Search results for "overnutrition"

showing 10 items of 19 documents

The Microbiota and Malnutrition: Impact of Nutritional Status During Early Life

2019

According to the developmental origins of health and disease hypothesis, our health is determined by events experienced in utero and during early infancy. Indeed, both our prenatal and postnatal nutrition conditions have an impact on the initial architecture and activity of our microbiota. Recent evidence has underlined the importance of the composition of the early gut microbiota in relation to malnutrition, whether it be undernutrition or overnutrition, that is, in terms of both stunted and overweight development. It remains unclear how early microbial contact is linked to the risk of disease, as well as whether alterations in the microbiome underlie the pathogenesis of malnutrition or a…

0301 basic medicineMedicine (miscellaneous)DiseaseGut floraOverweight03 medical and health sciencesOvernutritionPregnancyEnvironmental healthmedicineHumansMicrobiomeInfant Nutritional Physiological PhenomenaPrenatal Nutritional Physiological Phenomena030109 nutrition & dieteticsNutrition and Dieteticsbiologybusiness.industryInfant NewbornInfantbiology.organism_classificationmedicine.diseaseCausalityObesityInfant Nutrition DisordersGastrointestinal MicrobiomeMalnutrition030104 developmental biologyFemalemedicine.symptombusinessAnnual Review of Nutrition
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Developmental programming of somatic growth, behavior and endocannabinoid metabolism by variation of early postnatal nutrition in a cross-fostering m…

2017

Background Nutrient deprivation during early development has been associated with the predisposition to metabolic disorders in adulthood. Considering its interaction with metabolism, appetite and behavior, the endocannabinoid (eCB) system represents a promising target of developmental programming. Methods By cross-fostering and variation of litter size, early postnatal nutrition of CB6F1-hybrid mice was controlled during the lactation period (3, 6, or 10 pups/mother). After weaning and redistribution at P21, all pups received standard chow ad libitum. Gene expression analyses (liver, visceral fat, hypothalamus) were performed at P50, eCB concentrations were determined in liver and visceral …

0301 basic medicinePhysiologyGene Expressionlcsh:MedicineAdipose tissueBiochemistryFatsMiceOvernutritionArcuate NucleusPregnancyLactationMedicine and Health SciencesCross-fosteringInsulin-Like Growth Factor Ilcsh:Sciencemedia_commonMultidisciplinaryAnimal BehaviorBrainNeurochemistryLipidsmedicine.anatomical_structureAdipose TissuePhysiological ParametersLiverAnimal SocialityFemaleAnatomyNeurochemicalsResearch Articlemedicine.medical_specialtymedia_common.quotation_subjectHypothalamusNutritional StatusIntra-Abdominal FatBiology03 medical and health sciencesInternal medicineGeneticsmedicineAnimalsHumansWeaningObesityNutritionBehaviorBody Weightlcsh:RBiology and Life SciencesAppetitemedicine.diseaseObesityDisease Models AnimalBiological Tissue030104 developmental biologyEndocrinologyDevelopmental plasticitylcsh:QZoologyBody mass indexEndocannabinoidsNeurosciencePLOS ONE
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Renal Programming by Transient Postnatal Overfeeding: The Role of Senescence Pathways

2020

Background: Early nutrition influences the risk of Chronic Kidney Diseases development in adulthood. Mechanisms underlying the early programming of altered renal function remain incompletely understood. This study aims at characterizing the role of cell senescence pathways in early programming of Chronic Kidney Disease after transient postnatal overfeeding. Material and Methods: Reduced litters of 3 mice pups and standard litters of 10 mice pups were obtained to induce overfed animals during lactation and control animals, respectively. Animals were sacrificed at 24 days (weaning) or at 7 months of life (adulthood). Body weight, blood pressure, kidney weight, and glomerular count were assess…

0301 basic medicineSenescencekidneymedicine.medical_specialtyPhysiologyRenal function030204 cardiovascular system & hematologyprogramming[SDV.MHEP.UN]Life Sciences [q-bio]/Human health and pathology/Urology and Nephrologylcsh:Physiology03 medical and health sciencesovernutrition0302 clinical medicineOvernutritionPhysiology (medical)Internal medicineLactationmedicineWeaningOriginal Research2. Zero hungerKidneypostnatal overfeedinglcsh:QP1-981biologySirtuin 1medicine.disease[SDV.MHEP.UN] Life Sciences [q-bio]/Human health and pathology/Urology and Nephrologychronic kidney disease; developmental origins of health and disease; kidney; overnutrition; postnatal overfeeding; programming[SDV.AEN] Life Sciences [q-bio]/Food and Nutrition030104 developmental biologymedicine.anatomical_structureEndocrinologybiology.proteindevelopmental origins of health and disease[SDV.AEN]Life Sciences [q-bio]/Food and Nutritionchronic kidney diseaseKidney diseaseFrontiers in Physiology
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Transient postnatal over nutrition induces long-term alterations in cardiac NLRP3-inflammasome pathway.

