Search results for "mutation."

showing 10 items of 2808 documents

Cutaneous mosaic syndromes associated with early postzygotic activating BRAF mutations

2017

IF 3.528; International audience

BRAF V600EBRAF G596[SDV.MHEP.DERM] Life Sciences [q-bio]/Human health and pathology/DermatologyBRAF K601N[ SDV.MHEP.DERM ] Life Sciences [q-bio]/Human health and pathology/Dermatology[SDV.MHEP.DERM]Life Sciences [q-bio]/Human health and pathology/DermatologyPostzygotic BRAF mutations
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A non common BRAF mutation c1799-1801 delTGA identified in sporadic colon rectal cancer of sicilian patients

2010

Anthracycline has been shown to induce heart failure. To monitor this toxic damage, echocardiographic parameters of left ventricular (LV) systolic function are usually used. Aim of this study was to evaluate in lymphoma’s patients the reliability of echocardiographic data in comparison with a LV systo-diastolic parameter function: the Tei index.

BRAF mutation sicilian patients
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BRAFV600E MUTATION, TISSUE INHIBITOR OF METALLOPROTEINASE-1 UPREGULATION AND NF-KB ACTIVATION: CLOSING THE LOOP ON THE PAPILLARY THYROID CANCER TRILO…

2011

BRAFV600E is the most common mutation in papillary thyroid carcinoma (PTC). Tissue inhibitor of metalloproteinases (TIMP-1) and Nuclear Factor (NF)-kB have been shown to play an important role in thyroid cancer. Our aim was to evaluate whether an interplay among these three factors exerts a functional role in PTCs. 56 PTC specimens were analyzed for BRAFV600E mutation, TIMP-1 expression and NF-kB activation by real-time allele-specific amplification, realtime quantitative PCR (qRT-PCR) and electroforetic mobility shift assay (EMSA), respectively. We show that BRAFV600E mutation occurs selectively in PTC nodules and determines up-regulation of TIMP-1 and hyperactivation of NF-kB. In addition…

BRAFV600EMETALLOPROTEINASE-1NF-KBTHYROID CANCERMUTATIONSettore MED/13 - Endocrinologia
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BRCA1/2 variants of unknown significance in hereditary breast and ovarian cancer (HBOC) syndrome: Looking for the hidden meaning

2021

Hereditary breast and ovarian cancer syndrome is caused by germline mutations in BRCA1/2 genes. These genes are very large and their mutations are heterogeneous and scattered throughout the coding sequence. In addition to the above-mentioned mutations, variants of uncertain/unknown significance (VUSs) have been identified in BRCA genes, which make more difficult the clinical management of the patient and risk assessment. In the last decades, several laboratories have developed different databases that contain more than 2000 variants for the two genes and integrated strategies which include multifactorial prediction models based on direct and indirect genetic evidence, to classify the VUSs a…

BRCA2 ProteinOvarian NeoplasmsBRCA1 ProteinBreast NeoplasmsHematologyBRCA1Multifactorial prediction modelBRCA2Risk AssessmentVariants of Uncertain SignificanceVUSOncologyMutationHereditary Breast and Ovarian Cancer SyndromeHumansFemaleGenetic Predisposition to DiseaseGerm-Line MutationCritical Reviews in Oncology/Hematology
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Pathogenic correlation between mosaic variegated aneuploidy 1 (MVA1) and a novel BUB1B variant: a reappraisal of a severe syndrome.

2022

Funder: Università degli Studi di Catania

BUB1B gene Epileptic seizure Microcephaly Mosaic variegated aneuploidy 1 (MVA1) syndrome Ovary cystMosaicismCell Cycle ProteinsOvary cystDermatologyGeneral MedicineSyndromeBUB1B geneProtein Serine-Threonine KinasesAneuploidyPsychiatry and Mental healthSeizuresMosaic variegated aneuploidy 1 (MVA1) syndromeMutationEpileptic seizureMicrocephalyHumansNeurology (clinical)Neurological sciences : official journal of the Italian Neurological Society and of the Italian Society of Clinical Neurophysiology
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The Agr communication system provides a benefit to the populations of Listeria monocytogenes in soil

2014

International audience; In this study, we investigated whether the Agr communication system of the pathogenic bacterium Listeria monocytogenes was involved in adaptation and competitiveness in soil. Alteration of the ability to communicate, either by deletion of the gene coding the response regulator AgrA (response-negative mutant) or the signal pro-peptide AgrD (signal-negative mutant), did not affect population dynamics in soil that had been sterilized but survival was altered in biotic soil suggesting that the Agr system of L. monocytogenes was involved to face the complex soil biotic environment. This was confirmed by a set of co-incubation experiments. The fitness of the response-negat…

