Search results for "muscle biopsy"

showing 10 items of 42 documents

Autosomal recessive micrencephaly with simplified gyral pattern, abnormal myelination and arthrogryposis.

1999

The clinical courses, neuroimaging and muscle biopsy findings of two infants born to an inbred Arab family are described. They had a syndrome of micrencephaly with simplified gyral pattern, abnormal myelin formation and arthrogryposis. Increased variation of fiber size was seen in the muscle biopsy, creatine kinase, however was normal. Large areas of muscle were replaced by adipofibrous tissue. The infants had dysmorphic features consistent with the fetal akinesia/hypokinesia sequence. The abnormalities were suggestive of microlissencephaly probably associated with a dysgenetic process in the muscles. The syndrome showed an autosomal recessive inheritance.

Pathologymedicine.medical_specialtyMicrocephalyLissencephalyChromosome DisordersGenes RecessiveCentral nervous system diseaseConsanguinityHypokinesiaBiopsymedicineHumansMuscle SkeletalMyelin SheathArthrogryposisArthrogryposisChromosome AberrationsMuscle biopsymedicine.diagnostic_testbusiness.industryInfant NewbornBrainInfantGeneral MedicineAnatomySyndromemedicine.diseaseMagnetic Resonance ImagingMicrencephalyPedigreeSpinal CordPediatrics Perinatology and Child HealthMicrocephalyFemaleNeurology (clinical)medicine.symptombusinessFollow-Up StudiesNeuropediatrics
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Branching enzyme deficiency/glycogenosis storage disease type IV presenting as a severe congenital hypotonia: muscle biopsy and autopsy findings, bio…

2010

The fatal infantile neuromuscular presentation of branching enzyme deficiency (glycogen storage disease type IV) due to mutations in the gene encoding the glycogen branching enzyme, is a rare but probably underdiagnosed cause of congenital hypotonia. We report an infant girl with severe generalized hypotonia, born at 33 weeks gestation who required ventilatory assistance since birth. She had bilateral ptosis, mild knee and foot contractures and echocardiographic evidence of cardiomyopathy. A muscle biopsy at 1 month of age showed typical polyglucosan storage. The autopsy at 3.5 months of age showed frontal cortex polymicrogyria and polyglucosan bodies in neurons of basal ganglia, thalamus, …

Pathologymedicine.medical_specialtyMuscle HypotoniaCardiomyopathyAutopsyGlycogen Storage Disease Type IVFatal Outcome14-alpha-Glucan Branching EnzymemedicineGlycogen branching enzymePolymicrogyriaHumansGlycogen storage disease type IVMuscle SkeletalGenetics (clinical)Muscle biopsymedicine.diagnostic_testbiologyInfant NewbornBrainInfantmedicine.diseaseNeurologyPediatrics Perinatology and Child Healthbiology.proteinMuscle HypotoniaFemaleNeurology (clinical)Differential diagnosisInfant PrematureNeuromuscular disorders : NMD
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Muscle degeneration in neuramindase 1 deficient mice results from infiltration of the muscle fibers by expanded connective tissue

2010

AbstractNeuraminidase 1 (NEU1) regulates the catabolism of sialoglycoconjugates in lysosomes. Congenital NEU1 deficiency in children is the basis of sialidosis, a severe neurosomatic disorder in which patients experience a broad spectrum of clinical manifestations varying in the age of onset and severity. Osteoskeletal deformities and muscle hypotonia have been described in patients with sialidosis. Here we present the first comprehensive analysis of the skeletal muscle pathology associated with loss of Neu1 function in mice. In this animal model, skeletal muscles showed an expansion of the epimysial and perimysial spaces, associated with proliferation of fibroblast-like cells and abnormal …

Pathologymedicine.medical_specialtyMuscle HypotoniaMuscle Fibers SkeletalNeuraminidaseConnective tissueApoptosisNEU1BiologyArticleMiceNecrosisNEU1SarcolemmaCell MovementSettore BIO/10 - BiochimicamedicineAnimalsSialidosisMuscular dystrophyMyopathyMolecular BiologySialidosiMetalloproteinaseCell ProliferationMice KnockoutMuscle biopsySialidosisECMmedicine.diagnostic_testSkeletal muscleFibroblastsMuscular Dystrophy Animalmedicine.diseaseLysosomeExtracellular MatrixMuscular Atrophymedicine.anatomical_structureConnective TissueImmunologyMolecular MedicineMuscle biopsymedicine.symptom
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Myopathology of non-infectious inflammatory myopathies - the current status.

