Search results for "neuron"

showing 10 items of 2611 documents

Different α2δ Accessory Subunits Regulate Distinctly Different Aspects of Calcium Channel Function in the Same Drosophila Neurons

2019

AbstractVoltage gated calcium channels (VGCCs) regulate neuronal excitability and translate activity into calcium dependent intracellular signaling. The pore forming α1subunit of high voltage activated (HVA) VGCCs operates not in isolation but associates with α2δ accessory subunits. α2δ subunits can affect calcium channel biophysical properties, surfacing, localization and transport, but theirin vivofunctions are incompletely understood. In vertebrates, it is largely unknown whether different combinations of the four α2δ and the 7 α1subunits mediate different or partially redundant functions or whether different α1/α2δ combinations regulate different aspects of VGCC function. This study cap…

Nervous systemmedicine.anatomical_structureVoltage-dependent calcium channelAxon terminalChemistryCalcium channelGenetic modelmedicineNeuronAxonSynaptic vesicleCell biology
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Emerging Therapeutic Strategies for Traumatic Spinal Cord Injury

2020

Spinal cord injury (SCI) is a debilitating neurologic condition with tremendous socioeconomic impact on affected individuals and the health care system. The treatment of SCI principally includes surgical treatment and marginal pharmacologic and rehabilitation therapies targeting secondary events with minor clinical improvements. This unsuccessful result mainly reflects the complexity of SCI pathophysiology and the diverse biochemical and physiologic changes that occur in the injured spinal cord. Once the nervous system is injured, cascades of cellular and molecular events are triggered at varying times. Although the cascade of tissue reactions and cell injury develops over a period of days …

Nervous systemmedicine.medical_specialtyCordmedicine.medical_treatmentSpinal cord injuryRegenerative MedicineMesenchymal Stem Cell TransplantationNeuroprotection03 medical and health sciences0302 clinical medicineNeural Stem CellsGlyburideGranulocyte Colony-Stimulating FactormedicineHumansHypoglycemic AgentsIntensive care medicineErythropoietinSpinal cord injurySpinal Cord InjuriesNeuronal PlasticityRehabilitationCombination treatmentsHepatocyte Growth Factorbusiness.industryNeurological RehabilitationDecompression SurgicalSpinal cordmedicine.diseaseNeuroregenerationNeuroprotectionClinical trialFibroblast Growth FactorsClinical trialmedicine.anatomical_structure030220 oncology & carcinogenesisIntercellular Signaling Peptides and ProteinsSurgeryNeuroregenerationSchwann CellsNeurology (clinical)business030217 neurology & neurosurgeryStem Cell TransplantationWorld Neurosurgery
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Genetic relationship between five psychiatric disorders estimated from genome-wide SNPs

2013

AM Vicente - Cross-Disorder Group of the Psychiatric Genomics Consortium Most psychiatric disorders are moderately to highly heritable. The degree to which genetic variation is unique to individual disorders or shared across disorders is unclear. To examine shared genetic etiology, we use genome-wide genotype data from the Psychiatric Genomics Consortium (PGC) for cases and controls in schizophrenia, bipolar disorder, major depressive disorder, autism spectrum disorders (ASD) and attention-deficit/hyperactivity disorder (ADHD). We apply univariate and bivariate methods for the estimation of genetic variation within and covariation between disorders. SNPs explained 17-29% of the variance in …

