Search results for "ligand"

showing 10 items of 2559 documents

Hunting for the high-affinity state of G-protein-coupled receptors with agonist tracers: Theoretical and practical considerations for positron emissi…

2019

Abstract The concept of the high‐affinity state postulates that a certain subset of G‐protein‐coupled receptors is primarily responsible for receptor signaling in the living brain. Assessing the abundance of this subset is thus potentially highly relevant for studies concerning the responses of neurotransmission to pharmacological or physiological stimuli and the dysregulation of neurotransmission in neurological or psychiatric disorders. The high‐affinity state is preferentially recognized by agonists in vitro. For this reason, agonist tracers have been developed as tools for the noninvasive imaging of the high‐affinity state with positron emission tomography (PET). This review provides an…

Central Nervous SystemBETA-ADRENERGIC-RECEPTORpositron emission tomographyagonist high-affinity stateD-2/3 AGONISTG-protein-coupled receptorsReview ArticleReceptors G-Protein-Coupledchemistry.chemical_compound0302 clinical medicineDrug DiscoveryReceptorNeurotransmitterReview Articles0303 health sciencesmedicine.diagnostic_testNONHUMAN PRIMATE BRAINEndocytosisTEST-RETEST REPRODUCIBILITYPositron emission tomographyG‐protein‐coupled receptors030220 oncology & carcinogenesisENDOGENOUS OPIOID RELEASEMolecular MedicineIN-VIVO BINDINGSignal TransductionAgonistNoninvasive imagingexperimental designmedicine.drug_classNeurotransmissionRAT-BRAINneurotransmittersagonist high‐affinity state03 medical and health sciencesIn vivomedicineAnimalsHumanshuman brain030304 developmental biologyG protein-coupled receptorPharmacologyDOPAMINE D2(HIGH) RECEPTORS5-HT1A RECEPTORSchemistryPositron-Emission TomographyPET RADIOLIGANDRadiopharmaceuticalsNeuroscienceMedicinal research reviews
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Mutations in spalt cause a severe but reversible neurodegenerative phenotype in the embryonic central nervous system ofDrosophila melanogaster

2002

The gene spalt is expressed in the embryonic central nervous system of Drosophila melanogaster but its function in this tissue is still unknown. To investigate this question, we used a combination of techniques to analyse spalt mutant embryos. Electron microscopy showed that in the absence of Spalt, the central nervous system cells are separated by enlarged extracellular spaces populated by membranous material at 60% of embryonic development. Surprisingly, the central nervous system from slightly older embryos (80% of development) exhibited almost wild-type morphology. An extensive survey by laser confocal microscopy revealed that thespalt mutant central nervous system has abnormal levels o…

Central Nervous SystemHeterozygoteTime FactorsFasciclin 2Cellular differentiationCentral nervous systemLigandsCell AdhesionImage Processing Computer-AssistedIn Situ Nick-End LabelingmedicineAnimalsDrosophila ProteinsCell LineageCell adhesionMolecular BiologyCells CulturedCytoskeletonHomeodomain ProteinsNeuronsMicroscopy ConfocalMicroscopy VideobiologyCell adhesion moleculeCell DifferentiationAnatomyCadherinsbiology.organism_classificationImmunohistochemistryPhenotypeCell biologyTransplantationMicroscopy ElectronDrosophila melanogasterPhenotypemedicine.anatomical_structureMutationDrosophila melanogasterTranscription FactorsDevelopmental BiologyDevelopment
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Lymphocyte apoptosis in children with central nervous system tuberculosis: a case control study

2011

Abstract Background Studies of the apoptosis mechanisms involved in the pathogenesis of tuberculosis have suggested that Mycobacterium tuberculosis can actively interfere with the apoptosis of infected cells. In vivo studies have been performed in adult populations but have not focused on this process in children. In the present study, we analyzed spontaneous T lymphocyte (PBT) apoptosis in the peripheral blood of children with central nervous system tuberculosis (CNS TB), before and after chemotherapy, and compared the results with healthy controls. Methods A case-control study was conducted from January 2002 to June 2009. It included 18 children with CNS TB and 17 healthy controls. Sponta…

Central Nervous SystemMaleFas Ligand ProteinTuberculosisSettore MED/17 - Malattie InfettiveTuberculosiT-Lymphocytesmedicine.medical_treatmentCentral nervous systemApoptosisLymphocyte ActivationMycobacterium tuberculosisPathogenesismeningoencephalitichildrenCentral Nervous System Bacterial InfectionsmedicineHumansfas ReceptorPediatrics Perinatology and Child HealthChildSettore MED/04 - Patologia GeneraleChemotherapybiologybusiness.industrylcsh:RJ1-570Case-control studylcsh:PediatricsMycobacterium tuberculosisT lymphocyteTuberculosis Central Nervous Systembiology.organism_classificationmedicine.diseaseapoptosimedicine.anatomical_structureApoptosisCase-Control StudiesChild PreschoolPediatrics Perinatology and Child HealthImmunologyFemalebusinessResearch ArticleBMC Pediatrics
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Noncompetitive agonism at nicotinic acetylcholine receptors; functional significance for CNS signal transduction.

