Search results for "lymphocyte"

showing 10 items of 2280 documents

Second-generation Langerhans cells originating from epidermal precursors are essential for CD8+ T cell priming.

2014

Abstract In vivo studies questioned the ability of Langerhans cells (LCs) to mediate CD8+ T cell priming. To address this issue, we used intradermal immunization with plasmid DNA, a system in which activation of CD8+ T cells depends on delayed kinetics of Ag presentation. We found that dendritic cells (DCs) located in the skin at the time of immunization have limited ability to activate CD8+ T cells. This activity was mediated by a second generation of DCs that differentiated in the skin several days after immunization, as well as by lymph node–resident DCs. Intriguingly, CD8+ T cell responses were not affected following treatment with clodronate liposomes, immunization of CCR2−/− mice, or …

Receptors CCR2T cellImmunologyPriming (immunology)CD11cchemical and pharmacologic phenomenaBiologyCD8-Positive T-LymphocytesLymphocyte ActivationMiceImmune systemGiant Cells LanghansmedicineImmunology and AllergyCytotoxic T cellAnimalsSkinMice KnockoutChemokine CCL20integumentary systemhemic and immune systemsCell DifferentiationDendritic CellsMolecular biologyCD11c AntigenCCL20Mice Inbred C57BLmedicine.anatomical_structureImmunologyIntercellular Signaling Peptides and ProteinsClodronic AcidCD8Ex vivoHeparin-binding EGF-like Growth FactorPlasmidsJournal of immunology (Baltimore, Md. : 1950)
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Amazing IL-9: revealing a new function for an “old” cytokine

2012

Th9 cells are a subset of CD4+ Th cells that produce the pleiotropic cytokine IL-9. IL-9/Th9 can function as both positive and negative regulators of immune response, but the role of IL-9/Th9 in tumor immunity is unknown. We examined the role of IL-9/Th9 in a model of pulmonary melanoma in mice. Lack of IL-9 enhanced tumor growth, while tumor-specific Th9 cell treatment promoted stronger antitumor responses in both prophylactic and therapeutic models. Th9 cells also elicited strong host antitumor CD8+ CTL responses by promoting Ccl20/Ccr6-dependent recruitment of DCs to the tumor tissues. Subsequent tumor antigen delivery to the draining LN resulted in CD8+ T cell priming. In agreement with…

Receptors CCR6Lung Neoplasmsmedicine.medical_treatmentBiologyCD8-Positive T-LymphocytesMiceImmunityCell Line TumormedicineAnimalsLack of knowledgeInterleukin 9MelanomaMice KnockoutAntigen PresentationImmunity CellularChemokine CCL20Antitumor immunityMelanomaInterleukin-9General MedicineDendritic CellsT-Lymphocytes Helper-Inducermedicine.diseaseCytokineImmunologyCommentaryImmunotherapySkin cancerFunction (biology)
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Crosstalk of regulatory T cells and tolerogenic dendritic cells prevents contact allergy in subjects with low zone tolerance

2012

Background Allergic contact dermatitis is one of the most common occupational diseases. A main protective mechanism in those who do not develop allergic contact dermatitis is tolerance induction by repeated exposure to low doses of contact allergen, which is termed low zone tolerance (LZT). The mechanisms that determine the tolerance induction in subjects with LZT are still elusive. Objective We performed analysis of the role of CD4 + CD25 + forkhead box protein 3 (FOXP3)–positive regulatory T (Treg) cells and dendritic cells (DCs) in mice with LZT. Methods Mechanisms of tolerance induction were analyzed in a murine model of LZT by using FOXP3 and IL-10 reporter mice, as well as mice that a…

Receptors CCR7Adoptive cell transferImmunologyMice Transgenicchemical and pharmacologic phenomenaCell CommunicationBiologyLymphocyte ActivationT-Lymphocytes RegulatoryMiceImmune ToleranceAnimalsImmunology and AllergyIL-2 receptorInterleukin-2 Receptor alpha SubunitFOXP3Forkhead Transcription Factorshemic and immune systemsDendritic CellsDendritic cellCD11c AntigenInterleukin-10Tolerance inductionInterleukin 10CTLA-4Dermatitis Allergic ContactImmunologyCD8Journal of Allergy and Clinical Immunology
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Regulatory T cells selectively preserve immune privilege of self-antigens during viral central nervous system infection.

