Search results for "neuron"

showing 10 items of 2611 documents

Role of the p75 neurotrophin receptor (p75NTR) in the basal forebrain cholinergic neurons (BFCNs)

2021

225 págs, figuras y tablas

NeurotrofinasNeuronas colinérgicas del prosencéfalo basalReceptor de neurotrofines p75NeurocienciasUNESCO::CIENCIAS MÉDICASReceptor de neurotrofinas p75UNESCO::CIENCIAS DE LA VIDANeurociènciessense organs:CIENCIAS MÉDICAS [UNESCO]Neuronas colonérgicas de prosencéfalo basal:CIENCIAS DE LA VIDA [UNESCO]
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Sensitivity of neuronal nicotinic acetylcholine receptors to the opiate antagonists naltrexone and naloxone: receptor blockade and up-regulation

2003

In HEK293 cells stably expressing alpha4beta2 nAChRs, naltrexone, but not naloxone, blocked alpha4beta2 nAChRs via an open-channel blocking mechanism. In primary hippocampal cultures, naltrexone inhibited alpha7 nAChRs up-regulated by nicotine, and in organotypic hippocampal cultures naltrexone caused a time-dependent up-regulation of functional alpha7 nAChRs that was detected after removal of the drug. These results indicate that naltrexone could be used as a smoking cessation aid.

NicotinePatch-Clamp TechniquesTime FactorsNarcotic AntagonistsClinical BiochemistryGene ExpressionPharmaceutical Science(+)-NaloxoneReceptors NicotinicPharmacologyHippocampal formationSensitivity and Specificitycomplex mixturesBiochemistryNaltrexoneCell LineNicotineStructure-Activity Relationshipmental disordersDrug DiscoverymedicineHumansMolecular BiologyAcetylcholine receptorNeuronsNaloxoneChemistryNarcotic antagonistmusculoskeletal neural and ocular physiologyOrganic ChemistryNaltrexoneUp-RegulationNicotinic agonistnervous systemMechanism of actionMolecular MedicineSmoking Cessationsense organsmedicine.symptommedicine.drugBioorganic & Medicinal Chemistry Letters
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Chapter 8 Nicotinic receptors of the vertebrate CNS: introductory remarks

1996

Publisher Summary This chapter focuses on the nicotinic receptors of the vertebrate central nervous system (CNS). In vertebrates, nicotinic cholinergic neurotransmission is found in both the CNS and the periphery (muscle endplate). Although muscle and neuronal nicotinic acetylcholine receptors (nAChR) have evolved from a common ancestor, it is striking that the muscle receptor has remained rather stable in evolution, whereas the neuronal receptor has evolved to a wide diversity of subtypes. As an attractive hypothesis, neurotransmitters and neurohormones may not only interact with their archetypic cognate receptors but also with other neuroreceptor, albeit in a modulatory fashion. By modula…

Nicotinic agonistmedicine.anatomical_structureCentral nervous systemAllosteric regulationmedicinePremovement neuronal activityBiologyReceptorNeurohormonesNeuroscienceCoincidence detection in neurobiologyAcetylcholine receptor
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Endothelial NT-3 Delivered by Vasculature and CSF Promotes Quiescence of Subependymal Neural Stem Cells through Nitric Oxide Induction

2014

SummaryInteractions of adult neural stem cells (NSCs) with supportive vasculature appear critical for their maintenance and function, although the molecular details are still under investigation. Neurotrophin (NT)-3 belongs to the NT family of trophic factors, best known for their effects in promoting neuronal survival. Here we show that NT-3 produced and secreted by endothelial cells of brain and choroid plexus capillaries is required for the quiescence and long-term maintenance of NSCs in the mouse subependymal niche. Uptake of NT-3 from irrigating vasculature and cerebrospinal fluid (CSF) induces the rapid phosphorylation of endothelial nitric oxide (NO) synthase present in the NSCs, lea…

Nitric Oxide Synthase Type IIICell SurvivalNeuroscience(all)BiologyNitric OxideNitric oxidechemistry.chemical_compoundMiceCerebrospinal fluidNeural Stem CellsNeurotrophin 3Subependymal zoneAnimalsCells CulturedCell ProliferationNeuronsGeneral NeuroscienceEndothelial CellsCell DifferentiationNeural stem cellCell biologynervous systemchemistrybiology.proteinPhosphorylationChoroid plexusStem cellNeuroscienceNeurotrophinNeuron
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Spiking patterns emerging from wave instabilities in a one-dimensional neural lattice.

2003

The dynamics of a one-dimensional lattice (chain) of electrically coupled neurons modeled by the FitzHugh-Nagumo excitable system with modified nonlinearity is investigated. We have found that for certain conditions the lattice exhibits a countable set of pulselike wave solutions. The analysis of homoclinic and heteroclinic bifurcations is given. Corresponding bifurcation sets have the shapes of spirals twisting to the same center. The appearance of chaotic spiking patterns emerging from wave instabilities is discussed.