Netherlands Twin Register (NTR)MedizinInheritance PatternsSocial SciencesAUTISM SPECTRUM DISORDERSnosologyheritabilityCOMMON SNPS0302 clinical medicineCrohn DiseaseSCHIZOPHRENIAChildPsychiatric geneticsGenetics & HeredityMAJOR DEPRESSIVE DISORDERRISK0303 health sciencesATTENTION-DEFICIT/HYPERACTIVITY DISORDER120 000 Neuronal CoherenceMental DisordersVariantsBIPOLAR DISORDERASSOCIATIONGenomic disorders and inherited multi-system disorders [DCN PAC - Perception action and control IGMD 3]Psychiatric DisordersCROHNS-DISEASE3. Good healthSchizophreniagenetic association studyMedical geneticsMajor depressive disorderSNPsAdultmedicine.medical_specialtygenetic etiologymedical geneticsDEFICIT HYPERACTIVITY DISORDERBiologyPolymorphism Single Nucleotidebehavioral disciplines and activitiesArticleGenomic disorders and inherited multi-system disorders DCN MP - Plasticity and memory [IGMD 3]HeritabilityGenetic Heterogeneity03 medical and health sciencesPrevalence of mental disordersmental disorders/dk/atira/pure/keywords/cohort_studies/netherlands_twin_register_ntr_[SDV.BBM] Life Sciences [q-bio]/Biochemistry Molecular BiologyGeneticsmedicineddc:61HumansAttention deficit hyperactivity disorderGenetic Predisposition to Disease[SDV.BBM]Life Sciences [q-bio]/Biochemistry Molecular BiologyDCN PAC - Perception action and control NCEBP 9 - Mental healthddc:610Medizinische Fakultät » Universitätsklinikum Essen » LVR-Klinikum Essen » Klinik für Psychiatrie Psychosomatik und Psychotherapie des Kindes- und JugendaltersBipolar disorderPsychiatry030304 developmental biologyDepressive Disorder MajorGenome HumanGenetic heterogeneitymedicine.diseaseschizophreniaAttention Deficit Disorder with HyperactivityChild Development Disorders PervasivePerturbações do Desenvolvimento Infantil e Saúde Mental030217 neurology & neurosurgeryGenome-Wide Association Study
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Psychiatric genome-wide association study analyses implicate neuronal, immune and histone pathways

2015

G.B. and S.N. acknowledge funding support for this work from the National Institute for Health Research (NIHR) Mental Health Biomedical Research Centre at South London and Maudsley NHS Foundation Trust and King's College London. P.H.L. is supported by US National Institute of Mental Health (NIMH) grant K99MH101367. Genome-wide association studies (GWAS) of psychiatric disorders have identified multiple genetic associations with such disorders, but better methods are needed to derive the underlying biological mechanisms that these signals indicate. We sought to identify biological pathways in GWAS data from over 60,000 participants from the Psychiatric Genomics Consortium. We developed an an…

Netherlands Twin Register (NTR)Statistical methodsAutismMedizinLOCIGenome-wide association studyheritabilityGenome-wide association studiesHistonesGenètica mèdica0302 clinical medicineHistone methylationDatabases Genetic2.1 Biological and endogenous factorsPsychologyGWASAetiologyPsychiatric geneticsR2Cbipolar disorderPsychiatry0303 health sciencesDisordersLociDepressionGeneral NeuroscienceMental DisordersMedical geneticsMETHYLATIONBrain3rd-DASSerious Mental IllnessPsychiatric Disorders3. Good healthHistoneMental HealthSchizophreniaMental DisorderCognitive Sciences[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]PromotersBDCBURDENRC0321 Neuroscience. Biological psychiatry. NeuropsychiatryHumanSignal Transductionmedicine.medical_specialtyDISORDERSGenomicsNetwork and Pathway Analysis Subgroup of Psychiatric Genomics ConsortiumBurdenBiologyMethylationArticleBiological pathwayPROMOTERS03 medical and health sciencesDatabasesGeneticmedicineGenetics/dk/atira/pure/keywords/cohort_studies/netherlands_twin_register_ntr_HumansGenetic Predisposition to Diseasehistone methylationBipolar disorderPsiquiatriaAUTISMPsychiatry030304 developmental biologyGenetic associationNeurodevelopmental disorders Donders Center for Medical Neuroscience [Radboudumc 7]Neurology & NeurosurgeryNeuroscience (all)Human GenomeNeurosciencesmedicine.diseaseBrain DisordersGood Health and Well BeingDE-NOVO MUTATIONSPerturbações do Desenvolvimento Infantil e Saúde MentalRC0321SchizophreniaGenome-wide Association StudiesDe-novo mutationsmajor depressionNeuroscience030217 neurology & neurosurgeryGenome-Wide Association Study
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Non-linear neuro-inspired circuits and systems: Processing and learning issues

