Search results for "messenger"

showing 10 items of 1493 documents

Specific and global regulation of mRNA stability during osmotic stress in Saccharomyces cerevisiae.

2009

Hyperosmotic stress yields reprogramming of gene expression in Saccharomyces cerevisiae cells. Most of this response is orchestrated by Hog1, a stress-activated, mitogen-activated protein kinase (MAPK) homologous to human p38. We investigated, on a genomic scale, the contribution of changes in transcription rates and mRNA stabilities to the modulation of mRNA amounts during the response to osmotic stress in wild-type and hog1 mutant cells. Mild osmotic shock induces a broad mRNA destabilization; however, osmo-mRNAs are up-regulated by increasing both transcription rates and mRNA half-lives. In contrast, mild or severe osmotic stress in hog1 mutants, or severe osmotic stress in wild-type cel…

BioquímicaMessenger RNASaccharomyces cerevisiae ProteinsTranscription GeneticOsmotic shockMRNA destabilizationRNA Stabilityp38 mitogen-activated protein kinasesSaccharomyces cerevisiaeMRNA stabilizationSaccharomyces cerevisiaeBiologybiology.organism_classificationMolecular biologyArticleGenètica molecularCell biologyOsmotic PressureGene Expression Regulation FungalGene expressionOsmotic pressureRNA MessengerMitogen-Activated Protein KinasesMolecular Biology
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Structure-activity relationship of staurosporine analogs in regulating expression of endothelial nitric-oxide synthase gene.

2000

In human umbilical vein endothelial cells and in human umbilical vein endothelial cell-derived EA.hy 926 cells, staurosporine (Stsp) and its glycosidic indolocarbazole analogs 7-hydroxystaurosporine (UCN-01) and 4'-N-benzoyl staurosporine (CGP 41251) enhanced nitric-oxide synthase (NOS) III mRNA expression (analyzed by RNase protection assay), protein expression (determined by Western blot), and activity [measured by rat fetal lung fibroblast (RFL-6) reporter cell assay] in a concentration- and time-dependent manner. In contrast, the bisindolylmaleimide analogs GF 109203X, Ro 31-8220 and Go 6983 had no effect on NOS III expression, and Go 6976, a methyl- and cyanoalkyl-substituted nonglycos…

BisindolylmaleimideNitric Oxide Synthase Type IIIBiologyEndothelial NOSNitric OxideGene Expression Regulation Enzymologicchemistry.chemical_compoundStructure-Activity RelationshipAlkaloidsmedicineCyclic GMP-Dependent Protein KinasesStaurosporineAnimalsHumansDrug InteractionsRNA MessengerEnzyme InhibitorsProtein kinase APromoter Regions GeneticProtein Kinase InhibitorsProtein kinase CCells CulturedProtein Kinase CPharmacologyKinaseTumor Necrosis Factor-alphaNitric Oxide Synthase Type IIIProtein-Tyrosine KinasesStaurosporineMolecular biologyCyclic AMP-Dependent Protein KinasesRatschemistryMolecular MedicineEndothelium VascularNitric Oxide SynthaseTyrosine kinaseProtein Kinasesmedicine.drugMolecular pharmacology
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Expression of homeobox-containing genes in the sea urchin (Parancentrotus lividus) embryo

1994

Two homeobox-containing genes that belong to different homeodomain classes have been isolated from a sea urchin geonomic library. One, PlHbox11, is the sea urchin homologue of the human and mouse Hox B3 gene, the other, PlHbox12, shows about 55% identity with paired class genes. Expression profile analysis of the two sea urchin Hbox genes suggests that they play different roles during embryogenesis. In fact, PlHbox11 transcripts are rare and are detected only in the pluteus larva and in the Aristotle's lantern and intestine of the adult. The PlHbox12 gene is, on the contrary, transiently expressed in the very early embryo already at the four cell stage; it accumulates at the 64 cell stage a…

