Search results for "lcsh:RC321-571"

showing 10 items of 504 documents

Bioenergetic Failure in Rat Oligodendrocyte Progenitor Cells Treated with Cerebrospinal Fluid Derived from Multiple Sclerosis Patients

2017

In relapsing-remitting multiple sclerosis (RRMS) subtype, the patient's brain itself is capable of repairing the damage, remyelinating the axon and recovering the neurological function. Cerebrospinal fluid (CSF) is in close proximity with brain parenchyma and contains a host of proteins and other molecules, which influence the cellular physiology, that may balance damage and repair of neurons and glial cells. The purpose of this study was to determine the pathophysiological mechanisms underpinning myelin repair in distinct clinical forms of MS and neuromyelitis optica (NMO) patients by studying the effect of diseased CSF on glucose metabolism and ATP synthesis. A cellular model with primary…

0301 basic medicineCell physiologyglucose metabolismneuromyelitis opticaTransferrin receptorBiologymultiple sclerosiscerebrospinal fluidlcsh:RC321-571myelin repair03 medical and health sciencesCellular and Molecular NeuroscienceMyelin0302 clinical medicineCerebrospinal fluidGene expressionmedicineAxonlcsh:Neurosciences. Biological psychiatry. NeuropsychiatryOriginal ResearchMultiple sclerosisoligodendrocyte progenitor cellsmedicine.disease3. Good health030104 developmental biologymedicine.anatomical_structureHypoxanthine-guanine phosphoribosyltransferaseImmunologyCancer researchgene expression030217 neurology & neurosurgeryNeuroscienceFrontiers in Cellular Neuroscience
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The FOXP2-Driven Network in Developmental Disorders and Neurodegeneration

2017

The transcription repressor FOXP2 is a crucial player in nervous system evolution and development of humans and songbirds. In order to provide an additional insight into its functional role we compared target gene expression levels between human neuroblastoma cells (SH-SY5Y) stably overexpressing either human FOXP2 cDNA or its orthologues from the common chimpanzee, Rhesus monkey, and marmoset, respectively. Subsequent RNA-seq led to identification of 27 genes with differential regulation under the control of human FOXP2, which were previously reported to have FOXP2-driven and/or songbird song-related expression regulation. Importantly, RT-qPCR and Western blotting indicated differential re…

0301 basic medicineCell signalingCytoskeleton organizationspeechbrainBiologyAxonogenesislcsh:RC321-57103 medical and health sciencesCellular and Molecular NeuroscienceHuntington's diseasemedicineGeneTranscription factorlcsh:Neurosciences. Biological psychiatry. Neuropsychiatryneuronal circuitryOriginal ResearchlanguageNeurodegenerationFOXP2medicine.diseaseschizophrenia030104 developmental biologyParkinson’s diseaseNeuroscienceAlzheimer’s diseaseNeuroscienceHuntington’s diseaseFrontiers in Cellular Neuroscience
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Molecular Mechanism Involved in the Pathogenesis of Early-Onset Epileptic Encephalopathy

2019

Recent studies have shown that neurologic inflammation may both precipitate and sustain seizures, suggesting that inflammation may be involved not only in epileptogenesis but also in determining the drug-resistant profile. Extensive literature data during these last years have identified a number of inflammatory markers involved in these processes of “neuroimmunoinflammation” in epilepsy, with key roles for pro-inflammatory cytokines such as: IL-6, IL-17 and IL-17 Receptor (IL-17R) axis, Tumor-Necrosis-Factor Alpha (TNF-α) and Transforming-Growth-Factor Beta (TGF-β), all responsible for the induction of processes of blood-brain barrier (BBB) disruption and inflammation of the Central Nervou…

0301 basic medicineCentral nervous systemInflammationContext (language use)ReviewEpileptogenesisNOlcsh:RC321-571pathogenic mechanismsPathogenesis03 medical and health sciencesCellular and Molecular NeuroscienceEpilepsy0302 clinical medicineImmune systemMedicinelcsh:Neurosciences. Biological psychiatry. NeuropsychiatryMolecular BiologyPathologicalchildhoodbusiness.industrybiological markermedicine.diseaseepileptic encephalopathy030104 developmental biologymedicine.anatomical_structureinflammationmedicine.symptombusinessNeurosciencebiological markers030217 neurology & neurosurgeryNeurosciencebiological markers epileptic encephalopathy inflammation pathogenic mechanisms childhoodFrontiers in Molecular Neuroscience
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Overview of General and Discriminating Markers of Differential Microglia Phenotypes.

