Search results for "mitochondrial dna"

showing 10 items of 259 documents

Reactive oxygen species derived from the mitochondrial respiratory chain are not responsible for the basal levels of oxidative base modifications obs…

2004

The mitochondrial electron transport chain (ETC) is the most important source of reactive oxygen species (ROS) in mammalian cells. To assess its relevance to the endogenous generation of oxidative DNA damage in the nucleus, we have compared the background (steady-state) levels of oxidative DNA base modifications sensitive to the repair glycosylase Fpg (mostly 7,8-dihydro-8-oxoguanine) in wild-type HeLa cells and HeLa rho0 cells. The latter are depleted of mitochondrial DNA and therefore are unable to produce ROS in the ETC. Although the levels of ROS measured by flow cytometry and redox-sensitive probes in rho0 cells were only 10-15% those of wild-type cells, steady-state levels of oxidativ…

Mitochondrial ROSCarbonyl Cyanide m-Chlorophenyl HydrazoneMitochondrial DNADNA damageCells[SDV]Life Sciences [q-bio]Oxidative phosphorylationMitochondrionBiologyBiochemistryElectron Transport03 medical and health sciences0302 clinical medicinePhysiology (medical)AnimalsHumansComputingMilieux_MISCELLANEOUS030304 developmental biologyCell Nucleus0303 health sciencesGuanosineNucleotidesEscherichia coli ProteinsDNAFlow CytometryMitochondriaNuclear DNAMitochondrial respiratory chainDNA-Formamidopyrimidine GlycosylaseBiochemistryDNA glycosylaseMacrolidesReactive Oxygen SpeciesOxidation-Reduction030217 neurology & neurosurgeryDNA DamageHeLa Cells
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Mitochondrial Oxidative Stress, Mitochondrial DNA Damage and Their Role in Age-Related Vascular Dysfunction

2015

The prevalence of cardiovascular diseases is significantly increased in the older population. Risk factors and predictors of future cardiovascular events such as hypertension, atherosclerosis, or diabetes are observed with higher frequency in elderly individuals. A major determinant of vascular aging is endothelial dysfunction, characterized by impaired endothelium-dependent signaling processes. Increased production of reactive oxygen species (ROS) leads to oxidative stress, loss of nitric oxide (•NO) signaling, loss of endothelial barrier function and infiltration of leukocytes to the vascular wall, explaining the low-grade inflammation characteristic for the aged vasculature. We here disc…

Mitochondrial ROSmedicine.medical_specialtyMitochondrial DNADNA RepairInflammationReviewBiologyMitochondrionmedicine.disease_causeDNA MitochondrialCatalysisAntioxidantsNitric oxideInorganic Chemistrylcsh:Chemistrychemistry.chemical_compoundInternal medicinemedicineAnimalsHumansPhysical and Theoretical ChemistryEndothelial dysfunctionMolecular Biologylcsh:QH301-705.5Spectroscopychemistry.chemical_classificationReactive oxygen speciesmitochondrial oxidative stressOrganic Chemistryagingmitochondrial DNA damageGeneral Medicinevascular dysfunctionmedicine.diseaseComputer Science ApplicationsMitochondriaOxidative StressEndocrinologyBiochemistrychemistrylcsh:Biology (General)lcsh:QD1-999Cardiovascular Diseasesmedicine.symptomReactive Oxygen SpeciesOxidative stressDNA DamageInternational Journal of Molecular Sciences
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Mitochondrially encoded cysteine predicts animal lifespan

2007

Summary The role of genetic factors in the determination of lifespan is undisputed. However, numerous successful efforts to identify individual genetic modulators of longevity have not yielded yet a quantitative measure to estimate the lifespan of a species from scratch, merely based on its genomic constitution. Here, we report on a meta-examination of genome sequences from 248 animal species with known maximum lifespan, including mammals, birds, fish, insects, and helminths. Our analysis reveals that the frequency with which cysteine is encoded by mitochondrial DNA is a specific and phylogenetically ubiquitous molecular indicator of aerobic longevity: long-lived species synthesize respirat…

Models MolecularGeneticsAgingMitochondrial DNAFree Radicalsmedia_common.quotation_subjectLongevityRespiratory chainLongevityComputational BiologyLocus (genetics)Cell BiologyMitochondrionBiologyProtein oxidationDNA MitochondrialGenomeMitochondriaMitochondrial ProteinsAnimalsHumansCysteineAnaerobic bacteriaPhylogenymedia_commonAging Cell
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Whole mitochondrial genomes unveil the impact of domestication on goat matrilineal variability