2018

In this chapter the main elements useful for the design and realization of the neural architectures reported in the following chapters will be presented. Considering spiking and non-spiking neurons, the models used for implementing each of them, the synaptic models, the basic learning and plasticity algorithms and the network architectures will be introduced and analysed. The key elements that led to their selection and application in the developed neuro-inspired systems will be discussed briefly.

Network architectureQuantitative Biology::Neurons and Cognitionbusiness.industryComputer scienceReservoir computingEnergy Engineering and Power TechnologyNonlinear systemBasic learningEngineering (all)Key (cryptography)Selection (linguistics)Mathematics (all)Biotechnology; Chemical Engineering (all); Mathematics (all); Materials Science (all); Energy Engineering and Power Technology; Engineering (all)Chemical Engineering (all)Artificial intelligenceMaterials Science (all)businessRealization (systems)Electronic circuitBiotechnology
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Are dendrites in Drosophila homologous to vertebrate dendrites?

2005

AbstractDendrites represent arborising neurites in both vertebrates and invertebrates. However, in vertebrates, dendrites develop on neuronal cell bodies, whereas in higher invertebrates, they arise from very different neuronal structures, the primary neurites, which also form the axons. Is this anatomical difference paralleled by principal developmental and/or physiological differences? We address this question by focussing on one cellular model, motorneurons of Drosophila and characterise the compartmentalisation of these cells. We find that motorneuronal dendrites of Drosophila share with typical vertebrate dendrites that they lack presynaptic but harbour postsynaptic proteins, display c…

NeuriteCompartmentalisationDendriteDendriteAnimals Genetically ModifiedMicePostsynaptic potentialbiology.animalmedicineAnimalsUrbilaterianMolecular BiologyMosaic analysisCytoskeletonCells CulturedMotor NeuronsDendritic spikeTransmitter receptorsbiologyVertebrateCell PolarityCell DifferentiationCell BiologyAnatomyDendritesbiology.organism_classificationBiological EvolutionCell biologyRatsmedicine.anatomical_structureDrosophila melanogasterDrosophilaSomaCalciumRabbitsCellular modelDevelopmental BiologyDevelopmental biology
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Microtubule Dynamics and Neuronal Excitability: Advances on Cytoskeletal Components Implicated in Epileptic Phenomena

2020

AbstractExtensive researches have deepened knowledge on the role of synaptic components in epileptogenesis, but limited attention has been devoted to the potential implication of the cytoskeleton. The study of the development of epilepsy and hyperexcitability states involves molecular, synaptic, and structural alterations of neuronal bioelectric activity. In this paper we aim to explore the neurobiological targets involved in microtubule functioning and cytoskeletal transport, i.e. how dynamic scaffolding of microtubules can influence neuronal morphology and excitability, in order to suggest a potential role for microtubule dynamics in the processes turning a normal neuronal network in a hy…

NeurodevelopmentHyperexcitabilityMicrotubuleBiologyMicrotubulesSettore BIO/09 - FisiologiaEpileptogenesisNeuroprotectionMicrotubule polymerizationCellular and Molecular NeuroscienceNeurotrophic factorsMicrotubulemedicineHumansPremovement neuronal activityCannabinoidCytoskeletonNeuronsEpilepsyNeurodegenerationCell BiologyGeneral Medicinemedicine.diseaseNeuroprotectionMicrotubule-Associated ProteinsNeuroscienceNeural developmentCellular and Molecular Neurobiology
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O-glycosylation of the tail domain of neurofilament protein M in human neurons and in spinal cord tissue of a rat model of amyotrophic lateral sclero…