2020

Inflammatory processes and microglia activation accompany most of the pathophysiological diseases in the central nervous system. It is proven that glial pathology precedes and even drives the development of multiple neurodegenerative conditions. A growing number of studies point out the importance of microglia in brain development as well as in physiological functioning. These resident brain immune cells are divergent from the peripherally infiltrated macrophages, but their precise in situ discrimination is surprisingly difficult. Microglial heterogeneity in the brain is especially visible in their morphology and cell density in particular brain structures but also in the expression of cell…

0301 basic medicineCentral nervous systemInflammationReviewBiologylcsh:RC321-571M1/M2 phenotype03 medical and health sciencesCellular and Molecular Neuroscience0302 clinical medicineImmune systemneurotoxicitymedicineCytotoxic T celllcsh:Neurosciences. Biological psychiatry. NeuropsychiatrypolarizationMicrogliaRegeneration (biology)Neurotoxicityinfiltrating macrophagesmedicine.diseasePhenotype030104 developmental biologymedicine.anatomical_structureinflammationCellular Neuroscienceregenerationmicroglial heterogeneitymedicine.symptomNeuroscience030217 neurology & neurosurgeryFrontiers in cellular neuroscience
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Brain Distribution and Modulation of Neuronal Excitability by Indicaxanthin From Opuntia Ficus Indica Administered at Nutritionally-Relevant Amounts

2018

Several studies have recently investigated the role of nutraceuticals in complex pathophysiological processes such as oxidative damages, inflammatory conditions and excitotoxicity. In this regard, the effects of nutraceuticals on basic functions of neuronal cells, such as excitability, are still poorly investigated. For this reason, the possible modulation of neuronal excitability by phytochemicals (PhC) could represent an interesting field of research given that excitotoxicity phenomena are involved in neurodegenerative alterations leading, for example, to Alzheimer's disease. The present study was focused on indicaxanthin from Opuntia ficus indica, a bioactive betalain pigment, with a pro…

0301 basic medicineCerebellumAgingCognitive NeuroscienceExcitotoxicityHippocampusindicaxanthinBiologyHippocampal formationmedicine.disease_causeNeuroprotectionmicroiontophoresisbrain localizationlcsh:RC321-57103 medical and health scienceschemistry.chemical_compound0302 clinical medicineexcitabilitymedicinelcsh:Neurosciences. Biological psychiatry. NeuropsychiatryOriginal ResearchnutraceuticalselectrophysiologyCortex (botany)brain localization; electrophysiology; excitability; indicaxanthin; microiontophoresis; neuroprotection; nutraceuticals030104 developmental biologymedicine.anatomical_structurechemistrynervous systemmicroiontophoresineuroprotectionNeuronIndicaxanthinNeuroscience030217 neurology & neurosurgeryNeuroscienceFrontiers in Aging Neuroscience
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The Role of Iron in Friedreich's Ataxia: Insights From Studies in Human Tissues and Cellular and Animal Models.

2019

Friedreich’s ataxia (FRDA) is a rare early-onset degenerative disease that affects both the central and peripheral nervous systems, and other extraneural tissues, mainly the heart and endocrine pancreas. This disorder progresses as a mixed sensory and cerebellar ataxia, primarily disturbing the proprioceptive pathways in the spinal cord, peripheral nerves and nuclei of the cerebellum. FRDA is an inherited disease with an autosomal recessive pattern caused by an insufficient amount of the nuclear-encoded mitochondrial protein frataxin, which is an essential and highly evolutionary conserved protein whose deficit results in iron metabolism dysregulation and mitochondrial dysfunction. The firs…

0301 basic medicineCerebellumAtaxiaFriedreich’s ataxiaReviewMitochondrionmedicine.disease_causelcsh:RC321-57103 medical and health sciencesiron0302 clinical medicineDegenerative diseasemedicineoxidative stresslcsh:Neurosciences. Biological psychiatry. Neuropsychiatrychemistry.chemical_classificationReactive oxygen speciesfrataxinbiologyCerebellar ataxialipid deregulationGeneral Neurosciencemedicine.diseaseanimal modelsCell biology030104 developmental biologymedicine.anatomical_structurechemistryFrataxinbiology.proteiniron chelatorsmedicine.symptom030217 neurology & neurosurgeryOxidative stressNeuroscienceFrontiers in neuroscience
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26th Annual Computational Neuroscience Meeting (CNS*2017): Part 2

2017

International audience; No abstract available

0301 basic medicineCerebellumComputer science[SDV]Life Sciences [q-bio]General Neurosciencelcsh:QP351-495Meeting Abstractslcsh:RC321-57103 medical and health sciencesCellular and Molecular Neurosciencelcsh:Neurophysiology and neuropsychology030104 developmental biologymedicine.anatomical_structuremedicineNeuronlcsh:Neurosciences. Biological psychiatry. NeuropsychiatryNeuroscienceComputingMilieux_MISCELLANEOUScomputational neuroscienceBMC Neuroscience
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The Importance of Cerebellar Connectivity on Simulated Brain Dynamics

