. A growing number of human genetic diseases involve splicing defects that are directly connected to their pathology. In myotonic dystrophy type 1 (DM1), several clinical manifestations have been proposed to be the consequence of tissue-specific missplicing of numerous genes. These events are triggered by an RNA gain-of-function and resultant deregulation of specific RNA-binding factors, such as the nuclear sequestration of muscleblind-like family factors (MBNL1-MBNL3). Thus, the identification of chemical modulators of splicing events could lead to the development of the…

Myotonic dystrophyNeuroscience (miscellaneous)lcsh:MedicineMedicine (miscellaneous)BiologySplicingMyotonic dystrophyGeneral Biochemistry Genetics and Molecular Biologychemistry.chemical_compoundMinigeneImmunology and Microbiology (miscellaneous)lcsh:PathologymedicineAnimalsMBNL1Resource ArticleGeneGeneticsDrug discoverylcsh:RAlternative splicingmedicine.diseasebiology.organism_classificationHigh-Throughput Screening AssaysAlternative SplicingDrosophila melanogasterchemistryIn vivo screeningRNA splicingDrosophila melanogasterLuciferaselcsh:RB1-214MinigeneDisease Models & Mechanisms
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Comprehensive three‐dimensional morphology of neoangiogenesis in pulmonary veno‐occlusive disease and pulmonary capillary hemangiomatosis

2019

Abstract Pulmonary veno‐occlusive disease (PVOD) is a rare lung disease characterized by fibrotic narrowing of pulmonary veins leading to pulmonary hypertension (PH) and finally to death by right heart failure. PVOD is often accompanied by pulmonary capillary hemangiomatosis (PCH), a marked abnormal proliferation of pulmonary capillaries. Both morphological patterns often occur together and are thought to be distinct manifestations of the same disease process and accordingly are classified together in group 1′ of the Nice classification of PH. The underlying mechanisms of these aberrant remodeling processes remain poorly understood. In this study, we investigated the three‐dimensional struc…

Pathologymedicine.medical_specialtyLung NeoplasmsHypertension Pulmonarypulmonary veno‐occlusive diseasePulmonary capillary hemangiomatosis030204 cardiovascular system & hematologypulmonary capillary hemangiomatosisPathology and Forensic Medicine03 medical and health sciencesThree dimensional morphology0302 clinical medicineRight heart failurepulmonary hypertensionmedicinelcsh:PathologyHumansHemangioma CapillaryLungNeovascularization Pathologicbusiness.industryBrief Definitive Reportintussusceptive neoangiogenesismedicine.diseasePulmonary hypertension3. Good healthmedicine.anatomical_structurePulmonary VeinsLung disease030220 oncology & carcinogenesisPulmonary Veno-Occlusive DiseaseImmunohistochemistryPulmonary Veno-Occlusive Diseasebusinesspulmonary vascular remodelinglcsh:RB1-214The Journal of Pathology: Clinical Research
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Tissue microarrays analysis in chondrosarcomas: light microscopy, immunohistochemistry and xenograft study

2008

Abstract Background Chondrosarcoma (Chs) is the third most frequent primary malignant tumour of bone and can be primary or secondary, the latter results mainly from the malignant transformation of a benign pre-existing tumour. Methods All the cases diagnosed as Chs (primary tumours, recurrences and/or metastasis and xenotransplanted Chs) from the files of our Department were collected. Only cases with paraffin blocks available were selected (Total 32 cases). Six Tissue Microarrays (TMAs) were performed and all the cases and biopsies were distributed into the following groups: a) only paraffin block available from primary and/or metastatic tumours (3 TMAs), b) paraffin block available from p…

Pathologymedicine.medical_specialtyTissue microarrayHistologyintegumentary systembusiness.industryCD99General Medicinemedicine.diseaseMalignant transformationStainingMetastasisPathology and Forensic MedicineProceedingshemic and lymphatic diseasesSurvivinlcsh:PathologymedicineImmunohistochemistryChondrosarcomabusinessskin and connective tissue diseaseslcsh:RB1-214Diagnostic Pathology
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Synovial cysts of the temporomandibular joint: an immunohistochemical characterization and literature review.

