Search results for "ldl"

showing 10 items of 664 documents

Accumulation of apoE-enriched triglyceride-rich lipoproteins in patients with coronary artery disease.

2005

Triglycerides (TGs) are vehicled by multiple particles with different abilities to promote atherosclerosis. Among plasma TG-rich lipoproteins (TRLs), subspecies may or may not contain apolipoprotein E (apoE) molecules: in this study, we evaluated the relative contribution of apoE-rich and apoE-poor TRLs to coronary atherosclerosis. We selected a group of males with premature coronary artery disease (CAD) without any of the classical nonlipid risk factors and/or high plasma lipid levels and evaluated the plasma concentration of TRL subspecies in comparison with healthy controls. Patients with CAD and controls had total cholesterol and TG levels within the normal range (despite slightly, even…

Apolipoprotein EAdultMalemedicine.medical_specialtyEndocrinology Diabetes and MetabolismCoronary Artery DiseaseChromatography AffinityApolipoproteins ECoronary artery diseasechemistry.chemical_compoundEndocrinologyApolipoproteins EInternal medicinemedicineHumansInsulinCoronary atherosclerosisTriglyceridesbiologyTriglycerideCholesterolCholesterol HDLLipoprotein(a)Cholesterol LDLMiddle Agedmedicine.diseaseEndocrinologyapoE triglyceride-rich lipoproteins coronary artery diseaseLogistic ModelschemistryMultivariate Analysisbiology.proteinlipids (amino acids peptides and proteins)Electrophoresis Polyacrylamide GelLipoproteinLipoprotein(a)Metabolism: clinical and experimental
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Relationship Between the Apolipoprotein E Genotype and LDL Particle Size in Patients With Obstructive Sleep Apnea.

2016

Obstructive sleep apnea (OSA) is associated with dyslipidemia and increased cardiovascular risk. We assessed the effects of apolipoprotein E ( APOE) genotype on low-density lipoprotein (LDL) and high-density lipoprotein (HDL) particle size and lipid subclasses (separated by gradient gel electrophoresis) in patients with OSA. Stable patients (n = 181) prospectively recruited underwent full polysomnography. Both LDL particle size and LDL I proportion were reduced from ∊3∊3 homozygotes to ∊2 carriers and to ∊4 carriers (analysis of variance: P = .024; P = .040, respectively); carriers of the ∊4 allele of the APOE genotype had significantly lower LDL particle size and LDL I proportion compared…

Apolipoprotein EAdultMalemedicine.medical_specialtyGenotypePolysomnographyApolipoprotein E4Statistics as TopicPolysomnography030204 cardiovascular system & hematology03 medical and health sciences0302 clinical medicineInsulin resistanceApolipoproteins EInternal medicineGenotypemedicineHumansGenetic Predisposition to Disease030212 general & internal medicineProspective StudiesParticle SizeAgedSleep Apnea Obstructivemedicine.diagnostic_testbusiness.industryGenetic Carrier ScreeningMiddle Agedmedicine.diseaseObstructive sleep apneaLipoproteins LDLEndocrinologyCardiovascular Diseaseslipids (amino acids peptides and proteins)FemaleMetabolic syndromeCardiology and Cardiovascular MedicinebusinessDyslipidemiaLipoproteinAngiology
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Low and very low density lipoprotein composition and resistance to copper-induced oxidation are not notably modified in smokers.

1997

To study whether tobacco use was associated with oxidative phenomena affecting lipoproteins, we estimated susceptibility of LDL and VLDL to an in vitro copper-mediated oxidation, and measured serum autoantibody titers against oxidized LDL in 45 middle-age healthy nonsmokers, 35 smokers and 37 ex-smokers of both sexes, taking into account the detailed lipid composition of the lipoproteins. VLDL from female smokers had higher triglyceride, phospholipid, apolipoprotein E and alpha-tocopherol content and showed a higher rate of copper-induced oxidation in comparison with those from nonsmokers (P < or = 0.05) whereas the relative composition of these particles in saturated, mono- or poly-unsatur…