1995

The alkaloids (-)physostigmine (Phy), galanthamine (Gal) and codeine (Cod), and several derivatives and homologous compounds, can act as noncompetitive agonists (NCA) of nicotinic acetylcholine receptors (nAChR) from Torpedo electrocytes, frog and mammalian muscle cells, clonal rat pheochromocytoma cells, cultured hippocampal neurons and several ectopic expression systems, by interacting with a binding site on the alpha-subunits of these nAChRs that is insensitive to the natural transmitter, acetylcholine (ACh), and ACh-competitive agonists and antagonists. Several endogenous ligands, including opioid-type compounds, can also act via this site, albeit at higher concentrations than is typica…

Central Nervous SystemPharmacologyReceptors NicotinicLigandsBiochemistrylaw.inventionEvolution MolecularlawMuscarinic acetylcholine receptormedicineAnimalsHumansNicotinic AgonistsBinding siteReceptorMolecular BiologyAcetylcholine receptorBinding SitesMolecular StructureChemistryCell BiologyAcetylcholineCell biologyNicotinic agonistnervous systemSignal transductionAcetylcholineTorpedomedicine.drugSignal TransductionJournal of receptor and signal transduction research
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Differential Promotion of Glutamate Transporter Expression and Function by Glucocorticoids in Astrocytes from Various Brain Regions

2005

Steroids that activate glucocorticoid receptors (GRs) and mineralocorticoid receptors have important regulatory effects on neural development, plasticity, and the body's stress response. Here, we investigated the role of corticosteroids in regulating the expression of the glial glutamate transporters glial glutamate transporter-1 (GLT-1) and glutamate-aspartate transporter (GLAST) in rat primary astrocytes. The synthetic glucocorticoid dexamethasone provoked a marked increase of GLT-1 transcription and protein levels in cortical astrocytes, whereas GLAST expression remained unaffected. Up-regulation of GLT-1 expression was accompanied by an enhanced glutamate uptake, which could be blocked …

Central Nervous SystemTime FactorsAmino Acid Transport System X-AGLigandsBiochemistryDexamethasoneRats Sprague-Dawleychemistry.chemical_compoundGlucocorticoid receptorMineralocorticoid receptorAdrenal Cortex HormonesCorticosteroneCerebellumGene expressionLuciferasesReceptorDNA Modification MethylasesKainic AcidReverse Transcriptase Polymerase Chain ReactionGlutamate receptorBrainImmunohistochemistryUp-RegulationMifepristoneAzacitidineNeurogliaGlucocorticoidmedicine.drugmedicine.medical_specialtymedicine.drug_classBlotting WesternDetergentsBiologyDecitabineTransfectionMembrane MicrodomainsInternal medicinemedicineAnimalsGlucocorticoidsMolecular BiologyDNA PrimersFluorescent DyesDose-Response Relationship DrugCell BiologyDNA MethylationRatsReceptors MineralocorticoidEndocrinologychemistryMineralocorticoidAstrocytesCorticosteroneJournal of Biological Chemistry
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Nicotinic receptor function in the mammalian central nervous system.

1995

The diversity of neuronal nicotinic receptors (nAChRs) in addition to their possible involvement in such pathological conditions as Alzheimer's disease have directed our research towards the characterization of these receptors in various mammalian brain areas. Our studies have relied on electrophysiological, biochemical, and immunofluorescent techniques applied to cultured and acutely dissociated hippocampal neurons, and have been aimed at identifying the various subtypes of nAChRs expressed in the mammalian central nervous system (CNS), at defining the mechanisms by which CNS nAChR activity is modulated, and at determining the ion permeability of CNS nAChR channels. Our findings can be sum…

Central nervous systemHippocampal formationNeurotransmissionIn Vitro TechniquesReceptors NicotinicLigandsHippocampusSynaptic TransmissionGeneral Biochemistry Genetics and Molecular BiologyStructure-Activity RelationshipHistory and Philosophy of SciencemedicineAnimalsMagnesiumPhosphorylationReceptorCells CulturedMammalsMolecular StructureChemistryGeneral NeuroscienceAcetylcholineOlfactory bulbElectrophysiologyNicotinic agonistmedicine.anatomical_structurenervous systemCalciumSignal transductionNeuroscienceIon Channel GatingSignal TransductionAnnals of the New York Academy of Sciences
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Anandamide-induced apoptosis in Chang liver cells involves ceramide and JNK/AP-1 pathway

2006

In the present study we demonstrate that anandamide, the most important endogenous cannabinoid, markedly induced apoptosis in Chang liver cells, an immortalized non-tumor cell line derived from normal liver tissue, while it induced only modest effects in a number of hepatoma cell lines. The apoptotic effect was reduced by methyl-beta-cyclodextrin, a membrane cholesterol depletor, suggesting an interaction between anandamide and the membrane microdomains named lipid rafts. Anandamide effects were mediated by the production of ceramide, as demonstrated by experiments performed with the sphingomyelinase inhibitor, desipramine, or with the sphingomyelinase activator, melittin. This conclusion w…

