Search results for "muro"

showing 10 items of 64 documents

Ablation of the Regulatory IE1 Protein of Murine Cytomegalovirus Alters In Vivo Pro-inflammatory TNF-alpha Production during Acute Infection

2012

Little is known about the role of viral genes in modulating host cytokine responses. Here we report a new functional role of the viral encoded IE1 protein of the murine cytomegalovirus in sculpting the inflammatory response in an acute infection. In time course experiments of infected primary macrophages (MΦs) measuring cytokine production levels, genetic ablation of the immediate-early 1 (ie1) gene results in a significant increase in TNFα production. Intracellular staining for cytokine production and viral early gene expression shows that TNFα production is highly associated with the productively infected MΦ population of cells. The ie1- dependent phenotype of enhanced MΦ TNFα production …

MaleCytomegalovirus InfectionMuromegalovirusViral Diseasesmedicine.medical_treatmentvirusesTNF TNF-alpha murine cytomegalovirus MCMV IEVirus ReplicationMice0302 clinical medicineGene expressionBiology (General)Mice Inbred BALB C0303 health scienceseducation.field_of_studyPhysicsvirus diseasesHerpesviridae InfectionsTransfection3. Good healthGenètica microbianaInterleukin 10PhenotypeInfectious DiseasesCytokineLiverCytokinesMedicineFemaleTumor necrosis factor alphaBIOMEDICINA I ZDRAVSTVO. Temeljne medicinske znanosti.Microbial geneticsSignal TransductionResearch ArticleDNA ReplicationGene Expression Regulation ViralQH301-705.5ImmunologyPopulationBiologyMicrobiologyCell LineImmediate-Early ProteinsViral Proteins03 medical and health sciencesIn vivoVirologyGeneticsmedicineAnimalseducationMolecular Biology030304 developmental biologyTumor Necrosis Factor-alphaMacrophagesBIOMEDICINE AND HEALTHCARE. Basic Medical Sciences.FísicaRC581-607Mice Inbred C57BLViral replicationDNA ViralImmunologyParasitologyImmunologic diseases. Allergy030215 immunology
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Antigen-presenting cells of haematopoietic origin prime cytomegalovirus-specific CD8 T-cells but are not sufficient for driving memory inflation duri…

2011

Expansion of the CD8 T-cell memory pool, also known as ‘memory inflation’, for certain but not all viral epitopes in latently infected host tissues is a special feature of the immune response to cytomegalovirus. The Ld-presented murine cytomegalovirus (mCMV) immediate–early (IE) 1 peptide is the prototype of an epitope that is associated with memory inflation. Based on the detection of IE1 transcripts in latently infected lungs it was previously proposed that episodes of viral gene expression and antigenic activity due to desilencing of a limited number of viral genes may drive epitope-specific memory inflation. This would imply direct antigen presentation through latently infected host tis…

MaleMice Inbred BALB CMuromegalovirusbiologyAntigen presentationAntigen-Presenting CellsPriming (immunology)CD8-Positive T-LymphocytesVirologyEpitopeImmediate-Early ProteinsVirus LatencyEpitopesMiceImmune systemAntigenVirologyImmunologyMHC class Ibiology.proteinAnimalsCytotoxic T cellFemaleAntigen-presenting cellImmunologic MemoryJournal of General Virology
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Single cell detection of latent cytomegalovirus reactivation in host tissue

2011

The molecular mechanisms leading to reactivation of latent cytomegalovirus are not well understood. To study reactivation, the few cells in an organ tissue that give rise to reactivated virus need to be identified, ideally at the earliest possible time point in the process. To this end, mouse cytomegalovirus (MCMV) reporter mutants were designed to simultaneously express the red fluorescent protein mCherry and the secreted Gaussia luciferase (Gluc). Whereas Gluc can serve to assess infection at the level of individual mice by measuring luminescence in blood samples or by in vivo imaging, mCherry fluorescence offers the advatage of detection of infection at the single cell level. To visualiz…

MaleMuromegalovirusCytomegalovirusGene Expressionmedicine.disease_causeVirusHerpesviridaeGreen fluorescent proteinMiceGaussiaMuromegalovirusSingle-cell analysisGenes ReporterVirologyVirus latencymedicineAnimalsHumansLuciferasesLungMice Inbred BALB CbiologyHerpesviridae Infectionsbiology.organism_classificationmedicine.diseaseVirologyVirus LatencyDisease Models AnimalLuminescent ProteinsCytomegalovirus InfectionsHost-Pathogen InteractionsFemaleVirus ActivationSingle-Cell AnalysismCherryJournal of General Virology
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The NK Cell Response to Mouse Cytomegalovirus Infection Affects the Level and Kinetics of the Early CD8+ T-Cell Response

