Search results for "liver neoplasm"

showing 10 items of 740 documents

The stable repression of mesenchymal program is required for hepatocyte identity: A novel role for hepatocyte nuclear factor 4α

2011

The concept that cellular terminal differentiation is stably maintained once development is complete has been questioned by numerous observations showing that differentiated epithelium may undergo an epithelial-to-mesenchymal transition (EMT) program. EMT and the reverse process, mesenchymal-to-epithelial transition (MET), are typical events of development, tissue repair, and tumor progression. In this study, we aimed to clarify the molecular mechanisms underlying these phenotypic conversions in hepatocytes. Hepatocyte nuclear factor 4α (HNF4α) was overexpressed in different hepatocyte cell lines and the resulting gene expression profile was determined by real-time quantitative polymerase…

Transcription FactorCellular differentiationMESH: Mice KnockoutMESH: HepatocytesMesodermMice0302 clinical medicineMESH: Liver NeoplasmsMESH: AnimalsHepatocyteHepatocyte Nuclear Factor 1-alphaMESH: Carcinoma HepatocellularRegulator geneHepatocyte differentiationMice KnockoutMESH: Mesoderm0303 health sciencesLiver NeoplasmsCell DifferentiationMESH: Transcription FactorsCell biologyHepatocyte nuclear factorsPhenotypeMESH: Models AnimalHepatocyte Nuclear Factor 4MESH: Epithelial CellsLiver Neoplasm030220 oncology & carcinogenesisModels AnimalMESH: Hepatocyte Nuclear Factor 4HumanMESH: Cell DifferentiationMESH: Cell Line TumorCarcinoma Hepatocellular[SDV.BC]Life Sciences [q-bio]/Cellular BiologyBiologyMESH: PhenotypeArticle03 medical and health scienceshepatocyte; mesenchymal program; SnailCell Line TumorAnimalsHumansMESH: Hepatocyte Nuclear Factor 1-alphaMESH: MiceTranscription factorAnimals; Carcinoma Hepatocellular; Cell Differentiation; Cell Line Tumor; Epithelial Cells; Hepatocyte Nuclear Factor 1-alpha; Hepatocyte Nuclear Factor 4; Hepatocytes; Humans; Liver Neoplasms; Mesoderm; Mice; Mice Knockout; Models Animal; Phenotype; Snail Family Transcription Factors; Transcription Factors; Hepatology030304 developmental biologyEpithelial CellMESH: HumansHepatologyAnimalMesenchymal stem cellEpithelial CellsSnail Family Transcription FactorMolecular biologyHepatocyte nuclear factor 4HepatocytesSnail Family Transcription FactorsChromatin immunoprecipitationTranscription Factors
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TGFβ-induced EMT requires focal adhesion kinase (FAK) signaling

2007

The epithelial-to-mesenchymal transition (EMT) is a crucial process, occurring both during development and tumor progression, by which an epithelial cell undergoes a conversion to a mesenchymal phenotype, dissociates from initial contacts and migrates to secondary sites. We recently reported that in hepatocytes the multifunctional cytokine TGFβ induces a full EMT characterized by (i) Snail induction, (ii) E-cadherin delocalization and down-regulation, (iii) down-regulation of the hepatocyte transcriptional factor HNF4α and (iv) up-regulation of mesenchymal and invasiveness markers. In particular, we showed that Snail directly causes the transcriptional down-regulation of E-cadherin and HN…

Transcriptional ActivationTGFβFAK; MT; Src; TGFβ; Animals; Biomarkers Tumor; Cadherins; Cell Line; Cell Transformation Neoplastic; Enzyme Activation; Epithelial Cells; Focal Adhesion Protein-Tyrosine Kinases; Hepatocytes; Liver Neoplasms; Mesoderm; Mice; Neoplasm Invasiveness; Signal Transduction; Transcriptional Activation; Transforming Growth Factor beta; Up-Regulation; src-Family Kinases; Cell BiologyCell LineMesodermFocal adhesionMiceTransforming Growth Factor betaBiomarkers TumorAnimalsHepatocyteNeoplasm InvasivenessNeoplasm InvasiveneEpithelial CellFocal Adhesion Protein-Tyrosine KinaseFAKbiologyAnimalCadherinLiver NeoplasmsMesenchymal stem cellEpithelial CellsCell BiologyTransforming growth factor betaTgf beta; fak; srcCadherinsUp-RegulationCell biologyEnzyme ActivationCell Transformation Neoplasticsrc-Family KinasesHepatocyte nuclear factor 4Liver NeoplasmTumor progressionMTFocal Adhesion Protein-Tyrosine KinasesCadherinHepatocytesCancer researchbiology.proteinsrc-Family KinaseSignal transductionSrcSignal TransductionProto-oncogene tyrosine-protein kinase SrcExperimental Cell Research
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Comparative toxicity and cell-tissue distribution study on nanoparticular iron complexes using avian embryos and HepG2-cells

