Search results for "mito"

showing 10 items of 2513 documents

Mitochondrial complex I impairment in leukocytes from polycystic ovary syndrome patients with insulin resistance.

2009

Insulin resistance is a feature of polycystic ovary syndrome (PCOS) and is related to mitochondrial function.Our objective was to assess mitochondrial function by evaluating mitochondrial oxygen (O(2)) consumption, reactive oxygen species (ROS) production, levels of glutathione (GSH), the oxidized glutathione/GSH ratio, TNFalpha levels, and membrane potential. Additionally, we have evaluated mitochondrial complex I as a target of the oxidative stress responsible for PCOS in polymorphonuclear cells.This was a prospective controlled study conducted in an academic medical center.The study population consisted of 20 lean reproductive-age women with PCOS and 20 body composition-matched controls.…

medicine.medical_specialtyendocrine system diseasesEndocrinology Diabetes and Metabolismmedicine.medical_treatmentClinical BiochemistryContext (language use)BiologyMitochondrionmedicine.disease_causeBiochemistrychemistry.chemical_compoundEndocrinologyInsulin resistanceOxygen ConsumptionInternal medicinemedicineLeukocytesHumanschemistry.chemical_classificationMembrane Potential MitochondrialReactive oxygen speciesElectron Transport Complex ITumor Necrosis Factor-alphaInsulinBiochemistry (medical)nutritional and metabolic diseasesGlutathionemedicine.diseasePolycystic ovaryGlutathionefemale genital diseases and pregnancy complicationsEndocrinologychemistryFemaleInsulin ResistanceReactive Oxygen SpeciesOxidative stressPolycystic Ovary Syndrome
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Endothelial Bmx tyrosine kinase activity is essential for myocardial hypertrophy and remodeling

2015

Cardiac hypertrophy accompanies many forms of heart disease, including ischemic disease, hypertension, heart failure, and valvular disease, and it is a strong predictor of increased cardiovascular morbidity and mortality. Deletion of bone marrow kinase in chromosome X (Bmx), an arterial nonreceptor tyrosine kinase, has been shown to inhibit cardiac hypertrophy in mice. This finding raised the possibility of therapeutic use of Bmx tyrosine kinase inhibitors, which we have addressed here by analyzing cardiac hypertrophy in gene-targeted mice deficient in Bmx tyrosine kinase activity. We found that angiotensin II (Ang II)-induced cardiac hypertrophy is significantly reduced in mice deficient i…

medicine.medical_specialtyendotheliumEndotheliumAngiogenesiscardiomyocyteCardiomegalyheartmTORC1030204 cardiovascular system & hematologyMitochondria Heart03 medical and health sciencesMice0302 clinical medicineInternal medicinemedicineAnimalsMyocytes Cardiac030304 developmental biologyMice Knockout0303 health sciencesMultidisciplinaryKinasebusiness.industryta1184Angiotensin IIBiological SciencesProtein-Tyrosine KinasesAngiotensin IImedicine.anatomical_structureEndocrinologyEtkcardiovascular systemCancer researchPhosphorylationCytokinesEndothelium VascularSignal transductionInflammation MediatorssignalingbusinessTyrosine kinaseSignal Transduction
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Mitomycin C bei oberflächlichen Hornhautablationen mit dem Excimer-Laser: Eigene Erfahrungen und Literaturübersicht

2005

Haze formation with loss of corneal transparency and surface irregularities and myopic regression are the major complications after corneal refractive surface surgery. The use of mitomycin C (MMC) with its antibiotic and antineoplastic properties is intended to inhibit wound healing mechanisms leading to subepithelial fibrosis. We report the use of MMC to achieve visual rehabilitation in the re-treatment of 3 eyes of 2 patients following refractive corneal surgery. According to the literature, the local use of MMC 0.02 % for 2 minutes is safe and enables one to treat and prevent stromal haze and myopic regression and allows a reduction of the postoperative topical pharmacotherapy. Results a…

medicine.medical_specialtygenetic structuresbusiness.industryMitomycin CVisual rehabilitationCorneal opacityeye diseasesCorneal transparencySurgeryOphthalmologyPharmacotherapyCorneal surgerySubepithelial fibrosisMedicinesense organsMajor complicationbusinessKlinische Monatsblätter für Augenheilkunde
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A Look into Liver Mitochondrial Dysfunction as a Hallmark in Progression of Brain Energy Crisis and Development of Neurologic Symptoms in Hepatic Enc…

