Search results for "leukemia"

showing 10 items of 976 documents

8,8-bis-(Dihydroconiferyl)-diferulate displayed impressive cytotoxicity towards a panel of human and animal cancer cells

2020

Abstract Background Recalcitrant cancers appear as a major obstacle to chemotherapy, prompting scientists to intensify the search for novel drugs to tackle the cell lines expressing multi-drug resistant (MDR) phenotypes. Purpose The purpose of this study was to evaluate the antiproliferative potential of a ferrulic acid derivative, 8,8-bis-(dihydroconiferyl)-diferulate (DHCF2) on a panel of 18 cancer cell lines, including various sensitive and drug-resistant phenotypes, belonging to human and animals. The mode of induction of cell death by this compound was further studied. Methods The antiproliferative activity, autophagy, ferroptotic and necroptotic cell death were evaluated by the resazu…

Programmed cell deathPharmaceutical ScienceApoptosisMice03 medical and health sciences0302 clinical medicineCell Line TumorDrug DiscoverymedicineAnimalsHumansCytotoxic T cellCytotoxicityCaspase030304 developmental biologyPharmacology0303 health sciencesLeukemiabiologyPlant ExtractsChemistrymedicine.diseaseAntineoplastic Agents PhytogenicDrug Resistance MultipleLeukemiaComplementary and alternative medicineDoxorubicinDrug Resistance NeoplasmCell cultureApoptosis030220 oncology & carcinogenesisCancer cellbiology.proteinCancer researchMolecular MedicinePhytomedicine
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α-Tocopherol impairs 7-ketocholesterol-induced caspase-3-dependent apoptosis involving GSK-3 activation and Mcl-1 degradation on 158N murine oligoden…

2011

Abstract In important and severe neurodegenerative pathologies, 7-ketocholesterol, mainly resulting from cholesterol autoxidation, may contribute to dys- or demyelination processes. On various cell types, 7-ketocholesterol has often been shown to induce a complex mode of cell death by apoptosis associated with phospholipidosis. On 158N murine oligodendrocytes treated with 7-ketocholesterol (20 μg/mL corresponding to 50 μM, 24–48 h), the induction of a mode of cell death by apoptosis characterised by the occurrence of cells with condensed and/or fragmented nuclei, caspase activation (including caspase-3) and internucleosomal DNA fragmentation was observed. It was associated with a loss of tr…

Programmed cell deathTime FactorsCell Survivalalpha-TocopherolApoptosisCaspase 3BiochemistryDephosphorylationGlycogen Synthase Kinase 3MiceMembrane MicrodomainsGSK-3AnimalsKetocholesterolsMolecular BiologyProtein kinase BCell ProliferationMembrane Potential MitochondrialPhospholipidosisGlycogen Synthase Kinase 3 betaCaspase 3ChemistryOrganic ChemistryCytochromes cCell BiologyCell biologyEnzyme ActivationOligodendrogliaProtein TransportProto-Oncogene Proteins c-bcl-2ApoptosisMyeloid Cell Leukemia Sequence 1 ProteinDNA fragmentationChemistry and Physics of Lipids
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Antiproliferative Properties of a Few Auranofin-Related Gold(I) and Silver(I) Complexes in Leukemia Cells and their Interferences with the Ubiquitin …

2020

A group of triethylphosphine gold(I) and silver(I) complexes, structurally related to auranofin, were prepared and investigated as potential anticancer drug candidates. The antiproliferative properties of these metal compounds were assessed against two leukemia cell lines, i.e., CCRF-CEM and its multidrug-resistant counterpart, CEM/ADR5000. Interestingly, potent cytotoxic effects were disclosed for both series of compounds against leukemia cells, with IC50 values generally falling in the low-micromolar range, the gold derivatives being on the whole more effective than the silver analogues. Some initial structure-function relationships were drawn. Subsequently, the ability of the study compo…

ProteasesProteasome Endopeptidase ComplexAuranofinSilverleukemia cellsPharmaceutical Sciencemetal complexesantiproliferative propertiesArticleAnalytical ChemistryMetallcsh:QD241-44103 medical and health sciencesInhibitory Concentration 500302 clinical medicineGold Compoundslcsh:Organic chemistryCell Line TumorDrug DiscoverymedicineCytotoxic T cellHumansPhysical and Theoretical Chemistry030304 developmental biologyCell Proliferationproteasome inhibition0303 health sciencesLeukemiaChemistryUbiquitinOrganic Chemistryauranofinmedicine.diseaseauranofin metal complexes proteasome inhibition leukemia cells antiproliferative propertiesDrug Resistance MultipleLeukemiaProteasomeBiochemistryChemistry (miscellaneous)Drug Resistance Neoplasm030220 oncology & carcinogenesisvisual_artvisual_art.visual_art_mediumauranofin;metal complexes; proteasome inhibition; leukemia cells; antiproliferative propertiesMolecular MedicineGoldSelectivitymedicine.drugMolecules
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Direct evidence that FK506 inhibition of FcεRI-mediated exocytosis from RBL mast cells involves calcineurin

