Search results for "oxidative stress."

showing 10 items of 1575 documents

Deficiency of Nrf2 accelerates the effector phase of arthritis and aggravates joint disease

2011

14 páginas, 8 figuras, 1 tabla.-- et al.

musculoskeletal diseasesGenetically modified mouseMedicinaNF-E2-Related Factor 2PhysiologyChemokine CXCL1Clinical BiochemistryNitric Oxide Synthase Type IIArthritisMice Transgenicmedicine.disease_causeenvironment and public healthBiochemistryNrf2MicemedicineAnimalsMolecular BiologyGeneral Environmental SciencebiologyInterleukin-6Effectorbusiness.industryArthritisInflammation and degenerationCell Biologyrespiratory systemmedicine.diseaseArthritis ExperimentalInfection and autoimmunity Auto-immunity transplantation and immunotherapy [NCMLS 1]Disease Models AnimalOxidative StressEicosanoidCyclooxygenase 2Rheumatoid arthritisTumor Necrosis FactorsImmunologyOsteocalcinbiology.proteinGeneral Earth and Planetary SciencesJointsTumor necrosis factor alphaImmune Regulation Auto-immunity transplantation and immunotherapy [NCMLS 2]businessOxidation-ReductionHeme Oxygenase-1Oxidative stress
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An update on the relationships between rheumatoid arthritis and atherosclerosis

2010

Rheumatoid arthritis is a chronic inflammatory disease. Cardiovascular events are the most important cause of mortality and morbidity in patients with rheumatoid arthritis. Beyond the traditional cardiovascular risk factors, chronic systemic inflammation has been shown to be a crucial factor in atherosclerosis development and progression from endothelial dysfunction to plaque rupture and thrombosis. Many studies have shown that atherosclerosis is not a passive event like accumulation of lipids in the vessel walls; by contrast, it represents an active inflammation of the vessels. Inflammatory cells such as macrophages, monocytes and T cells play important roles in the development of both rhe…

musculoskeletal diseasesT-LymphocytesAtherosclerosis Rheumatoid arthritis Inflammation Cardiovascular risk10265 Clinic for Endocrinology and DiabetologyInflammation610 Medicine & healthComorbiditySystemic inflammationModels BiologicalMonocytes2705 Cardiology and Cardiovascular MedicineArthritis RheumatoidRisk FactorsImmunopathologymedicineHumansEndothelial dysfunctionAutoimmune diseaseInflammationbusiness.industryVascular diseaseMacrophagesmedicine.diseaseAtherosclerosisThrombosisOxidative StressCardiovascular DiseasesRheumatoid arthritisImmunologyDisease Progressionmedicine.symptomInsulin ResistanceCardiology and Cardiovascular Medicinebusiness
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A global DNA repair mechanism involving the Cockayne syndrome B (CSB) gene product can prevent the in vivo accumulation of endogenous oxidative DNA b…

2002

The Cockayne syndrome B (CSB) gene product is involved in the repair of various types of base modifications in actively transcribed DNA sequences. To investigate its significance for the repair of endogenous oxidative DNA damage, homozygous csb(-/-)/ogg1(-/-) double knockout mice were generated. These combine the deficiency of CSB with that of OGG1, a gene coding for the mammalian repair glycosylase that initiates the base excision repair of 7,8-dihydro-8-oxoguanine (8-oxoG). Compared to ogg1(-/-) mice, csb(-/-)/ogg1(-/-) mice were found to accumulate with age severalfold higher levels of oxidited purine modifications in hepatocytes, splenocytes and kidney cells. In contrast, the basal (ste…

musculoskeletal diseasescongenital hereditary and neonatal diseases and abnormalitiesCancer ResearchDNA RepairTranscription GeneticDNA damageDNA repairBiologyGene productMicechemistry.chemical_compoundGeneticsAnimalsPoly-ADP-Ribose Binding ProteinsMolecular BiologyGeneDNA PrimersMice KnockoutBase SequenceHomozygoteDNA HelicasesDeoxyguanosinenutritional and metabolic diseasesBase excision repairMolecular biologyOxidative StressDNA Repair EnzymesBiochemistrychemistry8-Hydroxy-2'-DeoxyguanosineDNA glycosylaseDNADNA DamageNucleotide excision repairOncogene
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Effect of Non-Surgical Periodontal Treatment on Oxidative Stress Markers in Leukocytes and Their Interaction with the Endothelium in Obese Subjects w…

