Search results for "neurogenic inflammation"

showing 10 items of 29 documents

Facilitated neurogenic inflammation in unaffected limbs of patients with complex regional pain syndrome.

2003

Pain, edema, increased skin temperature, reddening and trophic changes characterize complex regional pain syndrome (CRPS). Recently, we have been able to show facilitated neurogenic inflammation on the affected limb. In the current study unaffected limbs were examined after resolution of the CRPS symptoms to assess possible generalized changes predisposing to CRPS. In 12 patients and in 12 healthy volunteers dermal microdialysis in combination with electrical C-fiber stimulation was employed to induce neuropeptide release. Dialysate protein concentration and axon reflex vasodilation were measured. Neither in patients nor in controls did electrical stimulation lead to protein extravasation, …

AdultMicrodialysisVasodilationStimulationEdemamedicineHumansNeurogenic inflammationbusiness.industryGeneral NeuroscienceNeuropeptidesExtremitiesMiddle Agedmedicine.diseaseExtravasationElectric StimulationVasodilationComplex regional pain syndromeAnesthesiaAxon reflexFemaleDisease Susceptibilitymedicine.symptomNeurogenic InflammationbusinessComplex Regional Pain SyndromesNeuroscience letters
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Methylprednisolone prevents nerve injury-induced hyperalgesia in neprilysin knockout mice.

2013

The pathophysiology of the complex regional pain syndrome involves enhanced neurogenic inflammation mediated by neuropeptides. Neutral endopeptidase (neprilysin, NEP) is a key enzyme in neuropeptide catabolism. Our previous work revealed that NEP knock out (ko) mice develop more severe hypersensitivity to thermal and mechanical stimuli after chronic constriction injury (CCI) of the sciatic nerve than wild-type (wt) mice. Because treatment with glucocorticoids is effective in early complex regional pain syndrome, we investigated whether methylprednisolone (MP) reduces pain and sciatic nerve neuropeptide content in NEP ko and wt mice with nerve injury. After CCI, NEP ko mice developed more se…

Agonistmedicine.medical_specialtymedicine.drug_classNeuropeptideSubstance PMethylprednisolonechemistry.chemical_compoundMiceInternal medicineMedicineAnimalsNeprilysinMice KnockoutNeurogenic inflammationbusiness.industryfungiNerve injuryMice Inbred C57BLAnesthesiology and Pain MedicineEndocrinologyNeuroprotective AgentsNeurologychemistryHyperalgesiaAnesthesiaHyperalgesiaNeprilysinNeurology (clinical)Sciatic nervemedicine.symptomSciatic NeuropathybusinessPainReferences
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Role of Sortilin in Models of Autoimmune Neuroinflammation

2015

Abstract The proneurotrophin receptor sortilin is a protein with dual functions, being involved in intracellular protein transport, as well as cellular signal transduction. The relevance of the receptor for various neuronal disorders, such as dementia, seizures, and brain injury, is well established. In contrast, little is known about the role of sortilin in immune cells and inflammatory diseases. The aim of our study was to elucidate the distribution of sortilin in different immune cell types in mice and humans and to analyze its function in autoimmune CNS inflammation. Sortilin was expressed most profoundly in murine and human macrophages and dendritic cells and to a much lesser extent in…

Central Nervous SystemCell typeEncephalomyelitis Autoimmune ExperimentalMultiple SclerosisT-LymphocytesEncephalomyelitisImmunologyAutoimmunityBiologyMiceImmune systemmedicineAnimalsHumansImmunology and AllergyReceptorNeuroinflammationMice KnockoutAutoimmune diseaseAntigen PresentationMacrophagesExperimental autoimmune encephalomyelitisDendritic Cellsmedicine.diseaseImmunity InnateMice Inbred C57BLAdaptor Proteins Vesicular TransportBrain InjuriesImmunologyNeurogenic InflammationSignal transductionSignal Transduction
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OTUB1 inhibits CNS autoimmunity by preventing IFN-γ-induced hyperactivation of astrocytes.

