Search results for "lipogenesis"

showing 10 items of 32 documents

Lipid composition and metabolism of European sea bass (Dicentrarchus labrax L.) fed diets containing wheat gluten and legumes meals as substitutes fo…

2013

The role of dietary protein source on growth performance and lipid metabolism in European sea bass (Dicentrarchus labrax) was investigated, by the evaluation of fat content in different tissues, plasma metabolites, liver lipogenic activity and fatty acid composition of dorsal muscle tissue.Five isonitrogenous (490gkg-1crude protein dry-matter basis) and isolipidic (170gkg-1lipid dry-matter basis) diets with graded levels of wheat gluten, soybean and pea meal in substitution of fish meal were evaluated in European sea bass (initial body weight 23.9±0.1) for 96days. There was a significant difference (p<0.05) in dorsal muscle and liver lipid contents. Plasma glucose and cholesterol levels …

Muscle tissuevegetable mealMealVegetable meal Liver lipogenesis Muscle PUFA Plasma metabolitesLipid metabolismMetabolismplasma metabolitesAquatic ScienceBiologybiology.organism_classificationFish mealmedicine.anatomical_structureSettore BIO/10 - Biochimicalipid metabolismmedicineSettore AGR/18 - Nutrizione E Alimentazione AnimaleDicentrarchusFood sciencevegetable meal; European sea bass; lipid metabolism; muscle PUFA; plasma metabolitesmuscle PUFASea bassEuropean sea bassVegetable meal Liver lipogenesis Muscle PUFA Plasma metabolitesLegume
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Mboat7 down-regulation by hyper-insulinemia induces fat accumulation in hepatocytes.

2020

Background: Naturally occurring variation in Membrane-bound O-acyltransferase domain-containing 7 (MBOAT7), encoding for an enzyme involved in phosphatidylinositol acyl-chain remodelling, has been associated with fatty liver and hepatic disorders. Here, we examined the relationship between hepatic Mboat7 down-regulation and fat accumulation. Methods: Hepatic MBOAT7 expression was surveyed in 119 obese individuals and in experimental models. MBOAT7 was acutely silenced by antisense oligonucleotides in C57Bl/6 mice, and by CRISPR/Cas9 in HepG2 hepatocytes. Findings: In obese individuals, hepatic MBOAT7 mRNA decreased from normal liver to steatohepatitis, independently of diabetes, inflammatio…