2020

Aim: The primary objective of this pilot study was to evaluate the effect of non-surgical periodontal treatment. The secondary aim was to evaluate the effect of dietary therapy on both parameters of oxidative stress in leukocytes and leukocyte-endothelial cell interactions in an obese population. Methods: This was a pilot study with a before-and-after design. Forty-nine obese subjects with periodontitis were randomized by means of the minimization method and assigned to one of two groups, one of which underwent dietary therapy while the other did not. All the subjects underwent non-surgical periodontal treatment. We determined periodontal, inflammatory and oxidative stress parameters&mdash

obesityPopulationlcsh:MedicineLeukocyte homeostasis030204 cardiovascular system & hematologyPharmacologymedicine.disease_causeArticleendothelial dysfunctionSuperoxide dismutase03 medical and health scienceschemistry.chemical_compound0302 clinical medicineoxidative stressMedicineEndothelial dysfunctioneducationperiodontitisreactive oxygen specieschemistry.chemical_classificationPeriodontitisReactive oxygen specieseducation.field_of_studydietary therapybiologybusiness.industrySuperoxidelcsh:R030206 dentistryGeneral Medicinemedicine.diseaseperiodontal treatmentchemistrybiology.proteinbusinessOxidative stressJournal of Clinical Medicine
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Study of the mechanisms of oxidative stress, mitochondrial function and endoplasmic reticulum stress in obesity. Role in subclinical atherosclerosis …

2019

El aumento alarmante de la prevalencia de obesidad a nivel mundial y la enorme carga de enfermedad asociada ha generado una creciente demanda de estrategias para frenar la epidemia y / o mitigar los efectos dañinos de la obesidad sobre la salud. Para ello se requiere un compromiso firme y sinérgico por parte de todas las fuerzas sociales, no sólo para desarrollar estrategias preventivas, sino también para contribuir al conocimiento de los eventos patológicos y los mecanismos involucrados en el desarrollo de esta enfermedad. Por ello, el objetivo principal de la presente tesis doctoral es profundizar en los mecanismos subyacentes de la obesidad, especialmente en aquellos relacionados con el …

obesityleukocytes activationUNESCO::CIENCIAS DE LA VIDA::Biología molecularchronic periodontitis:CIENCIAS MÉDICAS ::Medicina interna::Endocrinología [UNESCO]pinitolendothelial dysfunction:CIENCIAS DE LA VIDA::Biología molecular [UNESCO]mitochondriaUNESCO::CIENCIAS MÉDICAS ::Patología::Arteriosclerosisinflammationcardiovascular diseasebotanical compoundsoxidative stresscaloric restriction:CIENCIAS DE LA VIDA::Fisiología humana ::Fisiología endocrina [UNESCO]:CIENCIAS MÉDICAS ::Patología::Arteriosclerosis [UNESCO]metabolismUNESCO::CIENCIAS MÉDICAS ::Medicina interna::EndocrinologíaUNESCO::CIENCIAS DE LA VIDA::Fisiología humana ::Fisiología endocrina
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Oxidative stress and antioxidant levels in patients with anorexia nervosa: A systematic review and exploratory meta-analysis.

2015

Objective To systematically review and meta-analyze oxidative stress and antioxidant markers in anorexia nervosa (AN). Methods Electronic PubMed search from database inception until 12/31/2013. Out of 1062 hits, 29 studies comparing oxidative stress/antioxidant markers between patients with AN and healthy controls (HCs) with a total of 1,729 participants (AN-=-895, HCs-=-834) were eligible. Data about oxidative stress and antioxidant markers, independent of their source, were extracted. We calculated random effects standardized mean differences (SMDs) as effect size measures for outcomes reported in ≥5 studies; others were summarized descriptively. Results Compared to HCs, AN patients showe…

oxidative streantioxidantAnorexia NervosaAntioxidantsHealthy VolunteersOxidative StressYoung AdultCross-Sectional StudiesPsychiatry and Mental Healthhealthy controlsHumansFemaleanorexia nervosa; antioxidant; healthy controls; oxidative stress; Anorexia Nervosa; Antioxidants; Biomarkers; Cross-Sectional Studies; Female; Healthy Volunteers; Humans; Oxidative Stress; Young Adult; Psychiatry and Mental HealthBiomarkersThe International journal of eating disorders
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Oxidative Stress and Antioxidant Levels in Patients with Anorexia Nervosa after Oral Re-alimentation: A Systematic Review and Exploratory Meta-analys…

