Search results for "motor neurons"

showing 10 items of 89 documents

Aberrant arrested in maturation neuromuscular junctions in centronuclear myopathy

1994

Unusual ultrastructural changes of the nerve terminals have been found in an infant born with severe, fatal XLR form of centronuclear myopathy. Aberrant neuromuscular junctions in myotubes decorated by N-CAM were observed. The junction changes were manifested by simplification of postsynaptic membrane and paucity of secondary synaptic clefts. These resembles fetal neuromuscular junctions. The findings suggest that the expression of N-CAM by arrested myotubes may be promoted by abnormal nerve-muscle cell interactions, induced by motor endplate immaturity.

MaleCell Adhesion Molecules NeuronalCellNeuromuscular JunctionElectromyographyBiologyMicrotubulesMotor EndplateNeuromuscular junctionMotor EndplateMicrotubulemedicineHumansCentronuclear myopathyMotor NeuronsFetusTissue Embeddingmedicine.diagnostic_testElectromyographyMyogenesisMusclesInfantNeuromuscular Diseasesmedicine.diseaseCell biologyMicroscopy Electronmedicine.anatomical_structureNeurologySynapsesNeurology (clinical)NeuroscienceJournal of the Neurological Sciences
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Effects of Propofol on H-reflex in Humans

2001

Background Depression of spinal cord motoneuron excitability has been proposed to contribute to surgical immobility. The H-reflex, which measures alpha-motoneuron excitability, is depressed by volatile anesthetics, whereas the action of propofol is unknown. The objective of this study was to determine the effects of propofol anesthesia on the H-reflex. Methods In 13 patients (group 1), H-reflex was measured before (T0), 3 min after (T1), and 10 min after (T2) a 2-mg/kg bolus dose of propofol, followed by an infusion of 10 mg x kg(-1) x h(-1). Ten patients (group 2) were studied when propofol was given via a programmable pump set to a propofol blood concentration of 6 microg/ml, and 10 pati…

MaleCentral nervous systemH-ReflexmedicineHumansMuscle SkeletalPropofolMotor Neuronsbusiness.industryVolatile anestheticMiddle AgedSpinal cordElectric StimulationElectrophysiologyAnesthesiology and Pain Medicinemedicine.anatomical_structureMechanism of actionAction (philosophy)AnesthesiaFemaleH-reflexmedicine.symptomAnesthesia InhalationbusinessPropofolNeuroscienceAnesthetics Intravenousmedicine.drugAnesthesiology
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Estimation of the prevalence and incidence of motor neuron diseases in two Spanish regions: Catalonia and Valencia

2021

AbstractAccording to the degree of upper and lower motor neuron degeneration, motor neuron diseases (MND) can be categorized into amyotrophic lateral sclerosis (ALS), primary lateral sclerosis (PLS) or progressive muscular atrophy (PMA). Although several studies have addressed the prevalence and incidence of ALS, there is a high heterogeneity in their results. Besides this, neither concept has been previously studied in PLS or PMA. Thus, the objective of this study was to estimate the prevalence and incidence of MND, (distinguishing ALS, PLS and PMA), in the Spanish regions of Catalonia and Valencia in the period 2011–2019. Two population-based Spanish cohorts were used, one from Catalonia …

MaleMalalties neuromuscularsGene ExpressionSuperoxide Dismutase-10302 clinical medicinePrevalence030212 general & internal medicineAmyotrophic lateral sclerosisEstimation theoryPrimary Lateral Sclerosismedia_commonMotor neuronsMotor Neuronseducation.field_of_studyMultidisciplinaryIncidenceIncidence (epidemiology)QRMiddle AgedProgressive muscular atrophyNeuromuscular diseasesmedicine.anatomical_structureNeurones motoresNeurologyMedicineFemaleRiskSciencemedia_common.quotation_subjectPopulationBiologyArticleMuscular Atrophy Spinal03 medical and health sciencesmedicineHumansMotor Neuron DiseaseEspanyaEstimació Teoria de l'educationAgedEstimationSelection biasMotor neurons -- DiseasesModels StatisticalC9orf72 ProteinAmyotrophic Lateral SclerosisMotor neuronmedicine.diseaseRisk factorsSpainNeurones motores -- MalaltiesMutationBiomarkers030217 neurology & neurosurgeryDemography
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Inhibition of autophagy rescues muscle atrophy in a LGMDD2 Drosophila model

2021

Limb-girdle muscular dystrophy D2 (LGMDD2) is an ultrarare autosomal dominant myopathy caused by mutation of the normal stop codon of the TNPO3 nuclear importin. The mutant protein carries a 15 amino acid C-terminal extension associated with pathogenicity. Here we report the first animal model of the disease by expressing the human mutant TNPO3 gene in Drosophila musculature or motor neurons and concomitantly silencing the endogenous expression of the fly protein ortholog. A similar genotype expressing wildtype TNPO3 served as a control. Phenotypes characterization revealed that mutant TNPO3 expression targeted at muscles or motor neurons caused LGMDD2-like phenotypes such as muscle degener…