Blastomeresanimal structuresMolecular Sequence DataSettore BIO/11 - Biologia MolecolarePlant ScienceBiologyMicebiology.animalGeneticsAnimalsHumansAmino Acid SequenceRNA MessengerCloning MolecularHox geneGeneSea urchinRegulation of gene expressionSequence Homology Amino AcidEmbryogenesisGenes HomeoboxGene Expression Regulation DevelopmentalEmbryocell specificationGeneral MedicineBlastomereSequence Analysis DNAMolecular biologyhomeodomainInsect ScienceSea Urchinsembryonic structuresHomeoboxAnimal Science and Zoologyembryogenesispaired
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Type 1 diabetic mellitus patients with increased atherosclerosis risk display decreased CDKN2A/2B/2BAS gene expression in leukocytes

2019

Background Type 1 diabetes mellitus (T1DM) patients display increased risk of cardiovascular disease (CVD) and are characterized by a diminished regulatory T (Treg) cell content or function. Previous studies have shown an association between decreased CDKN2A/2B/2BAS gene expression and enhanced CVD. In the present study the potential relationship between CDKN2A/2B/2BAS gene expression, immune cell dysfunction and increased cardiovascular risk in T1DM patients was explored. Methods A cross-sectional study was performed in 90 subjects divided into controls and T1DM patients. Circulating leukocyte subpopulations analysis by flow cytometry, expression studies on peripheral blood mononuclear cel…

Blood Glucose0301 basic medicineendocrine system diseasesCellular differentiationlcsh:Medicine0302 clinical medicineRisk FactorsRAR-related orphan receptor gammaimmune system diseasesLeukocytesIL-2 receptorDiabetisFOXP3Cell DifferentiationGeneral MedicineType 1 diabetes030220 oncology & carcinogenesisCytokinesRNA Long Noncodingmedicine.symptomAdultmedicine.medical_specialtyCD14T cellsInflammationPeripheral blood mononuclear cellGeneral Biochemistry Genetics and Molecular Biology03 medical and health sciencesInternal medicinemedicineHumansRNA MessengerCyclin-Dependent Kinase Inhibitor p16Cyclin-Dependent Kinase Inhibitor p15Glycated HemoglobinInflammationType 1 diabetesbusiness.industryResearchlcsh:RAtherosclerosismedicine.diseaseCardiovascular riskDiabetes Mellitus Type 1030104 developmental biologyEndocrinologyGene Expression RegulationCase-Control StudiesbusinessJournal of Translational Medicine
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Chronic exposure of 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) induces an obesogenic effect in C57BL/6J mice fed a high fat diet

2017

IF 3.582; International audience; Contaminant involvement in the pathophysiology of obesity is widely recognized. It has been shown that low dose and chronic exposure to endocrine disruptor compounds (EDCs) potentiated diet- induced obesity. High and acute exposure to 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD), a persistent organic pollutant (POP) and an EDC with anti-estrogenic property, causes wasting syndrome . However at lower doses, the TCDD metabolic effects remain poorly understood. We investigated the obesogenic effect during chronic exposure of TCDD at 1μg/kg body weight (bw)/week in adult C57BL/6J mice fed with a high fat diet (HFD) and exposed from 10 to 42 weeks old to TCDD or e…

Blood GlucoseLeptinMale0301 basic medicineTCDDPolychlorinated DibenzodioxinsTime FactorsAdipose tissue010501 environmental sciencesToxicology01 natural sciencesBasic Helix-Loop-Helix Transcription FactorsInsulinAdiposity2. Zero hunger[ SDV.MHEP.EM ] Life Sciences [q-bio]/Human health and pathology/Endocrinology and metabolism[SDV.MHEP.EM]Life Sciences [q-bio]/Human health and pathology/Endocrinology and metabolism3. Good healthLiverEndocrine disruptorReceptors AndrogenCytokinesEnvironmental PollutantsFemaleInflammation Mediatorsmedicine.symptomStearoyl-CoA Desaturasemedicine.medical_specialtyLipolysisInflammationchronic exposureIntra-Abdominal FatDiet High-FatRisk Assessment03 medical and health sciencesSex FactorsobesogenInternal medicinemedicineAnimalsEndocrine systemObesityRNA MessengerWasting SyndromeTriglycerides0105 earth and related environmental sciencesbusiness.industrymedicine.diseaseObesityMice Inbred C57BL030104 developmental biologyEndocrinologyReceptors Aryl HydrocarbonInsulin ResistancebusinessBiomarkersObesogenDrug metabolism
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ERK1 and ERK2 activation modulates diet-induced obesity in mice

