Search results for "nitric Oxide"

showing 10 items of 1075 documents

Arginine and nitric oxide synthase: regulatory mechanisms and cardiovascular aspects

2014

L-Arginine (L-Arg) is a conditionally essential amino acid in the human diet. The most common dietary sources of L-Arg are meat, poultry and fish. L-Arg is the precursor for the synthesis of nitric oxide (NO); a key signaling molecule via NO synthase (NOS). Endogenous NOS inhibitors such as asymmetric-dimethyl-L-Arg inhibit NO synthesis in vivo by competing with L-Arg at the active site of NOS. In addition, NOS possesses the ability to be "uncoupled" to produce superoxide anion instead of NO. Reduced NO bioavailability may play an essential role in cardiovascular pathologies and metabolic diseases. L-Arg deficiency syndromes in humans involve endothelial inflammation and immune dysfunctions…

medicine.medical_specialtyArgininemedicine.disease_causeArginineCardiovascular SystemNitric oxidechemistry.chemical_compoundImmune system[SDV.MHEP.CSC]Life Sciences [q-bio]/Human health and pathology/Cardiology and cardiovascular systemInternal medicinemedicineHumansComputingMilieux_MISCELLANEOUSbiologySuperoxideNitric oxide synthasecardiovascularBiological TransportTetrahydrobiopterinmedicine.diseaseBiopterinDiet3. Good health[SDV.MHEP.CSC] Life Sciences [q-bio]/Human health and pathology/Cardiology and cardiovascular systemNitric oxide synthaseEndocrinologyAdipose TissuechemistryCardiovascular DiseasesDietary Supplementsbiology.proteinCitrullineMetabolic syndrometherapeutic potentialOxidative stressFood ScienceBiotechnologymedicine.drug
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Prenatal Brain Damage in Preeclamptic Animal Model Induced by Gestational Nitric Oxide Synthase Inhibition

2010

Cerebral palsy is a major neonatal handicap with unknown aetiology. There is evidence that prenatal brain injury is the leading cause of CP. Severe placental pathology accounts for a high percentage of cases. Several factors predispose to prenatal brain damage but when and how they act is unclear. The aim of this paper was to determine if hypoxia during pregnancy leads to damage in fetal brain and to evaluate the localization of this injury. An animal model of chronic hypoxia produced by chronic administration of a nitric oxide synthase inhibitor (L-NAME) was used to evaluate apoptotic activity in fetal brains and to localize the most sensitive areas. L-NAME reproduces a preeclamptic-like c…

medicine.medical_specialtyArticle SubjectPlacentaApoptosisBlood PressureBrain damagelcsh:Gynecology and obstetricsCrown-Rump LengthPre-EclampsiaPregnancyInternal medicinePlacentamedicineAnimalsRats WistarHypoxia Brainlcsh:RG1-991FetusPregnancyAnalysis of VarianceProteinuriabiologybusiness.industryObstetrics and GynecologyBrainOrgan SizeHypoxia (medical)medicine.diseaseRatsNitric oxide synthaseDisease Models AnimalFetal DiseasesEndocrinologymedicine.anatomical_structureNG-Nitroarginine Methyl Esterbiology.proteinGestationFemalemedicine.symptomNitric Oxide SynthasebusinessResearch Article
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Circadian rhythm in NO synthase I transcript expression and its photoperiodic regulation in the rat pineal gland.

2001

The photoneural regulation of nitric oxide synthase type I (NOS I) expression in the rat pineal was investigated using semiquantitative RT-PCR. NOS I transcript expression exhibited a daily rhythm with peak values during the night hours. The daily rhythm in NOS I transcript expression persisted under constant dark conditions and was abolished under constant light conditions. The extent of nocturnal NOS I expression was found to be dependent on the photoperiod. It was attenuated under 20 h light and 4 h dark (L:D 20:4) compared with 12 h light and 12 h dark (L:D 12:12). The present findings indicate that, in the rat pineal, NOS I transcript expression exhibits a true circadian rhythm. Furthe…

medicine.medical_specialtyArylamine N-AcetyltransferasePhotoperiodNitric Oxide Synthase Type IBiologyPineal GlandGene Expression Regulation EnzymologicRats Sprague-DawleyTranscription (biology)Internal medicineGene expressionmedicineAnimalsCircadian rhythmRNA MessengerMuridaephotoperiodismRegulation of gene expressionGeneral Neurosciencebiology.organism_classificationCircadian RhythmRatsNitric oxide synthaseEndocrinologybiology.proteinNitric Oxide SynthaseEndocrine glandNeuroreport
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Role of GSH in the modulation of NOS-2 expression in the weaned mammary gland

2005

GSH delivery to the lactating mammary gland is essential for the maintenance of lactation as its decrease leads to apoptosis and involution of the mammary gland. In fact, it has already been demonstrated that some of the changes in gene expression found in the lactating mammary gland after forced weaning are reproduced in rats treated with buthionine sulphoximine to deplete GSH levels. An oligonucleotide microarray experiment would give us a better knowledge of the mRNA expression patterns during lactation and after weaning and the possible functions of GSH in the modulation of these events.

