Search results for "oxidation"

showing 10 items of 1913 documents

Acute Metabolic Response, Energy Expenditure, and EMG Activity in Sitting and Standing

2017

Purpose While merely standing up interrupts sedentary behavior, it is important to study acute metabolic responses during single bouts of sitting and standing to understand the physiological processes affecting the health of office workers. Methods Eighteen healthy middle-age women 49.4 ± 7.9 yr old (range: 40–64) with a body mass index of 23.4 ± 2.8 kg·m−2 volunteered for this laboratory-based randomized crossover trial where they performed 2 h desk work in either sitting or standing postures after overnight fasting. Muscle activity (normalized to walking at 5 km·h−1), respiratory gas exchange, and blood samples were assessed after glucose loading (75 g). Results Compared with seated work,…

Blood GlucoseGlycerolHydrocortisoneElectromyographyBicepscarbohydrate oxidation0302 clinical medicineenergy expenditureInsulinOrthopedics and Sports Medicine030212 general & internal medicineglucose loadingsit-stand workstationta315aineenvaihduntaCross-Over Studiesmuscle activitymedicine.diagnostic_testArea under the curvefat oxidationMiddle AgedLipidsAnesthesiaFemaleOxidation-Reductionenergiankulutus (aineenvaihdunta)Adultmedicine.medical_specialtyPosturePhysical Therapy Sports Therapy and RehabilitationCarbohydrate metabolismSittingistuminen03 medical and health sciencesLumbarmedicineHumansMuscle SkeletallihasaktiivisuusElectromyographyPulmonary Gas Exchangebusiness.industry030229 sport sciencesCrossover studyseisominenQPverensokeriPhysical therapyEnergy MetabolismbusinessBody mass index
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Antioxidant treatment for impaired hypoxic ventilatory responses in experimental diabetes in the rat

2018

Inflammation, tissue hypoxia, and impaired hypoxic ventilatory response (HVR) are the intricately entwined features of diabetes which perpetuate the disease and its sequelae. Hyperglycemia, notably, is an oxygen consuming process due to enhanced cellular metabolism. Oxidative stress underlies diabetic pathogenesis and also is a crucial modulator of the hypoxic chemoreflex. The present study seeks to determine if suppressed ventilation in diabetes could be improved by antioxidant treatment. The study was performed in streptozotocin-induced diabetes in awake rats. Two weeks into full-fledged diabetes, the rats were divided into mangiferin (potent natural antioxidant)-treated and untreated, wi…

Blood GlucoseMale0301 basic medicinePulmonary and Respiratory MedicineAntioxidantPhysiologyXanthonesmedicine.medical_treatmentInflammationHypoxic ventilatory responsePharmacologymedicine.disease_causeThiobarbituric Acid Reactive SubstancesAntioxidantsDiabetes Mellitus ExperimentalSuperoxide dismutase03 medical and health scienceschemistry.chemical_compoundAntioxidant treatment; Diabetes; Hypoxic ventilatory response; Inflammation; Mangiferin; Oxidative stress.Diabetes mellitusmedicineAnimalsRats WistarHypoxiaMangiferinInflammationbiologySuperoxide DismutaseTumor Necrosis Factor-alphabusiness.industryRespirationGeneral NeuroscienceBrainmedicine.diseaseOxidative Stress030104 developmental biologychemistrybiology.proteinTumor necrosis factor alphaLipid Peroxidationmedicine.symptombusinessOxidative stressRespiratory Physiology & Neurobiology
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ERK1 and ERK2 activation modulates diet-induced obesity in mice

2017

IF 3.112; International audience; Obesity is a worldwide problem, and dietary lipids play an important role in its pathogenesis. Recently, Erk1 knock-out (ERK1(-/-)) mice have been shown to exhibit low preference for dietary fatty acids. Hence, we maintained Erk1(-/-) mice on a high-fat diet (HFD) to assess the implication of this mitogen-activated protein (MAP) kinase in obesity. The Erk1(-/-) mice, fed the HFD, were more obese than wild-type (WT) animals, fed the same diet. Erk1(-/-) obese mice gained more fat and liver mass than WT obese animals. No difference was observed in daily food and energy intake in HFD-fed both group of animals. However, feed efficiency was higher in Erk1(-/-) t…

