Search results for "macrophage"

showing 10 items of 781 documents

THU0582 Prognostic Factors of Macrophage Activation Syndrome in Adults: Analysis of 40 Cases in 2 Tertiary Referral Centers

2016

Background Macrophage activation syndrome (MAS) is a rare, life-threatening disease in which early diagnosis and aggressive therapeutic strategy may improve the outcome [1]. Due to its rarity, epidemiologic data are still lacking. Hyperferritinemia is frequently associated with MAS and might modulate the cytokines storm the latter contributing to the development of multi-organ failure [2]. Objectives In the current study, we aimed to investigate clinical data, treatments, and outcome of adult MAS patients secondary to autoimmune rheumatic disease. Methods We retrospectively investigated clinical data, treatments, and outcome of 40 adult MAS patients secondary to autoimmune rheumatic disease…

medicine.medical_specialtyImmunologyCatastrophic antiphospholipid syndromeGastroenterologyGeneral Biochemistry Genetics and Molecular Biologychemistry.chemical_compoundTocilizumabRheumatologyInternal medicinemedicineImmunology and AllergyMacrophage activation syndrome adult onset Still‟s disease hyperferritinemic syndromebusiness.industrySeptic shockProportional hazards modelMortality ratehyperferritinemic syndromeadult onset Still‟s diseasemedicine.diseaseComorbiditySurgeryMethylprednisolonechemistryMacrophage activation syndromeMacrophage activation syndromebusinessmedicine.drugAnnals of the Rheumatic Diseases
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Neurological impairment in experimental antiphospholipid syndrome is associated with increased ligand binding to hippocampal and cortical serotonergi…

2013

The antiphospholipid syndrome (APS) is an autoimmune disease where the presence of high titers of circulating autoantibodies causes thrombosis with consecutive infarcts. In experimental APS (eAPS), a mouse model of APS, behavioral abnormalities develop in the absence of vessel occlusion or infarcts. Using brain hemispheres of control and eAPS mice with documented neurological and cognitive deficits, we checked for lymphocytic infiltration, activation of glia and macrophages, as well as alterations of ligand binding densities of various neurotransmitter receptors to unravel the molecular basis of this abnormal behavior. Lymphocytic infiltrates were immunohistochemically characterized using a…

medicine.medical_specialtyImmunologyHippocampusAMPA receptorBiologySerotonergicHippocampusMiceNeurotransmitter receptorInternal medicinemedicineAnimalsImmunology and AllergyLymphocytesReceptor5-HT receptorAutoantibodiesBehavior AnimalMicrogliaGABAA receptorMacrophagesSomatosensory CortexHematologyAntiphospholipid SyndromeAntigens DifferentiationUp-RegulationDisease Models Animalmedicine.anatomical_structureEndocrinologynervous systemAstrocytesReceptor Serotonin 5-HT1ANervous System DiseasesImmunobiology
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Intraperitoneal injection of tetracyclines protects mice from lethal endotoxemia downregulating inducible nitric oxide synthase in various organs and…

1997

We have tested whether tetracyclines (TETs) are able to protect mice from lipopolysaccharide (LPS)-induced shock, a cytokine-mediated inflammatory reaction. Mice, injected with a single dose of tetracycline base (TETb; 1.5, 10 and 20 mg/kg of body weight) or doxycycline (DOXY; 1.5 mg/kg), were significantly protected from a lethal intraperitoneal injection of LPS (500 micrograms per mouse). TETs acted in early events triggered in response to LSP; in fact, they were no longer significantly protective if injected more than 1 h after the injection of endotoxin. LPS-treated mice protected by TETs showed a significant inhibition of tumor necrosis factor alpha (TNF-alpha), interleukin-1 alpha (IL…

medicine.medical_specialtyLipopolysaccharidemedicine.medical_treatmentIntraperitoneal injectionDown-RegulationAlpha (ethology)SpleenBiologyMicechemistry.chemical_compoundInternal medicinemedicineAnimalsPharmacology (medical)LungAntibacterial agentPharmacologyMice Inbred BALB CNitratesTumor Necrosis Factor-alphaTetracyclineShock SepticEndotoxemiaAnti-Bacterial AgentsNitric oxide synthaseInfectious DiseasesEndocrinologyCytokinemedicine.anatomical_structurechemistryDoxycyclineEnzyme InductionMacrophages Peritonealbiology.proteinCytokinesFemaleTumor necrosis factor alphaNitric Oxide SynthaseInjections IntraperitonealSpleenInterleukin-1Research ArticleAntimicrobial Agents and Chemotherapy
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Initiation and progression of atherosclerosis – enzymatic or oxidative modification of low-density lipoprotein?

