Search results for "multidrug"

showing 10 items of 118 documents

Lack of Cry1Fa binding to the midgut brush border membrane in a resistant colony of Plutella xylostella moths with a mutaton in the ABCC2 locus

2012

ABSTRACT Previous studies reported “mode 1” Bacillus thuringiensis resistance in a colony of diamondback moths (NO-QA), and recently, this resistance has been mapped to an ABC transporter ( ABCC2 ) locus. We report the lack of binding of Cry1Fa to insects derived from this colony and compare our data with those from other insects with ABCC2 -associated resistance.

BioquímicaBrush borderBiotecnologia agrícolaDrug ResistanceResistència als plaguicidesLocus (genetics)ATP-binding cassette transporterDrug resistanceApplied Microbiology and BiotechnologyLepidoptera genitaliaHemolysin ProteinsPlagues ControlBacterial ProteinsBacillus thuringiensisInvertebrate MicrobiologyAnimalsGeneticsBacillus thuringiensis ToxinsMicrovilliEcologybiologyfungiPlutellaMidgutbiology.organism_classificationMultidrug Resistance-Associated Protein 2EndotoxinsLepidopteraMutationMultidrug Resistance-Associated ProteinsProtein BindingFood ScienceBiotechnology
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Curcumin as a possible lead compound against hormone-independent, multidrug-resistant breast cancer

2009

We examine the possible evidence that the phytochemical curcumin may overcome resistance to hormonal and cytotoxic agents in breast cancer. We present our observations on MCF-7R, a multidrug-resistant (MDR) variant of the MCF-7 breast cancer cell line. In contrast to MCF-7, MCF-7R lacks aromatase and estrogen receptor alpha (ERalpha) and overexpresses the multidrug transporter ABCB1 and the products of different genes implicated in cell proliferation and survival, like c-IAP-1, NAIP, survivin, and COX-2. Nevertheless, in cytotoxicity and cell death induction assays, we found that the antitumor activity of curcumin is substantial both in MCF-7 and in MCF-7R. We elaborated the diketone system…

Breast cancer multidrug resistance hormone-independencecurcumin analoguesCurcuminAnaloguesAntineoplastic AgentsBreast NeoplasmsPharmacologyMultidrug resistanceGeneral Biochemistry Genetics and Molecular Biologychemistry.chemical_compoundBreast cancerBreast cancerHistory and Philosophy of ScienceCell Line TumorSurvivinmedicineHumansAnalogues; Breast cancer; Curcumin; Hormone-independence; Multidrug resistance;Aromataseskin and connective tissue diseasesCytotoxicitybiologyHormone-independenceGene Expression ProfilingGeneral Neurosciencemedicine.diseaseDrug Resistance MultipleMultiple drug resistancechemistryDrug Resistance NeoplasmApoptosisCurcuminbiology.proteinSettore BIO/14 - FarmacologiaEstrogen receptor alpha
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Drug Susceptibility Patterns in MDR-TB Patients: Challenges for Future Regimen Design. A Cross-Sectional Study

2015

Globally, there is substantial concern regarding the challenges of treating complex drug resistance patterns in multidrug resistant tuberculosis cases. Utilising data from three different settings (Estonia, Latvia, Romania) we sought to contrast drug susceptibility profiles for multidrug resistant tuberculosis cases, highlight the difficulties in designing universal regimen, and inform future regimen selection. Demographic and microbiological surveillance data for multidrug resistant tuberculosis cases from 2004-13 were analysed. High levels of additional resistance to currently recommended second line drugs were seen in all settings, with extensive variability between countries. Accurate d…

COUNTRIESEstoniaMaleDrug Resistance Multiple Bacterial/drug effectsFluoroquinolones/pharmacologyGeneral Science & TechnologyAntitubercular Agents/therapeutic useRomania/epidemiologyAntitubercular Agentslcsh:MedicineDrug Resistance Multiple BacterialMD MultidisciplinaryTuberculosis Multidrug-ResistantHumansMULTIDRUG-RESISTANT TUBERCULOSISlcsh:ScienceLatvia/epidemiologyDemographyScience & TechnologyRomanialcsh:REstonia/epidemiologyLatviaMultidisciplinary SciencesCross-Sectional StudiesScience & Technology - Other Topicslcsh:QFemaleTuberculosis Multidrug-Resistant/drug therapyResearch ArticleFluoroquinolonesPLoS ONE
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Laurus nobilis L. Seed Extract Reveals Collateral Sensitivity in Multidrug-Resistant P-Glycoprotein-Expressing Tumor Cells.