Research paperTGFβ Transforming Growth Factor BetaIntracellular SpaceCRISPR Clustered Regularly Interspaced Short Palindromic RepeatshHEPS Human HepatocytesMice0302 clinical medicineLPIAT1DAG Diacylglyceroli.p. Intraperitonealmedia_commonFatty AcidsGeneral Medicine3. Good health030220 oncology & carcinogenesisHOMA-IR homeostasis Model Assessment of Insulin ResistanceMPO morpholinolcsh:Medicine (General)medicine.medical_specialtyPE Phosphatidyl-EthanolamineNashGeneral Biochemistry Genetics and Molecular Biology03 medical and health sciencesTNFα tumor Necrosis Factor AlphaLDL Low Density LipoproteinsHyperinsulinismNAFLDSD Standard Dietmedia_common.cataloged_instanceHumansCPT1 Carnitine Palmitoyltransferase IPhosphatidylinositolGene SilencingEuropean unionVLDL Very Low Density Lipoproteinlcsh:RhHSC Human Hepatic Stellate Cellsmedicine.diseaseLipid MetabolismOA Oleic AcidCI Confidence IntervalMboat7 Membrane bound O-acyltransferase domain containing 7MCD methionine choline deficient diet030104 developmental biologyEndocrinologychemistryCDP Cytidine-DiphosphateFOXO1 Forkhead Box protein O1NAFLD nonalcoholic fatty liver diseaseSteatohepatitisBMI Body Mass IndexCL CardiolipinAcyltransferases0301 basic medicineAlcoholic liver diseaseCXCL10 C-X-C Motif Chemokine 10lcsh:Medicinechemistry.chemical_compoundNon-alcoholic Fatty Liver DiseaseIFG Impaired Fasting GlucoseAPOB Apolipoprotein BNonalcoholic fatty liver diseasePIP Phosphatidyl-Inositol-PhosphateSteatohepatitisqRT-PCR quantitative Real Time Polymerase Chain ReactionMice Knockoutlcsh:R5-920ORO Oil Red O StainingPI PhosphatidylinositolFatty liverTM6SF2 Transmembrane 6 Superfamily Member 2PhospholipidTAG TriglyceridesNASH Nonalcoholic SteatohepatitisLipogenesisLPA Lyso-Phosphatidic AcidPhosphatidylinositolSignal TransductionPS Phosphatidyl-SerinePA Palmitic AcidALD alcoholic liver diseasePC Phosphatidylcholinei.v. IntravenousFATP1 Fatty Acid Transport Protein 1Models BiologicalInternal medicinemedicineAnimalsNonalcoholic fatty liver diseasePPARα Peroxisome Proliferator-Activated Receptor alphaObesityG3P Glyceraldehyde-3-PhosphateSREBP1c Sterol Regulatory Element-Binding Protein 1HDL High Density Lipoproteinsbusiness.industryPI3K Phosphatidylinositol 3 KinaseMembrane ProteinsNHEJ Non-Homologues End JoiningPNPLA3 Patatin-like Phospholipase Domain-containing-3MTTP Microsomal Triglyceride Transfer ProteinLPIAT1 Lysophosphatidylinositol Acyltransferase 1TMC4 Transmembrane Channel-Like 4Disease Models AnimalGene Expression RegulationHepatocytesFOXA2 Forkhead Box A2mTOR mammalian target of RapamycinSteatosisInsulin ResistancebusinessPG Phosphatidyl-GlycerolFABP1 Fatty Acid-Binding Protein 1 FAS Fatty Acid SynthaseT2DM Type 2 Diabetes MellitusEBioMedicine
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Spirulina reduces diet-induced obesity through downregulation of lipogenic genes expression in Psammomys obesus

2020

The present study evaluates the protective effect of spirulina against diet-induced obesity and metabolic disorders in Psammomys obesus, an animal model of metabolic syndrome. Psammomys obesus live...

Spirulina (genus)medicine.medical_specialtybiologyPhysiology030209 endocrinology & metabolismGeneral Medicinemedicine.diseasebiology.organism_classificationObesity03 medical and health sciences0302 clinical medicineEndocrinologyInsulin resistanceDownregulation and upregulation030220 oncology & carcinogenesisPhysiology (medical)Internal medicineLipogenesismedicinePsammomysMetabolic syndromeGeneArchives of Physiology and Biochemistry
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Strikingly High Respiratory Quotients: A Further Characteristic of the Tumor Pathophysiome

2008

Conspicuously high respiratory quotients (RQs) are found in solid tumors in vivo. RQs in the range between 1.29 and 1.95 neither reflect the degree of substrate oxidation by tumor cells nor indicate the types of fuels involved in metabolic processes. Instead, such tumor RQs most probably are caused by (a) channeling of glycolytic end-products into lipogenesis, and by (b) CO2 release from the tumor following extracellular buffering of H+ -inos by bicarbonate. H+ -inos exported from the intracellular space into the interstitial compartment titrate extracellular bicarbonate to CO2 and H2O with the aid of the ectoenzyme carbonic anhydrase IX, which is activated at low pH. Strikingly high (RQs) …

Tumor microenvironmentchemistry.chemical_compoundchemistryBiochemistryIn vivoBicarbonateLipogenesisExtracellularGlycolysisRespiratory systemBiologyIntracellular
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Role of LECT2 in non alcoholic fatty liver disease

