Search results for "oxidative phosphorylation"

showing 10 items of 284 documents

Mitochondrial function and energy metabolism in neuronal HT22 cells resistant to oxidative stress

2014

Background and Purpose The hippocampal cell line HT22 is an excellent model for studying the consequences of endogenous oxidative stress. Extracellular glutamate depletes cellular glutathione by blocking the glutamate/cystine antiporter system xc−. Glutathione depletion induces a well-defined programme of cell death characterized by an increase in reactive oxygen species and mitochondrial dysfunction. Experimental Approach We compared the mitochondrial shape, the abundance of mitochondrial complexes and the mitochondrial respiration of HT22 cells, selected based on their resistance to glutamate, with those of the glutamate-sensitive parental cell line. Key Results Glutamate-resistant mitoch…

PharmacologyOligomycinATP synthaseCellular respirationOxidative phosphorylationMitochondrionBiologymedicine.disease_causechemistry.chemical_compoundMitochondrial permeability transition poreBiochemistrychemistrymedicinebiology.proteinATP–ADP translocaseOxidative stressBritish Journal of Pharmacology
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The effect of sea water on cytochrome oxidase and oxidative phosphorylation.

1961

Piccole quantita di acqua di mare causano una forte inibizione della fosforilazione ossidativa, mentre l'effetto sulla attivita citocromossidasica e piuttosto modesto. L'ione responsabile di questo effetto e il Calcio.

PharmacologybiologyChemistryWaterCell BiologyOxidative phosphorylationOxidative PhosphorylationElectron Transport Complex IVCellular and Molecular NeuroscienceMetabolismBiochemistrybiology.proteinMolecular MedicineCytochrome c oxidaseCytochromesSeawaterMolecular BiologyExperientia
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Cold exposure enhances fat utilization but not non-esterified fatty acids, glycerol or catecholamines availability during submaximal walking and runn…

2013

Cold exposure modulates the use of carbohydrates and fat during exercise. This phenomenon has mostly been observed in controlled cycling studies, but not during walking and running when core temperature and oxygen consumption are controlled, as both may alter energy metabolism. This study aimed at examining energy substrate availability and utilization during walking and running in the cold when core temperature and oxygen consumption are maintained. Ten lightly clothed male subjects walked or ran for 60-min, at 50% and 70% of maximal oxygen consumption, respectively, in a climatic chamber set at 0°C or 22°C. Thermal, cardiovascular, and oxidative responses were measured every 15-…

Physiologykylmäaltistuscold exposurerasvahapotchemistry.chemical_elementBlood lipidsOxidative phosphorylationglycerol030204 cardiovascular system & hematologyBiologyOxygenthermal responseslcsh:Physiologyrasva-aineenvaihdunta03 medical and health scienceschemistry.chemical_compound0302 clinical medicineAnimal sciencePhysiology (medical)fatHeart rateGlycerolglyseroliOriginal Research Articleta315Exerciselcsh:QP1-981kuormauskuormitusfat metabolismVO2 max030229 sport sciencesRespiratory quotientGlucosechemistryBiochemistrykatakolamiinitEnergy sourceEnergy Metabolismkatekoliamiinit
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Cytotoxic effects of oxysterols associated with human diseases: Induction of cell death (apoptosis and/or oncosis), oxidative and inflammatory activi…

2009

Oxysterols resulting from spontaneous or enzymatic oxidation of cholesterol are present in numerous foodstuffs and have been identified at increased levels in the plasma and the vascular walls of patients with cardiovascular diseases, especially in atherosclerotic lesions. Consequently, their role in lipid disorders is widely suspected, but they may also contribute to the development of important degenerative diseases such as Alzheimer's disease, Parkinson's disease, multiple sclerosis, osteoporosis, age-related macular degeneration, and cataract. Since these pathologies can be associated with the presence of apoptotic cells, oxidative and inflammatory processes, and lipid disorders, the ab…

Programmed cell deathClinical BiochemistryInflammationApoptosisOxidative phosphorylationPharmacologyBiologyLipidosesBiochemistrychemistry.chemical_compoundMacular DegenerationNeoplasmspolycyclic compoundsmedicineCytotoxic T cellHumansMolecular BiologyKetocholesterolsPhospholipidsPhospholipidosisInflammationCholesterolGeneral MedicineAtherosclerosisHydroxycholesterolsOxidative StresschemistryApoptosisImmunologyMolecular Medicinelipids (amino acids peptides and proteins)Signal transductionmedicine.symptomMolecular aspects of medicine
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Side effects of oxysterols: cytotoxicity, oxidation, inflammation, and phospholipidosis.

