Search results for "leukocyte"

showing 10 items of 970 documents

Effect of bakuchiol on leukocyte functions and some inflammatory responses in mice.

1996

Abstract The effects of bakuchiol, a meroterpenoid isolated from the leaves of Psoralea glandulosa L., on phospholipase A2 (PLA2) activity from different sources, human neutrophil responses, zymosan air pouch and topical inflammation in mice, were investigated. This natural product was a weak inhibitor of secretory and intracellular PLA2 but dose-dependently reduced the formation of LTB4 and TXB2 by human neutrophils and platelet microsomes, respectively. In addition, bakuchiol inhibited degranulation in human neutrophils, whereas superoxide generation was not affected. In mice, bakuchiol decreased cell migration, myeloperoxidase activity and eicosanoid levels in the air pouch inflammation …

MaleLeukotriene B4Cell SurvivalNeutrophilsPharmaceutical ScienceInflammationPharmacologyBiologyIn Vitro TechniquesLeukotriene B4DinoprostonePhospholipases Achemistry.chemical_compoundMicePhospholipase A2PhenolsSuperoxidesmedicineLeukocytesAnimalsEdemaHumansBakuchiolPeroxidasePharmacologyInflammationZymosanAnti-Inflammatory Agents Non-SteroidalDegranulationZymosanThromboxane B2Phospholipases A2EicosanoidchemistryImmunologybiology.proteinlipids (amino acids peptides and proteins)medicine.symptomLeukocyte ElastaseEicosanoid ProductionThe Journal of pharmacy and pharmacology
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The systemic lupus erythematosus IRF5 risk haplotype is associated with systemic sclerosis.

2013

Systemic sclerosis (SSc) is a fibrotic autoimmune disease in which the genetic component plays an important role. One of the strongest SSc association signals outside the human leukocyte antigen (HLA) region corresponds to interferon (IFN) regulatory factor 5 (IRF5), a major regulator of the type I IFN pathway. In this study we aimed to evaluate whether three different haplotypic blocks within this locus, which have been shown to alter the protein function influencing systemic lupus erythematosus (SLE) susceptibility, are involved in SSc susceptibility and clinical phenotypes. For that purpose, we genotyped one representative single-nucleotide polymorphism (SNP) of each block (rs10488631, r…

MaleLinkage disequilibrium:Phenomena and Processes::Genetic Phenomena::Phenotype [Medical Subject Headings]Polimorfismo de nucleótido simpleSLElcsh:MedicineAutoimmunityGenome-wide association studyLinkage DisequilibriumScleroderma:Phenomena and Processes::Genetic Phenomena::Genotype::Haplotypes [Medical Subject Headings]:Organisms::Eukaryota::Animals::Chordata::Vertebrates::Mammals::Primates::Haplorhini::Catarrhini::Hominidae::Humans [Medical Subject Headings]Gene Frequency:Named Groups::Persons::Population Groups::Continental Population Groups::European Continental Ancestry Group [Medical Subject Headings]Risk FactorsIRF5Genetics of the Immune SystemLupus Erythematosus Systemic:Diseases::Skin and Connective Tissue Diseases::Skin Diseases::Scleroderma Systemic [Medical Subject Headings]skin and connective tissue diseaseslcsh:ScienceMultidisciplinary:Diseases::Immune System Diseases::Autoimmune Diseases::Lupus Erythematosus Systemic [Medical Subject Headings]Predisposición genética a la enfermedad:Phenomena and Processes::Genetic Phenomena::Genetic Linkage::Linkage Disequilibrium [Medical Subject Headings]:Phenomena and Processes::Genetic Phenomena::Genotype::Genetic Predisposition to Disease [Medical Subject Headings]PhenotypeInterferon Regulatory FactorsSYSTEMIC SCLEROSISMedicineEvaluation of complex medical interventions Auto-immunity transplantation and immunotherapy [NCEBP 2]FemaleIRF5; SLE; TYPE I INTERFERON; SYSTEMIC SCLEROSISHaplotiposResearch ArticleFactores de riesgoImmunology:Chemicals and Drugs::Amino Acids Peptides and Proteins::Peptides::Intracellular Signaling Peptides and Proteins::Adaptor Proteins Signal Transducing::Interferon Regulatory Factors [Medical Subject Headings]:Check Tags::Male [Medical Subject Headings]:Health Care::Environment and Public Health::Public Health::Epidemiologic Factors::Causality::Risk Factors [Medical Subject Headings]Single-nucleotide polymorphismHuman leukocyte antigenBiologyPolymorphism Single NucleotideWhite PeopleAutoimmune DiseasesRheumatologyLupus eritematoso sistémicoGeneticsHumansGenetic Predisposition to DiseaseGrupo de ascendencia continental europeaAlleleBiologyAllele frequencyAllelesGenetic Association Studies:Phenomena and Processes::Genetic Phenomena::Genetic Structures::Genome::Genome Components::Genes::Alleles [Medical Subject Headings]Scleroderma SystemicHaplotypelcsh:R:Phenomena and Processes::Genetic Phenomena::Genetic Structures::Genome::Genome Components::Genetic Loci [Medical Subject Headings]Human Genetics:Phenomena and Processes::Genetic Phenomena::Genetic Variation::Polymorphism Genetic [Medical Subject Headings]Factores reguladores del interferónHaplotypesDesequilibrio de ligamiento:Check Tags::Female [Medical Subject Headings]Genetic LociTYPE I INTERFERONGenetics of DiseaseImmunologyGenetic PolymorphismClinical Immunologylcsh:Q:Phenomena and Processes::Genetic Phenomena::Gene Frequency [Medical Subject Headings]Population GeneticsIRF5PLoS ONE
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Tumor necrosis factor alpha promoter polymorphism at position -238 is associated with chronic active hepatitis C infection