2007

In this study the toxicity and intracellular availability of iron from iron dextran (FeD), iron sucrose (FeS), and iron gluconate (FeG) was compared in organs of avian (turkey) embryos and in isolated cells (HepG2) in cell culture. Iron uptake was more pronounced in embryonic liver than in renal tissue. Cellular iron uptake in liver and kidney was more or less similar for the different compounds. Only some experiments showed slightly greater iron concentrations in liver and kidney with FeG compared with FeD and FeS. Significant differences were found in the survival ratios of the eggs and the embryo weights depending on the type of iron complex administered. The rank order of toxicities was…

TurkeysCarcinoma HepatocellularEmbryo NonmammalianLiver cytologyChick EmbryoBiologyKidneyIron sucroseIn ovoFerric CompoundsGluconatesAndrologyGlucaric AcidIn vivoCell Line TumorPhysiology (medical)medicineAnimalsHumansFerric Oxide SaccharatedBody WeightLiver NeoplasmsBiochemistry (medical)Public Health Environmental and Occupational HealthKidney metabolismGeneral MedicineIn vitroSurvival RateLiverBiochemistryCell cultureToxicityHematinicsNanoparticlesIron-Dextran Complexmedicine.drugTranslational Research
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Uncommon evolutions and complications of common benign liver lesions

2018

International audience; Frequently encountered on abdominal imaging studies, the majority of common benign liver lesions are asymptomatic, confidently diagnosed by imaging, and do not require further workup, follow-up, or treatment. The increasing use of multimodality liver imaging, has allowed the recognition of uncommon evolutions of common benign liver lesions such as size changes, fibrotic regression, and content and vascularization changes, and their complications such as rupture, hemorrhage, thrombosis, extrinsic compression, and malignancy. The purpose of this pictorial review is to describe and illustrate the incidence and diagnostic features of these uncommon evolutions and complic…

Uncommon evolutionsmedicine.medical_specialtyBenign liver lesionComplications[SDV]Life Sciences [q-bio]UrologyComputed tomographyMalignancyAsymptomaticExtrinsic compression03 medical and health sciences0302 clinical medicineInternal medicinemedicineBenign liver lesionsHumansRadiology Nuclear Medicine and imagingComputed tomographyRadiological and Ultrasound Technologymedicine.diagnostic_testbusiness.industryLiver DiseasesLiver NeoplasmsGastroenterologyMagnetic resonance imagingHepatologymedicine.diseaseMagnetic Resonance ImagingThrombosis3. Good healthLiver030220 oncology & carcinogenesis030211 gastroenterology & hepatologyRadiologyDifferential diagnosismedicine.symptomTomography X-Ray ComputedbusinessComplicationUncommon evolutionAbdominal Radiology
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Epidemiology of Nonalcoholic Fatty Liver Disease and Nonalcoholic Steatohepatitis: Implications for Liver Transplantation.

2018

Nonalcoholic fatty liver disease (NAFLD) affects 25% of the global adult population with a range of 13.5% in Africa and 31.8% in the Middle East. Nonalcoholic fatty liver disease is closely associated with a constellation of metabolic comorbidities which include: obesity, type 2 diabetes mellitus, hypertension, and hypercholesteremia. In fact, the increasing number of metabolic comorbidities not only increases the prevalence of NAFLD but also places patients at higher risk for progressive liver disease. As such, NAFLD is presently among the top etiologies for hepatocellular carcinoma and an indication for liver transplantation (LT) in the United States. Therefore, the following recommendati…

United StateLiver Cirrhosismedicine.medical_specialtyCarcinoma HepatocellularTissue and Organ ProcurementWaiting ListsLiver Cirrhosimedicine.medical_treatmentComorbidity030230 surgeryLiver transplantationdigestive system03 medical and health sciencesLiver disease0302 clinical medicineNon-alcoholic Fatty Liver DiseaseRisk FactorsInternal medicineEpidemiologyNonalcoholic fatty liver diseasePrevalenceMedicineHumansMetabolic SyndromeTransplantationbusiness.industryRisk FactorIncidencePatient SelectionLiver Neoplasmsnutritional and metabolic diseasesType 2 Diabetes Mellitusmedicine.diseaseComorbidityObesitydigestive system diseasesUnited StatesLiver TransplantationLiver NeoplasmWaiting ListDisease Progression030211 gastroenterology & hepatologyMetabolic syndromebusinessHumanTransplantation
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Infrequent promoter methylation of the MGMT gene in liver metastases from uveal melanoma.

