Search results for "oxides"

showing 10 items of 547 documents

Impairment of the extrusion transporter for asymmetric dimethyl-L-arginine: a novel mechanism underlying vasospastic angina.

2012

Abstract A 37-year old male patient presented with frequent angina attacks (up to 40/day) largely resistant to classical vasodilator therapy. The patient showed severe coronary and peripheral endothelial dysfunction, increased platelet aggregation and increased platelet-derived superoxide production. The endothelial nitric oxide synthase (eNOS)-inhibitor N G -nitro- l -arginine methyl ester (L-NAME) reduced superoxide formation in platelets identifying “uncoupled” eNOS as a superoxide source. Oral l -arginine normalized coronary and peripheral endothelial dysfunction and reduced platelet aggregation and eNOS-derived superoxide production. Plasma concentrations of the endogenous NOS inhibito…

AdultBlood PlateletsMalemedicine.medical_specialtyArginineNitric Oxide Synthase Type IIIBiophysicsCoronary VasospasmVasodilationArginineBiochemistryPeripheral blood mononuclear cellAngina Pectorischemistry.chemical_compoundEnosSuperoxidesInternal medicinemedicineHumansPlateletEndothelial dysfunctionEnzyme InhibitorsMolecular BiologybiologyChemistrySuperoxideCell Biologymedicine.diseasebiology.organism_classificationEndocrinologyNG-Nitroarginine Methyl EsterEndothelium VascularIntracellularBiochemical and biophysical research communications
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Hematologic effects of recombinant human granulocyte colony-stimulating factor in patients with malignancy.

1989

Abstract The effect of recombinant human granulocyte colony-stimulating factor (G-CSF) on hematologic parameters was evaluated in a phase I clinical study in 18 patients with advanced malignancy. G-CSF was administered once daily as a 30-minute infusion for 14 days; three patients each were treated at increasing dose levels of 1, 3, 10, 30, and 60 micrograms kg-1 day-1. A transient decrease in neutrophil and monocyte counts was observed immediately after the G-CSF infusion, followed by a dose-dependent increase of up to 15-fold. G-CSF-induced neutrophils exhibited an increased O2- radical production, and serum levels of enzymes related to granulocyte turnover, including lysozyme and elastas…

AdultBlood Plateletsmedicine.medical_specialtySide effectImmunologyAntineoplastic AgentsPlatelet Membrane GlycoproteinsGranulocyteMalignancyBiochemistryLeukocyte CountColony-Stimulating FactorsSuperoxidesInternal medicineGranulocyte Colony-Stimulating FactormedicineHumansPlateletBone painAgedbusiness.industryPlatelet CountMonocyteElastaseReceptors Interleukin-2Cell BiologyHematologyMiddle Agedmedicine.diseaseRecombinant ProteinsGranulocyte colony-stimulating factorHematopoiesisEndocrinologymedicine.anatomical_structureImmunologyDrug Evaluationmedicine.symptombusinessBlood
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Solitary necrotic nodules of the liver: cross-sectional imaging findings and follow-up in nine patients.

2008

OBJECTIVE. The purpose of our study was to retrospectively evaluate the sonographic, CT, and MRI findings (number, diameter, lobar location, depth from the hepatic capsule, and appearance of lesions) in a series of nine patients with pathologically proven solitary necrotic nodules of the liver and the natural evolution at follow-up in four of the nine patients.CONCLUSION. Solitary necrotic nodules are usually small, solitary lesions, mainly located under the liver capsule of the right lobe. They are hypoechoic on sonography, hypoattenuating on CT, have low signal intensity on both T1- and T2-weighted MRI with lack of enhancement after IV contrast administration, and at follow-up have a tend…

AdultGadolinium DTPAMalemedicine.medical_specialtyIohexolIronContrast MediaCross-sectional imagingNecrosisImage Interpretation Computer-AssistedmedicineHumansRadiology Nuclear Medicine and imagingLiver neoplasmMagnetite NanoparticlesAgedRetrospective StudiesFibrous capsule of Glissonmedicine.diagnostic_testbusiness.industryHepatic capsuleLiver NeoplasmsMagnetic resonance imagingDextransOxidesUltrasonography DopplerGeneral MedicineMiddle Agedmedicine.diseaseMagnetic Resonance ImagingLobeFerrosoferric Oxideliver metastases; liver neoplasm; solitary necrotic nodulesmedicine.anatomical_structureLiverFemaleRadiologyTomographyNuclear medicinebusinessTomography Spiral ComputedCalcificationFollow-Up StudiesAJR. American journal of roentgenology
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Genetic alterations and oxidative metabolism in sporadic colorectal tumors from a Spanish community