2012

ABSTRACT Natural killer (NK) cells and CD8 + T cells play a prominent role in the clearance of mouse cytomegalovirus (MCMV) infection. The role of NK cells in modulating the CD8 + T-cell response to MCMV infection is still the subject of intensive research. For analyzing the impact of NK cells on mounting of a CD8 + T-cell response and the contribution of these cells to virus control during the first days postinfection (p.i.), we used C57BL/6 mice in which NK cells are specifically activated through the Ly49H receptor engaged by the MCMV-encoded ligand m157. Our results indicate that the requirement for CD8 + T cells in early MCMV control inversely correlates with the engagement of Ly49H. W…

MaleMuromegalovirusImmunologyNK cellsCD8-Positive T-LymphocytesBiologym157MicrobiologyRodent DiseasesMice03 medical and health sciencesInterleukin 210302 clinical medicineVirologyAnimalsCytotoxic T cellmouse cytomegalovirus; NK cells; T-cell response; Ly49H; m157IL-2 receptor030304 developmental biologyMice Inbred BALB C0303 health sciencesJanus kinase 3ZAP70BIOMEDICINE AND HEALTHCARE. Basic Medical Sciences.Herpesviridae InfectionsNatural killer T cellMouse cytomegalovirus3. Good healthKiller Cells NaturalMice Inbred C57BLKineticsT-cell responseInsect ScienceImmunologyInterleukin 12CytokinesPathogenesis and ImmunityFemaleLy49HBIOMEDICINA I ZDRAVSTVO. Temeljne medicinske znanosti.CD8030215 immunology
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Liver Sinusoidal Endothelial Cells Are a Site of Murine Cytomegalovirus Latency and Reactivation▿

2009

ABSTRACT Latent cytomegalovirus (CMV) is frequently transmitted by organ transplantation, and its reactivation under conditions of immunosuppressive prophylaxis against graft rejection by host-versus-graft disease bears a risk of graft failure due to viral pathogenesis. CMV is the most common cause of infection following liver transplantation. Although hematopoietic cells of the myeloid lineage are a recognized source of latent CMV, the cellular sites of latency in the liver are not comprehensively typed. Here we have used the BALB/c mouse model of murine CMV infection to identify latently infected hepatic cell types. We performed sex-mismatched bone marrow transplantation with male donors …

MaleMuromegalovirusMyeloidGenes ViralViral pathogenesisImmunologymedicine.disease_causeMicrobiologyHerpesviridaeVirusMiceAntigenBetaherpesvirinaeVirologyVirus latencymedicineAnimalsMice Inbred BALB CbiologyGene Expression ProfilingEndothelial Cellsbiology.organism_classificationmedicine.diseaseVirologyVirus LatencyHaematopoiesismedicine.anatomical_structureLiverInsect ScienceImmunologyPathogenesis and ImmunityFemaleVirus Activation
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Two Antigenic Peptides from Genes m123 and m164 of Murine Cytomegalovirus Quantitatively Dominate CD8 T-Cell Memory in the H-2 d Haplotype

2001

ABSTRACT The importance of CD8 T cells for the control of cytomegalovirus (CMV) infection has raised interest in the identification of immunogenic viral proteins as candidates for vaccination and cytoimmunotherapy. The final aim is to determine the viral “immunome” for any major histocompatibility complex class I molecule by antigenicity screening of proteome-derived peptides. For human CMV, there is a limitation to this approach: the T cells used as responder cells for peptide screening are usually memory cells that have undergone in vivo selection. On this basis, pUL83 (pp65) and pUL123 (IE1 or pp68 to -72) were classified as immunodominant proteins. It is an open question whether this li…

MuromegalovirusAdoptive cell transferAntigenicityImmunologyCD8-Positive T-LymphocytesBiologymedicine.disease_causeMajor histocompatibility complexMicrobiologyImmediate-Early ProteinsViral Matrix ProteinsMiceOpen Reading FramesViral ProteinsImmune systemVirologymedicineAnimalsCytotoxic T cellAntigens ViralMice Inbred BALB CH-2 AntigensCytomegalovirusHerpesviridae InfectionsPhosphoproteinsVirologyPeptide FragmentsHaplotypesInsect ScienceProteomeImmunologybiology.proteinPathogenesis and ImmunityFemaleImmunologic MemorySpleenCD8Journal of Virology
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Control of murine cytomegalovirus in the lungs: Relative but not absolute immunodominance of the immediate-early 1 nonapeptide during the antiviral c…

1998

Effective control by the immune system is a hallmark of cytomegalovirus (CMV) infection. Accordingly, human CMV disease is a medical problem restricted to the immunologically immature or immunocompromised host (for a review, see reference 21). Murine models have implicated natural killer (NK) cells and CD8 T cells in the control of CMV infection. While NK cells mediate early protection in genetically resistant mouse inbred strains (4, 5, 31, 51), CD8 T cells establish enduring protective memory and function as principal antiviral effectors in susceptible strains (31). Specifically, in the BALB/c strain, major histocompatibility complex (MHC) class I-restricted antiviral CD8 T cells resolve …