Blood GlucoseMale0301 basic medicinemedicine.medical_treatmentMice ObeseBiochemistryMicechemistry.chemical_compoundPhosphorylationBeta oxidationCells CulturedMice KnockoutMitogen-Activated Protein Kinase 1Mitogen-Activated Protein Kinase 3Reverse Transcriptase Polymerase Chain ReactionGeneral MedicineLipidsFatty acid synthaseLiverLipogenesisHomeostatic model assessmentmedicine.medical_specialtyBlotting WesternBiologyDiet High-FatReal-Time Polymerase Chain Reaction03 medical and health sciencesInsulin resistanceInternal medicinemedicineAnimals[SDV.BBM]Life Sciences [q-bio]/Biochemistry Molecular BiologyRNA MessengerObesity[ SDV.BBM ] Life Sciences [q-bio]/Biochemistry Molecular BiologyInflammationTriglycerideLipogenesisInsulinBody WeightLipid Metabolismmedicine.diseaseObesityMice Inbred C57BL030104 developmental biologyEndocrinologychemistrybiology.proteinMAP kinaseInsulin ResistanceBiochimie
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POSTPRANDIAL HYPERGLYCEMIA IS A DETERMINANT OF PLATELET ACTIVATION IN EARLY 2 DIABETES MELLITUS

2010

BACKGROUND: Chronic hyperglycemia is a major contributor to in vivo platelet activation in diabetes mellitus. OBJECTIVES: To evaluate the effects of acarbose, an alpha-glucosidase inhibitor, on platelet activation and its determinants in newly diagnosed type 2 diabetic patients. METHODS: Forty-eight subjects (26 males, aged 61 +/- 8 years) with early type 2 diabetes (baseline hemoglobin A(1c) < or = 7% and no previous hypoglycemic treatment) were randomly assigned to acarbose up to 100 mg three times a day or placebo, and evaluated every 4 weeks for 20 weeks. The main outcome measures were urinary 11-dehydro-thromboxane (TX)B(2) (marker of in vivo platelet activation) and 8-iso-prostaglandi…

Blood GlucoseMaleTime FactorsSettore MED/09 - Medicina InternaDinoprostpostprandial hyperglycemia; platelet activationMedicineEnzyme InhibitorsSettore MED/49 - Scienze Tecniche Dietetiche Applicatepostprandial hyperglycemiaAcarboseplateletHemoglobin AHematologyMiddle AgedPostprandial PeriodP-SelectinPostprandialTreatment OutcomeC-Reactive ProteinItalyFemaleBiological MarkersAcarboseType 2medicine.drugacarbose platelet activation postprandial hyperglycemia type 2 diabetes mellitusmedicine.medical_specialtySettore BIO/14 - FARMACOLOGIAUrinary systemCD40 LigandGlycosylatedArginineExcretionBlood Glucose; Time Factors; Lipid Peroxidation; Middle Aged; Hemoglobin A Glycosylated; Postprandial Period; Diabetes Mellitus Type 2; Enzyme Inhibitors; Hypoglycemic Agents; P-Selectin; Platelet Activation; Aged; CD40 Ligand; Treatment Outcome; Male; Female; Thromboxane B2; Dinoprost; Italy; Arginine; Acarbose; Double-Blind Method; Humans; Biological Markers; Hyperglycemia; alpha-Glucosidases; C-Reactive ProteinDouble-Blind MethodInternal medicineDiabetes mellitusDiabetes MellitusHypoglycemic AgentsHumansGlycoside Hydrolase InhibitorsPlatelet activationGlycemicAgedGlycated Hemoglobinbusiness.industryType 2 Diabetes Mellitusalpha-Glucosidasesmedicine.diseasePlatelet ActivationThromboxane B2EndocrinologyDiabetes Mellitus Type 2HyperglycemiaLipid PeroxidationbusinessBiomarkers
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Antioxidant effect of Ajuga iva aqueous extract in streptozotocin-induced diabetic rats.