2012

Abstract Regulatory T cells (Tregs) are important for the attenuation of immune reactions. During viral CNS infections, however, an indiscriminate maintenance of CNS immune privilege through Treg-mediated negative regulation could prevent autoimmune sequelae but impair the control of viral replication. We analyzed in this study the impact of Tregs on the development of acute viral encephalomyelitis, T cell-mediated antiviral protection, and prevention of CNS autoimmunity following intranasal infection with the gliatropic mouse hepatitis virus strain A59. To assess the contribution of Tregs in vivo, we specifically depleted CD4+Foxp3+ T cells in a diphtheria toxin-dependent manner. We found …

Receptors CXCR3T cellImmunologychemical and pharmacologic phenomenaAutoimmunityBiologyCD8-Positive T-Lymphocytesmedicine.disease_causeCXCR3Lymphocyte ActivationAutoantigensT-Lymphocytes RegulatoryLymphocyte DepletionAutoimmunity03 medical and health sciencesMice0302 clinical medicineCentral Nervous System InfectionsImmune privilegeImmunitymedicineImmunology and AllergyAnimalsHumansEncephalomyelitisAdministration Intranasal030304 developmental biologyCell Proliferation0303 health sciencesImmunity CellularMice Inbred BALB CMurine hepatitis virusFOXP3hemic and immune systemsForkhead Transcription Factors3. Good healthmedicine.anatomical_structureViral replicationImmunologyAcute DiseaseCD4 AntigensLymph NodesCoronavirus InfectionsCD8030215 immunologyJournal of immunology (Baltimore, Md. : 1950)
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C-X-C Motif Chemokine Receptor 4 Blockade Promotes Tissue Repair After Myocardial Infarction by Enhancing Regulatory T Cell Mobilization and Immune-R…

2019

Background: Acute myocardial infarction (MI) elicits an inflammatory response that drives tissue repair and adverse cardiac remodeling. Inflammatory cell trafficking after MI is controlled by C-X-C motif chemokine ligand 12 (CXCL12) and its receptor, C-X-C motif chemokine receptor 4 (CXCR4). CXCR4 antagonists mobilize inflammatory cells and promote infarct repair, but the cellular mechanisms are unclear. Methods: We investigated the therapeutic potential and mode of action of the peptidic macrocycle CXCR4 antagonist POL5551 in mice with reperfused MI. We applied cell depletion and adoptive transfer strategies using lymphocyte-deficient Rag1 knockout mice; DEREG mice, which express a diphth…

Receptors CXCR4Regulatory T cellCXCR4 antagonistSus scrofaAnti-Inflammatory AgentsMyocardial InfarctionNeovascularization PhysiologicMice TransgenicInflammation030204 cardiovascular system & hematologyT-Lymphocytes RegulatoryVentricular Function Left03 medical and health sciencesChemokine receptor0302 clinical medicineImmune systemPhysiology (medical)medicineAnimalsMyocardial infarction030304 developmental biology0303 health sciencesMobilizationVentricular Remodelingbusiness.industryMyocardiumProteinsDendritic CellsRecovery of FunctionRegulatory T cellsTissue repairmedicine.diseaseMyocardial ContractionBlockadeMice Inbred C57BLDisease Models Animalmedicine.anatomical_structureCancer researchmedicine.symptomCardiology and Cardiovascular MedicinebusinessSignal TransductionCirculation
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Reduced virus specific T helper cell induction by autologous dendritic cells in patients with chronic hepatitis B-restoration by exogenous interleuki…

2002

SUMMARYInsufficient stimulatory capacities of autologous dendritic cells (DC) may contribute in part to impaired T cell stimulation and therefore viral persistence in patients with chronic hepatitis B virus (HBV) infection. In order to characterize the antigen presenting functions of DC from chronic HBV carriers and controls antigen specific T cell responses were analysed. CD34+ peripheral blood progenitor cells were differentiated to immature DC in the presence of GM-CSF, IL-6/IL-6R fusion protein and stem cell factor. Proliferative CD4+ T cell responses and specific cytokine release were analysed in co-cultures of DC pulsed with HBV surface and core antigens or tetanus toxoid and autologo…

Recombinant Fusion ProteinsT cellImmunologyAntigen presentationBiologyHepatitis B ChronicImmune systemAntigenClinical StudiesTetanus ToxoidmedicineHumansImmunology and AllergyCells CulturedAntigen PresentationLymphokinesStem Cell FactorHepatitis B Surface AntigensInterleukin-6Granulocyte-Macrophage Colony-Stimulating FactorCell DifferentiationConvalescenceDendritic CellsT-Lymphocytes Helper-InducerT helper cellDendritic cellHepatitis Bmedicine.diseaseHepatitis B Core AntigensInterleukin-12VirologyCoculture Techniquesmedicine.anatomical_structureImmunologyInterleukin 12
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Soluble interleukin-6 receptor (sIL-6R) makes IL-6R negative T cell line respond to IL-6: it inhibits TNF production