2015

Background The current extensive use of the domestic goat (Capra hircus) is the result of its medium size and high adaptability as multiple breeds. The extent to which its genetic variability was influenced by early domestication practices is largely unknown. A common standard by which to analyze maternally-inherited variability of livestock species is through complete sequencing of the entire mitogenome (mitochondrial DNA, mtDNA). Results We present the first extensive survey of goat mitogenomic variability based on 84 complete sequences selected from an initial collection of 758 samples that represent 60 different breeds of C. hircus, as well as its wild sister species, bezoar (Capra aega…

Most recent common ancestor[SDV]Life Sciences [q-bio]PopulationMolecular Sequence DataMtDNA haplogroupsCapra aegagrusBiologyDNA MitochondrialHaplogroupDomesticationQH301Settore AGR/17 - Zootecnica Generale E Miglioramento GeneticoCapra hircusGeneticsAnimalsCapra aegagruDomesticationeducationQH426Phylogeny2. Zero hungereducation.field_of_studyOrigin of Capra hircusGenomeMtDNA haplogroupPhylogenetic treeGoatsHaplotypeGenetic VariationDNAOrigin of Capra hircuMitochondrialCapra aegagrus; Domestication; Goat mitochondrial genome; MtDNA haplogroups; Origin of Capra hircus; Biotechnology; GeneticsHaplotypesEvolutionary biologyGoat mitochondrial genomeGenome MitochondrialCapra aegagrus; Domestication; Goat mitochondrial genome; MtDNA haplogroups; Origin of Capra hircus; Animals; DNA Mitochondrial; Female; Genetic Variation; Genome Mitochondrial; Goats; Haplotypes; Molecular Sequence Data; Phylogeny; Biotechnology; GeneticsmtDNA haplogroupsFemaleResearch ArticleHuman mitochondrial DNA haplogroupBiotechnology
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STR genotyping and mtDNA sequencing of latent fingerprint on paper

2003

A systematic study was conducted to investigate whether DNA can be successfully extracted from latent fingerprints deposited on ordinary paper and analysed using short tandem repeat profiling and mitochondrial DNA sequencing. In order to evaluate the performance of latent fingerprint analysis in a criminal case, experiments with varying conditions were carried out to improve our understanding of low copy number (LCN) DNA typing. After optimising the extraction methods to achieve increased sensitivity, the examination of touched paper can routinely yield the STR profile of the individual who has touched it. A fingerprint can therefore be considered as a potential source of DNA for genetic id…

PaperMitochondrial DNAGenotypeSequence analysisComputational biologyBiologyDNA MitochondrialPolymerase Chain ReactionLatent fingerprintPathology and Forensic Medicinelaw.inventionlawHumansTypingDermatoglyphicsGenotypingAllelesPolymerase chain reactionElectrophoresis Agar GelGeneticsSequence Analysis DNADNA FingerprintingTandem Repeat SequencesMicrosatelliteLow copy numberLawForensic Science International
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Population differentiation in the marginal populations of the great tit (Paridae: Parus major)

2007

The major subspecies group of the great tit, Parus major, has experienced demographic and spatial expansions during the last century in several sites at the edges of its distribution range. These expansions, although temporarily very even, have resulted in dissimilar patterns of molecular diversity. Populations locating at regions of contact to other subspecies groups (in Amur, Kirghizia–Kazakhstan, and Iran) show divergence from central population by nuclear and mitochondrial markers. In Amur, gene flow from minor group could be detected based on the existence of private minor alleles in the major population. In Kirghizia and Kazakhstan, the bokharensis and major groups share almost all th…

Paruseducation.field_of_studyMitochondrial DNARange (biology)EcologyPopulationPopulation geneticsZoologyBiologySubspeciesbiology.organism_classificationGene flowMicrosatelliteeducationEcology Evolution Behavior and SystematicsBiological Journal of the Linnean Society
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Mitochondrial and chromosomal DNA alterations in human chromophobe renal cell carcinomas