Nonlinear Sciences::Chaotic DynamicsNonlinear systemClassical mechanicsQuantitative Biology::Neurons and CognitionArtificial neural networkControl theoryLattice (order)ChaoticCountable setHomoclinic orbitNonlinear Sciences::Pattern Formation and SolitonsBifurcationMathematicsPhysical review. E, Statistical, nonlinear, and soft matter physics
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A note on finite-time Lyapunov dimension of the Rossler attractor

2018

For the R\"ossler system we verify Eden's conjecture on the maximum of local Lyapunov dimension. We compute numerically finite-time local Lyapunov dimensions on the R\"ossler attractor and embedded unstable periodic orbits. The UPO computation is done by Pyragas time-delay feedback control technique.

Nonlinear Sciences::Chaotic DynamicsQuantitative Biology::Neurons and CognitionFOS: Physical sciencesChaotic Dynamics (nlin.CD)Nonlinear Sciences - Chaotic Dynamics
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Visual information flow in Wilson-Cowan networks.

2020

In this paper, we study the communication efficiency of a psychophysically tuned cascade of Wilson-Cowan and divisive normalization layers that simulate the retina-V1 pathway. This is the first analysis of Wilson-Cowan networks in terms of multivariate total correlation. The parameters of the cortical model have been derived through the relation between the steady state of the Wilson-Cowan model and the divisive normalization model. The communication efficiency has been analyzed in two ways: First, we provide an analytical expression for the reduction of the total correlation among the responses of a V1-like population after the application of the Wilson-Cowan interaction. Second, we empiri…

Normalization (statistics)PhysiologyComputer scienceComputationPopulationModels Biological050105 experimental psychologyRetina03 medical and health sciencesWilson–Cowan equations0302 clinical medicineMulti-informationtotal correlationHumans0501 psychology and cognitive sciencesVisual PathwaysEfficient coding hypothesisEfficient representation principleeducationVisual Cortexeducation.field_of_studyNormalization modelGeneral Neuroscience05 social sciencesUnivariateFOS: Biological sciencesQuantitative Biology - Neurons and CognitionDivisive normalizationVisual PerceptionNeurons and Cognition (q-bio.NC)Total correlationNeural Networks ComputerNerve NetAlgorithm030217 neurology & neurosurgeryImage compressionJournal of neurophysiology
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Neuronal ceroid-lipofuscinoses: The current status

1992

In view of the epidemiological connotation of childhood neuronal ceroid-lipofuscinosis (NCL) as one of the most frequent progressive lysosomal diseases and neurodegenerative disorders in children, the recognition of the individual clinical forms of childhood NCL is still based on invasive diagnostic electronmicroscopy which, currently, may be applied also for prenatal diagnosis. Like other inherited disorders, the NCL group has finally also benefited from the genetic breakthroughs of localization of the genes for infantile NCL and juvenile NCL on chromosomes 1 and 16, respectively. This review concerns recent advances in morphological studies, broadening of the clinical spectrum of childhoo…

NosologyPrenatal diagnosisGeneral MedicineBiologymedicine.diseaseGene LocalizationDegenerative diseaseDevelopmental NeuroscienceNeuronal Ceroid-LipofuscinosesPediatrics Perinatology and Child HealthmedicineHumansNeuronal ceroid lipofuscinosisNeurology (clinical)NeuroscienceNeuronal Ceroid-LipofuscinosesBrain and Development
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The

2016

ABSTRACT Members of the Junctophilin (JPH) protein family have emerged as key actors in all excitable cells, with crucial implications for human pathophysiology. In mammals, this family consists of four members (JPH1-JPH4) that are differentially expressed throughout excitable cells. The analysis of knockout mice lacking JPH subtypes has demonstrated their essential contribution to physiological functions in skeletal and cardiac muscles and in neurons. Moreover, mutations in the human JPH2 gene are associated with hypertrophic and dilated cardiomyopathies; mutations in JPH3 are responsible for the neurodegenerative Huntington's disease-like-2 (HDL2), whereas JPH1 acts as a genetic modifier …

NotchGenotypeCardiomyopathyGenes InsectAnimals Genetically ModifiedAnimalsDrosophila ProteinsAllelesMammalsNeuronsHuntingtin ProteinReceptors NotchMusclesMyocardiumMembrane ProteinsReproducibility of ResultsDrosHuntington's diseaseDisease Models AnimalDrosophila melanogasterPhenotypeGene Knockdown TechniquesMutationNerve DegenerationPhotoreceptor Cells InvertebrateRNA InterferenceJunctophilinDrosophilaTrinucleotide Repeat ExpansionSignal TransductionResearch ArticleDisease modelsmechanisms
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The renin–angiotensin system in gastrointestinal functions

2023

In this chapter, we describe the role played by classical and alternative renin–angiotensin system (RAS) in the physiological regulation of gastrointestinal functions. RAS modulates gut motility and mucosal functions, including secretion, fluid, and nutrient absorption. We focus mainly on the activation of angiotensin II type 1 and 2 receptors located on the smooth muscle and epithelial cells or on the enteric neurons. Few studies indicate that alternative RAS may counteract classical RAS functions. Although data on the enteric RAS system are still scarce, they encourage further investigations in consideration also of a potential involvement in gastrointestinal disorders.

Nutrient absorptionGastrointestinal tractAngiotensin II receptors (ATRs)Gastrointestinal secretionGastrointestinal mucosaSettore BIO/09 - FisiologiaGut motilityEnteric neurons
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