2015

Oxidative stress markers seem to be higher in patients with anorexia nervosa (AN) than healthy controls, but the potentially beneficial effects of weight gain is not known. We calculated random effects standardised mean differences (SMDs) as effect size measures of oxidative stress marker changes after re-alimentation reported in two or more studies, summarising others descriptively. Seven longitudinal studies (n = 104) were included. After a median follow-up period of 8 weeks, AN patients significantly increased their body mass index (15.1 ± 2.1 to 17.1 ± 2.2, p < 0.0001). This weight gain was followed by a significant increase in serum levels of the antioxidant albumin (studies = 6, SM…

oxidative streantioxidantSuperoxide DismutaseMedicine (all)weight gainCatalaseAntioxidantsanorexia nervosaOxidative StressClinical PsychologyPsychiatry and Mental HealthAlbuminsApolipoprotein B-100albumin; anorexia nervosa; antioxidant; ApoB; oxidative stress; refeeding; weight gain; Albumins; Anorexia Nervosa; Antioxidants; Apolipoprotein B-100; Biomarkers; Catalase; Humans; Longitudinal Studies; Superoxide Dismutase; Weight Gain; Oxidative Stress; Clinical Psychology; Psychiatry and Mental Health; Medicine (all)HumansLongitudinal StudiesApoBApoB; albumin; anorexia nervosa; antioxidant; oxidative stress; refeeding; weight gainBiomarkersalbuminrefeeding
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Correlation of Metabolic Syndrome with Redox Homeostasis Biomarkers: Evidence from High-Fat Diet Model in Wistar Rats

2022

Metabolic Syndrome (MetS) is an extremely complex disease. A non-balanced diet such as high-fat diet (HFD) induces metabolic dysfunction that could modify redox homeostasis. We here aimed at exploring redox homeostasis in male Wistar rats, following 8 weeks of HFD, correlating the eventual modification of selected biomarkers that could be associated with the clinical manifestations of MetS. Therefore, we selected parameters relative to both the glucose tolerance and lipid altered metabolism, but also oxidative pattern. We assessed some biomarkers of oxidative stress i.e., thiols balance, lipid peroxidation and antioxidant barriers, via the use of specific biochemical assays, individuating e…

oxidative streglucose toleranceSettore BIO/16 - Anatomia UmanaPhysiologyClinical Biochemistrynon-alcoholic fatty liver diseaseCell BiologySettore BIO/09 - FisiologiaBiochemistryoxidative stress; anti-oxidant barriers; glucose tolerance; lipid metabolism; non-alcoholic fatty liver disease; adipose tissue distributionanti-oxidant barrierlipid metabolismMolecular Biologyadipose tissue distributionAntioxidants
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Changes in plasmatic redox status following low-moderate interval exercise training in rhythmic gymnastics trainers

2013

oxidative stress antioxidant capacity exercise training exercise intensity rhytmic gymnastics
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SICILIAN MANGO PEEL INDUCES CELLULAR STRESS ACCOMPANIED TO MITOCHONDRIAL DYSFUNCTION IN COLON CANCER CELLS

2021

Currently, cellular stresses as the oxidative, metabolic and genotoxic stress are considered the cause of many different human pathologies as neurodegenerative diseases (e.g.,Alzheimer’s, Parkinson’s, amyotrophic lateral sclerosis), alcoholic liver disease, chronic obstructive pulmonary disease, and also cancer. Although the role of cellular stress has been largely debated in cancer, nowadays some therapies aim to target the intracellular pro-oxidant/anti-oxidant balance triggering the tumor commitment to cell death. Therefore, it has become more necessary an improved understanding of cancer response to cellular stress that could be advantageous to develop cancer tailored therapies. In this…

oxidative stress colon cancer cells mitochondria.
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