1998

eRI-mediated exocytosis of preformed mediators from mast cells and basophils (e.g. histamine, serotonin, beta-hexosaminidase) is sensitive to the immunosuppressants cyclosporin A and FK506 (IC 50 200 and 4 n M , respectively) but not rapamycin. The mechanism of inhibition does not appear to involve tyrosine phosphorylation, hydrolysis of inositol phosphates or calcium flux. Here we report experiments using a molecular approach to assess the role of calcineurin, a serine/threonine phosphatase thought to be the primary pharmacological target of these drugs. Calcineurin’s activity requires association of its catalytic (A) subunit with an intrinsic regulatory (B) subunit. We hypothesized that c…

Protein subunitDermatologyBiologyTransfectionSensitivity and SpecificityCell DegranulationExocytosisTacrolimusExocytosischemistry.chemical_compoundCyclosporin aCalcium fluxTumor Cells CulturedmedicineAnimalsMast CellsDose-Response Relationship DrugReceptors IgECalcineurinTyrosine phosphorylationGeneral MedicineMast cellRatsCell biologyCalcineurinElectroporationmedicine.anatomical_structureLeukemia Basophilic AcutechemistryBiochemistryImmunosuppressive AgentsHistamineArchives of Dermatological Research
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Comparative Proteome Profiling and Functional Analysis of Chronic Myelogenous Leukemia Cell Lines

2007

The aim of the present study was the molecular profiling of different Ph+ chronic myelogenous leukemia (CML) cell lines (LAMA84, K562, and KCL22) by a proteomic approach. By employing two-dimensional gel electrophoresis combined with mass spectrometry analysis, we have identified 191 protein spots corresponding to 142 different proteins. Among these, 63% were cancer-related proteins and 74% were described for the first time in leukemia cells. Multivariate analysis highlighted significant differences in the global proteomic profile of the three CML cell lines. In particular, the detailed analysis of 35 differentially expressed proteins revealed that LAMA84 cells preferentially expressed prot…

Proteomicschronic myelogenous leukemia cell lineBiologyProteomicsBiochemistrySettore BIO/13 - Biologia ApplicataCell MovementCell Line TumorEthidiumLeukemia Myelogenous Chronic BCR-ABL Positivehemic and lymphatic diseases[SDV.BBM] Life Sciences [q-bio]/Biochemistry Molecular BiologymedicineHumansElectrophoresis Gel Two-DimensionalNeoplasm InvasivenessGel electrophoresisdrug resistanceProteomic ProfileGene Expression Regulation LeukemicGene Expression ProfilingGeneral Chemistrytumor invasionmedicine.diseasePhenotypeMolecular biologyAcridine OrangeGene expression profilingLeukemiaPhenotypeDrug Resistance Neoplasmproteome profilingMultivariate AnalysisDisease ProgressionK562 CellsChronic myelogenous leukemiaK562 cellsJournal of Proteome Research
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Real-time detection of BRAF V600E mutation from archival hairy cell leukemia FFPE tissue by nanopore sequencing

2018

The MinION is a miniaturized high-throughput next generation sequencing platform of novel conception. The use of nucleic acids derived from formalin-fixed paraffin-embedded samples is highly desirable, but their adoption for molecular assays is hurdled by the high degree of fragmentation and by the chemical-induced mutations stemming from the fixation protocols. In order to investigate the suitability of MinION sequencing on formalin-fixed paraffin-embedded samples, the presence and frequency of BRAF c.1799T > A mutation was investigated in two archival tissue specimens of Hairy cell leukemia and Hairy cell leukemia Variant. Despite the poor quality of the starting DNA, BRAF mutation was su…

Proto-Oncogene Proteins B-raf0301 basic medicineDNA Mutational AnalysisComputational biologyBiologybraf; ffpe; hairy cell leukemia; minion; nanopore sequencing; ngs; molecular biology; geneticsPolymerase Chain ReactionPolymorphism Single NucleotideDNA sequencingNanopores03 medical and health sciencesngsBiomarkers TumorGeneticsmedicinehairy cell leukemiaHumansDigital polymerase chain reactionHairy cell leukemiaGenetic TestingMolecular BiologyHairy Cell Leukemia VariantLeukemia Hairy CellMolecular pathologyPoint mutationHigh-Throughput Nucleotide SequencingDNA NeoplasmSequence Analysis DNAGeneral Medicinemedicine.diseaseminion030104 developmental biologyMolecular Diagnostic TechniquesMinionnanopore sequencingMutationNanopore sequencingbrafffpeMolecular Biology Reports
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Surgical management of invasive pulmonary aspergillosis in neutropenic patients.