Apolipoprotein EAdultMalemedicine.medical_specialtyVery low-density lipoproteinAdolescentmedicine.medical_treatmentClinical BiochemistryPhospholipidLipoproteins VLDLBiochemistrychemistry.chemical_compoundInternal medicinemedicineHumansVitamin ELipoprotein oxidationAgedAutoantibodieschemistry.chemical_classificationTriglycerideVitamin EBiochemistry (medical)SmokingProteinsGeneral MedicineMiddle AgedLipidsLipoproteins LDLEndocrinologychemistryBiochemistryLow-density lipoproteinlipids (amino acids peptides and proteins)FemaleOxidation-ReductionCopperPolyunsaturated fatty acidClinica chimica acta; international journal of clinical chemistry
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Functional Role of Lipoprotein Receptors in Alzheimers Disease

2008

The LDL receptor gene family constitutes a class of structurally closely related cell surface receptors fulfilling diverse functions in different organs, tissues, and cell types. The LDL receptor is the prototype of this family, which also includes the VLDLR, ApoER2/LRP8, LRP1 and LRP1B, as well as Megalin/GP330, SorLA/LR11, LRP5, LRP6 and MEGF7. Recently several lines of evidence have positioned the LDL receptor gene family as one of the key players in Alzheimer's disease (AD) research. Initially this receptor family was of high interest due to its key function in cholesterol/apolipoprotein E (ApoE) uptake, with the epsilon4 allele of ApoE as the strongest genetic risk factor for late-onse…

Apolipoprotein EAmyloid beta-PeptidesbiologyChemistryEndosomeLRP1BLRP1Cell biologyAmyloid beta-Protein PrecursorApolipoproteins ECholesterolReceptors LDLNeurologyAlzheimer DiseaseCell surface receptormental disordersLDL receptorAmyloid precursor proteinbiology.proteinAnimalsHumansNeurology (clinical)ReceptorCurrent Alzheimer Research
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Uptake mechanism of ApoE-modified nanoparticles on brain capillary endothelial cells as a blood-brain barrier model.

2012

Background The blood-brain barrier (BBB) represents an insurmountable obstacle for most drugs thus obstructing an effective treatment of many brain diseases. One solution for overcoming this barrier is a transport by binding of these drugs to surface-modified nanoparticles. Especially apolipoprotein E (ApoE) appears to play a major role in the nanoparticle-mediated drug transport across the BBB. However, at present the underlying mechanism is incompletely understood. Methodology/Principal Findings In this study, the uptake of the ApoE-modified nanoparticles into the brain capillary endothelial cells was investigated to differentiate between active and passive uptake mechanism by flow cytome…

Apolipoprotein EDrugs and DevicesDrug Research and DevelopmentLipoproteinsMaterials Sciencelcsh:MedicinePlasma protein bindingBiologyBlood–brain barrierBiochemistryFlow cytometryApolipoproteins EMaterial by AttributeMiceApolipoproteins EDrug Delivery Systemsddc:570Cell Line TumormedicineAnimalsHumansNanotechnologyPharmacokineticsReceptorlcsh:ScienceBiologySerum AlbuminBrain DiseasesMultidisciplinaryMicroscopy Confocalmedicine.diagnostic_testlcsh:RBrainEndothelial CellsProteinsBiological TransportFlow CytometryCell biologymedicine.anatomical_structureBlood-Brain BarrierNanoparticles for drug delivery to the brainLDL receptorNanoparticlesMedicinelcsh:QProtein BindingResearch ArticleBiotechnologyPLoS ONE
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Functional role of the low-density lipoprotein receptor-related protein in Alzheimer's disease.

