Search results for "neutrophils"

showing 10 items of 316 documents

Leukocyte subtypes, gelatinases, and their tissue inhibitors in a group of subjects with asymptomatic carotid atherosclerosis

2022

In a cohort of subjects with asymptomatic carotid atherosclerosis (ACA), we have evaluated the neutrophil and lymphocyte count and their ratio (NLR), the gelatinases (MMP-2 and MMP-9) and their tissue inhibitors (TIMP-1 and TIMP-2). At baseline, no difference was observed between ACA subjects and subject control group regarding neutrophil and lymphocyte count while was evident in ACA subjects a significant increase in MMP-2, MMP-9 and TIMP-2 associated to a significant decrease in TIMP-1. Dividing the ACA according to the number of cardiovascular risk factors (CRFs) we have observed an increase in lymphocyte count in the subgroup with 3–5 CRFs. Evaluating the leukocyte subtypes according to…

Carotid Artery Diseasescardiovascular risk factorslymphocytesTissue Inhibitor of Metalloproteinase-2Tissue Inhibitor of Metalloproteinase-1PhysiologyHematologyTIMP-2Asymptomatic carotid atherosclerosisTIMP-1Matrix Metalloproteinase 9neutrophilsinsulin resistancePhysiology (medical)LeukocytesHumansMatrix Metalloproteinase 2Cardiology and Cardiovascular MedicinegelatinasesBiomarkersResearch ArticleClinical Hemorheology and Microcirculation
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Superoxide generation by human neutrophils induced by low doses of Escherichia coli hemolysin.

1991

Escherichia coli hemolysin (Hly) was isolated from bacterial culture supernatants by polyethylene glycol precipitation and centrifugation in glycerol density gradients. The toxin preparations contained less than 1 mol of lipopolysaccharide per 10 mol of protein, and they had no fatty acids. The capacity of purified hemolysin to stimulate superoxide anion production in polymorphonuclear leukocytes was monitored kinetically in a lumimeter by using the lucigenin assay and was correlated with the kinetics of transmembrane pore formation. When applied to leukocytes suspended in protein-free buffer, very low concentrations (0.02 to 0.1 HU/ml) of the toxin strongly stimulated the production of sup…

Cell Membrane PermeabilityNeutrophilsImmunologyBacterial ToxinsBiologymedicine.disease_causeHemolysin ProteinsMicrobiologychemistry.chemical_compoundHemolysin ProteinsBacterial ProteinsSuperoxidesmedicineEscherichia coliHumansCentrifugationLucigeninEscherichia coliSuperoxideToxinEscherichia coli ProteinsHemolysinFlow CytometryRespiratory burstKineticsInfectious DiseaseschemistryBiochemistryTetradecanoylphorbol AcetateParasitologyPropidiumResearch ArticleInfection and immunity
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Hypersusceptibility of neutrophil granulocytes towards lethal action of free fatty acids contained in enzyme-modified atherogenic low density lipopro…

2008

Abstract Objective The bulk of LDL entrapped in the arterial intima is modified by hydrolytic enzymes, leading to extensive cleavage of cholesterylesters and liberation of fatty acids. The latter induce apoptosis in endothelial cells but are far less cytotoxic towards macrophages. We have compared the cytotoxic effects of enzymatically modified LDL (E-LDL) on macrophages and polymorphonuclear granulocytes (PMN). Methods and results E-LDL displayed toxicity towards PMN at far lower concentrations than towards monocyte-derived macrophages. Native or oxidized LDL had no effect. Free fatty acids contained in E-LDL were the cause of the observed toxicity, which could be mimicked by linoleic acid…

Cell Membrane PermeabilityTime FactorsCell SurvivalNeutrophilsLinoleic acidGranulocyteFatty Acids NonesterifiedHemolysisLinoleic Acidchemistry.chemical_compoundAdenosine TriphosphateSuperoxidesmedicineAnimalsHumansPropidium iodideCells CulturedPeroxidaseRespiratory BurstArachidonic AcidCell DeathL-Lactate DehydrogenaseSuperoxideHydrolysisMacrophagesSterol EsteraseAtherosclerosisRespiratory burstLipoproteins LDLOleic acidmedicine.anatomical_structurechemistryBiochemistryLow-density lipoproteinlipids (amino acids peptides and proteins)Arachidonic acidCalciumRabbitsCardiology and Cardiovascular MedicineOleic AcidPeptide HydrolasesAtherosclerosis
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Long-chain fatty alcohols from pomace olive oil modulate the release of proinflammatory mediators

