Search results for "nadph oxidases"

showing 10 items of 78 documents

Dynamic changes in the subcellular distribution of the tobacco ROS-producing enzyme RBOHD in response to the oomycete elicitor cryptogein.

2014

Highlight text The oomycete elicitor cryptogein triggers the relocation of RBOHD from intracellular compartments to the plasma membrane in tobacco cells. This suggests that intracellular trafficking is a potential determinant of RBOHD activity.

DETERGENT-RESISTANT MEMBRANESPhysiologyNicotiana tabacum[SDV]Life Sciences [q-bio]BY-2 cellsPlant SciencecryptogeinCell membranechemistry.chemical_compoundAPOPLASTIC OXIDATIVE BURSTCELL-SURFACEDISEASE RESISTANCE[MATH]Mathematics [math]Plant Proteinsreactive oxygen speciesFungal proteinNADPH oxidaseMicroscopy Confocalbiologyfood and beveragesElicitorCell biologymedicine.anatomical_structureBiochemistryprotein trafficking.[SDE]Environmental SciencessymbolsNADPH OXIDASE RBOHDprotein traffickingResearch PaperPhytophthoraCycloheximiderespiratory burst oxidase homolog D (RBOHD)Real-Time Polymerase Chain ReactionFungal Proteinssymbols.namesakeNICOTIANA-BENTHAMIANAMicroscopy Electron TransmissionTobaccomedicine[SDV.BV]Life Sciences [q-bio]/Vegetal Biology[INFO]Computer Science [cs]NITRIC-OXIDENicotiana tabacumCell MembraneNADPH OxidasesGolgi apparatusbiology.organism_classificationSubcellular localizationLIPID RAFTSchemistryPLASMA-MEMBRANEbiology.proteinPLANT DEFENSE
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Mitochondrial redox signaling: Interaction of mitochondrial reactive oxygen species with other sources of oxidative stress.

2014

Significance: Oxidative stress is a well established hallmark of cardiovascular disease and there is strong evidence for a causal role of reactive oxygen and nitrogen species (RONS) therein. Recent Advances: Improvement of cardiovascular complications by genetic deletion of RONS producing enzymes and overexpression of RONS degrading enzymes proved the involvement of these species in cardiovascular disease at a molecular level. Vice versa, overexpression of RONS producing enzymes as well as deletion of antioxidant enzymes was demonstrated to aggravate cardiovascular complications. Critical Issues: With the present overview we present and discuss different pathways how mitochondrial RONS inte…

Heart DiseasesPhysiologyClinical BiochemistryMitochondrionmedicine.disease_causeBiochemistrychemistry.chemical_compoundmedicineAnimalsHumansXanthine oxidaseMolecular BiologyGeneral Environmental Sciencechemistry.chemical_classificationInflammationReactive oxygen speciesNADPH oxidasebiologyNADPH OxidasesCell BiologyForum Review ArticlesAngiotensin IICell biologyMitochondriaNitric oxide synthaseCrosstalk (biology)Oxidative StressBiochemistrychemistrybiology.proteinGeneral Earth and Planetary SciencesReactive Oxygen SpeciesOxidation-ReductionOxidative stressSignal TransductionAntioxidantsredox signaling
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Flow cytometric investigation of neutrophil oxidative burst and apoptosis in physiological and pathological situations

2009

Flow cytometric analysis provides a rapid screen for abnormalities of polymorphonuclear neutrophils (PMN) function and reflect their behavior in vivo more accurately. This review summarizes the major fluorescent probes used to study PMN oxidative burst and apoptosis using flow cytometry (FCM). We also provide examples of FCM studies in physiological and pathological situations, illustrating the advantages of FCM for assessment of PMN oxidative burst and PMN apoptosis. These data point to the role of FCM in detecting primary immunodeficiencies such as IRAK4 deficiency and support the use of the assessment of the PMN oxidative burst for routine testing in patients with bacterial infections. W…