MuromegalovirusAdoptive cell transferImmunologyViral Pathogenesis and ImmunityBone Marrow CellsImmunodominanceVirus ReplicationMajor histocompatibility complexMicrobiologyImmediate-Early ProteinsMiceImmune systemAntigenVirologyMHC class IAnimalsCytotoxic T cellLungAntigen PresentationMice Inbred BALB CbiologyImmunodominant EpitopesAntigen processingvirus diseasesHerpesviridae InfectionsVirologyKineticsInsect ScienceImmunologyTrans-Activatorsbiology.proteinFemaleT-Lymphocytes Cytotoxic
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Adoptive CD8 T Cell Control of Pathogens Cannot Be Improved by Combining Protective Epitope Specificities

2008

Adoptive transfer of CD8 T cells has the potential to cure infectious or malignant diseases that are refractory to conventional chemotherapy. A practically important but still unanswered question is whether mixtures of protective CD8 T cells with different epitope specificities mediate more efficient effector cell functions than do the monospecific individual CD8 T cell populations. In this study, we have addressed this issue for models of viral and bacterial infection. CD8 T cell-mediated cytotoxicity in vitro and protection in vivo were assessed to test whether CD8 T cell lines cooperate in target cell lysis and control of infection, respectively. Our data clearly show that mixtures of cy…

MuromegalovirusAdoptive cell transferT cellEpitopes T-LymphocyteBacteremiaStreptamerCD8-Positive T-LymphocytesBiologyEpitopeMicemedicineAnimalsImmunology and AllergyCytotoxic T cellViremiaAntigen-presenting cellT lymphocyteAdoptive TransferListeria monocytogenesVirologyDisease Models AnimalInfectious Diseasesmedicine.anatomical_structureCytomegalovirus InfectionsImmunologyFemaleCD8The Journal of Infectious Diseases
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In vivo impact of cytomegalovirus evasion of CD8 T-cell immunity: Facts and thoughts based on murine models

2010

Cytomegaloviruses (CMVs) co-exist with their respective host species and have evolved to avoid their elimination by the hosts' immune effector mechanisms and to persist in a non-replicative state, known as viral latency. There is evidence to suggest that latency is nevertheless a highly dynamic condition during which episodes of viral gene desilencing, which can be viewed as incomplete reactivations, cause intermittent antigenic activity that stimulates CD8 memory-effector T cells and drives their clonal expansion. These T cells are supposed to terminate reactivation before completion of the productive viral cycle. In this view, CMVs do not "evade" their respective host's immune response bu…

MuromegalovirusCancer ResearchT cellAntigen presentationReceptors Antigen T-CellCytomegalovirusCD8-Positive T-LymphocytesBiologyMiceImmune systemAntigenVirologyVirus latencymedicineAntigenic variationAnimalsCytotoxic T cellViral InterferenceImmune EvasionAntigen PresentationHistocompatibility Antigens Class IHerpesviridae Infectionsmedicine.diseaseVirologyVirus LatencyDisease Models AnimalInfectious Diseasesmedicine.anatomical_structureCytomegalovirus InfectionsImmunologyVirus ActivationVirus Research
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Excessive CpG 1668 stimulation triggers IL-10 production by cDC that inhibits IFN-alpha responses by pDC.

2008

Upon stimulation with a wide range of concentrations of CpG oligodeoxynucleotide 2216 (CpG 2216), plasmacytoid DC are induced to produce type I IFN (IFN-alpha/beta). In contrast, CpG 1668 shows a bell-shaped dose-response correlation, i.e. only intermediate but not high doses of CpG 1668 induce IFN-alpha/beta. Interestingly, high-dose CpG 1668 completely inhibited IFN-alpha responses induced by CpG 2216. Experiments using supernatant of high-dose CpG-1668-treated cells indicated that secreted inhibitor(s) mediated the IFN-alpha shut-off. Among modulating cytokines, IL-10 turned out to be one important negative regulator. In line with this, supernatants of IL-10-deficient DC cultures stimula…

MuromegalovirusCpG OligodeoxynucleotideImmunologyStimulationmedicine.disease_causeNegative regulatorAutoimmunityMiceAdjuvants ImmunologicmedicineImmunology and AllergyAnimalsCells CulturedbiologyTLR9Interferon-alphaDendritic Cellsbiology.organism_classificationMolecular biologyInterleukin-10Interleukin 10CpG siteOligodeoxyribonucleotidesVesicular stomatitis virusToll-Like Receptor 9ImmunologyCytokinesEuropean journal of immunology
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