2020

The liver is the privileged site of metabolic regulations that must be tightly coordinated to prevent metabolic imbalance to preserve homeostasis. Alteration of these processes can promote steatosis, an essential feature of Non-Alcoholic Fatty Liver Diseases (NAFLD), representing the new global epidemic threat. During this thesis work, we demonstrated that LECT2, a liver secreted factor, is increased in both Human and mice harboring fatty liver. In a physiological context, we demonstrated that LECT2 controls liver lipogenesis through the LXR signalling pathway. At the molecular level, the absence of LECT2 induces a specific accumulation of 25-OHC oxysterol which leads to a significant incre…

[SDV.MHEP] Life Sciences [q-bio]/Human health and pathologySteatosisLect2LiverStéatoseLipogenesisLipogenèseLxrFoie[SDV.MHEP]Life Sciences [q-bio]/Human health and pathology
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Regulation of Farnesyl Diphosphate Synthase Gene Expression by Fatty Acids

2003

Cholesterol biosynthesis depends on the activity of regulatory enzymes, including the peroxisomal Farnesyl Diphosphate Synthase (FPPS ). Cholesterol regulates its own synthesis rate. Hence, as a response to cholesterol depletion, a feed back mechanism is activated, whereby sterol regulatory binding proteins (SREBPla, 1c and 2 ) are subjected to sequential proteolytic activation, which permits their interaction with specific DNA response elements from responsive genes. In turn, the transcriptional activity of cholesterol biosynthesis genes is induced. Conversely, cholesterol accumulation decreases SREBP maturation and transcription of controlled genes. In addition, polyunsaturated fatty acid…

chemistry.chemical_classificationbiologyCholesterolPeroxisomeSterolSterol regulatory element-binding proteinchemistry.chemical_compoundFarnesyl diphosphate synthasechemistryBiochemistryLipogenesisbiology.proteinlipids (amino acids peptides and proteins)GenePolyunsaturated fatty acid
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Coffee Restores Expression of lncRNAs Involved in Steatosis and Fibrosis in a Mouse Model of NAFLD

2021

Background and aim: Coffee intake exerts protective effects against non-alcoholic fatty liver disease (NAFLD), although without fully cleared mechanisms. In this study we aimed to assess whether coffee consumption may influence the expression of long non-coding RNAs (lncRNAs) in the liver. Methods: C57BL/6J mice were fed a 12-week standard diet (SD), high-fat diet (HFD) or HFD plus decaffeinated coffee solution (HFD + coffee). Expression of specific lncRNAs involved in NAFLD was analyzed by real-time PCR. For the most differentially expressed lncRNAs, the analysis was also extended to their mRNA targets. Results: Decaffeinated coffee intake reduced body weight gain, prevented NAFLD, lowered…

lncRNA.Liver CirrhosisMalemedicine.medical_specialtyGm16551; H19; NAFLD; coffee; lncRNA; Animals; Coffee; Disease Models Animal; Fatty Liver; Gene Expression; Liver; Liver Cirrhosis; Male; Mice; Mice Inbred C57BL; Non-alcoholic Fatty Liver Disease; RNA Long NoncodingCoenzyme ACircadian clockcoffeeGene ExpressionBiologyInbred C57BLArticlechemistry.chemical_compoundMicelncRNADownregulation and upregulationFibrosisSettore BIO/13 - Biologia ApplicataNon-alcoholic Fatty Liver DiseaseInternal medicineNAFLDmedicineAnimalsTX341-641Messenger RNANutrition and DieteticsH19Nutrition. Foods and food supplyAnimalGm16551Fatty liverNAFLD; coffee; lncRNA; Gm16551; H19nutritional and metabolic diseasesmedicine.diseaseMice Inbred C57BLFatty LiverDisease Models AnimalEndocrinologychemistryLiverLipogenesisDisease ModelsRNARNA Long NoncodingLong NoncodingSteatosisFood Science
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Multigenerational study of the obesogen effects of bisphenol S after a perinatal exposure in C57BL6/J mice fed a high fat diet.