2005

Mammalian neurofilaments (NFs) are modified by post-translational modifications that are thought to regulate NF assembly and organization. Whereas phosphorylation has been intensely studied, the role of another common modification, the attachment of O-linked N-acetylglucosamine (GlcNAc) to individual serine and threonine residues, is hardly understood. We generated a novel monoclonal antibody that specifically recognizes an O-glycosylated epitope in the tail domain of NF-M and allows determination of the glycosylation state at this residue. The antibody displays strong species preference for human NF-M, shows some reactivity with rat but not with mouse or bovine NF-M. By immunohistochemistr…

NeurofilamentGlycosylationGlycosylationMolecular Sequence DataHyperphosphorylationBiologyMitogen-activated protein kinase kinaseBiochemistryAnimals Genetically Modifiedchemistry.chemical_compoundEpitopesMiceWestern blotNeurofilament ProteinsCell Line TumorAcetylglucosaminidasemedicineAnimalsHumansAmino Acid SequenceProtein kinase AMolecular BiologyMitogen-Activated Protein Kinase KinasesNeuronsmedicine.diagnostic_testKinaseAmyotrophic Lateral SclerosisAntibodies MonoclonalCell BiologyAxonsCell biologyProtein Structure TertiaryRatsDisease Models AnimalchemistryBiochemistrySpinal CordNIH 3T3 CellsPhosphorylationCattleThe Journal of biological chemistry
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Transient cortical circuits match spontaneous and sensory-driven activity during development.

2020

At the earliest developmental stages, spontaneous activity synchronizes local and large-scale cortical networks. These networks form the functional template for the establishment of global thalamocortical networks and cortical architecture. The earliest connections are established autonomously. However, activity from the sensory periphery reshapes these circuits as soon as afferents reach the cortex. The early-generated, largely transient neurons of the subplate play a key role in integrating spontaneous and sensory-driven activity. Early pathological conditions—such as hypoxia, inflammation, or exposure to pharmacological compounds—alter spontaneous activity patterns, which subsequently in…

NeurogenesisSensory systemApoptosisClaustrumBiologyArticleMiceCortex (anatomy)SubplateNeuroplasticityNeural PathwaysmedicineAnimalsHumansCerebral CortexCortical circuitsMultidisciplinaryNeuronal PlasticityCortical architectureNeurogenesisMagnetic Resonance Imagingmedicine.anatomical_structureCortical networkThalamic NucleiSchizophreniaNeuroscienceScience (New York, N.Y.)
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Reawakening the sleeping beauty in the adult brain: neurogenesis from parenchymal glia

2015

Life-long neurogenesis is highly restricted to specialized niches in the adult mammalian brain and therefore the brain's capacity for spontaneous regeneration is extremely limited. However, recent work has demonstrated that under certain circumstances parenchymal astrocytes and NG2 glia can generate neuronal progeny. In the striatum, stroke or excitotoxic lesions can reawaken in astrocytes a latent neurogenic program resulting in the genesis of new neurons. By contrast, in brain areas that fail to mount a neurogenic response following injury, such as the cerebral cortex, forced expression of neurogenic reprogramming factors can lineage convert local glia into induced neurons. Yet, injury-in…

NeurogenesisStriatumBiologyParenchymaGeneticsmedicineAnimalsHumansRegenerationNeuronsRegulation of gene expressionRegeneration (biology)NeurogenesisGene Expression Regulation DevelopmentalAnatomyCellular Reprogrammingmedicine.anatomical_structurenervous systemCerebral cortexAstrocytesBrain InjuriesNeurogliaNeurogliaNeuroscienceReprogrammingDevelopmental BiologyCurrent Opinion in Genetics & Development
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