2008

Oxysterols are 27-carbon atom molecules resulting from autoxidation or enzymatic oxidation of cholesterol. They are present in numerous foodstuffs and have been demonstrated to be present at increased levels in the plasma of patients with cardiovascular diseases and in atherosclerotic lesions. Thus, their role in lipid disorders is widely suspected, and they might also be involved in important degenerative diseases such as Alzheimer's disease, osteoporosis, and age-related macular degeneration. Since atherosclerosis is associated with the presence of apoptotic cells and with oxidative and inflammatory processes, the ability of some oxysterols, especially 7-ketocholesterol and 7β-hydrox…

Programmed cell deathPhysiologyImmunologyBiophysicsInflammationApoptosisOxidative phosphorylationPharmacologyLipidosesBiochemistryPhospholipidosischemistry.chemical_compoundmedicinepolycyclic compoundsAnimalsHumansGeneral Pharmacology Toxicology and PharmaceuticsCytotoxicitylcsh:QH301-705.5PhospholipidsPhospholipidosisInflammationlcsh:R5-920ChemistryCholesterolGeneral NeuroscienceCell BiologyGeneral MedicineOxysterolsAtherosclerosisHydroxycholesterolsBiochemistrylcsh:Biology (General)Apoptosislipids (amino acids peptides and proteins)medicine.symptomSignal transductionlcsh:Medicine (General)Oxidation-ReductionBrazilian journal of medical and biological research = Revista brasileira de pesquisas medicas e biologicas
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Saccharomyces cerevisiae Cytosolic Thioredoxins Control Glycolysis, Lipid Metabolism, and Protein Biosynthesis under Wine-Making Conditions.

2019

Thioredoxins are small proteins that regulate the cellular redox state, prevent oxidative damage, and play an active role in cell repair. Oxidative stress has proven to be of much relevance in biotechnological processes when the metabolism of Saccharomyces cerevisiae is mainly respiratory. During wine yeast starter production, active dry yeast cytosolic thioredoxin Trx2p is a key player in protecting metabolic enzymes from being oxidized by carbonylation. Less is known about the role of redox control during grape juice fermentation. A mutant strain that lacked both cytosolic thioredoxins, Trx1p and Trx2p, was tested for grape juice fermentation. Its growth and sugar consumption were greatly…

ProteomicsSaccharomyces cerevisiae ProteinsSaccharomyces cerevisiaethioredoxin-thioredoxin reductase systemsyeastsWineOxidative phosphorylationSaccharomyces cerevisiaeApplied Microbiology and Biotechnology03 medical and health sciencesCytosolThioredoxinsYeastsMetabolomicsVitis030304 developmental biology0303 health sciencesEcologybiology030306 microbiologyChemistryfood and beveragesMembrane ProteinsLipid metabolismMetabolismPeroxiredoxinsglycolysisbiology.organism_classificationLipid MetabolismmetabolomicsYeastYeast in winemakingOxidative StressBiochemistryProtein BiosynthesisFermentationFood MicrobiologyFermentationThioredoxinThioredoxin-thioredoxin reductase systemsGlycolysisOxidation-ReductionGene DeletionFood ScienceBiotechnology
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Mitochondrial proteomics profile points oxidative phosphorylation as main target for beauvericin and enniatin B mixture

2020

Abstract Beauvericin (BEA) and enniatin B (EN B) are non-legislated Fusarium mycotoxins usually found in cereal and cereal-based products all around the world. By the proteomic analysis of mitochondria enriched extracts from Jurkat cells exposed for 24 h to three concentrations of BEA:EN B (0.01–0.1–0.5 μM), a number of 1821 proteins (202 mitochondrial) were identified and relatively quantified. 340 proteins (59 mitochondrial) were statistically significant altered in our samples (Anova p-value ≤ 0.05 and fold change (FC) ≥1.5). The protein mitochondrial translational release factor 1 like (MTRF1L) was the most abundant protein in the three mycotoxin exposures studied. The mycotoxins mixtur…

ProteomicsTranscription GeneticOxidative phosphorylationMitochondrionToxicologyProteomicsRibosomeJurkat cellsOxidative PhosphorylationElectron TransportMitochondrial ProteinsJurkat Cells03 medical and health scienceschemistry.chemical_compound0404 agricultural biotechnologyDepsipeptidesHumans030304 developmental biology0303 health sciencesfood and beverages04 agricultural and veterinary sciencesGeneral Medicine040401 food scienceBeauvericinFold changeMitochondriachemistryBiochemistryBacterial outer membraneFood ScienceFood and Chemical Toxicology
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Mitochondrial complex I: new insights from inhibitor assays

2000

The NADH:ubiquinone oxidoreductase (complex I) of the mitochondrial respiratory chain is by far the most complicated of the proton-translocating enzymes involved in the oxidative phosphorylation. Many clues regarding both electron transfer and proton translocation are still unknown. In this sense, inhibitor assays are relevant and useful pieces for elaborating a suitable model to explain the elusive bioenergetic mechanism of this enzyme. This short review presents the most recent advances in inhibitor studies and highlights the major controversies.

