Search results for "mito"

showing 10 items of 2513 documents

Mitochondrial disruption and apoptosis in lymphocytes of an HIV infected patient affected by lactic acidosis after treatment with highly active antir…

2003

Aims: Highly active antiretroviral therapy (HAART) can induce an increase in lactic acid concentrations that seems to be caused by mitochondrial dysfunction induced by the interaction of nucleoside reverse transcriptase inhibitors (NRTIs) with DNA polymerase γ in the mitochondria. Mitochondrial alterations have been described in liver and muscle cells of NRTI treated human immunodeficiency virus (HIV) infected patients. Because lymphocytes are the main target for HIV and because mitochondria are involved in apoptosis, we studied mitochondrial morphology and apoptosis in the lymphocytes of an HIV infected patient with severe lactic acidosis after treatment with stavudine, didanosine, and ind…

Adultmedicine.medical_specialtyAnti-HIV AgentsLymphocyteApoptosisHIV InfectionsCase ReportsMitochondrionBiologyPathology and Forensic Medicinechemistry.chemical_compoundimmune system diseasesIndinavirAntiretroviral Therapy Highly ActiveInternal medicinemedicineHumansLymphocytesDidanosineAcridine orangeStavudinevirus diseasesGeneral Medicinemedicine.diseaseMitochondriaEndocrinologymedicine.anatomical_structurechemistryApoptosisLactic acidosisImmunologyAcidosis LacticFemalemedicine.drugJournal of Clinical Pathology
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A Phase II Trial of Mitoxantrone plus Cyclophosphamide and 5-Fluorouracil in Modulation with Levo-Folinate for Advanced Breast Cancer Patients

1995

Advanced breast cancer remains a major clinical problem. Current chemotherapy regimens are able to induce a clinical response in many patients but do not appear to influence significantly patients' survival. The use of new drugs such as mitoxantrone with a predicted lower toxicity and biochemical modulation of 5-fluorouracil with levo-folinate are extensively studied research areas that could combine good therapeutic efficacy with the maintenance of an acceptable quality of life. 34 patients with advanced breast carcinoma were included in the study. Only 4 women had received prior chemotherapy for advanced disease. Treatment plan was: 5-fluorouracil 400 mg/m2 + l-leucovorin 100 mg/m2 days 1…

Adultmedicine.medical_specialtyCyclophosphamidemedicine.medical_treatmentLeucovorinBreast NeoplasmsGastroenterologyDrug Administration ScheduleInternal medicineAntineoplastic Combined Chemotherapy ProtocolsGranulocyte Colony-Stimulating FactormedicineHumansPharmacology (medical)Neoplasm MetastasisInfusions IntravenousCyclophosphamideAgedAntibacterial agentPharmacologyMitoxantroneCardiotoxicityChemotherapyLeukopeniaDose-Response Relationship Drugbusiness.industryMiddle AgedSurgeryInfectious DiseasesOncologyFluorouracilToxicityFemaleFluorouracilMitoxantronemedicine.symptombusinessmedicine.drugJournal of Chemotherapy
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Chromosomal aberrations in patients with primary biliary cirrhosis.

1990

Chromosomal aberrations in untreated lymphocyte cultures, bleomycin (BLM)-induced aberrations and sister chromatid exchanges (SCE) in the peripheral blood lymphocytes of 11 patients suffering from primary biliary cirrhosis (PBC) and 14 matched control individuals were analysed. The lymphocytes of the PBC patients had on average a lower mitotic index (2.3) compared with controls (3.5) in the untreated cultures. The mean baseline rate of aberrations of the cultured lymphocytes of the patients was 5.3 aberrations per 100 metaphases (%); this was significantly different (P = 0.0291) from that of the controls with a mean of 2.3%. In lymphocytes of the patients and controls, most of the aberratio…

Adultmedicine.medical_specialtyMitotic indexLymphocyteBiliary cirrhosisBiologyBleomycinGastroenterologychemistry.chemical_compoundBleomycinPrimary biliary cirrhosisInternal medicineGeneticsmedicineMitotic IndexSister chromatidsHumansLymphocytesGenetics (clinical)Cells CulturedAgedChromosome AberrationsLiver Cirrhosis BiliaryKaryotypeMiddle Agedmedicine.diseaseMolecular medicinemedicine.anatomical_structurechemistryKaryotypingImmunologyFemaleSister Chromatid ExchangeHuman genetics
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Immunoblotting as a confirmatory test for antimitochondrial antibodies in primary biliary cirrhosis.