1998

Tumor necrosis factor alpha (TNF-alpha) is involved in the pathogenesis of chronic hepatitis C virus infection. The gene for TNF-alpha is encoded in the major histocompatibility locus (MHC). Two polymorphisms at positions -308 and -238 in the TNF-alpha promoter region might influence TNF-alpha expression. These promoter polymorphisms have been linked previously to a number of infectious diseases. TNF-alpha promoter polymorphisms at positions -238 and -308 were studied by DNA sequencing and sequence-specific oligonucleotide hybridization in 82 individuals with chronic hepatitis C and 99 control subjects. Subjects had been HLA class I and class II typed in a previous study. The frequency of t…

MaleLinkage disequilibriumGenotypeHepatitis C virusHepacivirusHuman leukocyte antigenmedicine.disease_causeGene FrequencyVirologymedicineHumansProspective StudiesAllelePromoter Regions GeneticAllelesHepatitisPolymorphism GeneticbiologyTumor Necrosis Factor-alphaHistocompatibility Antigens Class IHistocompatibility Antigens Class IIPromoterHepatitis CHepatitis C Chronicmedicine.diseasebiology.organism_classificationVirologyInfectious DiseasesImmunologyFemaleJournal of Medical Virology
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Smoking and FOS expression from blood leukocyte transcripts in patients with coronary artery disease.

2011

International audience; OBJECTIVE: Analysis of the leukocyte transriptome, in particular the Finkel-Biskis-Jinkins Osteosarcoma (c-Fos) gene, which has a prominent role in inflammation, provides new insights into atherosclerosis mechanisms. Although smoking is a major risk factor, the links between smoking status and coronary artery disease (CAD) remains unclear. We aimed to analyze the relationship between smoking status and c-Fos expression in circulating leukocytes of patients with CAD. METHODS: c-Fos expression was measured by RT-Q-PCR, from blood leukocytes of 239 consecutive patients after acute myocardial infarction (MI). The patients were asked about their smoking status and stratif…