2009

The purpose of this study was to investigate the possible antioxidant effect of an aqueous extract of Ajuga iva (Ai) in streptozotocin (STZ)-induced diabetic rats. Twelve diabetic rats were divided into two groups fed a casein diet supplemented or not with Ai (0.5%), for 4 weeks. In vitro, the Ai extract possessed a very high antioxidant effect (1 mg/ml was similar to those of trolox 300 mmol/l). The results indicated that plasma thiobarbituric acid reactive substances (TBARS) values were reduced by 41% in Ai-treated compared with untreated diabetic rats. TBARS concentrations were lower 1.5-fold in liver, 1.8-fold in heart, 1.9-fold in muscle and 2.1-fold in brain in Ai-treated than untreat…

Blood GlucoseMalemedicine.medical_specialtyAntioxidantThiobarbituric acidmedicine.medical_treatmentGlutathione reductasePharmaceutical ScienceEnzyme-Linked Immunosorbent AssayNitric OxideThiobarbituric Acid Reactive SubstancesAntioxidantsStreptozocinLipid peroxidationchemistry.chemical_compoundInternal medicineDrug DiscoverymedicineTBARSAnimalsInsulinRats WistarPharmacologychemistry.chemical_classificationPlant ExtractsGlutathione peroxidaseBody WeightGlutathioneOrgan SizeCarotenoidsLipidsRatsEndocrinologyComplementary and alternative medicinechemistryBiochemistryMolecular MedicineTroloxLipid PeroxidationPhytomedicine : international journal of phytotherapy and phytopharmacology
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Fructose-enriched diet modifies antioxidant status and lipid metabolism in spontaneously hypertensive rats

2005

Abstract Objective High-fructose consumption in industrial countries has been shown to induce metabolic abnormalities or syndrome X. Changes in antioxidant defense are unknown in hypertension associated with metabolic disorders induced by high-fructose feeding. Methods Twenty spontaneously hypertensive rats were assigned to one of two groups; one received a fructose-enriched diet (60% fructose) and the other a starch diet. After a 13-wk diet period, total antioxidant status was assessed in the blood and liver by monitoring the rate of free radical-induced red blood cell hemolysis. Antioxidants (enzymes and vitamins) were determined in blood and liver. Gene expression of antioxidant enzymes …

Blood GlucoseMalemedicine.medical_specialtyErythrocytesAntioxidantEndocrinology Diabetes and Metabolismmedicine.medical_treatmentalpha-TocopherolGene ExpressionAscorbic AcidFructoseThiobarbituric Acid Reactive SubstancesAntioxidantsLipid peroxidationSuperoxide dismutasechemistry.chemical_compoundRats Inbred SHRInternal medicinemedicineAnimalsInsulinRNA MessengerVitamin Achemistry.chemical_classificationGlutathione PeroxidaseNutrition and DieteticsbiologySuperoxide DismutaseGlutathione peroxidaseStarchFructoseLipid MetabolismAscorbic acidDietRatsRed blood cellEndocrinologymedicine.anatomical_structureLiverchemistryHypertensionbiology.proteinLipid PeroxidationPeroxidaseNutrition
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Protein oxidation in a group of subjects with metabolic syndrome.

2013

Abstract Aims To examine the protein oxidation, marker of the oxidative stress, in metabolic syndrome (MS). Methods We enrolled 106 subjects (45 women and 61 men) with MS of which 43 (14 women and 27 men) were with diabetes mellitus and 63 (31 women and 32 men) were without diabetes mellitus, and 54 subjects (19 women and 35 men) as control group. The protein oxidation, expressed as carbonyl groups, was measured by an enzyme-like immunosorbent assay (ELISA) kit (BioCell PC test kit, Enzo Life Sciences AG, Switzerland). Results In the whole group of MS subjects, in comparison with control group, a significant increase in carbonyl groups was present. The same datum was also evident between co…

Blood GlucoseMalemedicine.medical_specialtyWaistEndocrinology Diabetes and MetabolismIndiaBlood PressureProtein oxidationmedicine.disease_causeNitric OxideProtein CarbonylationInternal medicineDiabetes mellitusinsulin resistanceInternal MedicinemedicineHumansInflammationMetabolic Syndromeoxidative strebusiness.industryGeneral MedicineMiddle Agedmedicine.diseaseOxidative StressEndocrinologyBlood pressureDiabetes Mellitus Type 2FemaleMetabolic syndromeWaist CircumferencebusinessOxidation-ReductionOxidative stressBiomarkersDiabetesmetabolic syndrome
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Associations of resting and peak fat oxidation with sex hormone profile and blood glucose control in middle-aged women.