2020

The brain shows a complex multiscale organization that prevents a direct understanding of how structure, function and dynamics are correlated. To date, advances in neural modeling offer a unique opportunity for simulating global brain dynamics by embedding empirical data on different scales in a mathematical framework. The Virtual Brain (TVB) is an advanced data-driven model allowing to simulate brain dynamics starting from individual subjects' structural and functional connectivity obtained, for example, from magnetic resonance imaging (MRI). The use of TVB has been limited so far to cerebral connectivity but here, for the first time, we have introduced cerebellar nodes and interconnecting…

0301 basic medicineCerebellumEmpirical dataComputer scienceThe Virtual Brainlcsh:RC321-57103 medical and health sciencesFunctional brainCellular and Molecular Neuroscience0302 clinical medicinemultiscale approachbrain dynamicsmedicineFunctional connectomestructural connectivitylcsh:Neurosciences. Biological psychiatry. NeuropsychiatryComputingMilieux_MISCELLANEOUSOriginal ResearchSignal processingFunctional connectivity[SCCO.NEUR]Cognitive science/Neurosciencefunctional connectivity030104 developmental biologyBrain statemedicine.anatomical_structureDynamics (music)Neuroscience030217 neurology & neurosurgeryNeurosciencecerebro-cerebellar loopFrontiers in Cellular Neuroscience
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Local field potential activity dynamics in response to deep brain stimulation of the subthalamic nucleus in Parkinson's disease

2020

Abstract Local field potentials (LFPs) may afford insight into the mechanisms of action of deep brain stimulation (DBS) and potential feedback signals for adaptive DBS. In Parkinson's disease (PD) DBS of the subthalamic nucleus (STN) suppresses spontaneous activity in the beta band and drives evoked resonant neural activity (ERNA). Here, we investigate how STN LFP activities change over time following the onset and offset of DBS. To this end we recorded LFPs from the STN in 14 PD patients during long (mean: 181.2 s) and short (14.2 s) blocks of continuous stimulation at 130 Hz. LFP activities were evaluated in the temporal and spectral domains. During long stimulation blocks, the frequency …

0301 basic medicineChange over timeMaleDeep brain stimulationSteady state (electronics)Parkinson's diseasemedicine.medical_treatmentDeep Brain StimulationParkinson's disease610 Medicine & healthStimulationFeedback markersLocal field potentialHigh frequency oscillationsArticlelcsh:RC321-57103 medical and health sciences0302 clinical medicineSubthalamic NucleusmedicineHumansBeta (finance)Adaptive deep brain stimulation610 Medicine & healthEvoked PotentialsBeta oscillationslcsh:Neurosciences. Biological psychiatry. NeuropsychiatryAgedLocal field potentialsChemistryParkinson DiseaseMiddle Agedmedicine.diseasenervous system diseasesSubthalamic nucleus030104 developmental biologysurgical procedures operativeNeurologynervous systemParkinson’s diseaseFemaleEvoked resonant neural activityGamma activityBeta RhythmNeuroscience030217 neurology & neurosurgery
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Environmental Tobacco Smoke During the Early Postnatal Period of Mice Interferes With Brain 18 F-FDG Uptake From Infancy to Early Adulthood – A Longi…

2020

Exposure to environmental tobacco smoke (ETS) is associated with high morbidity and mortality, mainly in childhood. Our aim was to evaluate the effects of postnatal ETS exposure in the brain 2-deoxy-2-[18F]-fluoro-D-glucose (18F-FDG) uptake of mice by positron emission tomography (PET) neuroimaging in a longitudinal study. C57BL/6J mice were exposed to ETS that was generated from 3R4F cigarettes from postnatal day 3 (P3) to P14. PET analyses were performed in male and female mice during infancy (P15), adolescence (P35), and adulthood (P65). We observed that ETS exposure decreased 18F-FDG uptake in the whole brain, both left and right hemispheres, and frontal cortex in both male and female i…

0301 basic medicineCingulate cortexmedicine.medical_specialtyCerebellumpositron emission tomographyglucose metabolismbrainThalamusHippocampusenvironmental tobacco smokeStriatum18F-FDG uptakelcsh:RC321-571Midbrain03 medical and health sciences0302 clinical medicineInternal medicineCortex (anatomy)medicinelcsh:Neurosciences. Biological psychiatry. NeuropsychiatryOriginal ResearchBasal forebrainpassive smokeneuroimagingbusiness.industryGeneral Neurosciencelongitudinal studyCÉREBRO030104 developmental biologyEndocrinologymedicine.anatomical_structurebusiness030217 neurology & neurosurgeryNeuroscienceFrontiers in Neuroscience
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