2006

Alzheimer’s disease (AD) is the most common age-related neurodegenerative disorder, characterized by neuronal loss, neurofibrillary tangle formation and the extracellular deposition of amyloid-β (Aβ) plaques. The amyloid precursor protein (APP) and the enzymes responsible for Aβ generation seem to be the base elements triggering the destructive processes. Initially, the low-density lipoprotein receptor-related protein (LRP) was genetically linked to AD and later it emerged to impact on many fundamental events related to this disease. LRP is not only involved in Aβ clearance but is also the major receptor of several AD-associated ligands, e.g. apolipoprotein E and α&lt;sub&gt;2&lt;/sub&gt;-m…

Apolipoprotein EFunctional rolemedicine.medical_specialtyPathologybiologyChemistryDiseaseLRP1Amyloid beta-Protein PrecursorEndocrinologyNeurologyAlzheimer DiseaseInternal medicineLDL receptormedicineExtracellularAmyloid precursor proteinbiology.proteinNeurofibrillary tangle formationAnimalsHumansNeurology (clinical)LDL-Receptor Related ProteinsNeuro-degenerative diseases
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Lipids, Lipoproteins and Apolipoproteins A&lt;sub&gt;I&lt;/sub&gt; A&lt;sub&gt;II&lt;/sub&gt;, B, C&lt;sub&gt;II&lt;/sub&gt;, C&lt;sub&gt;III&lt;/sub…

1991

In this study lipid and apolipoprotein patterns were investigated at birth and compared with those of adults. In cord sera, cholesterol, triglycerides, low-density lipoprotein (LDL) cholesterol and high-density lipoprotein (HDL) cholesterol were 38.2, 46.2, 50.5, and 31.9%, respectively, of adult values. Apolipoprotein A&lt;sub&gt;II&lt;/sub&gt;, B and C&lt;sub&gt;III&lt;/sub&gt; were 48.6, 30.6 and 44.5% of adult values, while apo A&lt;sub&gt;I&lt;/sub&gt;, apo C&lt;sub&gt;II&lt;/sub&gt; and apo E showed values approaching those of adults (63.4, 73.3 and 89.7%, respectively). Also cholesterol/HDL cholesterol and LDL cholesterol/HDL cholesterol ratios were lower in newborns. In cord sera, l…

Apolipoprotein ELdl cholesterolmedicine.medical_specialtyApolipoprotein BbiologyCholesterolLipid metabolismMetabolismPositive correlationchemistry.chemical_compoundEndocrinologychemistryInternal medicinePediatrics Perinatology and Child Healthmedicinebiology.proteinlipids (amino acids peptides and proteins)Developmental BiologyLipoproteinNeonatology
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Influence of microsomal triglyceride transfer protein promoter polymorphism -493 GT on fasting plasma triglyceride values and interaction with treatm…

2005

Familial hypercholesterolaemia (FH) is an autosomal dominant disease characterized by elevated levels of low-density lipoprotein-cholesterol (LDL-C). Phenotypic expression is highly variable, being influenced by diet, age, gender, body mass index, apolipoprotein E genotype and type of LDL-receptor gene mutation. Microsomal triglyceride (TG) transfer protein (MTP) is a protein involved in lipid metabolism. Polymorphism MTP -493 GT has been shown to modulate lipid levels in several populations. To analyse the effect of this polymorphism in the lipid phenotype expression of FH and treatment response, we studied a sample of 222 Spanish FH patients, of whom 147 were studied before and after trea…

Apolipoprotein EMaleAtorvastatinPolymerase Chain ReactionMicrosomal triglyceride transfer proteinBody Mass Indexchemistry.chemical_compoundAtorvastatinGeneral Pharmacology Toxicology and PharmaceuticsPromoter Regions GeneticGenetics (clinical)Polymorphism Single-Stranded ConformationalGeneticsbiologyAutosomal dominant traitFastingLipoproteins LDLCholesterolPhenotypeMolecular Medicinelipids (amino acids peptides and proteins)Femalemedicine.drugmedicine.medical_specialtyHeterozygoteGenotypeLipoproteinsHyperlipoproteinemia Type IIApolipoproteins ESex FactorsInternal medicineGeneticsmedicineHumansPyrrolesMolecular BiologyAllelesTriglyceridesPolymorphism GeneticTriglycerideCholesterolGenetic VariationCholesterol LDLDNALipid MetabolismEndocrinologychemistryHeptanoic AcidsPharmacogeneticsMutationbiology.proteinHydroxymethylglutaryl-CoA Reductase InhibitorsCarrier ProteinsBody mass indexPharmacogeneticsPharmacogenetics and genomics
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Genetic polymorphisms affecting the phenotypic expression in familial hypercholesterolemia