1993

Primary biliary cirrhosis is characterised by the presence of antimitochondrial antibodies which are directed against components of mitochondrial dehydrogenase complexes. The specificity of antimitochondrial antibodies for primary biliary cirrhosis as detected by immunoblotting was investigated. Commercially available preparations of pyruvate and oxo-glutarate dehydrogenases and beef-heart mitochondria were used as source of antigens. Sera from 47 primary biliary cirrhosis patients (46 of whom were antimitochondrial antibody positive by immunofluorescence), 16 non-primary biliary cirrhosis patients (antimitochondrial antibody positive by immunofluorescence), 23 liver-kidney microsomal antib…

Adultmedicine.medical_specialtyPathologyBiliary cirrhosisImmunoblottingFluorescent Antibody TechniquePyruvate Dehydrogenase ComplexBiologyImmunofluorescenceGastroenterologyMitochondria HeartPrimary biliary cirrhosisAntigenAntibody SpecificityInternal medicinemedicineAnimalsHumansKetoglutarate Dehydrogenase ComplexAutoantibodiesHepatitismedicine.diagnostic_testLiver Cirrhosis BiliaryGastroenterologyAutoantibodyMiddle AgedPyruvate dehydrogenase complexmedicine.diseaseMitochondriabiology.proteinCattleFemaleAntibodyResearch ArticleGut
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Metformin modulates human leukocyte/endothelial cell interactions and proinflammatory cytokines in polycystic ovary syndrome patients

2015

Abstract Objective We aim to assess the effect of metformin treatment on metabolic parameters, endothelial function and inflammatory markers in polycystic ovary syndrome (PCOS) subjects. Methods The study population consisted of 40 reproductive-age women with PCOS, who underwent treatment with metformin during a 12-week period, and their corresponding matched controls (n = 44). We evaluated endocrinological parameters, adhesion molecules (vascular cell adhesion molecule 1 (VCAM-1), intercellular cell adhesion molecule 1 (ICAM-1) and E-selectin) and proinflammatory cytokines (interleukin 6 (IL-6) and tumor necrosis factor alpha (TNFα)) in serum. In addition, interactions between human umbili…

Adultmedicine.medical_specialtyTime FactorsAdolescentendocrine system diseasesEndotheliumType 2 diabetesProinflammatory cytokineYoung AdultInsulin resistanceInternal medicineCell AdhesionHuman Umbilical Vein Endothelial CellsLeukocytesmedicineHumansLeukocyte RollingEndothelium Leukocyte Metformin Mitochondria PCOS Type 2 diabetesCells CulturedCell adhesion moleculebusiness.industryEndothelial Cellsnutritional and metabolic diseasesmedicine.diseasePolycystic ovaryCoculture TechniquesMetforminMetforminEndothelial stem cellTreatment Outcomemedicine.anatomical_structureEndocrinologyDiabetes Mellitus Type 2SpainCytokinesFemaleInflammation MediatorsCardiology and Cardiovascular MedicinebusinessCell Adhesion MoleculesBiomarkersPolycystic Ovary Syndromemedicine.drugAtherosclerosis
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Induction of oxidative stress and human leukocyte/endothelial cell interactions in polycystic ovary syndrome patients with insulin resistance.

2011

Insulin resistance is a feature of polycystic ovary syndrome (PCOS) and is related to mitochondrial and endothelial function.We tested whether hyperandrogenic insulin-resistant women with PCOS, who have an increased risk of vascular disease, display impaired leukocyte-endothelium interactions, and mitochondrial dysfunction.This was a prospective controlled study conducted in an academic medical center.The study population consisted of 43 lean reproductive-age women with PCOS and 39 controls subjects.We evaluated anthropometric and metabolic parameters, adhesion molecules, and interactions between leukocytes and human umbilical vein endothelial cells. Mitochondrial function was studied by as…

Adultmedicine.medical_specialtyUmbilical VeinsAdolescentendocrine system diseasesNeutrophilsEndocrinology Diabetes and Metabolismmedicine.medical_treatmentClinical BiochemistryVascular Cell Adhesion Molecule-1Cell SeparationBiologymedicine.disease_causeBiochemistrychemistry.chemical_compoundYoung AdultEndocrinologyInsulin resistanceOxygen ConsumptionInternal medicineE-selectinmedicineLeukocytesHumanschemistry.chemical_classificationReactive oxygen speciesInterleukin-6Tumor Necrosis Factor-alphaInsulinBiochemistry (medical)Endothelial Cellsnutritional and metabolic diseasesGlutathionemedicine.diseasePolycystic ovaryGlutathionefemale genital diseases and pregnancy complicationsMitochondriaEndothelial stem cellOxidative StressEndocrinologychemistrybiology.proteinFemaleInsulin ResistanceE-SelectinReactive Oxygen SpeciesOxidative stressPolycystic Ovary Syndrome
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Effects of metformin on mitochondrial function of leukocytes from polycystic ovary syndrome patients with insulin resistance.