Bacillus-subtilisMutantlcsh:QR1-502Genetic Fitnessmicrobial ecologymedicine.disease_causelcsh:MicrobiologyQuorum-sensing systemsOriginal Research ArticlePseudomonas-aeruginosaSoil Microbiology2. Zero hunger0303 health sciencesMutationeducation.field_of_studycompetitivenessMicrobiology and Parasitologycell communicationMicrobiologie et ParasitologiefitnessAgricultural sciences[SDV.MP]Life Sciences [q-bio]/Microbiology and ParasitologyInfectious DiseasesSoil microbiologyMicrobiology (medical)PopulationImmunologyLactobacillus-plantarum[SDV.SA.SDS]Life Sciences [q-bio]/Agricultural sciences/Soil studyBiologyMicrobiologyMicrobiologysoil03 medical and health sciences[ SDV.SA.AGRO ] Life Sciences [q-bio]/Agricultural sciences/AgronomyBacterial ProteinsListeria monocytogenesmedicineAgr system;cell communication;competitiveness;fitness;Listeria monocytogenes;soil;biotic interaction;quorum-sensing systems;expression;farm environment;dairy farm;bacterial-populations;pseudomonas-aeruginosa;microbial world;lactobacillus-plantarum;staphylococcus-aureus;bacillus-subtilisStaphylococcus-aureuseducationGene030304 developmental biology[ SDV ] Life Sciences [q-bio]Bacterial-populations030306 microbiologybiotic interactionFarm environmentListeria monocytogenesResponse regulatorMutationDairy farmGenetic Fitnessmicrobial worldSciences agricolesAgr system
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Electrophysiological evidence for heptameric stoichiometry of ion channels formed by Staphylococcus aureus alpha-toxin in planar lipid bilayers.

2000

Staphylococcal alpha-toxin forms homo-oligomeric channels in lipid bilayers and cell membranes. Here, we report that electrophysiological monitoring of single-channel function using a derivatized cysteine substitution mutant allows accurate determination of the subunit stoichiometry of the oligomer in situ. The electrophysiological phenotype of channels formed in planar lipid bilayers with the cysteine replacement mutant I7C is equal to that of the wild type. When pores were formed with I7C, alterations of several channel properties were observed upon modification with SH reagents. Decreases in conductance then occurred that were seen only as negative voltage was applied. At the level of si…

Bacterial ToxinsLipid BilayersWild typeConductanceBiologyMicrobiologyOligomerIon ChannelsElectrophysiologychemistry.chemical_compoundHemolysin ProteinsStructure-Activity RelationshipMembranechemistryBiochemistryMutationBiophysicsCysteineLipid bilayerMolecular BiologyIon channelStaphylococcus aureus alpha toxinCysteineMolecular microbiology
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Multiply Transitive Permutation Groups

1982

Since the beginnings of finite group theory, the multiply transitive permutation groups have exercised a certain fascination. This is mainly due to the fact that apart from the symmetric and alternating groups not many of them were known. Only very recently final results about multiply transitive permutation groups have been proved, using the classification of all finite simple groups (see 7.5).

Base (group theory)CombinatoricsTransitive relationFinite group theoryPermutation graphClassification of finite simple groupsPermutation groupCyclic permutationMathematics
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On finite products of totally permutable groups

1996

In this paper the structure of finite groups which are the product of two totally permutable subgroups is studied. In fact we can obtain the -residual, where is a formation, -projectors and -normalisers, where is a saturated formation, of the group from the corresponding subgroups of the factor subgroups.

Base (group theory)Pure mathematicsGroup (mathematics)Symmetric groupGeneral MathematicsProduct (mathematics)Structure (category theory)Permutable primeCyclic permutationMathematicsBulletin of the Australian Mathematical Society
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Beckwith–Wiedemann syndrome: multiple molecular mechanisms

2006

Beckwith–Wiedemann syndrome (BWS) is a congenital overgrowth condition with an increased risk of developing embryonic tumours, such as Wilms' tumour. The cardinal features are abdominal wall defects, macroglossia and gigantism. BWS is generally sporadic; only 10–15% of cases are familial. A variety of molecular aberrations have been associated with BWS. The only mutations within a gene are loss-of-function mutations in the CDKN1C gene, which codes for an imprinted cell-cycle regulator. CDKN1C mutations appear to be particularly associated with umbilical abnormalities, but not with increased predisposition to Wilms' tumour. In the remaining BWS subgroups, a disturbance of the tight epigeneti…

Beckwith-Wiedemann SyndromeGenotypeTranscription GeneticBeckwith–Wiedemann syndromeBioinformaticsModels BiologicalEpigenesis GeneticGenomic ImprintingGenotypeMacroglossiaAnimalsHumansMedicineEpigeneticsCyclin-Dependent Kinase Inhibitor p57Molecular BiologyModels Geneticbusiness.industryDNA Methylationmedicine.diseasePhenotypeGigantismPhenotypeMutationDNA methylationMolecular Medicinemedicine.symptombusinessGenomic imprintingExpert Reviews in Molecular Medicine
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