MaleMESH : RNA MessengerMESH: Chi-Square DistributionMESH : LeukocytesMESH : Prospective StudiesMESH : AgedMyocardial InfarctionSmoking PreventionMESH: Risk Assessmentc-FosMESH : Coronary Angiography0302 clinical medicineMESH : Genetic MarkersProspective StudiesMESH: Coronary Artery DiseaseAged 80 and over0303 health sciencesMESH: Middle AgedGenes fosMESH: Smoking Cessation3. Good healthMESH : SmokingMESH: Myocardial InfarctionOsteosarcomaSmoking statusCardiology and Cardiovascular MedicineGenetic Markersmedicine.medical_specialtyRisk AssessmentMESH: Leukocytes03 medical and health sciences[SDV.MHEP.CSC]Life Sciences [q-bio]/Human health and pathology/Cardiology and cardiovascular systemHumansMESH : Middle AgedRNA MessengerMESH : Coronary Artery DiseaseRisk factorMESH : Aged 80 and overAgedChi-Square DistributionMESH: HumansMESH : Chi-Square DistributionMESH : Smoking CessationMESH : Humansmedicine.diseaseMESH: Coronary AngiographyLinear ModelsMESH: FemaleBlood leukocyte transcriptomeMESH : Genes fos030204 cardiovascular system & hematologyMESH: Genetic MarkersBioinformaticsCoronary AngiographyCoronary artery diseaseMESH: Linear ModelsCoronary artery diseaseMESH: Aged 80 and overRisk FactorsMESH: Risk FactorsMESH : Linear ModelsLeukocytesMESH : FemaleMESH : Risk AssessmentMESH: Agedc-FosbiologySmoking[ SDV.MHEP.CSC ] Life Sciences [q-bio]/Human health and pathology/Cardiology and cardiovascular systemMiddle AgedMESH : Risk Factors[SDV.MHEP.CSC] Life Sciences [q-bio]/Human health and pathology/Cardiology and cardiovascular systemCardiologyFemaleFrancemedicine.symptomInflammation MediatorsMESH: SmokingMESH : MaleMESH: Inflammation MediatorsInflammationMESH: Genes fosMESH: Multivariate AnalysisMESH : Inflammation MediatorsInternal medicinemedicineIn patientMESH : France030304 developmental biologyMESH: RNA Messengerbusiness.industryMESH : Multivariate AnalysisMESH: MaleMESH: Prospective StudiesMESH: FranceMultivariate Analysisbiology.proteinSmoking CessationMESH : Myocardial Infarctionbusiness
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Human CD8+ T-cells Recognizing Peptides from Mycobacterium tuberculosis (Mtb) Presented by HLA-E Have an Unorthodox Th2-like, Multifunctional, Mtb In…

2015

Mycobacterial antigens are not exclusively presented to T-cells by classical HLA-class Ia and HLA-class II molecules, but also through alternative antigen presentation molecules such as CD1a/b/c, MR1 and HLA-E. We recently described mycobacterial peptides that are presented in HLA-E and recognized by CD8+ T-cells. Using T-cell cloning, phenotyping, microbiological, functional and RNA-expression analyses, we report here that these T-cells can exert cytolytic or suppressive functions, inhibit mycobacterial growth, yet express GATA3, produce Th2 cytokines (IL-4,-5,-10,-13) and activate B-cells via IL-4. In TB patients, Mtb specific cells were detectable by peptide-HLA-E tetramers, and IL-4 and…

MaleMacrophageQH301-705.5ImmunologyAntigen presentationBacterial ProteinMycobacterium tuberculosiHuman leukocyte antigenGATA3 Transcription FactorCD8-Positive T-LymphocytesMicrobiologyMicrobiologyMycobacterium tuberculosisImmune systemTh2 CellsGeneticHLA-EBacterial ProteinsVirologyGeneticsCytotoxic T cellHumansBiology (General)Th2 CellCytokineMolecular BiologyAntigen PresentationbiologyMacrophagesHistocompatibility Antigens Class ICD8-Positive T-LymphocyteMycobacterium tuberculosisRC581-607biology.organism_classificationBacterial Proteins; CD8-Positive T-Lymphocytes; Cytokines; Female; GATA3 Transcription Factor; Histocompatibility Antigens Class I; Humans; Macrophages; Male; Mycobacterium tuberculosis; Peptides; Th2 Cells; Antigen Presentation; Microbiology; Parasitology; Virology; Immunology; Genetics; Molecular BiologyPhenotypeVirology3. Good healthPeptideCytokinesParasitologyFemaleImmunologic diseases. AllergyPeptidesCD8HumanResearch ArticlePLoS Pathogens
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Molecular mechanisms of NET formation and degradation revealed by intravital imaging in the liver vasculature