2018

International audience; Background and aims: The prevalence of obesity is increasing worldwide at an alarming rate. Altered early nutrition, in particular postnatal overfeeding (PNOF), is a risk factor for impaired cardiac function in adulthood. In the understanding of the initiation or progression of heart diseases, NLRP3 inflammasome and non-coding RNAs have been proposed as key players. In this context, the aim of this study was to decipher the role of NLRP3 inflammasome and its post transcriptional control by micro-RNAs in the regulation of cardiac metabolic function induced by PNOF in mice. Methods and results: Based on a model of mice exposed to PNOF through litter size reduction, we …

0301 basic medicineTime FactorsLitter SizeInflammasomesEndocrinology Diabetes and Metabolismmedicine.medical_treatmentMedicine (miscellaneous)InflammasomeOvernutritionInsulinNutrition and Dieteticsintegumentary systembiologyInflammasomeMicro-RNAsTransfection[SDV.MHEP.CSC] Life Sciences [q-bio]/Human health and pathology/Cardiology and cardiovascular systemAnimal Nutritional Physiological PhenomenaSignal transductionCardiology and Cardiovascular Medicinemedicine.drugSignal TransductionCardiac function curvemedicine.medical_specialtyHeart DiseasesCardiac dysfunctionsNutritional StatusContext (language use)Cell LineProto-Oncogene Protein c-ets-103 medical and health sciences[SDV.MHEP.CSC]Life Sciences [q-bio]/Human health and pathology/Cardiology and cardiovascular systemInternal medicineNLR Family Pyrin Domain-Containing 3 ProteinmedicineAnimalsPost-transcriptional regulationNutritionbusiness.industryInsulinMyocardiumRatsMice Inbred C57BLInsulin receptorDisease Models AnimalMicroRNAs030104 developmental biologyEndocrinologyAnimals Newbornbiology.proteinbusinessNutrition, metabolism, and cardiovascular diseases : NMCD
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Manejo de la hiperglucemia con fármacos no insulínicos en pacientes adultos con diabetes tipo 2

2019

Resumen: El adecuado tratamiento de la diabetes mellitus tipo 2 (DM2) incluye la alimentación saludable y el ejercicio (150 min/semana) como pilares básicos. Para el tratamiento farmacológico, la metformina es el fármaco de elección inicial, salvo contraindicación o intolerancia; en caso de mal control, se dispone de 8 familias terapéuticas (6 orales y 2 inyectables) como posibles combinaciones. Se presenta un algoritmo y unas recomendaciones para el tratamiento de la DM2. En prevención secundaria cardiovascular se recomienda asociar un inhibidor del cotransportador sodio-glucosa tipo 2 (iSGLT2) o un agonista del receptor de glucagon-like peptide-1 (arGLP1) en pacientes con obesidad. En pre…

Agonistmedicine.medical_specialtymedicine.drug_class030204 cardiovascular system & hematologyOverweight:Phenomena and Processes::Physiological Phenomena::Nutritional Physiological Phenomena::Diet::Diabetic Diet [Medical Subject Headings]GastroenterologyDiabetes tipo 2:Organisms::Eukaryota::Animals::Chordata::Vertebrates::Mammals::Primates::Haplorhini::Catarrhini::Hominidae::Humans [Medical Subject Headings]03 medical and health sciences0302 clinical medicineFármacos antidiabéticos:Diseases::Nutritional and Metabolic Diseases::Metabolic Diseases::Glucose Metabolism Disorders::Hyperglycemia [Medical Subject Headings]Internal medicineDiabetes mellitusmedicineGliclazide030212 general & internal medicineHiperglucemiaContraindicationlcsh:R5-920business.industryAntidiabetic drugs:Diseases::Endocrine System Diseases::Diabetes Mellitus::Diabetes Mellitus Type 2 [Medical Subject Headings]nutritional and metabolic diseases:Psychiatry and Psychology::Behavior and Behavior Mechanisms::Psychology Social::Life Style [Medical Subject Headings]Type 2 diabetesGeneral Medicinemedicine.diseaseSulfonylureaObesityHumanos:Diseases::Nutritional and Metabolic Diseases::Nutrition Disorders::Overnutrition::Obesity [Medical Subject Headings]MetforminHyperglycemia:Chemicals and Drugs::Chemical Actions and Uses::Pharmacologic Actions::Physiological Effects of Drugs::Hypoglycemic Agents [Medical Subject Headings]medicine.symptomlcsh:Medicine (General)Family Practicebusinessmedicine.drug
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Postnatal Overfeeding Causes Early Shifts in Gene Expression in the Heart and Long-Term Alterations in Cardiometabolic and Oxidative Parameters