1992

Renal cell tumours are characterized by the loss of chromosome 3p and trisomy of 5q segments (common, non-papillary renal cell carcinoma), or by trisomy of chromosomes 7 and 17 and loss of the Y chromosome (papillary renal cell carcinoma), or by random karyotype changes and mitochondrial DNA alterations (renal oncocytoma). We have studied by means of RFLP analysis the genomic and mitochondrial DNA in 11 chromophobe renal cell carcinomas, which have a unique morphology among kidney cancers. We found a loss of the constitutional heterozygosity at chromosomal regions 3p, 5q, 17p, and 17q, a combination of allelic losses that has not been found in other types of renal cell tumours. Three of the…

Pathologymedicine.medical_specialtyMitochondrial DNAChromophobe Renal Cell CarcinomaChromophobe cellBiologyurologic and male genital diseasesDNA MitochondrialChromosomesPathology and Forensic MedicineLoss of heterozygosityRenal cell carcinomamedicineHumansRenal oncocytomaCarcinoma Renal CellChromosome AberrationsKidneyStaining and LabelingPapillary renal cell carcinomasDNA Neoplasmmedicine.diseaseKidney NeoplasmsBlotting Southernmedicine.anatomical_structurePolymorphism Restriction Fragment LengthThe Journal of Pathology
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Mitochondrial Toxicity in HAART: An Overview of In Vitro Evidence

2011

The combined antiretroviral therapeutic approach currently employed for the treatment of HIV infection, known as Higly Active Antiretroviral Therapy (HAART), has dramatically reduced AIDS-related morbidity and mortality. However, the adverse reactions associated with the long term use of this therapy have now become a major issue and researchers have focused on understanding the cellular mechanisms underlying these drug-induced detrimental effects which englobe a large list of different events including rash and hypersensibility reactions, hepatotoxicity, metabolic disturbances including lipodystrophy, and other metabolic syndrome-like disturbances such as hyperlactatemia, hyperlipedimia, i…

PharmacologyMitochondrial DNAAnti-HIV AgentsMitochondrionBiologyPharmacologymedicine.diseaseDNA MitochondrialReverse transcriptaseMitochondriaMitochondrial toxicityInsulin resistancePharmacotherapyAntiretroviral Therapy Highly ActiveDrug DiscoverymedicineHumansDrug Therapy CombinationHyperlactatemiaLipodystrophyCurrent Pharmaceutical Design
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Mitochondrial biogenesis in health and disease. Molecular and therapeutic approaches.

2014

Mitochondrial biogenesis (MB) is the essential mechanism by which cells control the number of mitochondria. Cells respond to different physiologic, metabolic, and pathologic changes by regulating this organelle with high morphological and functional adaptability. A considerable number of proteins, transcription factors, upstream regulatory proteins and secondary mechanisms are involved in MB and the stabilization of new mitochondrial DNA. These MB activators and regulators, including the main participating proteins (e.g. PGC-1α and mtTFA), are candidates for therapeutic intervention in diverse diseases, like neurodegenerative disorders, metabolic syndrome, sarcopenia, cardiac pathophysiolo…

PharmacologyMitochondrial DNAMitochondrial DiseasesMechanism (biology)Health StatusDiseaseBiologyTFAMMitochondrionBioinformaticsmedicine.diseaseCell biologyMitochondriaMitochondrial biogenesisSarcopeniaDrug DiscoverymedicineAnimalsHumansEnergy MetabolismTranscription factorSignal TransductionTranscription FactorsCurrent pharmaceutical design
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Enhanced oxidative stress and increased mitochondrial mass during Efavirenz-induced apoptosis in human hepatic cells

2010

BACKGROUND AND PURPOSE Efavirenz (EFV) is widely used in the treatment of HIV-1 infection. Though highly efficient, there is growing concern about EFV-related side effects, the molecular basis of which remains elusive. EXPERIMENTAL APPROACH In vitro studies were performed to address the effect of clinically relevant concentrations of EFV (10, 25 and 50 µM) on human hepatic cells. KEY RESULTS Cellular proliferation and viability were reduced in a concentration-dependent manner. Analyses of the cell cycle and several cell death parameters (chromatin condensation, phosphatidylserine exteriorization, mitochondrial proapoptotic protein translocation and caspase activation) revealed that EFV trig…

PharmacologyMitochondrial DNAProgrammed cell deathMitochondrionBiologymedicine.diseasemedicine.disease_causeCell biologyMitochondrial toxicitychemistry.chemical_compoundchemistryBiochemistryApoptosismedicineCardiolipinOxidative stressMitochondrial DNA replicationBritish Journal of Pharmacology
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