CeramideProgrammed cell deathFas Ligand ProteinCell SurvivalPolyunsaturated AlkamidesLiver cytologyp38 mitogen-activated protein kinasesBlotting WesternApoptosisArachidonic AcidsBiologyCeramidesCell LineMembrane Potentialschemistry.chemical_compoundCell Line TumorProto-Oncogene ProteinsGeneticsHumansEnzyme InhibitorsMembrane GlycoproteinsBcl-2-Like Protein 11Dose-Response Relationship DrugDesipramineJNK Mitogen-Activated Protein KinasesMembrane ProteinsFree Radical ScavengersGeneral MedicineAnandamideEndocannabinoid systemAcetylcysteineCell biologyEnzyme ActivationTranscription Factor AP-1cannabinoids apoptosis tumor cells JNK/AP1LiverchemistryApoptosisCaspasesMitochondrial MembranesTumor Necrosis FactorsApoptosis Regulatory ProteinsSphingomyelinEndocannabinoidsSignal TransductionInternational Journal of Molecular Medicine
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Computational identification of chemical compounds with potential anti-Chagas activity using a classification tree

2021

Chagas disease is endemic to 21 Latin American countries and is a great public health problem in that region. Current chemotherapy remains unsatisfactory; consequently the need to search for new drugs persists. Here we present a new approach to identify novel compounds with potential anti-chagasic action. A large dataset of 584 compounds, obtained from the Drugs for Neglected Diseases initiative, was selected to develop the computational model. Dragon software was used to calculate the molecular descriptors and WEKA software to obtain the classification tree. The best model shows accuracy greater than 93.4% for the training set; the tree was also validated using a 10-fold cross-validation p…

Chagas diseaseComputer scienceTrypanosoma cruziAntiprotozoal AgentsQuantitative Structure-Activity RelationshipBioengineeringLigandsMachine learningcomputer.software_genre01 natural sciencesConstant false alarm rateSoftwareMolecular descriptorDrug DiscoveryChagas Diseaseclassification treeVirtual screeningMolecular Structure010405 organic chemistrybusiness.industryDecision tree learningGeneral Medicinevirtual screening0104 chemical sciences010404 medicinal & biomolecular chemistryIdentification (information)Tree (data structure)Anti-chagasic actionTest setMolecular MedicineArtificial intelligencebusinesscomputerSoftware
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Scorpiand-like azamacrocycles prevent the chronic establishment of Trypanosoma cruzi in a murine model.

2013

Chagas disease is today one of the most important neglected diseases for its upcoming expansion to non-endemic areas and has become a threat to blood recipients in many countries. In this study, the trypanocidal activity of ten derivatives of a family of aza-scorpiand like macrocycles is evaluated against Trypanosoma cruzi in vitro and in vivo murine model in which the acute and chronic phases of Chagas disease were analyzed. The compounds 4, 3 and 1 were found to be more active against the parasite and less toxic against Vero cells than the reference drug benznidazole, 4 being the most active compound, particularly in the chronic phase. While all these compounds showed a remarkable degree …

Chagas diseaseMacrocyclic CompoundsTrypanosoma cruziAntiprotozoal AgentsLigandsMicrobiologyMiceIn vivoDrug DiscoveryChlorocebus aethiopsmedicineEscherichia coliAnimalsHumansTrypanosoma cruziVero CellsCells CulturedPharmacologychemistry.chemical_classificationAza CompoundsMice Inbred BALB CbiologyMolecular StructureSuperoxide DismutaseOrganic ChemistryGeneral Medicinebiology.organism_classificationmedicine.diseaseIn vitroDisease Models AnimalEnzymechemistryMechanism of actionBenznidazoleImmunologyChronic DiseaseVero cellFemalemedicine.symptommedicine.drugEuropean journal of medicinal chemistry
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A rare example of nickel(ii) chains based on a heteroscorpionate-like ligand with quadruple imidazolyl interactions

2014

The first nickel(ii) complex with the heteroscorpionate-like bridging ligand DIMMAL (2-di1H-2-imidazolylmethylmalonate), [Ni(DIMMAL)(H2O)3]n·3nH2O (1), is a one-dimensional coordination polymer whose structure shows regular Ni(ii) chains with H-bonding inter-chain interactions and a rare example of a Quadruple Imidazolyl Embrace (QIE). The Ni(ii) chain shows a weak antiferromagnetic interaction that can be modelled with a regular S = 1 chain model including a zero field splitting with g = 2.270, J = -1.5 cm(-1) and D = -2.26 cm(-1).

Chain modelCoordination polymerLigandStereochemistrychemistry.chemical_elementBridging ligandZero field splittingInorganic Chemistrychemistry.chemical_compoundNickelCrystallographychemistryChain (algebraic topology)AntiferromagnetismDalton Trans.
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