2013

Synovial cysts of the temporomandibular joint (TMJ) are very rare, and to date, only 12 cases of a synovial cyst in the TMJ region have been reported in the literature. In this paper, we present the clinicopathological and immunohistochemical characteristics of one such lesion affecting a 48-year-old woman, presented with a mass in the left preauricular region. We describe the usefulness of immunohistochemical analysis for recognizing the synovial lining, which allowed for clear differentiation between ganglion and synovial cysts. Immunohistochemical analyses can be used to diagnose synovial cysts with certainty; however, using at least two markers is advisable to distinguish the two existi…

Pathologymedicine.medical_specialtybusiness.industryCase ReportGeneral MedicineOdontologiaPatologiaTemporomandibular jointGanglionArticulacions MalaltiesLesionmedicine.anatomical_structureSynovial Cellstomatognathic systemPreauricular regionmedicinelcsh:PathologySynovial cystImmunohistochemistrymedicine.symptombusinessCàncerSynovial lininglcsh:RB1-214
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Pentamidine rescues contractility and rhythmicity in a Drosophila model of myotonic dystrophy heart dysfunction

2015

Up to 80% of individuals with myotonic dystrophy type 1 (DM1) will develop cardiac abnormalities at some point during the progression of their disease, the most common of which is heart blockage of varying degrees. Such blockage is characterized by conduction defects and supraventricular and ventricular tachycardia, and carries a high risk of sudden cardiac death. Despite its importance, very few animal model studies have focused on the heart dysfunction in DM1. Here, we describe the characterization of the heart phenotype in a Drosophila model expressing pure expanded CUG repeats under the control of the cardiomyocyte-specific driver GMH5-Gal4. Morphologically, expression of 250 CUG repeat…

[SDV]Life Sciences [q-bio]Myotonic dystrophyMedicine (miscellaneous)lcsh:MedicineVentricular tachycardiaImmunology and Microbiology (miscellaneous)DiastoleHeart RateDrosophila ProteinsMyocytes CardiacGeneticsbiologyRNuclear ProteinsHeartPhenotype3. Good healthCell biology[SDV] Life Sciences [q-bio]Drosophila melanogasterPhenotypeDrosophilaDrosophila melanogasterDrosophila ProteinResearch Articlelcsh:RB1-214congenital hereditary and neonatal diseases and abnormalitiesSystoleLongevityNeuroscience (miscellaneous)In situ hybridizationMyotonic dystrophyGeneral Biochemistry Genetics and Molecular BiologyMuscleblindContractilitymedicinelcsh:PathologyAnimalsPentamidineHeart dysfunctionfungilcsh:RArrhythmias Cardiacbiology.organism_classificationmedicine.diseaseMyocardial ContractionSurvival AnalysisDisease Models AnimalTrinucleotide repeat expansionTrinucleotide Repeat Expansion
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DOUBLE TRANSVERSE FORAMEN IN INFANTILE CERVICAL VERTEBRAE OF THE LATE 17TH AND EARLY 18TH CENTURIES. Doble foramen transverso en las vértebras infant…

2016

La presencia de doble foramen transverso es una variante anatómica que se observa con frecuencia en el raquis cervical inferior y que presenta implicaciones clínicas en relación con la arteria vertebral. Aunque en la actualidad existe cierta controversia acerca de si el origen de esta variante cervical es congénito o adquirido, y a pesar de que la presencia de doble foramen transverso en población infantil descartaría un origen degenerativo-artrósico, se encuentra una falta de información acerca de su presencia en vértebras cervicales infantiles. En este sentido, nuestro objetivo ha sido analizar la presencia de doble foramen transverso en vértebras cervicales infantiles procedentes de rest…

childcongenital abnormalitiesbusiness.industryGeography Planning and Developmentlcsh:Rniñolcsh:MedicineAnatomyDevelopmentspineraquismedicine.anatomical_structureSpainForamenlcsh:PathologyMedicinebusinessCervical vertebraelcsh:RB1-214Revista Argentina de Anatomía Clínica
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Progenitor death drives retinal dysplasia and neuronal degeneration in a mouse model of Atrip-Seckel syndrome