2007

Besides the classical inflammatory myopathies (IM), dermatomyositis (DM), polymyositis, and inclusion body myositis, the much larger spectrum of IM includes focal and nodular myositis, granulomatous myositis, macrophagic myofasciitis, graft vs. host myositis, eosinophilic myositis, and other immune-associated conditions, some of them only recently described. In addition, paraneoplastic, statin-induced and critical illness myopathies have been considered immune-associated IM. Infectious, i.e., bacterial, viral, and parasitic IM are much less frequent in the northern hemisphere. In IM, muscle biopsy is an essential diagnostic procedure to initiate therapy. The myopathological spectrum encompa…

Pathologymedicine.medical_specialtyMuscle biopsymedicine.diagnostic_testMyositisbusiness.industryMacrophagic myofasciitisCell BiologyDermatomyositismedicine.diseasePolymyositisImmunohistochemistryPathology and Forensic MedicineAtrophyEosinophilicImmunologymedicineHumansInclusion body myositisbusinessMyositisPathology, research and practice
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G.P.192

2014

After uneventful pregnancies, two newborn siblings, a girl and a boy – another sibling was stillborn – developed inspiratory stridor, hypertrophy of the right cardiac ventricle, reduction in spontaneous movements and mildly elevated creatine kinase. Muscle biopsies at ages of three months and seven weeks were performed and respectively, revealed a 'prepathological' pattern of infantile neurogenic atrophy suggesting spinal muscular atrophy (SMA). However, molecular analyses of SMN (SMA) and IGHMBP2 (SMARD1) genes did not disclose any mutations. Further histochemical staining of the skeletal muscle and heart demonstrated almost complete absence of cytochrome c oxidase while SDH was preserved.…

Pathologymedicine.medical_specialtyMuscle biopsymedicine.diagnostic_testSkeletal muscleSpinal muscular atrophyBiologySpinal cordmedicine.diseaseSMA*Muscle hypertrophymedicine.anatomical_structureNeurologyPediatrics Perinatology and Child Healthmedicinebiology.proteinCytochrome c oxidaseCreatine kinaseNeurology (clinical)Genetics (clinical)Neuromuscular Disorders
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Camptocormia associated with focal myositis in multiple-system atrophy

2005

Camptocormia (CC) or pronounced forward flexion of the trunk is a common symptom of Parkinson's disease. We describe 2 patients with probable, respectively possible multiple-system atrophy and CC. Magnetic resonance imaging of the erector trunci showed focal patchy hyperintensities with gadolinium enhancement and muscle biopsy was indicative of variably pronounced focal myositis. CC was progressive and the major handicap for both patients after 1 and 1.5 years of follow-up, respectively. The therapeutic response was poor. Similarities with the dropped-head syndrome suggest that the muscle pathology may be either the primary cause of CC, a focal reaction to the CC posture, or a coincident sy…

Pathologymedicine.medical_specialtyMuscle biopsymedicine.diagnostic_testbusiness.industryMagnetic resonance imagingmedicine.diseaseTrunkHyperintensityCamptocormiaAtrophyNeurologyBiopsymedicineNeurology (clinical)businessMyositisMovement Disorders
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Capillaries within human skeletal muscle fibers.