medicine.medical_specialtyButhionine SulphoximineMammary glandNitric Oxide Synthase Type IIBiologyNitric OxideBiochemistrychemistry.chemical_compoundMammary Glands AnimalInternal medicineLactationGene expressionmedicineAnimalsLactationWeaningInvolution (medicine)Gene Expression ProfilingGlutathioneGlutathioneIsoenzymesEndocrinologymedicine.anatomical_structureGene Expression RegulationchemistryApoptosisFemaleBiochemical Society Transactions
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Effects of proinsulin C-peptide on nitric oxide, microvascular blood flow and erythrocyte Na+,K+-ATPase activity in diabetes mellitus type I

2000

This study was conducted to evaluate the influence of proinsulin C-peptide on erythrocyte Na(+),K(+)-ATPase and endothelial nitric oxide synthase activities in patients with type I diabetes. In a randomized double-blind study design, ten patients with type I diabetes received intravenous infusions of either human C-peptide or physiological saline on two different occasions. C-peptide was infused at a rate of 3 pmol.min(-1).kg(-1) for 60 min, and thereafter at 10 pmol.min(-1).kg(-1) for 60 min. At baseline and after 60 and 120 min, laser Doppler flow (LDF) was measured following acetylcholine iontophoresis or mild thermal stimulation (44 degrees C), and venous blood samples were collected to…

medicine.medical_specialtyC-peptideArbitrary unitNitric Oxide Synthase Type IIIVenous bloodGeneral MedicineNitric oxidechemistry.chemical_compoundRed blood cellEndocrinologymedicine.anatomical_structurechemistryInternal medicinemedicineAcetylcholinemedicine.drugProinsulinClinical Science
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Dihydrocucurbitacin B, isolated from Cayaponia tayuya, reduces damage in adjuvant-induced arthritis

2005

23,24-Dihydrocucurbitacin B, from the anti-rheumatic plant Cayaponia tayuya, was tested on arthritis induced by adjuvant to corroborate the anti-inflammatory properties of this plant. Arthritis was induced in Lewis rats; the resulting arthritic rats were then treated with dihydrocucurbitacin B (1 mg/kg orally, daily, 1 week). The effect of dihydrocucurbitacin B on the synthesis, release, and activity of pro-inflammatory enzymes (elastase, cyclooxygenase-2, and nitric oxide synthase-2) as well as its effect on different mediators (tumor necrosis factor-alpha and interleukin-1beta) were determined. Dihydrocucurbitacin B modified the evolution of the clinical symptoms, reducing the swelling an…

medicine.medical_specialtyCell Survivalmedicine.medical_treatmentAnti-Inflammatory AgentsAdministration OralNitric Oxide Synthase Type IIPainArthritisPlant RootsDinoprostoneCell LineNitric oxidechemistry.chemical_compoundSuperoxidesInternal medicinemedicineAnimalsLymphocytesNitritesPharmacologyDose-Response Relationship DrugPancreatic ElastasebiologyPlant Extractsbusiness.industryMacrophagesElastasemedicine.diseasebiology.organism_classificationArthritis ExperimentalTriterpenesCayaponia tayuyaRatsEnzyme ActivationNitric oxide synthaseCucurbitaceaeEndocrinologyCytokinechemistryCyclooxygenase 2Rats Inbred LewAntirheumatic AgentsToxicitybiology.proteinCytokinesFemaleTumor necrosis factor alphabusinessPhytotherapyEuropean Journal of Pharmacology
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Hyperglycemia and oxidative stress in cultured endothelial cells – a comparison of primary endothelial cells with an immortalized endothelial cell li…

2012

Diabetes mellitus is a major risk factor for the development of cardiovascular disease and oxidative stress plays an important role in this process. Therefore, we investigated the effects of hyperglycemia on the formation of reactive oxygen species (ROS) and nitric oxide/cGMP signaling in two different endothelial cell cultures. Human umbilical vein endothelial cells (HUVEC) and EA.hy 926 cells showed increased oxidative stress and impaired NO-cGMP signaling in response to hyperglycemia. The major difference between the two different cell types was the dramatic decrease in viability in HUVEC whereas EA.hy cells showed rather increased growth under hyperglycemic conditions. Starvation led to…

medicine.medical_specialtyCell typeEndotheliumCell SurvivalEndocrinology Diabetes and MetabolismPrimary Cell CultureBiologyNitric Oxidemedicine.disease_causeUmbilical veinEndocrinologyInternal medicineHuman Umbilical Vein Endothelial CellsInternal MedicinemedicineHumansEndothelial dysfunctionCyclic GMPCells CulturedCell Line Transformedchemistry.chemical_classificationReactive oxygen speciesCell DeathDose-Response Relationship Drugmedicine.diseaseEndothelial stem cellOxidative StressGlucoseEndocrinologymedicine.anatomical_structurechemistryCell cultureHyperglycemiaEndothelium VascularReactive Oxygen SpeciesOxidative stressJournal of Diabetes and its Complications
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Intermittent ethanol exposure induces inflammatory brain damage and causes long-term behavioural alterations in adolescent rats