1997

Deletions of loci on chromosomes 5q, 17p, 18q, and 22q, together with the incidence of p53 mutations and amplification of the double minute-2 gene were investigated in the sporadic colorectal tumors of 44 patients from a Spanish community. Chromosome deletions were analyzed by means of loss of heterozygosity analysis using a restriction fragment length polymorphism assay. Allelic losses were also detected by polymerase chain reaction (PCR)-single-stranded conformation polymorphism (SSCP) analysis of a polymorphic site in intron 2 of the p53 gene. The percentages of genetic deletions on the screened chromosomes were 39.3% (5q), 58.3% (17p), 40.9% (18q), and 40% (22q). Mutations in p53 exons …

AdultGenetic MarkersMaleGenome instabilityHeterozygoteLipid PeroxidesCancer ResearchChromosomes Human Pair 22DNA Mutational AnalysisAdenocarcinomaBiologymedicine.disease_causeLoss of heterozygosityProto-Oncogene ProteinsGene duplicationmedicineHumansMolecular BiologyGenePolymorphism Single-Stranded ConformationalAgedSequence DeletionGene AmplificationDeoxyguanosineNuclear ProteinsProto-Oncogene Proteins c-mdm2Single-strand conformation polymorphismDNA NeoplasmMiddle AgedGenes p53GlutathioneMolecular biology8-Hydroxy-2'-DeoxyguanosineChromosomes Human Pair 1SpainGenetic markerChromosomes Human Pair 5FemaleRestriction fragment length polymorphismChromosomes Human Pair 18Colorectal NeoplasmsCarcinogenesisOxidation-Reduction
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Comparison of diffusion, cytotoxicity and tissue inflammatory reactions of four commercial bleaching products against human dental pulp stem cells

2018

AbstractMultiple side effects related to bleaching were found to occur in the dental pulp tissue, including decreased cell metabolism and viability. In this work we evaluated the in vitro diffusion capacity, cytotoxicity and biocompatibility of four commercial bleaching products on stem cells from human dental pulp (hDPSCs). Two commercial bleaching gels hydrogen peroxide-based (HP), Norblanc Office 37.5% (Nor-HP) and Opalescence Boost 40% (Opal-HP) were applied for 30 min to enamel/dentine discs. Another two gels from the same manufacturers, 16% carbamide peroxide-based (CP), Norblanc Home (Nor-CP) and Opalescence CP 16% (Opal-CP), were applied for 90 min. The diffusion of HP was analysed …

AdultMale0301 basic medicineNecrosisBiocompatibilityScienceCarbamide PeroxideArticleDiffusionTooth whiteningYoung Adult03 medical and health scienceschemistry.chemical_compoundDental biomaterials0302 clinical medicineAnti-Infective Agentsstomatognathic systemDental pulp stem cellsTooth BleachingmedicineAnimalsHumansRats WistarTooth Bleaching AgentsHydrogen peroxideCytotoxicityDental PulpInflammationMultidisciplinaryEnamel paintStem CellsQRHydrogen PeroxideMolecular biologyPeroxidesRatsstomatognathic diseases030104 developmental biologychemistryvisual_artToxicityMicroscopy Electron Scanningvisual_art.visual_art_mediumMedicinePulp (tooth)Femalemedicine.symptom030217 neurology & neurosurgeryScientific Reports
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How we prevent and treat differentiation syndrome in patients with acute promyelocytic leukemia

2014

Abstract Differentiation syndrome (DS), formerly known as retinoic acid syndrome, is a relatively common and potentially severe complication seen in patients with acute promyelocytic leukemia treated with all-trans retinoic acid and/or arsenic trioxide. The full-blown syndrome consists of unexplained fever, weight gain, dyspnea with pulmonary infiltrates, pleuropericardial effusion, hypotension, and renal failure. Most measures currently used for management of DS have very little evidence-based support, and therefore, many remain controversial. Despite the lack of evidence supporting DS prophylaxis, several groups have adopted a preventive strategy with corticosteroids, especially for patie…

AdultMaleAcute promyelocytic leukemiamedicine.medical_specialtyDrug-Related Side Effects and Adverse ReactionsPremedicationImmunologyTretinoinBiochemistryArsenicalslaw.inventionchemistry.chemical_compoundArsenic TrioxideLeukemia Promyelocytic AcutelawTretinoinInternal medicinemedicineHumansArsenic trioxideIntensive care medicineDexamethasonebusiness.industryOxidesSyndromeCell BiologyHematologymedicine.diseaseIntensive care unitDiscontinuationRetinoic acid syndromeLeukemiachemistryFemalebusinessmedicine.drugBlood
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Oxidative stress markers at birth: Analyses of a neonatal population

2015

In order to further understand neonatal stress and, thus, control it efficaciously, there is a need for more information on the manifestations of stress at the molecular level in the newborn, with particular regard to oxidants, and anti-oxidant and anti-stress mechanisms, including mitochondrial heat shock protein-chaperones such as Hsp60. We investigated patterns of anti-oxidants, biomarkers of oxidative stress, and Hsp60 levels in sera from newborns and found significant associations between glutathione (GSH) levels and gestational age, delivery modality, and lipid hydroperoxydes (LOOH) level. LOOH levels and spontaneous (vaginal) delivery were independently associated with increased GSH …