2008

Uveal melanoma is associated with a high mortality rate once metastases occur, with over >90% of metastatic patients dying within less than 1 year from metastases to the liver. The intraarterial hepatic (iah) administration of the alkylating agent fotemustine holds some promise with response rates of 36% and median survival of 15 months. Here, we investigated whether the DNA-repair-protein MGMT may be involved in the variability of response to fotemustine and temozolomide in uveal melanoma. Epigenetic inactivation of MGMT has been demonstrated to be a predictive marker for benefit from alkylating agent therapy in glioblastoma. We found a methylated MGMT promoter in 6% of liver metastases fr…

Uveal NeoplasmsCancer ResearchPathologymedicine.medical_specialtyProtein Array AnalysisAntineoplastic AgentsBiologyMetastasischemistry.chemical_compoundPredictive Value of TestsmedicineBiomarkers TumorHumansPromoter Regions GeneticneoplasmsDNA Modification MethylasesMelanomaTemozolomidePredictive markerTissue microarrayMelanomaTumor Suppressor ProteinsLiver NeoplasmsCancerDNA Methylationmedicine.diseaseImmunohistochemistrydigestive system diseasesNitrogen mustardDNA Repair EnzymesOncologychemistryCancer researchFotemustinemedicine.drugInternational journal of cancer
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Liver specific overexpression of platelet-derived growth factor-B accelerates liver cancer development in chemically induced liver carcinogenesis

2010

A genetic basis of hepatocellular carcinoma (HCC) has been well-established and major signaling pathways, such as p53, Wnt-signaling, transforming growth factor-β (TGF-β) and Ras pathways, have been identified to be essential to HCC development. Lately, the family of platelet-derived growth factors (PDGFs) has shifted to the center of interest. We have reported on spontaneously developing liver fibrosis in PDGF-B transgenic mice. Since HCC rarely occurs in healthy liver, but dramatically increases at the cirrhosis stage of which liver fibrosis is a preliminary stage, we investigated liver cancer development in chemically induced liver carcinogenesis in these mice. HCC induction was performe…

Vascular Endothelial Growth Factor AAlkylating AgentsCancer Researchmedicine.medical_specialtyCarcinoma HepatocellularCirrhosisPlatelet-derived growth factorBlotting WesternMice TransgenicBiologymedicine.disease_causeImmunoenzyme TechniquesMicechemistry.chemical_compoundLiver Neoplasms ExperimentalInternal medicinemedicineAnimalsHumansDiethylnitrosamineRNA MessengerReverse Transcriptase Polymerase Chain ReactionCancerProto-Oncogene Proteins c-sismedicine.diseaseFibroblast Growth FactorsPlatelet Endothelial Cell Adhesion Molecule-1Vascular endothelial growth factorEndocrinologyOncologychemistryPhenobarbitalbiology.proteinAnticonvulsantsCarcinogenesisLiver cancerPlatelet-derived growth factor receptorTransforming growth factorInternational Journal of Cancer
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Formation of pre-neoplastic hepatocellular foci by vinyl fluoride in newborn rats

1981

Vinyl CompoundsHealth Toxicology and MutagenesisLiver NeoplasmsPharmacology toxicologyNeoplasms ExperimentalGeneral MedicinePharmacologyToxicologyRatschemistry.chemical_compoundAnimals NewbornchemistryAnimalsOrganic chemistryFemalePrecancerous ConditionsFluorideVinyl fluorideArchives of Toxicology
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Epidemiology, Risk Factors, and Natural History of Hepatocellular Carcinoma

2002

The incidence of hepatocellular carcinoma is increasing in many countries. The estimated number of new cases annually is over 500,000, and the yearly incidence comprises between 2.5 and 7% of patients with liver cirrhosis. The incidence varies between different geographic areas, being higher in developing areas; males are predominantly affected, with a 2:3 male/female ratio. The heterogeneous geographic distribution reflects the epidemiologic impact of the main etiologic factors and environmental risk, which are the hepatitis B (HBV) and hepatitis C (HCV) viruses. The percentage of cases of hepatocellular carcinoma attributable to HBV worldwide is 52.3% and is higher in Asia where the serop…

Viral Hepatitis VaccinesHepatitis B virusmedicine.medical_specialtyAflatoxin B1Carcinoma HepatocellularCirrhosisPopulationHepacivirusmedicine.disease_causeGastroenterologyGeneral Biochemistry Genetics and Molecular BiologyHistory and Philosophy of ScienceRisk FactorsInternal medicinemedicineCarcinomaHumanseducationHepatitis B viruseducation.field_of_studybusiness.industryGeneral NeuroscienceIncidence (epidemiology)Liver NeoplasmsHepatitis CHepatitis BHepatitis Bmedicine.diseaseHepatitis Cdigestive system diseasesAlcoholsHepatocellular carcinomabusinessAnnals of the New York Academy of Sciences
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Waiting-time and quality of care deserved to patients with early stage hepatocellular carcinoma undergoing RFA treatment

2017

Waiting timemedicine.medical_specialtyCarcinoma HepatocellularSettore MED/09 - Medicina InternaHepatologybusiness.industryLiver NeoplasmsSpecialties of internal medicineGeneral Medicinemedicine.diseaseSurgeryRC581-951Liver NeoplasmHepatocellular carcinomamedicineCatheter AblationHumansHepatectomyCatheter Ablation; Hepatectomy; Humans; Quality of Health Care; Carcinoma Hepatocellular; Liver Neoplasms; HepatologyStage (cooking)Quality of carebusinessHumanQuality of Health Care
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