Proton translocationchemistry.chemical_classificationNADH-Ubiquinone OxidoreductaseMechanism (biology)Cell BiologyPlant ScienceGeneral MedicineOxidative phosphorylationBiologyMitochondrial respiratory chainEnzymeBiochemistrychemistryOxidoreductaseMitochondrial Complex IProtoplasma
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Oxidative Stress and Respiratory System: Pharmacological and Clinical Reappraisal of N-Acetylcysteine

2014

Abstract The large surface area for gas exchange makes the respiratory system particularly susceptible to oxidative stress-mediated injury. Both endogenous and exogenous pro-oxidants (e.g. cigarette smoke) trigger activation of leukocytes and host defenses. These mechanisms interact in a ìmultilevel cycleî responsible for the control of the oxidant/antioxidant homeostasis. Several studies have demonstrated the presence of increased oxidative stress and decreased antioxidants (e.g. reduced glutathione [GSH]) in subjects with chronic obstructive pulmonary disease (COPD), but the contribution of oxidative stress to the pathophysiology of COPD is generally only minimally discussed. The aim of t…

Pulmonary and Respiratory MedicineChronic ObstructiveAntioxidantantioxidantNeutrophilsmedicine.medical_treatmentAntioxidant; Copd exacerbation; Lung function; Small airways; Acetylcysteine; Antioxidants; Bronchitis Chronic; Disease Progression; Expectorants; Forced Expiratory Volume; Hospitalization; Humans; Macrophages; Neutrophils; Pulmonary Disease Chronic Obstructive; Reactive Oxygen Species; Respiratory Physiological Phenomena; Oxidative Stress; Pulmonary and Respiratory MedicineAntioxidant; Copd exacerbation; Lung function; Small airways; Acetylcysteine; Antioxidants; Bronchitis Chronic; Disease Progression; Expectorants; Forced Expiratory Volume; Hospitalization; Humans; Macrophages; Neutrophils; Pulmonary Disease Chronic Obstructive; Reactive Oxygen Species; Respiratory Physiological Phenomena; Oxidative StressOxidative phosphorylationReviewSettore MED/10 - Malattie Dell'Apparato Respiratoriomedicine.disease_causeAntioxidantsAcetylcysteinePulmonary Diseasechemistry.chemical_compoundPulmonary Disease Chronic ObstructiveCOPD exacerbationForced Expiratory VolumemedicineHumansRespiratory systemChronicBronchitisExpectorantschemistry.chemical_classificationCOPDReactive oxygen speciessmall airwaysbusiness.industryMacrophageslung functionGlutathionemedicine.diseaseAcetylcysteineBronchitis ChronicHospitalizationOxidative StresschemistryImmunologyDisease ProgressionRespiratory Physiological PhenomenabusinessReactive Oxygen SpeciesOxidative stressmedicine.drug
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Oxidative and nitrosative stress in the diaphragm of patients with COPD.

2007

COPD is associated with an increased load on the diaphragm. Since chronic muscle loading results in changes in antioxidant capacity and formation of reactive oxygen and reactive nitrogen species, we hypothesized that COPD has a similar effect on the diaphragm, which is related to the severity of COPD. Catalase activity was determined spectrophotometrically. Levels of 4-hydroxy-2-nonenal (HNE)-protein adducts and 3-nitrotyrosine (NT) formation were measured using western blotting. Levels of malondialdehyde (MDA) were assessed by high-performance liquid chromatography. We found that catalase activity was approximately 89% higher in the diaphragm of severe COPD patients (FEV1 37+/-5% predicted…

Pulmonary and Respiratory Medicinemedicine.medical_specialtyrespiratory musclesNitrosationDiaphragmOxidative phosphorylationmedicine.disease_causeSeverity of Illness IndexNOLipid peroxidationPulmonary Disease Chronic Obstructivechemistry.chemical_compoundInternal medicineHumansCOPDMedicineReactive nitrogen speciesOriginal ResearchCOPDoxidantsbiologybusiness.industryHealth PolicyPublic Health Environmental and Occupational HealthGeneral MedicineMiddle Agedmedicine.diseaseMalondialdehydeDiaphragm (structural system)Surgeryrespiratory tract diseasesOxidative StressantioxidantsEndocrinologychemistryCatalasebiology.proteinbusinessOxidative stressInternational journal of chronic obstructive pulmonary disease
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