MaleMutantBiochemistryAnimals Genetically ModifiedMutant proteinAutophagyGeneticsmedicineAnimalsHumansGene silencingMuscular dystrophyMyopathyMolecular BiologyMotor NeuronsbiologyMusclesAutophagyChloroquinebeta Karyopherinsmedicine.diseasebiology.organism_classificationMuscle atrophyCell biologySurvival RateDisease Models AnimalMuscular AtrophyDrosophila melanogasterPhenotypeMuscular Dystrophies Limb-GirdleInsect HormonesFemalemedicine.symptomDrosophila melanogasterLocomotionBiotechnologyThe FASEB Journal
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Membrane-Derived Phospholipids Control Synaptic Neurotransmission and Plasticity

2015

Synaptic communication is a dynamic process that is key to the regulation of neuronal excitability and information processing in the brain. To date, however, the molecular signals controlling synaptic dynamics have been poorly understood. Membrane-derived bioactive phospholipids are potential candidates to control short-term tuning of synaptic signaling, a plastic event essential for information processing at both the cellular and neuronal network levels in the brain. Here, we showed that phospholipids affect excitatory and inhibitory neurotransmission by different degrees, loci, and mechanisms of action. Signaling triggered by lysophosphatidic acid (LPA) evoked rapid and reversible depress…

MalePatch-Clamp TechniquesQH301-705.5NeurotransmissionBiologyInhibitory postsynaptic potentialSynaptic TransmissionGeneral Biochemistry Genetics and Molecular BiologyMicePregnancySynaptic augmentationMetaplasticityAnimalsRats WistarBiology (General)Motor Neuronsrho-Associated KinasesNeuronal PlasticityGeneral Immunology and MicrobiologyCalcineurinGeneral NeuroscienceReceptors GABA-ACell biologySynaptic fatigueBiochemistrySynapsesSynaptic plasticityExcitatory postsynaptic potentialFemalelipids (amino acids peptides and proteins)Synaptic signalingLysophospholipidsrhoA GTP-Binding ProteinGeneral Agricultural and Biological SciencesResearch Article
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Phenytoin-induced glutathione depletion in rat peripheral nerve

1995

Abstract Administration of high doses (150–250 mg/kg body weight) of phenytoin (DPH) promote a 40% decrease in glutathione (GSH) content of rat sciatic nerve. This DPH-induced GSH depletion is accompanied with an electrophysiological impairment of peripheral neuromuscular function. H7 (20 mg/kg body weight IP, 30 min prior to DPH), a protein kinase C inhibitor, was able to prevent the DPH-induced GSH depletion only at the lower DPH dose used. This same inhibitor completely prevented the electrophysiological impairment at the lower DPH dose, and only partially at the higher DPH dose used. These results confirm the hypothesis of a DPH-dependent activation of PKC (that might be triggered by, o…

MalePhenytoinAntioxidantmedicine.medical_treatmentAction PotentialsIn Vitro TechniquesPharmacologymedicine.disease_causeBiochemistryPiperazineschemistry.chemical_compound1-(5-Isoquinolinesulfonyl)-2-MethylpiperazinePhysiology (medical)polycyclic compoundsmedicineAnimalsEnzyme InhibitorsRats WistarMuscle SkeletalEvoked PotentialsProtein Kinase CProtein kinase CMotor NeuronsAnalysis of Variancetechnology industry and agricultureNeurotoxicityGlutathioneIsoquinolinesmedicine.diseaseGlutathioneSciatic NerveRatsKineticschemistryBiochemistryPhenytoinAnticonvulsantslipids (amino acids peptides and proteins)Sciatic nerveOxidative stressIntracellularmedicine.drugFree Radical Biology and Medicine
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Effects of caffeine on neuromuscular function in a non‐fatigued state and during fatiguing exercise

2020

New findings What is the central question of the study? What are the effects of caffeine on neuromuscular function in a non-fatigued state and during fatiguing exercise? What is the main finding and its importance? In a non-fatigued state, caffeine decreased the duration of the silent period evoked by transcranial magnetic stimulation. Caffeine-induced reduction of inhibitory mechanisms in the central nervous system before exercise was associated with an increased performance. Individuals who benefit from caffeine ingestion may experience lower perception of effort during exercise and an accelerated recovery of M-wave amplitude postfatigue. This study elucidates the mechanisms of action of …

MalePhysiologyväsymysmedicine.medical_treatmentliikunta030204 cardiovascular system & hematologyRC1200H-Reflexchemistry.chemical_compound0302 clinical medicineTriceps surae muscletranscranial magnetic stimulationMedicinerasitusMotor NeuronsNutrition and DieteticsMotor CortexGeneral MedicineTranscranial Magnetic Stimulationhermo-lihastoimintamedicine.anatomical_structureNeuromuscular AgentsMuscle FatigueCaffeineMuscle ContractionAdultmedicine.medical_specialtyQP301.H75_Physiology._Sport.Postureperipheral fatiguePlacebo03 medical and health sciencesPhysical medicine and rehabilitationDouble-Blind MethodCaffeinePhysiology (medical)Humansrate of perceived exertionExercisesoleussuorituskykySoleus musclePyramidal tractsbusiness.industryEvoked Potentials MotorQPCrossover studycentral fatigueTranscranial magnetic stimulationchemistrykofeiiniSilent periodbusiness030217 neurology & neurosurgeryExperimental Physiology
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Motoneurons of the stapedius muscle in the guinea pig middle ear: Afferent and efferent transmitters