2019

Astrocytes are critical regulators of neuroinflammation in multiple sclerosis (MS) and its animal model experimental autoimmune encephalomyelitis (EAE). Growing evidence indicates that ubiquitination of signaling molecules is an important cell‐intrinsic mechanism governing astrocyte function during MS and EAE. Here, we identified an upregulation of the deubiquitinase OTU domain, ubiquitin aldehyde binding 1 (OTUB1) in astrocytes during MS and EAE. Mice with astrocyte‐specific OTUB1 ablation developed more severe EAE due to increased leukocyte accumulation, proinflammatory gene transcription, and demyelination in the spinal cord as compared to control mice. OTUB1‐deficient astrocytes were hy…

Central Nervous SystemEncephalomyelitis Autoimmune ExperimentalNeuroimmunomodulationmedicine.medical_treatmentexperimental autoimmune encephalomyelitisAutoimmunityBiologymultiple sclerosisubiquitinationGeneral Biochemistry Genetics and Molecular BiologyProinflammatory cytokineneuroinflammationInterferon-gammaMice03 medical and health sciences0302 clinical medicineastrocytemedicineAnimalsMolecular BiologyCells CulturedNeuroinflammation030304 developmental biologyMice Knockout0303 health sciencesGeneral Immunology and MicrobiologySuppressor of cytokine signaling 1General NeuroscienceExperimental autoimmune encephalomyelitisArticlesmedicine.disease3. Good healthCell biologyMice Inbred C57BLCysteine EndopeptidasesCytokinemedicine.anatomical_structureAnimals NewbornAstrocytesSTAT proteinOTUB1FemaleNeurogenic InflammationJanus kinase030217 neurology & neurosurgeryAstrocyte
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Understanding the Role of T Cells in CNS Homeostasis.

2015

T cells within the central nervous system (CNS) have been generally considered pathogenic, especially in the context of neuroinflammatory disease. However, recent findings have revealed varied functions for T cells in the healthy CNS, as well as more complex roles for these cells in infection and injury than previously appreciated. Here we review evidence indicating important roles for different T cell subsets in the maintenance of CNS homeostasis. We examine the contribution of T cells in limiting inflammation and damage upon CNS injury, infection, and in neurodegeneration, and discuss the current understanding of the cellular and molecular mechanisms involved. Insight into these processes…

Central Nervous SystemT cellT-LymphocytesImmunologyCentral nervous systemContext (language use)InflammationDiseaseBiologyLymphocyte Depletion03 medical and health sciences0302 clinical medicineT-Lymphocyte SubsetsmedicineImmunology and AllergyAnimalsHomeostasisHumansNeurodegenerationmedicine.diseaseCns injurymedicine.anatomical_structureImmunologymedicine.symptomNeurogenic Inflammation030217 neurology & neurosurgeryHomeostasis030215 immunologyTrends in immunology
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The assessment of gingivitis using remote photoplethysmography

2020

Gum disease caused by bacterial infection, systemic conditions or neurogenic inflammation remains a prevalent and important concern of public health in both, developing and developed countries, potentially causing tooth loss if left untreated. Therefore, a simple, cost-effective, and contactless diagnostic tool is needed in dentistry. The present study aims to develop and investigate the feasibility of a motion artefact-free, miniature imaging photoplethysmography system which is dedicated for assessment of gingivitis in the dental office. The device comprised of a sub-miniature monochromatic camera equipped with miniature lens, 4-LED ring illuminator, cross polarizers and bandpass optical …

GingivitisNeurogenic inflammationGingival tissuebusiness.industryPhotoplethysmogrammedicineGingival inflammationmedicine.symptombusinessPerfusionBiomedical engineeringBiophotonics—Riga 2020
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Heat Shock Proteins Alterations in Rheumatoid Arthritis

2022

Rheumatoid arthritis (RA) is a chronic inflammatory and autoimmune disease characterized by the attack of the immune system on the body’s healthy joint lining and degeneration of articular structures. This disease involves an increased release of inflammatory mediators in the affected joint that sensitize sensory neurons and create a positive feedback loop to further enhance their release. Among these mediators, the cytokines and neuropeptides are responsible for the crippling pain and the persistent neurogenic inflammation associated with RA. More importantly, specific proteins released either centrally or peripherally have been shown to play opposing roles in the pathogenesis of this dise…