2004

The clinical expression of heterozygous familial hypercholesterolemia (FH) is highly variable even in patients carrying the same LDL receptor (LDL-R) gene mutation. This variability might be due to environmental factors as well as to modifying genes affecting lipoprotein metabolism. We investigated Apo E (2, 3, 4), MTP (-493G/T), Apo B (-516C/T), Apo A-V (-1131T/C), HL (-514C/T and -250G/A), FABP-2 (A54T), LPL (D9N, N291S, S447X) and ABCA1 (R219K) polymorphisms in 221 unrelated FH index cases and 349 FH relatives with defined LDL-R gene mutations. We found a significant and independent effect of the following polymorphisms on: (i) plasma LDL-C (Apo E, MTP and Apo B); (ii) plasma HDL-C (HL, …

Apolipoprotein EMaleSettore MED/09 - Medicina InternaApolipoprotein BFamilial hypercholesterolemiaGene mutationPolymerase Chain ReactionCoronary artery diseasecoronary artery disease; familial hypercholesterolemia; genetic polymorphisms; plasma lipidsCohort Studieschemistry.chemical_compoundGenotypePlasma lipidsOdds RatiobiologyFamilial hypercholesterolemia Plasma lipids Genetic polymorphisms Coronary artery diseaseIncidenceMiddle AgedPhenotypelipids (amino acids peptides and proteins)FemaleCardiology and Cardiovascular MedicineAdultmedicine.medical_specialtyMolecular Sequence DataFamilial hypercholesterolemiaPlasma lipidGenetic polymorphismsRisk AssessmentHyperlipoproteinemia Type IIFamilial hypercholesterolemia; Plasma lipids; Genetic polymorphisms; Coronary artery diseasePredictive Value of TestsInternal medicinemedicineConfidence IntervalsHumansGenetic Predisposition to DiseaseGenetic polymorphismPolymorphism GeneticBase SequenceCholesterolCholesterol HDLCase-control studyCholesterol LDLmedicine.diseaseEndocrinologyApolipoproteinschemistrySettore MED/03 - Genetica MedicaGene Expression RegulationReceptors LDLCase-Control StudiesLDL receptorbiology.protein
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Serum lipid responses to phytosterol-enriched milk in a moderate hypercholesterolemic population is not affected by apolipoprotein E polymorphism or …

2011

Background/Objectives: The importance of both low-density lipoprotein cholesterol (LDLc) size and the apolipoprotein E (Apo E) in the atherogenic process is known, but there is little information with regard to the effect of phytosterols (PS) on these parameters. The aim of this study was to evaluate the influence of PS on lipid profile and LDLc size according to Apo E genotype. Subjects/Methods: This was a randomized parallel trial employing 75 mild-hypercholesterolemic subjects and consisting of two 3-month intervention phases. After 3 months of receiving a standard healthy diet, subjects were divided into two intervention groups: a diet group (n = 34) and a diet+PS group (n = 41) that re…

Apolipoprotein EMalemedicine.medical_specialtyGenotype030309 nutrition & dieteticsPopulationHypercholesterolemiaMedicine (miscellaneous)030204 cardiovascular system & hematology03 medical and health scienceschemistry.chemical_compound0302 clinical medicineApolipoproteins EDouble-Blind MethodInternal medicineHyperlipidemiamedicineAnimalsHumansParticle SizeeducationApolipoprotein e polymorphismTriglycerides0303 health scienceseducation.field_of_studyNutrition and DieteticsPolymorphism GeneticCholesterolPhytosterolCholesterol HDLPhytosterolsCholesterol LDLMiddle Agedmedicine.diseaseLipidsLipoproteins LDLEndocrinologyCholesterolMilkTreatment OutcomechemistryLow-density lipoproteinFood FortifiedFemalelipids (amino acids peptides and proteins)Lipoprotein
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