LipopolysaccharidesHistologyNeutrophilsSimian Acquired Immunodeficiency SyndromeApoptosisBiologyPathology and Forensic MedicineFlow cytometryAdjuvants ImmunologicIn vivomedicineAnimalsHumansReceptorNeutrophil oxidative burstPathologicalRespiratory Burstmedicine.diagnostic_testToll-Like ReceptorsImidazolesNADPH OxidasesCell BiologyFlow CytometryPhenotypeRespiratory burstInterleukin-1 Receptor-Associated KinasesOligodeoxyribonucleotidesApoptosisImmunologyReactive Oxygen SpeciesCytometry Part A
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Gp91phox-containing NAD(P)H oxidase increases superoxide formation by doxorubicin and NADPH

2006

Doxorubicin is a highly effective antineoplastic drug associated with a dose-dependent cardiotoxicity that may result in irreversible cardiomyopathy and heart failure. Gene variants of the superoxide-generating enzyme NAD(P)H oxidase have recently been associated with this phenotype. We investigated the mechanism of this association using lucigenin-enhanced chemiluminescence, spectrophotometry, electrochemical sensor, and electron paramagnetic resonance spectroscopy. Superoxide production was measured in female wild-type and NAD(P)H oxidase-deficient (gp91phox knockout) mice. The magnitude of the increase in superoxide production on the addition of doxorubicin was much higher in hearts of w…

LuminescenceGene ExpressionAntineoplastic AgentsPolymerase Chain ReactionBiochemistryMicechemistry.chemical_compoundSuperoxidesPhysiology (medical)medicineAnimalsDoxorubicinNADPH-Ferrihemoprotein ReductaseMice Knockoutchemistry.chemical_classificationCardiotoxicityOxidase testMembrane GlycoproteinsDose-Response Relationship DrugSuperoxideMyocardiumNADPH OxidasesMolecular biologyMice Inbred C57BLEnzymechemistryBiochemistryDoxorubicinNAD(P)H oxidaseNADPH Oxidase 2Knockout mouseNAD+ kinaseNADPmedicine.drugFree Radical Biology and Medicine
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Impact of Donor Human Milk in the Preterm Very Low Birth Weight Gut Transcriptome Profile by Use of Exfoliated Intestinal Cells

2019

[Background] Own mother’s milk (OMM) is the optimal nutrition for preterm infants. However, pasteurized donor human milk (DHM) is a valid alternative. We explored the differences of the transcriptome in exfoliated epithelial intestinal cells (EEIC) of preterm infants receiving full feed with OMM or DHM.

Male0301 basic medicineDonor milkGene Expressionintestinal cellsmedicine.disease_causeTranscriptome0302 clinical medicinemother’s milkGene expressionInfant Very Low Birth Weightoxidative stressgeneticsProspective StudiesIntestinal Mucosa2. Zero hungerPrincipal Component AnalysisNutrition and DieteticsCaseinsIntestinal cells3. Good healthdonor milkGestationFemalemedicine.symptomPrematuritylcsh:Nutrition. Foods and food supplyInfant PrematureGestational Agelcsh:TX341-641InflammationBiologyArticleAndrology03 medical and health sciences030225 pediatricsMother’s milkGeneticsmedicineHumansGeneInflammationMilk HumanprematurityInfant NewbornNADPH OxidasesEpithelial CellsNeutrophil cytosolic factor 1Low birth weight030104 developmental biologyMilk BanksOxidative stressinflammationCyclooxygenase 1LactalbuminTranscriptomeOxidative stressFood ScienceNutrients
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CD40L controls obesity-associated vascular inflammation, oxidative stress, and endothelial dysfunction in high fat diet-treated and db/db mice

2018

Abstract Aims CD40 ligand (CD40L) signaling controls vascular oxidative stress and related dysfunction in angiotensin-II-induced arterial hypertension by regulating vascular immune cell recruitment and platelet activation. Here we investigated the role of CD40L in experimental hyperlipidemia. Methods and results Male wild type and CD40L−/− mice (C57BL/6 background) were subjected to high fat diet for sixteen weeks. Weight, cholesterol, HDL, and LDL levels, endothelial function (isometric tension recording), oxidative stress (NADPH oxidase expression, dihydroethidium fluorescence) and inflammatory parameters (inducible nitric oxide synthase, interleukin-6 expression) were assessed. CD40L exp…