2015

The frequent failure of standard cancer chemotherapy requires the development of novel drugs capable of killing otherwise drug-resistant tumors. Here, we have investigated a chloroform extract of Laurus nobilis seeds. Fatty acids and 23 constituents of the volatile fraction were identified by gas chromotography/flame ionization detection (GC/FID) and gas chromatography/mass spectrometry (GC/MS), in good agreement with (1)H NMR (nuclear magnetic resonance) spectrum. Multidrug-resistant P-glycoprotein-expressing CEM/ADR5000 leukemia cells were hypersensitive (collaterally sensitive) toward this extract compared to drug-sensitive CCRF-CEM cells, whereas CEM/ADR5000 cells were 2586-fold resista…

Cancer ResearchATP Binding Cassette Transporter Subfamily BCell SurvivalABC transporter Cancer Lauraceae Multidrug resistance Oncobiogram SpiceMedicine (miscellaneous)Multidrug resistanceLaurusGas Chromatography-Mass SpectrometryFlow cytometryNOchemistry.chemical_compoundLauraceaeLaurus nobilisfoodCell Line TumormedicineHumansDoxorubicinP-glycoproteinCancerNutrition and DieteticsChromatographyLeukemiabiologymedicine.diagnostic_testChemistryPlant ExtractsOncobiogramMolecular biologyAntineoplastic Agents Phytogenicfood.foodMultiple drug resistanceOleic acidSpiceEucalyptolOncologyCell cultureDoxorubicinDrug Resistance NeoplasmSeedsbiology.proteinABC transportermedicine.drugNutrition and cancer
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Expression of the IAPs in multidrug resistant tumor cells

2003

We have investigated the expression of the IAPs (inhibitory of apoptosis proteins) in the human HL-60 leukemia and in its multidrug resistant, P-glycoprotein (P-gp) over-expressing variant, HL-60R. HL-60R exhibits resistance to apoptosis induced from P-gp substrate drugs and also from other triggers (cisplatin, TNF-alpha, Fas ligation, TRAIL, IFN-gamma and serum starvation) not related to the multidrug transporter. Except for c-IAP-1 mRNA, HL-60R significantly over-expressed both the mRNAs and the proteins of all the IAPs studied, i.e. c-IAP-1, c-IAP-2, XIAP, NAIP and survivin. Determination of the DNA-binding capacity of NF-kappaB (p50 or p65 subunits) indicated that, while HL-60 cells sho…

Cancer ResearchBlotting WesternCellApoptosisHL-60 CellsBiologyInhibitor of Apoptosis Proteinsmultidrug resistanceSurvivinmedicineHumansRNA MessengerCisplatinOncogeneReverse Transcriptase Polymerase Chain ReactionNF-kappa BProteinsGeneral MedicineIAPCell cycleapoptosiMolecular biologyDrug Resistance MultipleXIAPGene Expression Regulation Neoplasticmedicine.anatomical_structureOncologyDrug Resistance NeoplasmApoptosisCancer researchNAIPmedicine.drugOncology Reports
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Apoptotic effects of thiazolobenzimidazole derivatives on sensitive and multidrug resistant leukaemic cells

2001

We investigated the cytotoxic activity of eight thiazolobenzimidazole derivatives on sensitive HL60 and multidrug-resistant (MDR) (HL60R) leukaemia cell lines. The antitumour effects of these compounds were compared with those of RS-TBZ, a thiazolobenzimidazole derivative, previously described in our reports, that was able to induce apoptosis more markedly in MDR cells than in the parental sensitive cell lines. Only two compounds in this study proved to have interesting effects: (a) the S-enantiomer of TBZ, that was able to induce apoptosis in MDR cells in a slightly more selective manner than TBZ (racemic form); and (b) TBZ-4-OCH3 (TBZ-4-OCH3), that showed cytotoxic and apoptotic effects o…

Cancer ResearchHL60Antineoplastic AgentsHL-60 CellsApoptosisBiologyMultidrug resistanceCaspase 8Anticancer drugschemistry.chemical_compoundAntigenCytotoxic T cellHumansLeukaemiafas ReceptorProgenitor cellLeukemiaCell CycleCaspase InhibitorsDrug Resistance MultipleMultiple drug resistanceThiazolobenzimidazoleThiazolesAnticancer drugs; Apoptosis; Leukaemia; Multidrug resistance; Thiazolobenzimidazole;OncologychemistryCell cultureApoptosisDrug Resistance NeoplasmImmunologyCancer researchBenzimidazoles
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Control of spasticity in a multiple sclerosis model using central nervous system-excluded CB1 cannabinoid receptor agonists

2014

The purpose of this study was the generation of central nervous system (CNS)-excluded cannabinoid receptor agonists to test the hypothesis that inhibition of spasticity, due to CNS autoimmunity, could be controlled by affecting neurotransmission within the periphery. Procedures included identification of chemicals and modeling to predict the mode of exclusion; induction and control of spasticity in the ABH mouse model of multiple sclerosis; conditional deletion of CB1 receptor in peripheral nerves; side-effect profiling to demonstrate the mechanism of CNS-exclusion via drug pumps; genome-wide association study in N2(129×ABH) backcross to map polymorphic cannabinoid drug pump; and sequencing…