2000

The receptor for interleukin-6 (IL-6) consists of two subunits: a ligand specific IL-6Ralpha and gp130 that is responsible for signal-transduction. A soluble form of the ligand specific chain was described that when complexed to IL-6 is capable of binding to the membrane-bound gp130 subunit and thus can elicit signal-transduction. This soluble receptor can act on cells that express only the gp130 but not the ligand-specific subunit of the IL-6R. This phenomenon, called trans-signaling, introduced a novel aspect of cytokine action. In this study we examined the response of Jurkat cells, that are known not to express IL-6Ralpha, to IL-6, the soluble IL-6 receptor (sIL-6R) and a covalent compl…

Recombinant Fusion ProteinsT-Lymphocytesmedicine.medical_treatmentT cellImmunologyBiologyJurkat cellsCell LineInterferon-gammaJurkat CellsAntigens CDCytokine Receptor gp130medicineHumansImmunology and AllergyInterferon gammaElméleti orvostudományokReceptorLymphotoxin-alphaMembrane GlycoproteinsInterleukin-6Tumor Necrosis Factor-alphaReceptors InterleukinOrvostudományokLigand (biochemistry)Glycoprotein 130Receptors Interleukin-6Molecular biologyCytokinemedicine.anatomical_structureSolubilityTumor necrosis factor alphamedicine.drug
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Specific Regulation of T Helper Cell 1–mediated Murine Colitis by CEACAM1

2004

Carcinoembryonic antigen-related cellular adhesion molecule 1 (CEACAM1) is a cell surface molecule that has been proposed to negatively regulate T cell function. We have shown that CEACAM1 is associated with specific regulation of T helper cell (Th)1 pathways, T-bet–mediated Th1 cytokine signaling, and Th1-mediated immunopathology in vivo. Mice treated with anti–mouse CEACAM1-specific monoclonal antibody (mAb) CC1 during the effector phase exhibited a reduced severity of trinitrobenzene sulfonic acid colitis in association with decreased interferon (IFN)-γ production. Although oxazolone colitis has been reported as Th2 mediated, mice treated with the CC1 mAb or a CEACAM1-Fc chimeric protein…

Recombinant Fusion Proteinsmedicine.medical_treatmentT cellImmunologyBiologyArticleOxazoloneInterferon-gammaMice03 medical and health scienceschemistry.chemical_compound0302 clinical medicineAntigeninflammatory bowel diseaseInterferonmedicineAnimalsImmunology and AllergyColitisCell adhesionCEACAM1030304 developmental biologyInflammationMice KnockoutMice Inbred BALB C0303 health sciencesT cell immunityOxazoloneAntibodies MonoclonalT-Lymphocytes Helper-InducerT helper cellTh1 CellsColitismedicine.diseaseMolecular biologyCarcinoembryonic AntigenImmunoglobulin Fc Fragments3. Good healthMice Inbred C57BLDisease Models Animalmedicine.anatomical_structureCytokinechemistry030220 oncology & carcinogenesisFemaleTh1 cytokineInterleukin-1hapten-induced colitismedicine.drugJournal of Experimental Medicine
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Exogenous introduction of an immunodominant peptide from the non-structural IE1 protein of human cytomegalovirus into the MHC class I presentation pa…

2008

Exogenous introduction of particle-associated proteins of human cytomegalovirus (HCMV) into the major histocompatibility complex (MHC) class I presentation pathway by subviral dense bodies (DB) is an effective way to sensitize cells against CD8 T-cell (CTL) recognition and killing. Consequently, these particles have been proposed as a platform for vaccine development. We have developed a strategy to refine the antigenic composition of DB. For proof of principle, an HCMV recombinant (RV-VM3) was generated that encoded the immunodominant CTL determinant IE1TMY from the IE1 protein in fusion with the major constituent of DB, the tegument protein pp65. To generate RV-VM3, a bacterial artificial…

Recombinant Fusion ProteinsvirusesCytomegalovirusImmunodominanceMajor histocompatibility complexImmediate-Early Proteinslaw.inventionViral ProteinsAntigenlawVirologyMHC class IHumansAntigen PresentationbiologyHistocompatibility Antigens Class IVirionvirus diseasesViral VaccinesGenetic TherapyFusion proteinVirologyPeptide FragmentsCTL*Cytomegalovirus Infectionsbiology.proteinRecombinant DNACD8T-Lymphocytes CytotoxicJournal of General Virology
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Lymphocyte subset reference values in peripheral blood: European survey

1992

Reference valuesImmunologyCell BiologyGeneral MedicineBiologyPeripheral bloodLymphocyte subsetsBiology of the Cell
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