2022

Background and Aims Menopause may reduce fat oxidation. We investigated whether sex hormone profile explains resting fat oxidation (RFO) or peak fat oxidation (PFO) during incremental cycling in middle-aged women. Secondarily, we studied associations of RFO and PFO with glucose regulation. Method and Results We measured RFO and PFO of 42 women (age 52–58 years) with indirect calorimetry. Seven participants were pre- or perimenopausal, 26 were postmenopausal, and nine were postmenopausal hormone therapy users. Serum estradiol (E2), follicle-stimulating hormone, progesterone, and testosterone levels were quantified with immunoassays. Insulin sensitivity (Matsuda index) and glucose tolerance (…

Blood GlucoseestradioliNutrition and Dieteticsvaihdevuodetglucose toleranceEndocrinology Diabetes and MetabolismhapettuminenmenopauseMedicine (miscellaneous)fat oxidationGlycemic ControlMiddle Agedinsuliiniresistenssirasva-aineenvaihduntahormonaaliset tekijätGlucoseglukoosi-intoleranssiestradiolBody Compositioninsulin sensitivityHumansFemaleInsulin ResistanceCardiology and Cardiovascular MedicineGonadal Steroid HormonesNutrition, metabolism, and cardiovascular diseases : NMCD
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Oxidant stress: the role of nutrients in cell-lipoprotein interactions

1999

Oxidant stress is increasingly becoming an important hypothesis to explain the genesis of several pathologies, including cancer, atherosclerosis and also ageing. Beside a few rare genetic defects, dietary factors are thought to play a key role in the regulation of the production of reactive oxygenated species. An imbalance between nutrients, and in particular those involved in antioxidant status, could explain the onset of an enhanced production of free radicals. We will briefly review information concerning oxidation of lipids and lipoproteins which lead to atherothrombosis. We also present new findings supporting a role for blood platelets in generating oxidant species. New data are also …

Blood PlateletsAntioxidantCellsLipoproteinsmedicine.medical_treatmentMedicine (miscellaneous)Butyratemedicine.disease_causeLipid peroxidationchemistry.chemical_compoundmedicineAnimalsHumansNutritional Physiological Phenomenachemistry.chemical_classificationNutrition and DieteticsCholesterolFatty acidLipoproteins LDLOxidative StressCholesterolBiochemistrychemistryLipid PeroxidationHomeostasisOxidative stressLipoproteinProceedings of the Nutrition Society
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Oleanonic acid, a 3-oxotriterpene from Pistacia, inhibits leukotriene synthesis and has anti-inflammatory activity.

2001

One of the best known bioactive triterpenoids is oleanolic acid, a widespread 3-hydroxy-17-carboxy oleanane-type compound. In order to determine whether further oxidation of carbon 3 affects anti-inflammatory activity in mice, different tests were carried out on oleanolic acid and its 3-oxo-analogue oleanonic acid, which was obtained from Pistacia terebinthus galls. The last one showed activity on the ear oedema induced by 12-deoxyphorbol-13-phenylacetate (DPP), the dermatitis induced by multiple applications of 12-O-tetradecanoyl-13-acetate (TPA) and the paw oedemas induced by bradykinin and phospholipase A2. The production of leukotriene B4 from rat peritoneal leukocytes was reduced by ol…

Blood PlateletsLeukotrienesLeukotriene B4medicine.drug_classNeutrophilsBradykininTetrazolium SaltsIn Vitro TechniquesLeukotriene B4Anti-inflammatorychemistry.chemical_compoundMiceStructure-Activity RelationshipPhospholipase A2medicineAnimalsEdemaHumansCyclooxygenase InhibitorsHypersensitivity DelayedEar ExternalOleanolic AcidOleanolic acidPeroxidasePharmacologyInflammationLeukotrienebiologyFootAnti-Inflammatory Agents Non-SteroidalBiological activityTriterpenesRatsThiazoleschemistryBiochemistryArachidonate 5-lipoxygenasePistaciabiology.proteinFemaleDrug Screening Assays AntitumorOxidation-ReductionEuropean journal of pharmacology
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