Search results for "lepra"

showing 10 items of 15 documents

Tuberculoid leprosy and Type 1 lepra reaction.

2008

Summary A patient is described with tuberculoid leprosy and Type 1 (lepra) reaction from Sicily a non-endemic region, who lived previously in Manila from 2000 to 2005. The skin lesions became acutely inflamed and edematous. The plaques were painless to touch or pinprick, and there was swelling of the nerves in the fibro-osseous tunnels under the surface of the skin, including both the ulnar nerve at the elbow, and the posterior tibial nerve (medial malleolus). During the course of electro-neurographic studies, conduction velocity in the motory nerves indicated a slowing-down. The diagnosis of leprosy was confirmed by residence in an endemic area for about 5 years, by simultaneous skin lesio…

AdultMalePathologymedicine.medical_specialtyPosterior tibial nerveBiopsyPhilippinesElbowNeural ConductionTuberculoid leprosyLeprostatic AgentsClofazimineNerve conduction velocityDiagnosis DifferentialmedicineHumansUlnar nerveSicilyTravelmedicine.diagnostic_testbusiness.industryPublic Health Environmental and Occupational HealthTuberculoid leprosy Type 1 lepra reaction.medicine.diseaseLeprosy TuberculoidInfectious Diseasesmedicine.anatomical_structureSkin biopsyLeprosyDifferential diagnosisRifampinbusinessDapsone
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Mycobacterial antigen(s) induce anergy by altering TCR- and TCR/CD28-induced signalling events: insights into T-cell unresponsiveness in leprosy.

2009

Present study investigates the role of Mycobacterium leprae (M. leprae) antigens on TCR- and TCR/CD28-induced signalling leading to T-cell activation and further correlates these early biochemical events with T-cell anergy, as prevailed in advanced stages of leprosy. We observed that both whole cell lystae (WCL) and soluble fraction of M. leprae sonicate (MLSA) not only inhibited TCR, thapsigargin and ionomycin induced calcium fluxes by diminishing the opening of calcium channels, but also TCR- or TCR/CD28-induced proximal signalling events like phosphorylation of Zap-70 and protein kinase-C (PKC) activity. Study of TCR- and TCR/CD28-induced downstream signals revealed that M. leprae antige…

Antigens Differentiation T-LymphocyteMAP Kinase Signaling SystemT cellT-LymphocytesImmunologyReceptors Antigen T-Cellchemical and pharmacologic phenomenaBiologyLymphocyte ActivationJurkat cellsp38 Mitogen-Activated Protein Kinaseschemistry.chemical_compoundJurkat CellsCD28 AntigensAntigens CDLeprosyCalcium fluxmedicineHumansLectins C-TypeEnzyme InhibitorsPromoter Regions GeneticMolecular BiologyMycobacterium lepraeProtein Kinase CCell ProliferationClonal AnergyAntigens BacterialMitogen-Activated Protein Kinase 3ZAP-70 Protein-Tyrosine KinaseIonophoresNFATC Transcription FactorsIonomycinT-cell receptorInterleukin-2 Receptor alpha SubunitCD28hemic and immune systemsNFATbiology.organism_classificationCell biologyMycobacterium lepraemedicine.anatomical_structurechemistryGene Expression RegulationIonomycinImmunologyInterleukin-2ThapsigarginCalciumMolecular immunology
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Interleukin-15 in mycobacterial infection of antigen-presenting cells.

1999

Interleukin-15 (IL-15) shares many biological functions with IL-2 but also exhibits unique effects. Some of these represent the potent chemoattractant activity and expansion of distinct T-cell subsets, particularly memory T cells. IL-15 may therefore modulate the quality and quantity of cellular immune responses directed against intracellular pathogens. Immunohistochemical examination of skin lesions obtained from patients with the lepromatous or the tuberculoid form of Hansen's disease revealed intralesional IL-15 protein in both forms of the disease. In addition to Mycobacterium leprae, a number of different mycobacterial species are capable of effectively inducing IL-15 secretion in infe…

Cytotoxicity ImmunologicInterleukin-15Mycobacterium InfectionsbiologyIntracellular parasiteImmunologyAntigen-Presenting CellsGeneral Medicinebiology.organism_classificationMycobacteriumImmune systemAntigenInterleukin 15ImmunologyLeukocytes MononuclearHumansSecretionChemoattractant activityAntigen-presenting cellMycobacterium lepraeCells CulturedScandinavian journal of immunology
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On the Age of Leprosy

2014

Leprosy is a chronic infection of the skin and nerves caused by Mycobacterium leprae and the newly discovered Mycobacterium lepromatosis. Human leprosy has been documented for millennia in ancient cultures. Recent genomic studies of worldwide M. leprae strains have further traced it along global human dispersals during the past ∼ 100,000 years. Because leprosy bacilli are strictly intracellular, we wonder how long humans have been affected by this disease-causing parasite. Based on recently published data on M. leprae genomes, M. lepromatosis discovery, leprosy bacilli evolution, and human evolution, it is most likely that the leprosy bacilli started parasitic evolution in humans or early h…

Immune defenselcsh:Arctic medicine. Tropical medicinelcsh:RC955-962EpidemiologyImmunologyReviewDermatologymedicine.disease_causeMicrobiologyMycobacteriumLeprosymedicineHumansMycobacterium lepraeBiologyPhylogenyMycobacterium lepromatosisClinical GeneticsbiologyHuman evolutionary geneticslcsh:Public aspects of medicinePublic Health Environmental and Occupational Healthlcsh:RA1-1270Genomicsbiology.organism_classificationmedicine.diseaseBiological EvolutionMycobacterium lepraeChronic infectionInfectious DiseasesHuman evolutionImmunologyHost-Pathogen InteractionsMedicineClinical ImmunologyLeprosyPublic HealthMycobacteriumPLoS Neglected Tropical Diseases
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Phenolic-glycolipid-1 and lipoarabinomannan preferentially modulate TCR- and CD28-triggered proximal biochemical events, leading to T-cell unresponsi…