2017

IF 3.112; International audience; Obesity is a worldwide problem, and dietary lipids play an important role in its pathogenesis. Recently, Erk1 knock-out (ERK1(-/-)) mice have been shown to exhibit low preference for dietary fatty acids. Hence, we maintained Erk1(-/-) mice on a high-fat diet (HFD) to assess the implication of this mitogen-activated protein (MAP) kinase in obesity. The Erk1(-/-) mice, fed the HFD, were more obese than wild-type (WT) animals, fed the same diet. Erk1(-/-) obese mice gained more fat and liver mass than WT obese animals. No difference was observed in daily food and energy intake in HFD-fed both group of animals. However, feed efficiency was higher in Erk1(-/-) t…

Blood GlucoseMale0301 basic medicinemedicine.medical_treatmentMice ObeseBiochemistryMicechemistry.chemical_compoundPhosphorylationBeta oxidationCells CulturedMice KnockoutMitogen-Activated Protein Kinase 1Mitogen-Activated Protein Kinase 3Reverse Transcriptase Polymerase Chain ReactionGeneral MedicineLipidsFatty acid synthaseLiverLipogenesisHomeostatic model assessmentmedicine.medical_specialtyBlotting WesternBiologyDiet High-FatReal-Time Polymerase Chain Reaction03 medical and health sciencesInsulin resistanceInternal medicinemedicineAnimals[SDV.BBM]Life Sciences [q-bio]/Biochemistry Molecular BiologyRNA MessengerObesity[ SDV.BBM ] Life Sciences [q-bio]/Biochemistry Molecular BiologyInflammationTriglycerideLipogenesisInsulinBody WeightLipid Metabolismmedicine.diseaseObesityMice Inbred C57BL030104 developmental biologyEndocrinologychemistrybiology.proteinMAP kinaseInsulin ResistanceBiochimie
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Evidence for a dysfunction and disease-promoting role of the circadian clock in the diabetic retina.

2021

Diabetic retinopathy is a major complication of chronic hyperglycemia and a leading cause of blindness in developed countries. In the present study the interaction between diabetes and retinal clocks was investigated in mice. It was seen that in the db/db mouse - a widely used animal model of diabetic retinopathy - clock function and circadian regulation of gene expression was disturbed in the retina. Remarkably, elimination of clock function by Bmal1-deficiency mitigates the progression of pathophysiology of the diabetic retina. Thus high-fat diet was seen to induce histopathology and molecular markers associated with diabetic retinopathy in wild type but not in Bmal1-deficient mice. The d…

Blood GlucoseMalemedicine.medical_specialtyCircadian clockCLOCK ProteinsDiseaseChronobiology DisordersPathogenesisCellular and Molecular Neurosciencechemistry.chemical_compoundMiceDiabetes mellitusInternal medicineCircadian ClocksRNA Ribosomal 18SMedicineAnimalsRNA MessengerRetinaDiabetic Retinopathybusiness.industryGlyceraldehyde-3-Phosphate DehydrogenasesRetinalDiabetic retinopathymedicine.diseaseSensory SystemsPathophysiologyCircadian RhythmMice Inbred C57BLOphthalmologyDisease Models Animalmedicine.anatomical_structureEndocrinologychemistryMicroscopy FluorescenceFemalebusinessExperimental eye research
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Fructose-enriched diet modifies antioxidant status and lipid metabolism in spontaneously hypertensive rats

2005

Abstract Objective High-fructose consumption in industrial countries has been shown to induce metabolic abnormalities or syndrome X. Changes in antioxidant defense are unknown in hypertension associated with metabolic disorders induced by high-fructose feeding. Methods Twenty spontaneously hypertensive rats were assigned to one of two groups; one received a fructose-enriched diet (60% fructose) and the other a starch diet. After a 13-wk diet period, total antioxidant status was assessed in the blood and liver by monitoring the rate of free radical-induced red blood cell hemolysis. Antioxidants (enzymes and vitamins) were determined in blood and liver. Gene expression of antioxidant enzymes …