Male0301 basic medicinePhysiologyAnti-Inflammatory AgentsNitric Oxide Synthase Type II030204 cardiovascular system & hematologyWeight Gainmedicine.disease_causeAntioxidantschemistry.chemical_compound0302 clinical medicineHyperlipidemiaEndothelial dysfunctionMice KnockoutbiologyLeptinLipidsVasodilationNitric oxide synthaseInflammation Mediatorsmedicine.symptomCardiology and Cardiovascular Medicinemedicine.medical_specialtyCD40 LigandHyperlipidemiasInflammationDiet High-Fat03 medical and health sciencesPhysiology (medical)Internal medicinemedicineAnimalsHumansObesityPlatelet activationInflammationTNF Receptor-Associated Factor 6Interleukin-6Cholesterolbusiness.industryMyocardiumNADPH OxidasesPlatelet Activationmedicine.diseaseMice Inbred C57BLDisease Models AnimalOxidative Stress030104 developmental biologyEndocrinologyDiabetes Mellitus Type 2chemistrybiology.proteinEndothelium VascularbusinessBiomarkersOxidative stressCardiovascular Research
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Roles of sedentary aging and lifelong physical activity in exchange of glutathione across exercising human skeletal muscle.

2014

Reactive oxygen species (ROS) are important signaling molecules with regulatory functions, and in young and adult organisms, the formation of ROS is increased during skeletal muscle contractions. However, ROS can be deleterious to cells when not sufficiently counterbalanced by the antioxidant system. Aging is associated with accumulation of oxidative damage to lipids, DNA, and proteins. Given the pro-oxidant effect of skeletal muscle contractions, this effect of age could be a result of excessive ROS formation. We evaluated the effect of acute exercise on changes in blood redox state across the leg of young (23±1 years) and older (66±2 years) sedentary humans by measuring the whole blood co…

MaleAgingAntioxidantmedicine.medical_treatmentSkeletal muscleFree radicalsBiochemistryAntioxidantschemistry.chemical_compoundSuperoxide Dismutase-1Glutathione Peroxidase GPX1Exercise/physiologyGlutathione Peroxidase/biosynthesisWhole bloodchemistry.chemical_classificationNADPH oxidasebiologyAgingraMotor Activity/physiologyMiddle AgedCatalaseGlutathionemedicine.anatomical_structureNADPH Oxidases/biosynthesisOxidation-ReductionMuscle Contraction/physiologyMuscle ContractionAdultmedicine.medical_specialtyCell signalingCatalase/biosynthesisGlutathione/bloodSuperoxide Dismutase/biosynthesisPhosphoproteins/biosynthesisMotor ActivityYoung AdultReactive Oxygen Species/metabolismPhysiology (medical)Internal medicinemedicineHumansMuscle SkeletalExerciseAgedLeg/physiologyReactive oxygen speciesGlutathione PeroxidaseLegAntioxidants/analysisSuperoxide DismutaseSkeletal muscleNADPH OxidasesGlutathionePhosphoproteinsMuscle Skeletal/physiologyOxidative StressEndocrinologyEnzymechemistrybiology.proteinLipid PeroxidationSedentary BehaviorReactive oxygen speciesReactive Oxygen SpeciesFree radical biologymedicine
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Anti-hypertensive effects of Rosuvastatin are associated with decreased inflammation and oxidative stress markers in hypertensive rats

2009

International audience; Among their pleiotropic effects, statins exert antioxidant and anti-inflammatory properties. The aim of this study was to evaluate in normotensive (WKY) and in spontaneously hypertensive rats (SHR) the effect of rosuvastatin (ROSU) treatment on (1) plasma inflammation markers and endogenous NO synthase inhibitor (ADMA) levels, (2) reactive oxygen species (ROS) generated by circulating leukocytes and (3) vascular oxidative stress and tissue inflammation markers. Plasma cytokines were higher in SHR than in WKY, except for IL-4, which was lower in SHR than in WKY. SHR monocytes exhibited higher production of ROS than did WKY monocytes. In the experimental conditions, RO…