2020

ABSTRACT Seckel syndrome is a type of microcephalic primordial dwarfism (MPD) that is characterized by growth retardation and neurodevelopmental defects, including reports of retinopathy. Mutations in key mediators of the replication stress response, the mutually dependent partners ATR and ATRIP, are among the known causes of Seckel syndrome. However, it remains unclear how their deficiency disrupts the development and function of the central nervous system (CNS). Here, we investigated the cellular and molecular consequences of ATRIP deficiency in different cell populations of the developing murine neural retina. We discovered that conditional inactivation of Atrip in photoreceptor neurons …

lcsh:MedicineMedicine (miscellaneous)315BlindnessMicechemistry.chemical_compoundImmunology and Microbiology (miscellaneous)Cell DeathneurodevelopmentStem CellsNeurodegenerationapoptosisneurodegenerationSyndromeCell biologyDNA-Binding Proteinsdna damage responsemedicine.anatomical_structurePhotoreceptor Cells VertebrateResearch Articlelcsh:RB1-214NeurogenesisNeuroscience (miscellaneous)Embryonic DevelopmentBiologyRetinaGeneral Biochemistry Genetics and Molecular Biologylcsh:PathologymedicineAnimalsAbnormalities MultipleProgenitor cellVision OcularAdaptor Proteins Signal TransducingCell ProliferationProgenitorRetinalcsh:RRetinalEmbryo Mammalianmedicine.diseasephotoreceptorDisease Models AnimalSeckel syndromechemistryvisual system developmentNerve DegenerationRetinal dysplasiaRetinal DysplasiaTumor Suppressor Protein p53Primordial dwarfismDNA DamageDisease Models & Mechanisms
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Increased autophagy and apoptosis contribute to muscle atrophy in a myotonic dystrophy type 1 Drosophila model

2015

ABSTRACT Muscle mass wasting is one of the most debilitating symptoms of myotonic dystrophy type 1 (DM1) disease, ultimately leading to immobility, respiratory defects, dysarthria, dysphagia and death in advanced stages of the disease. In order to study the molecular mechanisms leading to the degenerative loss of adult muscle tissue in DM1, we generated an inducible Drosophila model of expanded CTG trinucleotide repeat toxicity that resembles an adult-onset form of the disease. Heat-shock induced expression of 480 CUG repeats in adult flies resulted in a reduction in the area of the indirect flight muscles. In these model flies, reduction of muscle area was concomitant with increased apopto…

lcsh:MedicineMedicine (miscellaneous)Genes InsectApoptosisDystrophyInhibitor of Apoptosis ProteinsAnimals Genetically ModifiedCTG repeat expansion0302 clinical medicineImmunology and Microbiology (miscellaneous)Drosophila ProteinsMyotonic DystrophyMyocyte0303 health sciencesTOR Serine-Threonine KinasesMyotonin-protein kinaseNuclear ProteinsMuscle atrophyUp-RegulationCell biologyMuscular AtrophyDrosophila melanogastermedicine.anatomical_structureFemalemedicine.symptomSignal TransductionResearch Articlelcsh:RB1-214congenital hereditary and neonatal diseases and abnormalitiesProgrammed cell deathNeuroscience (miscellaneous)BiologyMyotonic dystrophyMyotonin-Protein KinaseMuscleblindGeneral Biochemistry Genetics and Molecular Biology03 medical and health sciencesAutophagylcsh:PathologymedicineAnimalsHumans030304 developmental biologylcsh:RAutophagyDystrophySkeletal musclemedicine.diseaseMolecular biologyDisease Models AnimalMuscle atrophyTrinucleotide Repeat Expansion030217 neurology & neurosurgeryDisease Models & Mechanisms
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