2007

Adolescent brain development seems to be important for the maturation of brain structures and behaviour. Intermittent binge ethanol drinking is common among adolescents, and this type of drinking can induce brain damage. Because we have demonstrated that chronic ethanol treatment induces inflammatory processes in the brain, we investigate whether intermittent ethanol intoxication enhances cyclooxygenase-2 (COX-2) and inducible nitric oxide synthase (iNOS) in adolescent rats, and whether these mediators induce brain damage and cause permanent cognitive dysfunctions. Adolescent rats were exposed to ethanol (3.0 g/kg) for two consecutive days at 48-h intervals over 14 days. Levels of COX-2, iN…

medicine.medical_specialtyCerebellumProgrammed cell deathIndomethacinHippocampusNitric Oxide Synthase Type IIInflammationBrain damageMotor ActivityNeuropsychological TestsDiscrimination Learningchemistry.chemical_compoundindomethacinInternal medicineintermittent ethanol intoxicationmedicineAnimalsDrug InteractionsRats WistarAnalysis of VarianceNeocortexEthanolbiologyBehavior AnimalCell DeathEthanolCaspase 3General NeuroscienceAnti-Inflammatory Agents Non-SteroidalBrainRecognition PsychologyRatsNitric oxide synthasemedicine.anatomical_structureEndocrinologychemistryAnimals NewbornneurobehaviourCyclooxygenase 2inflammationAnesthesiabiology.proteinEncephalitisadolescencemedicine.symptomPsychologyPsychomotor Performance
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Analysis of Rabbit Vascular Responses to DBI, an Ingol Derivative Isolated from Euphorbia canariensis

1997

Abstract We have analysed the effects of 7,12-O-diacetyl-8-O-benzoil-2,3-diepiingol (DBI), an ingol derivative isolated from E. canariensis, on isometric tension developed by isolated rabbit basilar and carotid arteries. Concentration-response curves to DBI (10−8 - 3 × 10−5 m) were obtained cumulatively in both arteries at resting tension and active tone (KC1, 50 mm). At resting tension, DBI induced a concentration-dependent contraction, which was not inhibited in Ca2+-free medium. H7 (1-(5-isoquinoline sulphonyl)-2-methylpiperazine dichloride) (10−4 m) inhibited the DBI-induced contraction both in basilar and in carotid arteries. Calmidazolium (10−4 m) inhibited the maximum contraction of …

medicine.medical_specialtyContraction (grammar)LatexCarotid Artery CommonMuscle RelaxationPharmaceutical ScienceProstacyclinNitric OxideNitroarginineMuscle Smooth VascularEuphorbia canariensisCalmodulin1-(5-Isoquinolinesulfonyl)-2-MethylpiperazineInternal medicinemedicine.arterymedicineBasilar arteryAnimalsEnzyme InhibitorsProtein Kinase CPharmacologyPlants MedicinalLagomorphabiologyPlant Extractsbiology.organism_classificationEpoprostenolEndocrinologymedicine.anatomical_structureBasilar ArteryCirculatory systemcardiovascular systemCalciumRabbitsDiterpenesMuscle Contractionmedicine.drugBlood vesselArteryJournal of Pharmacy and Pharmacology
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Expressional down-regulation of neuronal-type nitric oxide synthase I by glucocorticoids in N1E-115 neuroblastoma cells.

1998

Neuronal-type nitric oxide synthase (NOS I) is involved in ischemia-induced brain damage, and glucocorticoids have been reported to protect from brain damage. This prompted us to investigate if the activity or expression of NOS I was influenced by glucocorticoids. We used the murine neuroblastoma cell line N1E-115 as our experimental model. Short-term incubation (30 min) of the N1E-115 cells with dexamethasone (10 nM to 1 microM) or hydrocortisone (100 nM to 10 microM) did not change the enzymatic activity of NOS I. However, the glucocorticoids inhibited NOS I mRNA expression in a concentration-dependent fashion (down to 53.3 +/- 2. 5% of control). In time-course experiments with 100 nM dex…

medicine.medical_specialtyDown-RegulationNitric Oxide Synthase Type IBiologyNitric OxideDexamethasonechemistry.chemical_compoundMiceNeuroblastomaInternal medicinemedicineTumor Cells CulturedAnimalsRNA MessengerGlucocorticoidsDexamethasonePharmacologyNeuronsMessenger RNAAntiglucocorticoidMifepristoneNitric oxide synthaseBlotEndocrinologychemistryCell culturebiology.proteinMolecular MedicineNitric Oxide SynthaseGlucocorticoidmedicine.drugMolecular pharmacology
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