MaleAntioxidantmedicine.medical_treatmentBlood Pressure030204 cardiovascular system & hematologymedicine.disease_causeBiochemistryRats Inbred WKYchemistry.chemical_compound0302 clinical medicineRats Inbred SHR[SDV.IDA]Life Sciences [q-bio]/Food engineeringRosuvastatin CalciumComputingMilieux_MISCELLANEOUSchemistry.chemical_classification0303 health sciencesSulfonamidesGeneral Medicine3. Good healthNAD(P)H oxidasecardiovascular systemmedicine.symptommedicine.drugmedicine.medical_specialtyhypertensionleukocytesInflammationArgininestatins03 medical and health sciences[SDV.MHEP.CSC]Life Sciences [q-bio]/Human health and pathology/Cardiology and cardiovascular systemInternal medicinemedicineAnimalsRosuvastatin[SPI.GPROC]Engineering Sciences [physics]/Chemical and Process Engineeringcardiovascular diseasesAntihypertensive Agents030304 developmental biologyInflammationReactive oxygen speciesCholesterolNAD(P)H oxidaseNADPH OxidasescytokinesRatsFluorobenzenesOxidative StressEndocrinologyBlood pressurePyrimidineschemistryInterleukin-4Hydroxymethylglutaryl-CoA Reductase InhibitorsReactive Oxygen SpeciesOxidative stress
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Expression of NO synthases and redox enzymes in umbilical arteries from newborns born small, appropriate, and large for gestational age.

2012

Modified expression of nitric oxide synthases (NOSs) and an imbalance between the pro-oxidative and the antioxidative system accompany endothelial dysfunction, the first stage of atherosclerosis. Humans born small (SGA) or large (LGA) for gestational age are at higher risk of developing atherosclerosis later in life than humans born appropriate for gestational age (AGA). We hypothesized that indicators of endothelial dysfunction could be detectable at birth. The purpose of this study was to find out whether the expression patterns of NO synthases (endothelial NOS (eNOS), inducible NOS (iNOS), and neuronal NOS (nNOS)), pro-oxidative enzymes (components of nicotinamide adenine dinucleotide ph…

MaleBlotting WesternGestational AgeBiologyReal-Time Polymerase Chain ReactionIsozymeGene Expression Regulation EnzymologicUmbilical ArteriesAndrologyRedox enzymesGlutathione Peroxidase GPX1No synthasemedicineBirth WeightHumansRNA MessengerAnalysis of VarianceGlutathione PeroxidaseSuperoxide DismutaseInfant NewbornGestational ageGene Expression Regulation DevelopmentalNADPH OxidasesOxidation reductionInfant Low Birth Weightmedicine.diseaseCatalaseEnzymesIsoenzymesLow birth weightPediatrics Perinatology and Child HealthImmunologyInfant Small for Gestational AgeSmall for gestational ageFemalemedicine.symptomNitric Oxide SynthaseOxidation-ReductionPediatric research
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Betulinic acid protects against cerebral ischemia–reperfusion injury in mice by reducing oxidative and nitrosative stress

2011

Increased production of reactive oxygen and nitrogen species following cerebral ischemia-reperfusion is a major cause for neuronal injury. In hypercholesterolemic apolipoprotein E knockout (ApoE-KO) mice, 2h of middle cerebral artery (MCA) occlusion followed by 22h of reperfusion led to an enhanced expression of NADPH oxidase subunits (NOX2, NOX4 and p22phox) and isoforms of nitric oxide synthase (neuronal nNOS and inducible iNOS) in the ischemic hemisphere compared with the non-ischemic contralateral hemisphere. This was associated with elevated levels of 3-nitrotyrosine, an indicator of peroxynitrite-mediated oxidative protein modification. Pre-treatment with betulinic acid (50mg/kg/day f…

MaleCancer ResearchPhysiologyClinical BiochemistryIschemiaPharmacologymedicine.disease_causeBiochemistryBrain IschemiaMicechemistry.chemical_compoundStress PhysiologicalEnosBetulinic acidmedicineAnimalsRNA MessengerBetulinic AcidMice KnockoutNADPH oxidasebiologyChemistryBrainNADPH Oxidasesbiology.organism_classificationmedicine.diseaseReactive Nitrogen SpeciesTriterpenesNitric oxide synthaseOxidative StressBiochemistryReperfusion Injurycardiovascular systembiology.proteinTyrosineP22phoxNitric Oxide SynthasePentacyclic TriterpenesReperfusion injuryOxidative stressNitric Oxide
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