2013

International audience; Background: Postnatal overfeeding (OF) in rodents induces a permanent moderate increase in body weight in adulthood. However, the repercussions of postnatal OF on cardiac gene expression, cardiac metabolism and nitro-oxidative stress are less well known. Methodology/Principal Findings: Immediately after birth, litters of C57BL/6 mice were either maintained at 10 (normal-fed group, NF), or reduced to 3 in order to induce OF. At weaning, mice of both groups received a standard diet. The cardiac gene expression profile was determined at weaning and cardiac metabolism and oxidative stress were assessed at 7 months. The cardiac expression of several genes, including membe…

Blood GlucoseAnatomy and PhysiologyTime FactorsMouseMicroarrays[SDV]Life Sciences [q-bio]Myocardial InfarctionGene Expressionlcsh:Medicine030204 cardiovascular system & hematologyCardiovascularmedicine.disease_causeCardiovascular SystemMiceOvernutrition0302 clinical medicineBlood plasmaInsulinlcsh:Science2. Zero hungerRegulation of gene expression0303 health sciencesMultidisciplinaryEjection fractionVentricular RemodelingHeartAnimal ModelsReactive Nitrogen Species[SDV.MHEP.CSC] Life Sciences [q-bio]/Human health and pathology/Cardiology and cardiovascular systemApelin[SDV] Life Sciences [q-bio]Body CompositionMedicineFemaleDisease SusceptibilityOxidation-ReductionResearch ArticlePhysiogenomicsmedicine.medical_specialtyDiastoleEndocrine SystemMyocardial Reperfusion InjuryBiology03 medical and health sciencesModel Organisms[SDV.MHEP.CSC]Life Sciences [q-bio]/Human health and pathology/Cardiology and cardiovascular systemInternal medicinemedicineAnimalsWeaningVentricular remodelingBiology030304 developmental biologyEndocrine Physiology[ SDV ] Life Sciences [q-bio]Gene Expression ProfilingMyocardiumBody Weightlcsh:RComputational Biologymedicine.diseaseOxidative StressEndocrinologyGene Expression Regulationlcsh:QOxidative stress
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Postnatal overfeeding in rats leads to moderate overweight and to cardiometabolic and oxidative alterations in adulthood.

2011

In contrast to the masses of data on obesity, few data are available concerning the cardiometabolic and oxidative consequences of moderate overweight. The model of postnatal overfeeding (OF) induces an increase in body weight at weaning that remains during adult life. Litters of Wistar rats were either maintained at 12 pups (normal-fed group, NF), or reduced to 3 pups at birth in order to induce OF. At 6 months of age, metabolic parameters, circulating oxidative stress and aortic and coronary vasoreactivity were assessed. Cardiac susceptibility to ischemia-reperfusion injury was also evaluated ex vivo as were markers of cardiac remodeling. OF led to an increase in body weight at weaning (+5…

Blood GlucoseLeptinleft ventricular end-systolic pressuremedicine.medical_treatment030204 cardiovascular system & hematologyOverweight+dP/dtmedicine.disease_causeBiochemistryCardiovascular System0302 clinical medicineOvernutritionHRleft ventricular developed pressureheart rateInsulinhydroperoxidesworking modeComputingMilieux_MISCELLANEOUSmatrix metallo-proteinase-2W0303 health sciencesANOVAMMP-2OFLeptinROOHinternational unitsGeneral MedicineLsuperoxide dismutase[SDV.MHEP.CSC] Life Sciences [q-bio]/Human health and pathology/Cardiology and cardiovascular systemleft ventricular maximal pressure developmentFemalemedicine.symptomleft ventricular end-diastolic pressureanalysis of variancemedicine.medical_specialtyLDHNFleft ventricular minimal pressure developmentIschemiaSNPbody mass indexheartReal-Time Polymerase Chain Reactionoxidative stress AchBMI03 medical and health sciences[SDV.MHEP.CSC]Life Sciences [q-bio]/Human health and pathology/Cardiology and cardiovascular systemLangendorff modeoverfeedingInternal medicineRLUBKmedicineWeaningAnimalsLVEDPSODRats WistarVentricular remodeling030304 developmental biologyDNA PrimersPostnatal overfeedingBase Sequencebusiness.industryInsulinsodium nitroprussiatelactate dehydrogenaseLVDPLVESPOverweightrelative light unitsmedicine.diseaseacetylcholinearbitrary unitsRatsIUOxidative StressEndocrinology−dP/dtAUnormal-fedbradykininbusinessEx vivoOxidative stressBiochimie
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Altered adipose tissue metabolism in offspring of dietary obese rat dams.