2020

Background: The relationship between liver disease and neuropathology in hepatic encephalopathy is well known, but the genesis of encephalopathy in liver failure is yet to be elucidated. Conceptually, the main cause of hepatic encephalopathy is the accumulation of brain ammonia due to impaired liver detoxification function or occurrence of portosystemic shunt. Yet, as well as taking up toxic ammonia, the liver also produces vital metabolites that ensure normal cerebral function. Given this, for insight into how perturbations in the metabolic capacity of the liver may be related to brain pathology, it is crucial to understand the extent of ammonia-related changes in the hepatic metabolism th…

medicine.medical_specialtyhyperammonemiaEncephalopathylcsh:MedicineMitochondrionliverArticle03 medical and health sciencesLiver disease0302 clinical medicineInternal medicinebrain energy crisisKetogenesisMedicineHepatic encephalopathy030304 developmental biology0303 health sciencesbusiness.industrylcsh:RHyperammonemiaGeneral Medicinemedicine.diseaseketogenesismitochondriaEndocrinologygluconeogenesisKetone bodies030211 gastroenterology & hepatologyPortosystemic shuntbusinessJournal of clinical medicine
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Modulation of IL-2, IFN-γ, TNF-α and IL-4 production in mice of different ages by thymopentin

1992

The effect of an immunomodulator drug thymopentin (TP5) on the production of various cytokines (IFN-gamma, IL-2, IL-4, TNF-alpha) in mice of different ages has been studied. TP5 enhanced IL-2, TNF-alpha and IFN-gamma production but reduced the IL-4 secretion by splenocytes from aged mice (greater than 120 week old) in vitro. However, it had no effect on the IL-2, IFN-gamma, TNF-alpha or IL-4 production by splenocytes from young and adult mice. TP5 injected subcutaneously was able to induce high levels of IL-2 production by splenocytes from all groups of mice. The TP5 effect on TNF-alpha and IFN-gamma was similar, even though it was significant only in old mice. Furthermore, TP5 was able to …

medicine.medical_specialtymedicine.medical_treatmentImmunologyMitogen stimulationInterferon-gammaMiceT-Lymphocyte SubsetsInternal medicinemedicineSplenocyteAnimalsThymopentinSecretionLymphocytesInterleukin 4PharmacologyMice Inbred BALB CTumor Necrosis Factor-alphabusiness.industryAge FactorsIn vitroCytokineEndocrinologyInterleukin-2FemaleInterleukin-4Thymopentinbusinessmedicine.drugInternational Journal of Immunopharmacology
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Mitochondrial complex I impairment in leukocytes from type 2 diabetic patients.

2011

Diabetes is associated with oxidative stress. This study evaluated the rates of oxidative stress and mitochondrial impairment in type 2 diabetes patients. The study population consisted of 182 diabetic patients and 50 body-composition- and age-matched controls. We assessed anthropometric and metabolic parameters and mitochondrial function by evaluating mitochondrial oxygen (O2) consumption, reactive oxygen species (ROS) production, glutathione (GSH) levels, GSH/GSSG ratio, mitochondrial membrane potential, and mitochondrial complex I activity in polymorphonuclear cells from diabetes type 2 patients. We found an increase in waist circumference and augmented serum levels of triglycerides, pro…

medicine.medical_specialtymedicine.medical_treatmentType 2 diabetesMitochondrionBiologymedicine.disease_causeBiochemistryArticlechemistry.chemical_compoundInsulin resistancePhysiology (medical)Internal medicineDiabetes mellitusRotenonemedicineLeukocytesHumanschemistry.chemical_classificationReactive oxygen speciesElectron Transport Complex IInsulinMiddle Agedmedicine.diseaseMitochondriaOxygenOxidative StressEndocrinologychemistryDiabetes Mellitus Type 2Glycated hemoglobinReactive Oxygen SpeciesOxidative stress
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Gene expression centroids that link with low intrinsic aerobic exercise capacity and complex disease risk

2010

A strong link exists between low aerobic exercise capacity and complex metabolic diseases. To probe this linkage, we utilized rat models of low and high intrinsic aerobic endurance running capacity that differ also in the risk for metabolic syndrome. We investigated in skeletal muscle gene-phenotype relationships that connect aerobic endurance capacity with metabolic disease risk factors. The study compared 12 high capacity runners (HCRs) and 12 low capacity runners (LCRs) from generation 18 of selection that differed by 615% for maximal treadmill endurance running capacity. On average, LCRs were heavier and had increased blood glucose, insulin, and triglycerides compared with HCRs. HCRs we…