2008

The objective of the present study was to identify efferent and afferent transmitters of motoneurons of the stapedius muscle of the middle ear in order to gain more insight into the neuronal regulation of the muscle. To identify motoneurons, we injected the fluorescent neuronal tracer Fluorogold (FG) into the muscle after preparation of the middle ear in adult guinea pigs. Upon terminal uptake and retrograde neuronal transport, we observed FG in neurons located medial and ventral to the nucleus of the facial nerve ipsilateral to the injection site. Immunohistochemical studies of these motoneurons showed that the majority contains calcitonin gene-related peptide. Our data further demonstrate…

MaleSerotoninStilbamidinesCalcitonin Gene-Related PeptideEfferentGuinea PigsEar MiddleNitric Oxide Synthase Type ISubstance PBiologyNitric OxideEfferent PathwaysStapedius muscleGuinea pigHearingNitrergic NeuronsmedicineAnimalsMolecular BiologyNeuronal transportMotor NeuronsAfferent PathwaysBrain MappingNeurotransmitter AgentsStaining and LabelingGeneral NeuroscienceNeuropeptidesStapediusAnatomyMotor neuronImmunohistochemistryRetrograde tracingFacial nerveStapesRhombencephalonFacial Nervemedicine.anatomical_structurenervous systemMiddle earNeurology (clinical)NeuroscienceDevelopmental BiologyBrain Research
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Structural and functional identification of two distinct inspiratory neuronal populations at the level of the phrenic nucleus in the rat cervical spi…

2018

The diaphragm is driven by phrenic motoneurons that are located in the cervical spinal cord. Although the anatomical location of the phrenic nucleus and the function of phrenic motoneurons at a single cellular level have been extensively analyzed, the spatiotemporal dynamics of phrenic motoneuron group activity have not been fully elucidated. In the present study, we analyzed the functional and structural characteristics of respiratory neuron population in the cervical spinal cord at the level of the phrenic nucleus by voltage imaging, together with histological analysis of neuronal and astrocytic distribution in the cervical spinal cord. We found spatially distinct two cellular populations…

MaleTime FactorsFunctional identificationAction PotentialsPhrenic motoneuron0302 clinical medicineNeural PathwaysMotor Neuronseducation.field_of_studyPhrenic nucleusGeneral Neurosciencemusculoskeletal neural and ocular physiologyRespiratory control05 social sciencesVoltage imagingAnatomymusculoskeletal systemDiaphragm (structural system)Neuroanatomical Tract-Tracing Techniquesmedicine.anatomical_structureInhalationCervical VertebraeFemaleOriginal ArticleAnatomyAstrocyteAstrocyteHistologyCordInterneuronPopulationDiaphragmBiologyIn Vitro Techniques050105 experimental psychologyInterneuron03 medical and health sciencesmedicineAnimals0501 psychology and cognitive sciencesPhrenic NucleusRats WistareducationCervical CordScalene motoneuronCervical spinal cordSpinal cordVoltage-Sensitive Dye Imagingnervous systemAnimals Newborn030217 neurology & neurosurgeryBrain StemBrain structurefunction
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Ipsilateral corticomotor responses are confined to the homologous muscle following cross-education of muscular strength

2017

Cross-education of strength occurs when strength-training 1 limb increases the strength of the untrained limb and is restricted to the untrained homologous muscle. Cortical circuits located ipsilateral to the trained limb might be involved. We used transcranial magnetic stimulation (TMS) to determine the corticomotor responses from the untrained homologous (biceps brachii) and nonhomologous (flexor carpi radialis) muscle following strength-training of the right elbow flexors. Motor evoked potentials were recorded from the untrained left biceps brachii and flexor carpi radialis during a submaximal contraction from 20 individuals (10 women, 10 men; aged 18–35 years; training group, n = 10; c…

MaleTime FactorsPhysiologyEndocrinology Diabetes and MetabolismCross-activationPhysical strengthRandom Allocation0302 clinical medicineCross activationMedicineInhibitionMotor NeuronsNutrition and DieteticsMotor CortexGeneral MedicineAnatomymusculoskeletal systemTranscranial Magnetic StimulationestotTreatment OutcomeFemalelihaskuntoMuscle ContractionAdultAgonistmedicine.medical_specialtyAdolescentmedicine.drug_classAgonistCross educationYoung Adult03 medical and health sciencesPhysical medicine and rehabilitationPhysiology (medical)Homologous chromosomeHumansMuscle StrengthMuscle SkeletalSynergistCortical circuitsExcitabilityElectromyographybusiness.industryNeural InhibitionResistance Training030229 sport sciencesEvoked Potentials MotorC600raajatbody regionsbusiness030217 neurology & neurosurgerylihasvoimaApplied Physiology, Nutrition, and Metabolism
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