2011

To investigate further the mechanisms of developmental programming, we analysed the effects of maternal overnutrition and of postnatal high-fat feeding on adipose tissue metabolism in the offspring. Postnatal changes in serum adiponectin, leptin and TAG [triacylglycerol (triglyceride)] levels, adipose tissue TAGs, fatty acids and enzyme activities were determined in offspring of cafeteria-diet-fed dams during gestation and lactation, weaned on to standard chow or on to cafeteria diet. Obese rats showed higher adiposity (+35% to 85%) as well as a significant increase in serum glucose, insulin, leptin, adiponectin and TAG levels (P<0.01) and adipose tissue LPL (lipoprotein lipase) and …

Blood Glucosemedicine.medical_specialtyOffspringAdipokineAdipose tissueBiologychemistry.chemical_compoundOvernutritionPregnancyInternal medicinemedicineAdipocytesAnimalsObesityRats WistarPrenatal Nutritional Physiological PhenomenaTriglycerideschemistry.chemical_classificationTriglycerideAdiponectinLeptinBody WeightFatty AcidsGeneral MedicineFeeding BehaviorOrgan Sizemedicine.diseaseHormonesRatsPregnancy ComplicationsEndocrinologychemistryAdipose TissuePrenatal Exposure Delayed EffectsFemalePolyunsaturated fatty acidClinical science (London, England : 1979)
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0053 : Short-term moderate diet restriction in adulthood can reverse alterations of cardiac function induced by postnatal overfeeding in mice

2015

Postnatal overfeeding (OF) in rodents induces early programming of cardio-metabolic risk: permanent moderate increase in body weight, metabolic disorders and progressive alterations of cardiac function in adulthood. Our aim was to determine whether moderate diet restriction, performed at the time where these disorders are acquired, could restore cardiac function and ameliorate post-ischemic recovery.Immediately after birth, litters of C57BL/6 mice were either maintained at 9 (normal-fed group, NF), or reduced to 3 in order to induce OF. At weaning, mice of both groups received a standard diet ad libitum (AL). At 6 months of age, half of the NF mice and OF mice were assigned to a moderate 20…

Cardiac function curvemedicine.medical_specialtyEjection fractionbusiness.industryCalorie restrictionmedicine.diseaseBody weightOvernutritionEndocrinologyInternal medicinemedicineStandard dietWeaningLeft Ventricular Fractional ShorteningbusinessCardiology and Cardiovascular MedicineArchives of Cardiovascular Diseases Supplements
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Plasma Ceramides, Mediterranean Diet, and Incident Cardiovascular Disease in the PREDIMED Trial

2017

Background: Although in vitro studies and investigations in animal models and small clinical populations have suggested that ceramides may represent an intermediate link between overnutrition and certain pathological mechanisms underlying cardiovascular disease (CVD), no prospective studies have investigated the association between plasma ceramides and risk of CVD. Methods: The study population consisted of 980 participants from the PREDIMED trial (Prevención con Dieta Mediterránea), including 230 incident cases of CVD and 787 randomly selected participants at baseline (including 37 overlapping cases) followed for ≤7.4 years. Participants were randomized to a Mediterranean diet supplemente…

Male0301 basic medicineTime FactorsMediterranean dietPhysiologyDisease030204 cardiovascular system & hematologyDiet Mediterraneanchemistry.chemical_compound0302 clinical medicinecardiovascular diseaseRisk FactorsTandem Mass SpectrometryNutsMedicineProspective StudiesIncidenceMiddle AgedstrokeCardiovascular DiseasesFemaleCardiology and Cardiovascular MedicineCeramideCeramidesRisk AssessmentArticle03 medical and health sciencesOvernutritionMediterranean dietPhysiology (medical)HumansMetabolomicsceramidecoronary heart diseaseOlive OilDieta mediterraneaAgedProportional Hazards Modelsbusiness.industryChromatography liquidProtective Factorsmedicine.diseasePredimedCoronary heart disease030104 developmental biologychemistrySpainMultivariate AnalysisLinear ModelsbusinessRisk Reduction BehaviorBiomarkersChromatography Liquid
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