medicine.medical_treatmentBiochemistryResearch Communicationschemistry.chemical_compound0302 clinical medicineRisk Factorslipid metabolismOligonucleotide Array Sequence Analysis0303 health sciencesExercise ToleranceImmunohistochemistryMitochondriamedicine.anatomical_structureFemaleBiotechnologymedicine.medical_specialtyOxidative phosphorylationBiology03 medical and health sciencesOxygen ConsumptionMetabolic DiseasesPhysical Conditioning AnimalInternal medicineGeneticsmedicineAnimalsoxygen metabolismAerobic exerciseGenetic Predisposition to Diseaseskeletal muscleMuscle SkeletalMolecular BiologyAerobic capacity030304 developmental biologyMyosin Heavy Chainscomplex metabolic diseaseFatty acid metabolismGene Expression ProfilingInsulinSkeletal musclemedicine.diseaseRatsDisease Models AnimalEndocrinologyGene Expression RegulationchemistryBasal metabolic rateMetabolic syndromeEnergy Metabolism030217 neurology & neurosurgeryThe FASEB Journal
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Interleukin 12 enhances deficient HCV-antigen-induced Th1-type immune response of peripheral blood mononuclear cells

1998

The aim of this study was to examine the possible immunomodulating effects of rhIL-12 on the immune response induced by different hepatitis C virus (HCV) antigens. Freshly isolated peripheral blood mononuclear cells (PBMC) of 33 patients with chronic HCV infection were stimulated with optimal concentrations of antigens from the NS3, NS4, NS5, and core region of HCV in the absence or presence of interleukin12 (IL-12). Stimulation by α-CD3 + α-CD28, lipopolysaccharide (LPS), and pokeweed mitogen (PWM) were used as controls. Proliferation and cytokine production were determined by 3H-thymidine uptake and enzyme-linked immunosorbent assay (ELISA) after 72 hr. After stimulation with antigen or a…

medicine.medical_treatmentHepatitis C virusPokeweed mitogenBiologymedicine.disease_causeVirologyPeripheral blood mononuclear cellInfectious DiseasesImmune systemCytokineAntigenVirologyImmunologyInterleukin 12medicineTumor necrosis factor alphaJournal of Medical Virology
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POLYPHENOLS FROM RED WINE MODULATE IMMUNE RESPONSIVENESS: BIOLOGICAL AND CLINICAL SIGNIFICANCE.

2008

Many studies have been conducted on the effects of red wine polyphenols on certain diseases, primarily, coronary heart disease (CHD) and, in this respect, evidence has been demonstrated that intake of red wine is associated with a reduction of CHD symptomatology. In this framework, the purpose of this review is to illustrate the effects of polyphenols on immune cells from human healthy peripheral blood. Data will show that polyphenols are able to stimulate both innate and adaptive immune responses. In particular, the release of cytokines such as interleukin (IL)-12, interferon (IFN)-gamma, and IL-10 as well as immunoglobulins may be important for host protection in different immune related …

medicine.medical_treatmentImmunoglobulinsCoronary DiseaseWineImmunoglobulin ENitric OxidePeripheral blood mononuclear cellp38 Mitogen-Activated Protein KinasesNitric oxidePOLYPHENOLSIMMUNE SYSTEMCYTOKINESIMMUNOGLOBULINSNITRIC OXIDEATHEROSCLEROSISRED WINEchemistry.chemical_compoundImmune systemPhenolsInterferonDrug DiscoverymedicineAnimalsHumansPharmacologyFlavonoidsSettore MED/04 - Patologia Generalebiologybusiness.industryImmunityfood and beveragesInterleukinPolyphenolsCytokinechemistryImmunologyChronic Diseasebiology.proteinLeukocytes MononuclearCytokinesAntibodybusinessmedicine.drug
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Absence of the lactase-persistence-associated allele in early Neolithic Europeans.

2007

Lactase persistence (LP), the dominant Mendelian trait conferring the ability to digest the milk sugar lactose in adults, has risen to high frequency in central and northern Europeans in the last 20,000 years. This trait is likely to have conferred a selective advantage in individuals who consume appreciable amounts of unfermented milk. Some have argued for the “culture-historical hypothesis,” whereby LP alleles were rare until the advent of dairying early in the Neolithic but then rose rapidly in frequency under natural selection. Others favor the “reverse cause hypothesis,” whereby dairying was adopted in populations with preadaptive high LP allele frequencies. Analysis based on the cons…

medicine.medical_treatmentPopulationLactoseBiologyDNA MitochondrialPolymorphism Single NucleotideBone and BonesWhite PeopleNOLactose IntolerancemedicineHumansAlleleeducationSelectionAllele frequencyAllelesHistory AncientLactaseGeneticseducation.field_of_studyMultidisciplinaryNatural selectionAncient DNAHaplotypeLactaseEmigration and ImmigrationBiological SciencesAncient DNA Dairying SelectionEuropeDairyingLactase persistenceAncient DNAGenetics PopulationTandem Repeat SequencesToothProceedings of the National Academy of Sciences of the United States of America
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