2015

AbstractNeutrophil extracellular traps (NETs) composed of DNA decorated with histones and proteases trap and kill bacteria but also injure host tissue. Here we show that during a bloodstream infection with methicillin-resistant Staphylococcus aureus, the majority of bacteria are sequestered immediately by hepatic Kupffer cells, resulting in transient increases in liver enzymes, focal ischaemic areas and a robust neutrophil infiltration into the liver. The neutrophils release NETs into the liver vasculature, which remain anchored to the vascular wall via von Willebrand factor and reveal significant neutrophil elastase (NE) proteolytic activity. Importantly, DNase although very effective at D…

MaleMethicillin-Resistant Staphylococcus aureusExtracellular TrapsProteasesHydrolasesKupffer CellsGeneral Physics and AstronomyBacteremiaBiologyHepatic Veinsmedicine.disease_causeExtracellular TrapsGeneral Biochemistry Genetics and Molecular BiologyHistonesMiceHepatic ArteryVon Willebrand factorProtein-Arginine Deiminase Type 4von Willebrand FactormedicineAnimalsMice KnockoutMultidisciplinaryDeoxyribonucleasesGeneral ChemistryNeutrophil extracellular trapsStaphylococcal Infectionsmedicine.disease3. Good healthCell biologyHistoneLiverNeutrophil InfiltrationStaphylococcus aureusNeutrophil elastaseImmunologybiology.proteinLeukocyte ElastaseInfiltration (medical)
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Dynamic in vivo Imaging of Microvasculature and Perfusion by Miniaturized Confocal Laser Microscopy

2008

<i>Introduction:</i> Microvasculature and associated pathologies mandate dynamic imaging. We evaluated a novel miniaturized confocal laser scanning probe for in vivo visualization of blood vessels, blood flow, cell tracking and perfusion in both healthy rodents and disease models.<i> Methods:</i> The hand-held confocal microscopy system allowed a 500- to 2,400-fold magnification at a dynamically variable imaging depth. Different intravital stains were used alone or in combination for tissue, nuclear, plasma and vascular endothelial cell staining and for blood flow visualization, and targeted staining for individual cell populations. <i>Results:</i> Precis…

MaleMice Inbred MRL lprPathologymedicine.medical_specialtyMaterials scienceLaser scanningDynamic imagingConfocalCell Communicationlaw.inventionMiceImaging Three-DimensionalIn vivoConfocal microscopylawMicroscopyLeukocytesmedicineAnimalsFluorescent DyesInflammationMicroscopy ConfocalMiniaturizationMicrocirculationBrainEndothelial CellsThrombosisLupus NephritisMice Inbred C57BLDisease Models AnimalMicrovesselsFemaleSurgeryIntracranial ThrombosisGerbillinaePerfusionBlood Flow VelocityPreclinical imagingBiomedical engineeringEuropean Surgical Research
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A novel class of potent nonglycosidic and nonpeptidic pan-selectin inhibitors.

2006

An early step of the inflammatory response, the rolling of leukocytes on activated endothelial cells, is mediated by selectin/carbohydrate interactions. The tetrasaccharide sialy Lewisx is a ligand for E-, P-, and L-selectin and therefore serves as a lead structure for the development of analogues. A combination of synthesis and structure-based design allowed rapid optimization. The current lead 2a was evaluated in our E-selectin cell flow chamber assay where it proved to inhibit rolling and adhesion with an IC50 of 28+/-7 microM. The assays used are predictive for the in vivo efficacy of test compounds as shown for 2a in a proteose peptone induced peritonitis model of acute inflammation in…