2009

Pomace olive oil is a by-product of olive oil extraction that is traditionally produced and consumed in Spain. The nonglyceride matter of this oil is a good source of interesting minor compounds, like long-chain fatty alcohols, which are present free or as part of waxes. In the present study, long-chain fatty alcohols were isolated from the nonglyceride fraction of pomace olive oil, and the composition was identified and quantified. The major components of long-chain fatty alcohols were tetracosanol, hexacosanol and octacosanol. We investigated the ability of long-chain fatty alcohols from pomace olive oil to inhibit the release of different proinflammatory mediators in vitro by cells invol…

Cell SurvivalNeutrophilsEndocrinology Diabetes and MetabolismClinical BiochemistryNitric Oxide Synthase Type IIBiochemistryProinflammatory cytokineMiceAnimalsPlant OilsPomace olive oilPhospholipases A2 SecretoryMolecular BiologyOlive OilCytokineCalcimycinInflammationNutrition and DieteticsChemistryMacrophagesPomaceNitric oxideRatsThromboxane B2BiochemistryLong-chain fatty alcoholsFatty AlcoholsInflammation MediatorsLong chainOlive oil
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Herpes virus entry mediator synergizes with Toll-like receptor mediated neutrophil inflammatory responses

2006

In microbial infections polymorphnuclear neutrophils (PMN) constitute a major part of the innate host defence, based upon their ability to rapidly accumulate in inflamed tissues and clear the site of infection from microbial pathogens by their potent effector mechanisms. The recently described transmembrane receptor herpes virus entry mediator (HVEM) is a member of the tumour necrosis factor receptor super family and is expressed on many haematopoietic cells, including T cells, B cells, natural killer cells, monocytes and PMN. Interaction of HVEM with the natural ligand LIGHT on T cells has a costimulatory effect, and increases the bactericidal activity of PMN. To further characterize the f…

Cell SurvivalNeutrophilsImmunologyInflammationBiologyLigandsCell DegranulationNeutrophil ActivationPhagocytosismedicineHumansImmunology and AllergyOpsoninCells CulturedRespiratory BurstToll-like receptorInnate immune systemEffectorInterleukin-8Toll-Like ReceptorsDegranulationOriginal ArticlesAcquired immune systemRespiratory burstCell biologyImmunologyInflammation Mediatorsmedicine.symptomReceptors Tumor Necrosis Factor Member 14Immunology
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Distinct Signaling Cascades of TREM-1, TLR and NLR in Neutrophils and Monocytic Cells

2013

Triggering receptor expressed on myeloid cells 1 (TREM-1) is an important mediator of innate inflammatory responses in microbial infections and sepsis. TREM-1 ligation on neutrophils (PMN) or monocytes results in the production of proinflammatory cytokines. Engagement of TREM-1 induces the activation of MAP kinases as well as rapid Ca<sup>2+</sup> mobilization. However, a detailed understanding of TREM-1 signaling pathways is currently lacking. We evaluated the TREM-1 signaling hierarchy in monocytic cells and found that the acute myeloid leukemia cell line MUTZ-3 expresses TREM-1 in a natural and functional manner. We compared essential signaling molecules of the TREM-1, TLR an…

Cell signalingMyeloidNeutrophilsp38 Mitogen-Activated Protein KinasesMonocytesProinflammatory cytokinePhosphatidylinositol 3-KinasesCell Line TumormedicineHumansImmunology and AllergyCalcium SignalingReceptors ImmunologicExtracellular Signal-Regulated MAP KinasesPI3K/AKT/mTOR pathwayCalcium signalingMembrane GlycoproteinsChemistryToll-Like ReceptorsMyeloid leukemiaImmunity InnateTriggering Receptor Expressed on Myeloid Cells-1Cell biologyLeukemia Myeloid Acutemedicine.anatomical_structureOrgan SpecificityCell cultureImmunologyCytokinesInflammation MediatorsSignal transductionResearch ArticleJournal of Innate Immunity
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Increased adhesion and activation of polymorphonuclear neutrophil granulocytes to endothelial cells under heavy metal exposure in vitro.

1994

Heavy metals have been implicated in the mechanisms of endothelial damage. Influences of heavy metal ions on diverse cell types have been studied using a variety of in vitro and in vivo methods. Polymorphonuclear neutrophil granulocytes (PMNs) have physiological and pathological functions, including the modulation of adhesion to and destruction of endothelial cells (ECs). PMNs were studied during interaction with human umbilical vein ECs under exposure to zinc, nickel and cobalt using an in vitro model. We studied adhesion processes with the help of a computer-controlled image-analyzing system and examined the activation of PMNs by quantification of leukotriene B4 (LTB4) release. The biphas…

Cell typeUmbilical VeinsLeukotriene B4NeutrophilsEnzyme-Linked Immunosorbent AssayPathology and Forensic MedicineMetalchemistry.chemical_compoundIn vivoNickelCell AdhesionImage Processing Computer-AssistedHumansMolecular BiologyCells CulturedPolymorphonuclear neutrophilChemistryHeavy metalsCell BiologyGeneral MedicineAdhesionCobaltIn vitroCell biologyZincBiochemistryvisual_artvisual_art.visual_art_mediumEndothelium VascularE-Selectinhuman activitiesCell Adhesion MoleculesPathobiology : journal of immunopathology, molecular and cellular biology
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Skull and vertebral bone marrow are myeloid cell reservoirs for the meninges and CNS parenchyma.