2006

AbstractAtherosclerosis is widely regarded as a chronic inflammatory disease that develops as a consequence of entrapment of low-density lipoprotein (LDL) in the arterial intima. Native LDL lacks inflammatory properties, so the lipoprotein must undergo biochemical alterations to become atherogenic. Among several other candidates, two different concepts of lipoprotein modification are propagated, the widespread oxidation hypothesis and the less common E-LDL hypothesis, which proposes that modification of LDL occurs through the action of ubiquitous hydrolytic enzymes (enzymatically modified LDL or E-LDL) rather than oxidation. By clearly distinguishing between the initiation and progression o…

medicine.medical_specialtyLipoprotein modificationHydrolasesClinical BiochemistryOxidative phosphorylationDiseaseModels Biologicalchemistry.chemical_compoundInternal medicinemedicineAnimalsHumansMacrophagechemistry.chemical_classificationVascular diseaseBiochemistry (medical)General MedicineSterol EsteraseAtherosclerosismedicine.diseaseLipoproteins LDLC-Reactive ProteinEndocrinologyEnzymechemistryLow-density lipoproteinlipids (amino acids peptides and proteins)Oxidation-ReductionPeptide HydrolasesLipoproteinClinical Chemistry and Laboratory Medicine (CCLM)
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Canakinumab in systemic juvenile idiopathic arthritis: real-world data from a retrospective Italian cohort

2021

Abstract Objective The objective of this study was to use real-world data to evaluate the effectiveness and safety of canakinumab in Italian patients with systemic JIA (sJIA). Methods A retrospective multicentre study of children with sJIA was performed. Clinical features, laboratory parameters and adverse events were collected at baseline, and 6 and 12 months after starting canakinumab. The primary outcome measure of effectiveness was clinically inactive disease (CID) off glucocorticoids (GCs) treatment at 6 months. Results A total of 80 children from 15 Italian centres were analysed. Of the 12 patients who started canakinumab in CID while receiving anakinra, all maintained CID. Of the 68 …

medicine.medical_specialtyMultivariate analysissystemic juvenile idiopathic arthritisArthritisJuvenileAntibodies Monoclonal HumanizedcanakinumabAntibodiessystemic juvenile idiopathic arthritis.Settore MED/38 - Pediatria Generale E SpecialisticaRheumatologyInternal medicineMonoclonalmedicinecanakinumab; clinically inactive disease; systemic juvenile idiopathic arthritis; Antibodies Monoclonal Humanized; Child; Glucocorticoids; Humans; Retrospective Studies; Arthritis Juvenile; Macrophage Activation SyndromeHumanscanakinumab clinically inactive disease systemic juvenile idiopathic arthritis Antibodies Monoclonal Humanized Child Glucocorticoids Humans Retrospective Studies Arthritis Juvenile Macrophage Activation SyndromePharmacology (medical)clinical inactive disease.Adverse effectChildHumanizedGlucocorticoidsRetrospective StudiesUnivariate analysisAnakinrabusiness.industryclinically inactive diseaseArthritisMacrophage Activation Syndromemedicine.diseaseArthritis JuvenileCanakinumabMacrophage activation syndromeCohortSystemic juvenile idiopathic arthritibusinessmedicine.drug
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The neuro-B cell link of peptidergic innervation in the Bursa Fabricii

1991

The Bursa Fabricii, restricted to birds, specifically provides the microenvironment for B-cell maturation. The presence of nerve fibers containing immunopotent neuropeptides in immune organs opens interesting perspectives on the understanding of neuroimmune communication. As an organ for the development of only B-lymphocytes is not known in mammals, the contribution of a peptidergic innervation to the microenvironment of B-cells is not known. Therefore, we studied the peptidergic innervation of the Bursa Fabricii as an organ of B-cell maturation. Four different neuropeptides were found in nerve fibers of the Bursa Fabricii: tachykinins (TK), vasoactive intestinal peptide (VIP), galanin (GAL…

medicine.medical_specialtyNeuroimmunomodulationCalcitonin Gene-Related PeptideImmunologyVasoactive intestinal peptideNeuropeptideGalaninNerve fiberBiologyCalcitonin gene-related peptideMonocytesImmunoenzyme TechniquesBehavioral NeuroscienceBursa of FabriciusNerve FibersTachykininsInternal medicinemedicineAnimalsGalaninMedullaB cellB-LymphocytesEndocrine and Autonomic SystemsMacrophagesNeuropeptidesCell DifferentiationCell biologyEndocrinologymedicine.anatomical_structurePeripheral nervous systemPeptidesChickensVasoactive Intestinal PeptideBrain, Behavior, and Immunity
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Fresh cell therapy followed by fatal coma