MaleModels MolecularInflammationEnzyme-Linked Immunosorbent AssayIn Vitro TechniquesPeritonitisLigandsMiceStructure-Activity RelationshipIn vivoDrug DiscoverymedicineCell AdhesionLeukocytespara-AminobenzoatesTetrasaccharideAnimalsIC50Binding SitesChemistryCell adhesion moleculeAnti-Inflammatory Agents Non-SteroidalCaseinsEndothelial CellsLigand (biochemistry)In vitroPeptide FragmentsMice Inbred C57BLBiochemistryAcute DiseaseSelectinsMolecular Medicinemedicine.symptomE-Selectin4-Aminobenzoic AcidSelectinAlgorithmsProtein BindingJournal of medicinal chemistry
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The Effects of Cold Exposure on Leukocytes, Hormones and Cytokines during Acute Exercise in Humans

2014

The purpose of the study was to examine the effects of exercise on total leukocyte count and subsets, as well as hormone and cytokine responses in a thermoneutral and cold environment, with and without an individualized pre-cooling protocol inducing low-intensity shivering. Nine healthy young men participated in six experimental trials wearing shorts and t-shirts. Participants exercised for 60 min on a treadmill at low (LOW: 50% of peak VO2) and moderate (MOD: 70% VO2peak) exercise intensities in a climatic chamber set at 22°C (NT), and in 0°C (COLD) with and without a pre-exercise low-intensity shivering protocol (SHIV). Core and skin temperature, heart rate and oxygen consumption were col…

MaleMuscle PhysiologyHydrocortisonePhysiologylcsh:MedicineCardiovascular PhysiologyNorepinephrine0302 clinical medicineHeart RateImmune PhysiologySex Hormone-Binding GlobulinMedicine and Health SciencesLeukocytesMedicineTestosteroneInsulin-Like Growth Factor ITreadmilllcsh:Scienceta315MultidisciplinaryThermogenesista314116. Peace & justiceCold shock responseEpinephrineShiveringCytokinesmedicine.symptomEnvironmental HealthResearch ArticleBody Temperature Regulationmedicine.drugAdultmedicine.medical_specialtyEpinephrineleukocytesPhysical ExertionAdrenocorticotropic hormoneYoung Adult03 medical and health sciencesAdrenocorticotropic Hormoneeffects of exerciseInternal medicineHeart rateHumansSports and Exercise MedicineExerciseHydrocortisoneEndocrine Physiologybusiness.industryCold-Shock Responsesytokiinitlcsh:RBiology and Life Sciences030229 sport sciencesMolecular DevelopmenthormonitHealth CareEndocrinologylcsh:QPhysiological Processesbusiness030217 neurology & neurosurgeryDevelopmental BiologyHormonePLoS ONE
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Monitoring White Blood Cell Mitochondrial Aldehyde Dehydrogenase Activity: Implications for Nitrate Therapy in Humans

2009

Recent animal data suggest that reduced lipoic acid (LA) prevents oxidative inhibition of the nitrate bioactivating enzyme, the mitochondrial aldehyde dehydrogenase (ALDH-2), and that pentaerythritol tetranitrate (PETN) does not induce nitrate tolerance because of its intrinsic antioxidative properties, thereby preserving ALDH-2 activity. We sought to determine whether ALDH-2 activity in circulating white blood cells (WBCs) can be used to monitor nitrate tolerance and whether LA can prevent nitroglycerin tachyphylaxis in humans. Eight healthy male volunteers received, in randomized order, a single dose of glyceryl trinitrate (GTN; 0.8 mg), PETN (80 mg), or GTN plus LA (600 mg) orally. GTN (…

MaleMyocardial IschemiaAldehyde dehydrogenasePentaerythritol tetranitrateVasodilationTachyphylaxisPharmacologymedicine.disease_causeMitochondria Heartchemistry.chemical_compoundAnimal dataWhite blood cellLeukocytesmedicineAnimalsHumansRats WistarPharmacologyNitratesbiologyAldehyde DehydrogenaseRatsEnzyme ActivationVasodilationLipoic acidmedicine.anatomical_structurechemistrybiology.proteinMolecular MedicineOxidative stresscirculatory and respiratory physiologyJournal of Pharmacology and Experimental Therapeutics
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