Blood GlucoseMalemedicine.medical_specialtyErythrocytesAntioxidantEndocrinology Diabetes and Metabolismmedicine.medical_treatmentalpha-TocopherolGene ExpressionAscorbic AcidFructoseThiobarbituric Acid Reactive SubstancesAntioxidantsLipid peroxidationSuperoxide dismutasechemistry.chemical_compoundRats Inbred SHRInternal medicinemedicineAnimalsInsulinRNA MessengerVitamin Achemistry.chemical_classificationGlutathione PeroxidaseNutrition and DieteticsbiologySuperoxide DismutaseGlutathione peroxidaseStarchFructoseLipid MetabolismAscorbic acidDietRatsRed blood cellEndocrinologymedicine.anatomical_structureLiverchemistryHypertensionbiology.proteinLipid PeroxidationPeroxidaseNutrition
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Effects of acute exercise, exercise training, and diabetes on the expression of lymphangiogenic growth factors and lymphatic vessels in skeletal musc…

2007

Blood and lymphatic vessels together form the circulatory system, allowing the passage of fluids and molecules within the body. Recently we showed that lymphatic capillaries are also found in the capillary bed of skeletal muscle. Exercise is known to induce angiogenesis in skeletal muscle, but it is not known whether exercise has effects on lymphangiogenesis or lymphangiogenic growth factors. We studied lymphatic vessel density and expression of the main lymphangiogenic growth factors VEGF-C and VEGF-D and their receptor VEGFR-3 in response to acute running exercise and endurance exercise training in the skeletal muscle of healthy and diabetic mice. VEGF-C mRNA expression increased after t…

Blood GlucoseMalemedicine.medical_specialtyTime FactorsPhysiologyPhysical ExertionVascular Endothelial Growth Factor CVascular Endothelial Growth Factor DPhysical exercisePolymerase Chain ReactionDiabetes Mellitus ExperimentalMiceEndurance trainingPhysiology (medical)Internal medicinemedicineLymphatic vesselAnimalsRNA MessengerLymphangiogenesisMuscle SkeletalLymphatic Vesselsbusiness.industryBody WeightSkeletal muscleVascular Endothelial Growth Factor Receptor-3ImmunohistochemistryLymphangiogenesisLymphatic systemEndocrinologymedicine.anatomical_structureVascular endothelial growth factor CGene Expression RegulationCirculatory systemCardiology and Cardiovascular MedicinebusinessAmerican journal of physiology. Heart and circulatory physiology
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Eicosapentaenoic acid modulates fatty acid metabolism and inflammation in Psammomys obesus.

2015

Abstract The desert gerbil, Psammomys obesus, is a unique polygenic animal model of metabolic syndrome (insulin resistance, obesity and type 2 diabetes), and these pathological conditions resemble to those in human beings. In this study, the animals were fed ad libitum either a natural diet (ND) which contained desertic halophile plants or a standard laboratory diet (STD) or a diet which contained eicosapentaenoic acid (EPA), hence, termed as EPA diet (EPAD). In EPAD, 50% of total lipid content was replaced by EPA oil. By employing real-time PCR, we assessed liver expression of key genes involved in fatty acid metabolism such as PPAR-α, SREBP-1c, LXR-α and CHREBP. We also studied the expres…

Blood GlucoseMalemedicine.medical_specialtymedicine.medical_treatmentInterleukin-1betaAdipose tissueGene ExpressionBiologyBiochemistrychemistry.chemical_compoundInsulin resistanceInternal medicinemedicineAnimalsInsulinPPAR alphaRNA MessengerTriglyceridesLiver X Receptorschemistry.chemical_classificationFatty acid metabolismReverse Transcriptase Polymerase Chain ReactionTumor Necrosis Factor-alphaInsulinBody WeightFatty AcidsGeneral Medicinebiology.organism_classificationmedicine.diseaseLipid MetabolismOrphan Nuclear ReceptorsEicosapentaenoic acidEndocrinologychemistryAdipose TissueEicosapentaenoic AcidLiverlipids (amino acids peptides and proteins)PsammomysMetabolic syndromeInflammation MediatorsGerbillinaeSterol Regulatory Element Binding Protein 1Polyunsaturated fatty acidBiochimie
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