2012

Abstract Background Advanced stages of leprosy show T cell unresponsiveness and lipids of mycobacterial origin are speculated to modulate immune responses in these patients. Present study elucidates the role of phenolicglycolipid (PGL-1) and Mannose-capped lipoarabinomannan (Man-LAM) on TCR- and TCR/CD28- mediated signalling. Results We observed that lipid antigens significantly inhibit proximal early signalling events like Zap-70 phosphorylation and calcium mobilization. Interestingly, these antigens preferentially curtailed TCR-triggered early downstream signalling events like p38 phosphorylation whereas potentiated that of Erk1/2. Further, at later stages inhibition of NFAT binding, IL-2…

LipopolysaccharidesEndocrinology Diabetes and MetabolismT-LymphocytesClinical BiochemistryPGL-1Man-LAMGene ExpressionLymphocyte ActivationJurkat cellsJurkat CellsEndocrinologyT-cell activationIL-2 receptorPhosphorylationExtracellular Signal-Regulated MAP KinasesPromoter Regions Geneticlcsh:RC620-627Protein Kinase CImmunity CellularZAP-70 Protein-Tyrosine KinaseCD28hemic and immune systemsCell biologyMycobacterium lepraelcsh:Nutritional diseases. Deficiency diseasesmedicine.anatomical_structureHost-Pathogen InteractionsProtein BindingMAP Kinase Signaling SystemT cellReceptors Antigen T-Cellchemical and pharmacologic phenomenaBiologyImmune systemCD28 AntigensLeprosymedicineHumansSecretionCalcium SignalingCell ProliferationBiochemistry medicalAntigens BacterialLipoarabinomannanNFATC Transcription FactorsResearchBiochemistry (medical)T-cell receptorInterleukin-2 Receptor alpha SubunitMycobacteriaGene Expression RegulationAnergyImmunologyLeukocytes MononuclearInterleukin-2GlycolipidsLipids in Health and Disease
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¿Es ó no contagiosa la lepra? : memoria-dictamen presentada al Excmo. Ayuntamiento de Valencia

1897

Malalties transmissibles Obres anteriors al 1900Lepra Obres anteriors al 1900
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Leprosy: report of a case with severe peripheral neuropathy

2009

Leprosy (Hansen's disease) is a chronic granulomatous infectious disease, caused by Mycobacterium leprae, with cutaneous and neurological manifestations. Leprosy is very rare in Europe but some cases are reported, especially among people coming from endemic areas. Here, we report a case of Hansen's disease and emphasize the importance of a prompt diagnosis and treatment also in non-endemic areas.

Malemedicine.medical_specialtyNeurologyAdolescentSensory Receptor CellsChronic granulomatousLeprosy neuropathyNeural ConductionDermatologyDiseaseLeprosymedicineHumansMuscle SkeletalSicilyMycobacterium lepraeMotor NeuronsbiologyElectromyographybusiness.industryPeripheral Nervous System DiseasesGeneral Medicinebiology.organism_classificationmedicine.diseaseDermatologySenegalPsychiatry and Mental healthPeripheral neuropathyInfectious disease (medical specialty)ImmunologySettore MED/26 - NeurologiaNeurology (clinical)LeprosyNeurosurgerybusinessNeurological Sciences
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Reconstructing the ancestor of Mycobacterium leprae: The dynamics of gene loss and genome reduction

2007

We have reconstructed the gene content and order of the last common ancestor of the human pathogens Mycobacterium leprae and Mycobacterium tuberculosis. During the reductive evolution of M. leprae, 1537 of 2977 ancestral genes were lost, among which we found 177 previously unnoticed pseudogenes. We find evidence that a massive gene inactivation took place very recently in the M. leprae lineage, leading to the loss of hundreds of ancestral genes. A large proportion of their nucleotide content (∼89%) still remains in the genome, which allowed us to characterize and date them. The age of the pseudogenes was computed using a new methodology based on the rates and patterns of substitution in the…

Most recent common ancestorGeneticsLetterLineage (genetic)PseudogeneComputational BiologyMycobacterium tuberculosisBiologybiology.organism_classificationGenomeEvolution MolecularMycobacterium lepraeMycobacterium tuberculosisPhylogeneticsGeneticsDNA FungalMycobacterium lepraeGeneGene DeletionGenome BacterialPhylogenyGenetics (clinical)Genome Research
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Die Lepra in Riga, ihre Ausbreitung und Bekämpfung: Vortrag, gehalten am 22. April 1911 in der Gesellschaft für kommunale Sozialpolitik in Riga

1911

"Als Manuskript gedruckt für die Mitglieder der Gesellschaft für kommunale Sozialpolitik in Riga."

Mycobacterial diseasesLepraLepra - izplatība un novēršana (Rīga)LeprosyMikobakteriālās slimības:MEDICINE::Microbiology immunology infectious diseases::Infectious diseases [Research Subject Categories]SpitālībaMikobaktēriju infekcijas
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Spatiotemporal mapping of the leprosy granuloma landscape

2022

Mycobacterium lepraeSettore MED/04 - Patologia GeneraleInfectious DiseasesGranulomaImmunological landscapeImmunologyImmunology and AllergyLymphocytes
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