1997

Background The aim of our study was to clarify the indications for operation in invasive pulmonary aspergillosis. Methods Nineteen patients with hematologic malignancy, in whom invasive pulmonary aspergillosis developed during the course of neutropenia, had operations. Neutropenia lasted 28 days (range, 15 to 45 days). The preoperative diagnosis of invasive pulmonary aspergillosis was based on computed tomographic scan findings (halo or air crescent signs). Results Eight patients underwent emergency operations, before marrow recovery, for prevention of massive hemoptysis. The criterion for operation was an aspergillosis lesion that contacted the pulmonary artery on computed tomography. A lo…

Pulmonary and Respiratory MedicineAdultMalemedicine.medical_specialtyHemoptysisNeutropeniaAdolescentNeutropeniaOpportunistic InfectionsAspergillosismedicine.arteryMedicineAspergillosisHumansChildMycosisAgedLungLeukemiaLung Diseases Fungalbusiness.industryRespiratory diseaseLength of StayMiddle Agedmedicine.diseaseSurgerymedicine.anatomical_structureChild PreschoolPulmonary arteryAcute DiseaseSurgeryFemaleCardiology and Cardiovascular MedicineComplicationbusinessMultiple MyelomaWedge resection (lung)The Annals of thoracic surgery
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Pulmonary hypertension associated with ponatinib therapy

2016

IF 8.332; International audience; A case of pulmonary hypertension associated with ponatinib therapy in a chronic myelogenous leukaemia patient.

Pulmonary and Respiratory Medicinemedicine.medical_specialtyCardiac outputeducationMEDLINEDasatinib030204 cardiovascular system & hematology[SDV.MHEP.PSR]Life Sciences [q-bio]/Human health and pathology/Pulmonology and respiratory tract03 medical and health scienceschemistry.chemical_compoundMyelogenous0302 clinical medicineResearch letterInternal medicinehemic and lymphatic diseasesMedicineChronic myelogenous leukaemiaIntensive care medicineTyrosine kinasehealth care economics and organizationsbusiness.industryInhibitorsPonatinibDisease progressionArterial-hypertensionmedicine.diseasePulmonary hypertension3. Good healthLeukemia030228 respiratory systemchemistrybusiness[ SDV.MHEP.PSR ] Life Sciences [q-bio]/Human health and pathology/Pulmonology and respiratory tract
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Rho protein inactivation induced apoptosis of cultured human endothelial cells.

2002

Small GTP-binding Rho GTPases regulate important signaling pathways in endothelial cells, but little is known about their role in endothelial cell apoptosis. Clostridial cytotoxins specifically inactivate GTPases by glucosylation [ Clostridium difficile toxin B-10463 (TcdB-10463), C. difficile toxin B-1470 (TcdB-1470)] or ADP ribosylation ( C. botulinum C3 toxin). Exposure of human umbilical cord vein endothelial cells (HUVEC) to TcdB-10463, which inhibits RhoA/Rac1/Cdc42, or to C3 toxin, which inhibits RhoA, -B, -C, resulted in apoptosis, whereas inactivation of Rac1/Cdc42 with TcdB-1470 was without effect, suggesting that Rho inhibition was responsible for endothelial apoptosis. Disruptio…

Pulmonary and Respiratory Medicinerac1 GTP-Binding Proteinrho GTP-Binding ProteinsProgrammed cell deathUmbilical VeinsEndotheliumPhysiologyBacterial ToxinsCASP8 and FADD-Like Apoptosis Regulating ProteinApoptosisBcl-2-associated X proteinBacterial ProteinsPhysiology (medical)Proto-Oncogene ProteinsmedicineCyclic AMPIn Situ Nick-End LabelingHumanscdc42 GTP-Binding ProteinCells Culturedbcl-2-Associated X ProteinAdenosine Diphosphate RibosebiologyCaspase 3Intracellular Signaling Peptides and ProteinsCell BiologyCaspase 9Cell biologyNeoplasm ProteinsEndothelial stem cellmedicine.anatomical_structureCdc42 GTP-Binding ProteinProto-Oncogene Proteins c-bcl-2Cell cultureApoptosisCaspasesbiology.proteinMyeloid Cell Leukemia Sequence 1 ProteinEndothelium VascularSignal transductionCarrier ProteinsrhoA GTP-Binding ProteinBH3 Interacting Domain Death Agonist ProteinSignal TransductionAmerican journal of physiology. Lung cellular and molecular physiology
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Quality assurance in RT-PCR-based BCR/ABL diagnostics--results of an interlaboratory test and a standardization approach.

2000

Here we describe the results of an interlaboratory test for RT-PCR-based BCR/ABL analysis. The test was organized in two parts. The number of participating laboratories in the first and second part was 27 and 20, respectively. In the first part samples containing various concentrations of plasmids with the ela2, b2a2 or b3a2 BCR/ABL transcripts were analyzed by PCR. In the second part of the test, cell samples containing various concentrations of BCR/ABL-positive cells were analyzed by RT-PCR. Overall PCR sensitivity was sufficient in approximately 90% of the tests, but a significant number of false positive results were obtained. There were significant differences in sensitivity in the cel…

Quality ControlCancer ResearchFusion Proteins bcr-ablBiologylaw.inventionlawhemic and lymphatic diseasesLeukemia Myelogenous Chronic BCR-ABL PositiveBiomarkers TumorHumansBase sequencePolymerase chain reactionDNA PrimersABLBase Sequencebusiness.industryReverse Transcriptase Polymerase Chain Reactionbreakpoint cluster regionHematologyMolecular biologyReverse transcriptaseReal-time polymerase chain reactionOncologyRNA extractionbusinessQuality assuranceLeukemia
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