2021

International audience; Background : Bisphenol S is an endocrine disruptor exhibiting metabolic disturbances, especially following perinatal exposures. To date, no data are available on the obesogen effects of BPS in a mutligenerational issue.Objectives : We investigated obesogen effects of BPS in a multigenerational study by focusing on body weight, adipose tissue and plasma parameters in male and female mice.Methods : Pregnant C57BL6/J mice were exposed to BPS (1.5 μg/kg bw/day ie a human equivalent dose of 0.12 μg/kg bw/day) by drinking water from gestational day 0 to post natal day 21. All offsprings were fed with a high fat diet during 15 weeks. Body weight was monitored weekly and fat…

medicine.medical_specialty010504 meteorology & atmospheric sciences[SDV]Life Sciences [q-bio]Health Toxicology and MutagenesisAdipose tissue010501 environmental sciencesToxicologyDiet High-Fat01 natural scienceschemistry.chemical_compoundMiceNEFAPhenolsPregnancyLow dose exposureInternal medicinemedicineLipolysisAnimalsObesogenSulfonesPeri-natal exposure0105 earth and related environmental sciences2. Zero hungerTriglyceridebusiness.industryGeneral Medicinemedicine.diseasePollutionEndocrinologyTransgenerational effectschemistryEndocrine disruptorPrenatal Exposure Delayed EffectsLipogenesisFemalebusinessDyslipidemiaObesogenEnvironmental pollution (Barking, Essex : 1987)
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Different effects of pioglitazone and rosiglitazone on lipid metabolism in mouse cultured liver explants.

2010

Background Pioglitazone (PIO) and rosiglitazone (ROSI) are widely used as oral antidiabetic agents for treatment of type 2 diabetes. Although these medications exert similar effects on blood glucose, recent clinical studies indicated that PIO has a more pronounced beneficial effect on lipid parameters than ROSI. In order to get further insight into the lipid effects of both drugs, we tested whether PIO, compared to ROSI, could exert direct effects on lipid liver metabolism in relation with plasma lipids. Methods We performed in vitro studies using mice liver slices incubated 21 h either with ROSI (1 µmol/L) or PIO (7.5 µmol/L). Results We showed that both glitazones slightly reduced HMG-CoA…

medicine.medical_specialtyApolipoprotein BEndocrinology Diabetes and MetabolismRosiglitazoneTissue Culture Techniqueschemistry.chemical_compoundMiceEndocrinologyInternal medicineInternal MedicinemedicineAnimalsHumansScavenger receptorGlycated HemoglobinbiologyPioglitazoneCholesterolbusiness.industryCholesterol HDLLipid metabolismLipaseLipid MetabolismMice Inbred C57BLPPAR gammaEndocrinologychemistryLiverLipogenesisbiology.proteinThiazolidinedionesHepatic lipaseRosiglitazonebusinessPioglitazonemedicine.drugDiabetes/metabolism research and reviews
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Chatting Between the Brain and White Adipose Tissues

2012

Over the past decades, numerous papers have been published demonstrating the importance of the relationships between the brain and white adipose tissues in regard to body weight and metabolism regulation. Indeed the brain mainly via the sympathetic nervous system control body fat mass both by regulating adipocytes metabolism (lipolysis and lipogenesis), secretory activity (leptin and other adipokines) as well as development. In turn fat mass will send information to some brain areas via sensory nerves as well as via changes in metabolic and hormonal signals. Altogether these data are in support of a feedback loop between white adipose tissues and the brain. This crosstalk plays a major role…

medicine.medical_specialtyAutonomic nervous systemEndocrinologyInternal medicineLeptinLipogenesismedicineAdipokineLipolysisAdipose tissueWhite adipose tissueBiologyEnergy homeostasis
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