Central Nervous SystemCannabinoid receptorEncephalomyelitis Autoimmune ExperimentalMultiple Sclerosismedicine.medical_treatmentCentral nervous systemPharmacologyBiologyBiochemistryMiceReceptor Cannabinoid CB1GeneticsmedicineAnimalsSpasticityMolecular BiologyCannabinoid Receptor AgonistsCannabinoidsMultiple sclerosisExperimental autoimmune encephalomyelitisCannabinoid Receptor Agonistsmedicine.disease3. Good healthmedicine.anatomical_structureAjulemic acidMuscle SpasticityFemaleCannabinoidmedicine.symptomMultidrug Resistance-Associated ProteinsBiotechnologymedicine.drug
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Successful control of an outbreak of colonization by Klebsiella pneumoniae carbapenemase-producing K. pneumoniae sequence type 258 in a neonatal inte…

2013

This article reports an outbreak of colonization by Klebsiella pneumoniae carbapenemase-producing K. pneumoniae (KPC-Kp) sequence type (ST) 258 in a neonatal intensive care unit (NICU) in Palermo, Italy. KPC-Kp ST258 was detected by an active surveillance culture programme. Between 18th September and 14th November 2012, KPC-Kp was isolated from 10 out of 54 neonates admitted in the outbreak period. No cases of infection were recorded. Male sex was associated with colonization, whereas administration of ampicilline-sulbactam plus gentamicin was protective. Infection control interventions interrupted the spread of KPC-Kp without the need to close the NICU to new admissions. (C) 2013 The Healt…

ColonizationMaleMicrobiology (medical)Settore MED/07 - Microbiologia E Microbiologia ClinicaPediatricsmedicine.medical_specialtyNeonatal intensive care unitKlebsiella pneumoniaeMultidrug resistanceSettore MED/42 - Igiene Generale E ApplicataST258beta-LactamasesDisease OutbreaksNeonatal intensive care unitSettore MED/38 - Pediatria Generale E SpecialisticaBacterial ProteinsDrug Resistance Multiple BacterialIntensive Care Units NeonatalAmpicillinpolycyclic compoundsmedicineHumansInfection controlColonizationInfection ControlKPC-Klebsiella pneumoniaebiologybusiness.industryInfant NewbornOutbreakGeneral MedicineSulbactambiochemical phenomena metabolism and nutritionbacterial infections and mycosesbiology.organism_classificationKlebsiella InfectionsKlebsiella pneumoniaeInfectious DiseasesItalyFemaleGentamicinbusinessMultilocus Sequence Typingmedicine.drugJournal of Hospital Infection
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A Machine Learning-Based Prediction Platform for P-Glycoprotein Modulators and Its Validation by Molecular Docking

2019

P-glycoprotein (P-gp) is an important determinant of multidrug resistance (MDR) because its overexpression is associated with increased efflux of various established chemotherapy drugs in many clinically resistant and refractory tumors. This leads to insufficient therapeutic targeting of tumor populations, representing a major drawback of cancer chemotherapy. Therefore, P-gp is a target for pharmacological inhibitors to overcome MDR. In the present study, we utilized machine learning strategies to establish a model for P-gp modulators to predict whether a given compound would behave as substrate or inhibitor of P-gp. Random forest feature selection algorithm-based leave-one-out random sampl…

Computer scienceFeature selectionP-glycoproteinMachine learningcomputer.software_genreArticledrug discoveryMachine Learningmultidrug resistancemedicineHumansDoxorubicinATP Binding Cassette Transporter Subfamily B Member 1lcsh:QH301-705.5P-glycoproteinbiologybusiness.industryDrug discoveryGeneral Medicinemolecular dockingchEMBLartificial intelligenceMultiple drug resistanceMolecular Docking Simulationlcsh:Biology (General)Docking (molecular)biology.proteinEffluxArtificial intelligencebusinesscomputerSoftwaremedicine.drugCells
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No-touch methods of terminal cleaning in the intensive care unit: results from the first large randomized trial with patient-centred outcomes

2017

N/A

Cross infectionmedicine.medical_specialtySepsiICU-acquired infections; Multidrug resistant organisms; Sepsis; Terminal cleaningIntensive Care UnitMEDLINEMultidrug resistant organismCritical Care and Intensive Care MedicineArticlelaw.inventionSepsis03 medical and health sciencesICU-acquired infection0302 clinical medicineRandomized controlled triallawSepsismedicineHumans030212 general & internal medicineTerminal cleaningIntensive care medicineCross Infectionbusiness.industryMultidrug resistant organismslcsh:Medical emergencies. Critical care. Intensive care. First aid030208 emergency & critical care medicinelcsh:RC86-88.9medicine.diseaseIntensive care unitTerminal cleaningIntensive Care UnitsEmergency medicineICU-acquired infections; Multidrug resistant organisms; Sepsis; Terminal cleaning; Humans; Cross Infection; Intensive Care Units; Critical Care and Intensive Care MedicinebusinessICU-acquired infectionsPatient centredHumanCritical Care
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