1986

A 60-year-old woman received a 3-day course of nine injections of “fresh” cells from fetal lamb ovary, placenta, brain (hypothalamus) and liver. There were no immediate complications, but a few days later she developed headache, fever and hemiparesis. She subsequently fell into a coma and died 3 weeks after her fresh cell therapy and 2 weeks after the onset of her clinical symptoms. Autopsy revealed perivenous leucoencephalopathy with a probably steroid-treatment-induced paucity of perivascular inflammation. Fresh cell therapy, clinical symptomatology and morphological findings suggest, though do not prove, that this patient's monophasic and probably immune-mediated disease is a rare and fa…

medicine.medical_specialtyNeurologyCell- and Tissue-Based TherapyImmunoglobulinsOvaryAutopsyImmunoenzyme TechniquesCell therapyPonsPlacentamedicineHumansComaMyelin SheathCerebral CortexComabusiness.industryMacrophagesEncephalomyelitis Acute DisseminatedMiddle AgedSurgeryMicroscopy Electronmedicine.anatomical_structureHemiparesisNeurologyBlood-Brain BarrierAnesthesiaFemaleNeurology (clinical)medicine.symptomComplicationbusinessJournal of Neurology
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Toll-like receptor 5 in obesity: The role of gut microbiota and adipose tissue inflammation

2015

Objective This study aimed at establishing bacterial flagellin-recognizing toll-like receptor 5 (TLR5) as a novel link between gut microbiota composition, adipose tissue inflammation, and obesity. Methods An adipose tissue microarray database was used to compare women having the highest (n = 4, H-TLR) and lowest (n = 4, L-TLR) expression levels of TLR5-signaling pathway genes. Gut microbiota composition was profiled using flow cytometry and FISH. Standard laboratory techniques were used to determine anthropometric and clinical variables. In vivo results were verified using cultured human adipocytes. Results The H-TLR group had higher flagellated Clostridium cluster XIV abundance and Firmicu…

medicine.medical_specialtyNutrition and DieteticsbiologyAdiponectinEndocrinology Diabetes and MetabolismAdipose tissue macrophagesLeptinMedicine (miscellaneous)Adipose tissueInflammationGut florabiology.organism_classification3. Good healthInsulin receptorEndocrinologyEndocrinologyTLR5Internal medicinemedicinebiology.proteinmedicine.symptomObesity
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Bisphosphonates and atherosclerosis: why?

2005

The increasing knowledge on bone calcification processes has revealed some similarities with vascular tissue, where calcifications of arteries and cardiac valves contribute to several cardiovascular problems, such as heart failure, systolic hypertension, and myocardial and peripheral ischemic disease. Bisphosphonates have been used extensively for over two decades for the treatment of diseases associated with excessive bone resorption, i.e., osteoporosis, osteolytic bone metastasis, hypercalcemia and Paget’s disease, by blocking osteoclastic function. Etidronate, pamidronate and clodronate has been shown to inhibit the development of experimental atherosclerosis, and proposed mechanisms fo…

medicine.medical_specialtyOsteoporosisMevalonic AcidApoptosisDisease030204 cardiovascular system & hematologyBone resorption03 medical and health sciences0302 clinical medicineRheumatologyCalcinosisInternal medicinemedicineHumansBone ResorptionFoam cell030203 arthritis & rheumatologyDiphosphonatesMolecular Structurebusiness.industryMacrophagesBone metastasisCalcinosismedicine.diseaseAtherosclerosisArterial calcificationEndocrinologyCholesterolHeart failureCancer researchbusinessLupus
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Interrelation of peptidergic innervation with mast cells and ED1-positive cells in rat thymus

1991

The peptidergic innervation of rat thymus has been investigated by immunohistochemical methods, focusing on the spatial interrelationship of peptidergic nerve fibers with mast cells and macrophages in the rat. An antiserum directed against the protein gene product 9.5 (PGP 9.5) regarded as a pan-neuronal marker revealed a rich innervation, especially in the subcapsular cortex, in interlobular septa, and of the vasculature in the cortex and the corticomedullary boundary. A minor proportion of PGP 9.5-immunoreactive (ir) fibers supplied the thymic parenchyma. The main component of peptidergic innervation consisted of fibers costaining for tachykinins (TKs) and calcitonin gene-related peptide …

medicine.medical_specialtyPathologyNeuroimmunomodulationImmunologyVasoactive intestinal peptideConnective tissueNerve fiberThymus GlandBiologyCalcitonin gene-related peptideBehavioral NeuroscienceCatecholaminesNerve FibersInternal medicineCortex (anatomy)medicineAnimalsMast CellsGalaninEndocrine and Autonomic SystemsMacrophagesNeuropeptidesAntibodies MonoclonalCell DifferentiationRats Inbred StrainsMast cellRatsReceptors NeurotransmitterEndocrinologymedicine.anatomical_structurePeripheral nervous systemUbiquitin ThiolesteraseBiomarkersBrain, Behavior, and Immunity
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