1991

Internalized capillaries, i.e. capillaries within muscle fibers, represent a rare myopathological feature. This was systematically studied in 923 muscle biopsy specimens and found in 24, chiefly in the gastrocnemius muscle, more rarely in the biceps and quadriceps muscles affecting males more often than females and most frequently associated with juvenile spinal muscular atrophy or Becker's muscular dystrophy. Internalized capillaries, often multiple, ran along the long axis of the muscle fiber within an "internalized" extracellular space and were almost exclusively seen in type I myofibers. Internalization seems to start at the site of fiber splitting while penetration through the intact s…

Pathologymedicine.medical_specialtymedia_common.quotation_subjectBiopsyeducationBiologyBicepsPathology and Forensic MedicineMuscle hypertrophyGastrocnemius muscleAtrophymedicineHumansMuscular dystrophyInternalizationmedia_commonMuscle biopsySarcolemmamedicine.diagnostic_testHistocytochemistryMusclesCell BiologyAnatomyHypertrophymedicine.diseaseImmunohistochemistryCapillariesMicroscopy ElectronAtrophyPathology, research and practice
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Methods for a prompt and reliable laboratory diagnosis of Pompe disease : report from an international consensus meeting

2008

Pompe disease is an autosomal recessive disorder of glycogen metabolism caused by a deficiency of the lysosomal enzyme acid alpha-glucosidase (GAA). It presents at any age, with variable rates of progression ranging from a rapidly progressive course, often fatal by one-year of age, to a more slowly, but nevertheless relentlessly progressive course, resulting in significant morbidity and premature mortality. In infants, early initiation of enzyme replacement therapy is needed to gain the maximum therapeutic benefit, underscoring the need for early diagnosis. Several new methods for measuring GAA activity have been developed. The Pompe Disease Diagnostic Working Group met to review data gener…

Pediatricsmedicine.medical_specialtyEndocrinology Diabetes and MetabolismDiseaseBiochemistryEarly initiationchemistry.chemical_compoundEndocrinologyInternal medicineGlycogen storage disease type IIGeneticsmedicineHumansMolecular BiologyAcarboseMuscle biopsyGlycogenmedicine.diagnostic_testClinical Laboratory TechniquesGlycogen Storage Disease Type IIbusiness.industryInfantEnzyme replacement therapymedicine.diseasePompe disease; laboratory diagnosisEndocrinologychemistryAcid alpha-glucosidaseGlucan 14-alpha-Glucosidasebusinessmedicine.drug
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P.10.18 Common data elements for muscle biopsy reporting

2013

Physicians commonly utilize the muscle biopsy to assist in the diagnosis of neuromuscular diseases. However, there is no current standard for evaluating or reporting on findings, and the resulting variability can impede accurate diagnoses and limit the utility of the muscle biopsy as a tool for clinical care, research, and stratifying patients for clinical trials. The National Institutes of Neurological Disorders and Stroke (NINDS) recently launched a Common Data Element (CDE) in an effort to standardize neuromuscular data collected in clinical reports. For this study, the authors adapted the NINDS Muscle Biopsy CDE to generate a form for prospective muscle biopsy reporting (CDE-R). This fo…

medicine.medical_specialtyMuscle biopsymedicine.diagnostic_testbusiness.industryConcordanceDiseaseMuscle disorderChecklistClinical trialNeurologyPediatrics Perinatology and Child HealthmedicinePhysical therapyNeurology (clinical)Medical diagnosisIntensive care medicinebusinessPathologicalGenetics (clinical)Neuromuscular Disorders
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Muscle pathology in 57 patients with myotonic dystrophy type 2

2004

We evaluated muscle biopsies from 57 patients with genetically confirmed myotonic dystrophy type 2/proximal myotonic myopathy (DM2/PROMM). Light microscopy showed myopathic together with “denervation-like” changes in almost all biopsies obtained from four different muscles: increased fiber size variation, internal nuclei, small angulated fibers, pyknotic nuclear clumps, and predominant type 2 fiber atrophy. Quantitative morphometry in 18 biopsies that were immunostained for myosin heavy chain confirmed a predominance of nonselective type 2 fiber atrophy. These histological changes were similar in all patients regardless of the site of biopsy, the predominant clinical symptoms and signs, and…

medicine.medical_specialtyPathologyMuscle biopsymedicine.diagnostic_testPhysiologybusiness.industryAnatomical pathologymedicine.diseaseMyotoniaMyotonic dystrophyProximal myotonic myopathyCellular and Molecular NeuroscienceAtrophyPhysiology (medical)BiopsymedicineHistopathologyNeurology (clinical)businessMuscle & Nerve
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