AdultMaleLipid Peroxidesanimal structuresHistologyNeonatal stressPopulationNeonatal strePhysiologyOxidative-stress markerDiseaseBiologymedicine.disease_causeMitochondrial Proteinschemistry.chemical_compoundLipid hydroperoxydemedicineHumanseducationOxidative-stress markerseducation.field_of_studyfungiInfant NewbornAnti-stress moleculeGestational ageChaperonin 60Cell BiologyGeneral MedicineGlutathioneHsp60GlutathioneNeonatal stress; Oxidative-stress markers; Lipid hydroperoxydes; Anti-stress molecules; Glutathione; Hsp60Oxidative StressAdult lifeLipid hydroperoxydeschemistryAnti-stress moleculesImmunologyFemaleHSP60BiomarkersOxidative stressNeonatal stressActa Histochemica
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Leukocytes in neuronal ceroid-lipofuscinoses: function and apoptosis

1997

The neuronal ceroid-lipofuscinoses (NCL) are a group of progressive encephalopathies with a fatal course that are mostly of autosomal recessive inheritance. The pathophysiological mechanisms causing the diseases are not known. The characteristic histomorphological feature of the NCL is an abnormal lysosomal accumulation of lipopigments in neural and extraneural cells, including peripheral blood leukocytes. We studied the function of peripheral venous blood immunocompetent cells in ten patients with NCL and in age- and sex-matched controls to determine how, if at all, the accumulation of intracytoplasmic storage material influences the functional capacity of affected tissue. Our results did …

AdultMaleProgrammed cell deathPathologymedicine.medical_specialtyAdolescentmedicine.medical_treatmentImmunoglobulinsApoptosisImmunoglobulin EImmunophenotypingPathogenesisDevelopmental NeuroscienceNeuronal Ceroid-LipofuscinosesSuperoxidesLeukocytesmedicineHumansChildRespiratory BurstbiologyInterleukin-6Interleukin-8General Medicinemedicine.diseasePathophysiologyCytokineApoptosisChild PreschoolPediatrics Perinatology and Child HealthImmunologybiology.proteinFemaleNeuronal ceroid lipofuscinosisNeurology (clinical)AntibodyBiomarkersCell DivisionInterleukin-1Brain and Development
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Evidence of noncentrosymmetry of human tooth hydroxyapatite crystals

2014

Herein, we investigate human single hydroxyapatite crystals (enamel and dentine) by convergent-beam electron diffraction (CBED) and automated electron-diffraction tomography (ADT). The CBED pattern shows the absence of the mirror plane perpendicular to the c axis leading to the P63 space group instead of the P63 /m space group considered for larger-scale crystals, this is confirmed by ADT. This experimental evidence is of prime importance for understanding the morphogenesis and the architectural organization of calcified tissues.

AdultMaleatomic structuresCatalysisX-Ray DiffractionHuman toothmedicineHydroxidesHumansDental Enamelatomic structures; biomineralization; human tooth crystals; noncentrosymmetry; piezoelectricityEnamel paintpiezoelectricityChemistryOrganic ChemistrySpectrometry X-Ray EmissionGeneral Chemistrybiomineralizationnoncentrosymmetrystomatognathic diseasesCrystallographymedicine.anatomical_structureDurapatiteElectron diffractionvisual_artDentinvisual_art.visual_art_mediumNanoparticleshuman tooth crystalsFemaleMirror plane
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Effects of the association of aging and obesity on lipids, lipoproteins and oxidative stress biomarkers: a comparison of older with young men.

2011

In this study, plasma lipids, lipoproteins and markers of oxidant/antioxidant status were investigated in young (n = 45) and older (n = 40) obese men and compared to those in young (n = 65) and older (n = 55) normal weight controls. The purpose was to determine whether obesity exacerbates or not lipid, lipoprotein abnormalities and oxidative stress in older men. Our findings showed that all obese patients had increased plasma triglyceride, cholesterol, LDL-cholesterol, -triglyceride and HDL-triglyceride levels concentrations compared to controls (P < 0.01). However, the younger obese men had relatively larger and accentuated changes in plasma lipids and lipoproteins than the older patients.…

AdultMalemedicine.medical_specialtyAgingLipid PeroxidesAntioxidantEndocrinology Diabetes and Metabolismmedicine.medical_treatmentLipoproteinsMedicine (miscellaneous)medicine.disease_causeAntioxidantsBody Mass IndexProtein Carbonylationchemistry.chemical_compoundInternal medicineMedicineHumansObesityAgedchemistry.chemical_classificationNutrition and DieteticsTriglyceridebusiness.industryCholesterolSuperoxide DismutaseGlutathione peroxidaseMiddle Agedmedicine.diseaseObesityLipidsOxidative StressEndocrinologychemistrylipids (amino acids peptides and proteins)Cardiology and Cardiovascular MedicinebusinessBody mass indexOxidative stressBiomarkersLipoproteinNutrition, metabolism, and cardiovascular diseases : NMCD
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