2021

Getting around the blood–brain barrier The meninges comprise three membranes that surround and protect the central nervous system (CNS). Recent studies have noted the existence of myeloid cells resident there, but little is known about their ontogeny and function, and whether other meningeal immune cell populations have important roles remains unclear (see the Perspective by Nguyen and Kubes). Cugurra et al. found in mice that a large proportion of continuously replenished myeloid cells in the dura mater are not blood derived, but rather transit from cranial bone marrow through specialized channels. In models of CNS injury and neuroinflammation, the authors demonstrated that these myeloid c…

Central Nervous SystemPathologymedicine.medical_specialtyMyeloidEncephalomyelitis Autoimmune ExperimentalNeutrophilsCentral nervous systemBone Marrow CellsBiologyArticleMonocytesMiceImmune systemMeningesBone MarrowCell MovementCentral Nervous System DiseasesParenchymamedicineAnimalsHomeostasisMyeloid CellsNeuroinflammationSpinal Cord InjuriesMultidisciplinaryInnate immune systemSkullMeningesBrainSpinemedicine.anatomical_structureSpinal CordBone marrowDura MaterScience (New York, N.Y.)
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Synthesis and pharmacological evaluation of 2'-hydroxychalcones and flavones as inhibitors of inflammatory mediators generation.

1995

2'-Hydroxy-3,4-dimethoxy-3',4'-dimethylchalcone (3a), 2'-hydroxy-3',4',3,4-tetramethoxychalcone (3b), and their corresponding flavones, 3',4'-dimethoxy-7,8-dimethylflavone (4a) and 3',4',7,8-tetramethoxyflavone (4b), were prepared from 3,4-dimethoxycinnamic acid and the respective phenol. The four compounds inhibited enzymic lipid peroxidation and showed weak peroxyl scavenging activity. They also reduced LTB 4 release from human neutrophils stimulated by A23187. The chalcone 3b was the only compound able to inhibit in a concentration-dependent way, synovial human recombinant phospholipase A 2 activity, human platelet TXB 2 generation, and human neutrophil degranulation. This chalcone exert…

ChalconeAntioxidantNeutrophilsmedicine.medical_treatmentFlavonoidChemical synthesisFlavonesCell DegranulationPhospholipases ALipid peroxidationchemistry.chemical_compoundMiceChalconeChalconesDrug DiscoverySynovial FluidmedicineAnimalsHumanschemistry.chemical_classificationFlavonoidsPhospholipase APancreatic ElastaseChemistryDegranulationFree Radical ScavengersPhospholipases A2BiochemistryMolecular MedicineEicosanoidsLipid PeroxidationInflammation MediatorsJournal of medicinal chemistry
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Phenylsulphonyl urenyl chalcone derivatives as dual inhibitors of cyclo-oxygenase-2 and 5-lipoxygenase

2005

Two series of phenylsulphonyl urenyl chalcone derivatives (UCH) with various patterns of substitution were tested for their effects on nitric oxide (NO) and prostaglandin E2 (PGE2) overproduction in RAW 264.7 macrophages. None of the tested compounds reduced NO production more than 50% at 10 microM but most of them inhibited the generation of PGE2 with IC50 values under the micromolar range. Me-UCH 1, Me-UCH 5, Me-UCH 9, Cl-UCH 1, and Cl-UCH 9 were selected to evaluate their influence on human leukocyte functions and eicosanoids generation. These derivatives selectively inhibited cyclo-oxygenase-2 (COX-2) activity in human monocytes being Me-UCH 5 the most potent (IC50 0.06 microM). Selecte…

ChalconeNeutrophilsNitric OxideLeukotriene B4DinoprostoneGeneral Biochemistry Genetics and Molecular BiologyCell LineNitric oxideMiceStructure-Activity Relationshipchemistry.chemical_compoundChalconesmedicineAnimalsHumansCyclooxygenase InhibitorsLipoxygenase InhibitorsGeneral Pharmacology Toxicology and PharmaceuticsProstaglandin E2IC50Molecular StructurebiologySuperoxideMacrophagesElastaseGeneral MedicinechemistryBiochemistryCyclooxygenase 2MyeloperoxidaseArachidonate 5-lipoxygenasebiology.proteinmedicine.drugLife Sciences
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