InflammationHeat shock proteins Hsp therapy Inflammation Neurogenic inflammation Rheumatoid arthritis VaccineOrganic ChemistryGeneral MedicineChaperonin 60CatalysisComputer Science ApplicationsInorganic ChemistryArthritis RheumatoidHumansHSP70 Heat-Shock ProteinsHSP90 Heat-Shock ProteinsPhysical and Theoretical ChemistryMolecular BiologySpectroscopyHeat-Shock Proteins
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Short-Term Long Chain Omega3 Diet Protects from Neuroinflammatory Processes and Memory Impairment in Aged Mice

2011

Regular consumption of food enriched in omega3 polyunsaturated fatty acids (oméga3 PUFAs) has been shown to reduce risk of cognitive decline in elderly, and possibly development of Alzheimer's disease. Docosahexaenoic acid (DHA) and eicosapentaenoic acid (EPA) are the most likely active components of oméga3-rich PUFAs diets in the brain. We therefore hypothesized that exposing mice to a DHA and EPA enriched diet may reduce neuroinflammation and protect against memory impairment in aged mice. For this purpose, mice were exposed to a control diet throughout life and were further submitted to a diet enriched in EPA and DHA during 2 additional months. Cytokine expression together with a thoroug…

MaleAnatomy and PhysiologyMouse[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionNeuroimmunologyNutritional Disorderslcsh:MedicineHippocampusHippocampusBiochemistryMiceLearning and Memory0302 clinical medicineImmune PhysiologyCognitive declinelcsh:Science2. Zero hungerchemistry.chemical_classification0303 health sciencesMultidisciplinaryFatty AcidsBrainfood and beveragesAnimal ModelsLipidsEicosapentaenoic acidmedicine.anatomical_structureEicosapentaenoic AcidNeurologyBiochemistryDocosahexaenoic acidAlimentation et NutritionCytokinesMedicineNeurogliaFemalelipids (amino acids peptides and proteins)Neurogenic InflammationResearch ArticlePolyunsaturated fatty acidmedicine.medical_specialtyDocosahexaenoic AcidsCognitive NeuroscienceImmunologyBiology03 medical and health sciencesModel OrganismsInternal medicineFatty Acids Omega-3medicineFood and NutritionAnimalsMemory impairmentWorking MemoryBiologyNeuroinflammationNutrition030304 developmental biologyInflammationMemory Disorderslcsh:RImmunityImmunologic SubspecialtiesDietMice Inbred C57BLNeuroanatomyEndocrinologychemistryDietary SupplementsClinical Immunologylcsh:Q[SDV.AEN]Life Sciences [q-bio]/Food and Nutrition030217 neurology & neurosurgeryNeurosciencePLoS ONE
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Effect of triterpenoids on the inflammation induced by protein kinase C activators, neuronally acting irritants and other agents.

2000

In order to establish the mode of the anti-inflammatory activity of triterpenoids, 11 naturally occurring compounds were assayed on mouse ear oedema induced by the protein kinase C activators, mezerein, 12-O-tetradecanoylphorbol-13-acetate (TPA), two 12-deoxyphorbol-13-monoesters (13-tetradecanoate (DPT) and 13-phenylacetate (DPP)) and bryostatin 1, and by resiniferatoxin, xylene and arachidonic acid. The effects on bradykinin-induced paw oedema and on the rat skin inflammation caused by hydrogen peroxide were also examined. The oedema induced by mezerein and DPT was reduced to different extents by the triterpenoids administered epicutaneously (0.5 mg per ear). Against DPT-induced oedema, l…

MezereinTime FactorsBryostatin 1ResiniferatoxinAnti-Inflammatory AgentsEnzyme ActivatorsPharmacologyBradykininchemistry.chemical_compoundGlucose OxidaseMiceAnimalsEdemaBryostatinRats WistarProtein kinase AProtein kinase CProtein Kinase CSkinPharmacologyNeurogenic inflammationArachidonic AcidMolecular StructureTerpenesBiological activityEarTriterpenesRatschemistryBiochemistryIrritantsDermatitis IrritantFemaleDiterpenesNeurogenic InflammationReactive Oxygen SpeciesEuropean journal of pharmacology
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Corrigendum to “Inhibition of neutral endopeptidase (NEP) facilitates neurogenic inflammation” [Exp. Neurol. 195 (2005) 179–184]

2006

Neurogenic inflammationDevelopmental NeuroscienceNeurologyChemistryImmunologyPharmacologyNeprilysinExperimental Neurology
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