2015

ObjectiveOxidative stress and mitochondrial dysfunction are implicated in polycystic ovary syndrome (PCOS). The present study assesses the effect of metformin treatment on mitochondrial function in polymorphonuclear cells from PCOS subjects. Additionally, we evaluate endocrine parameters and levels of interleukin 6 (IL6) and tumour necrosis factor alpha (TNFα).Design and methodsOur study population was comprised of 35 women of reproductive age diagnosed with PCOS and treated with metformin for 12 weeks, and their corresponding controls (n=41), adjusted by age and BMI. We evaluated the alteration of endocrinological and anthropometrical parameters and androgen levels. Mitochondrial O2 consum…

Adultmedicine.medical_specialtyendocrine system diseasesNeutrophilsEndocrinology Diabetes and MetabolismMitochondrionBiologymedicine.disease_causechemistry.chemical_compoundYoung AdultEndocrinologyDehydroepiandrosterone sulfateInsulin resistanceInternal medicinemedicineHumansHypoglycemic Agentschemistry.chemical_classificationReactive oxygen speciesInterleukin-6Tumor Necrosis Factor-alphanutritional and metabolic diseasesGeneral MedicineGlutathionemedicine.diseasePolycystic ovaryMetforminMetforminMitochondriaEndocrinologyTreatment OutcomechemistryFemaleInsulin ResistanceOxidative stressmedicine.drugPolycystic Ovary Syndrome
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Mitomycin C from birth to adulthood.

2016

Mitomycin C (MMC) intravesical therapy for “superficial” papillary bladder tumors was firstly introduced in the early seventies with promising results. In the following years, several pharmacokinetic studies investigated its mechanism of action to optimize the intravesical administration. Numerous studies confirmed thereafter both the ablative and the prophylactic efficacy and the low toxicity of MMC when intravesically given. In 1984, a complete response rate of 42% in 60 patients not responsive to thiotepa was reported with intravesical MMC at the dose of 40 mg diluted in 40 ml for 8 weeks. In the following decades, many large randomized studies showed the benefit of intravesical prophyla…

Adultmedicine.medical_specialtymedicine.drug_classmedicine.medical_treatmentMitomycinAntibiotics030232 urology & nephrologyUrologyThioTEPASettore MED/24 - Urologia03 medical and health sciencesYoung Adult0302 clinical medicinePharmacokineticsMedicineHumansYoung adultChildComplete responseAntibiotics Antineoplasticbusiness.industryMedicine (all)digestive oral and skin physiologyMitomycin CGeneral MedicineRegimenUrinary Bladder Neoplasms030220 oncology & carcinogenesisUrinary Bladder NeoplasmbusinessAdjuvantHumanmedicine.drugUrologia
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Upgrading cytochrome P450 activity in HepG2 cells co-transfected with adenoviral vectors for drug hepatotoxicity assessment

2011

In a number of adverse drug reactions leading to hepatotoxicity, drug metabolism is thought to be involved by the generation of reactive metabolites from non-toxic drugs. The use of hepatoma cell lines, such as HepG2 cell line, for the evaluation of drug-induced hepatotoxicity is hampered by their low cytochrome P450 expression which makes impossible the study of the toxicity produced by bioactivable compounds. Genetically manipulated cells constitute promising tools for hepatotoxicity applications. HepG2 cells were simultaneously transfected with recombinant adenoviruses encoding CYP1A2, CYP2C9 and CYP3A4 to confer them drug-metabolic competence. Upgraded cells (Adv-HepG2) were highly able…

Aflatoxin B1Cell SurvivalGenetic VectorsPharmacologyTransfectionToxicologyModels BiologicalCitric AcidCalcium in biologyAdenoviridaeCytochrome P-450 CYP1A2RotenoneCytochrome P-450 CYP3AHumansViability assayCytochrome P-450 CYP2C9Membrane Potential MitochondrialCYP3A4biologyChemistryCYP1A2Cytochrome P450Hep G2 CellsGeneral MedicineTransfectionBiochemistryHigh-content screeningbiology.proteinCalciumAryl Hydrocarbon HydroxylasesChemical and Drug Induced Liver InjuryDrug metabolismToxicology in Vitro
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Mitochondrial transcriptional study of the effect of aflatoxins, enniatins and carotenoids in vitro in a blood brain barrier model

2020

C. maxima (var. Delica), a variety of pumpkin, is well known for its high concentration on carotenoids, possessing dietary benefits and antioxidant properties. Aflatoxins and enniatins are common mycotoxins present in food and feed with an extended toxicity profile in humans and animals. Both types of substances reach a wide range of tissues and organs and have the capability to penetrate the blood brain barrier. Since carotenoids and mycotoxins have been reported to modify diverse mitochondrial processes individually, transcriptional in vitro studies on human epithelial cells ECV 304 were conducted to analyze the relative expression of 13 mitochondria related genes. ECV 304 cells were diff…

AflatoxinMitochondrial DNAAntioxidantmedicine.medical_treatmentAlzheimer Antioxidants Mycotoxicity Neurodegenerative diseases Carotenoids qPCR ECV 304MitochondrionToxicologyBlood–brain barrierAntioxidantsCell LineNOchemistry.chemical_compoundAflatoxinsCucurbitaDepsipeptidesHuman Umbilical Vein Endothelial CellsmedicineHumansECV 304MycotoxinMycotoxicityCarotenoidchemistry.chemical_classificationLS9_6Neurodegenerative diseasesfood and beveragesGeneral MedicineCarotenoidsIn vitroMitochondriaqPCRmedicine.anatomical_structureElectron Transport Chain Complex ProteinschemistryBiochemistryBlood-Brain BarrierAlzheimerFood Science
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