Search results for "pathogen"

showing 10 items of 1657 documents

Immune activation promotes evolutionary conservation of T-cell epitopes in HIV-1.

2013

The immune system should constitute a strong selective pressure promoting viral genetic diversity and evolution. However, HIV shows lower sequence variability at T-cell epitopes than elsewhere in the genome, in contrast with other human RNA viruses. Here, we propose that epitope conservation is a consequence of the particular interactions established between HIV and the immune system. On one hand, epitope recognition triggers an anti-HIV response mediated by cytotoxic T-lymphocytes (CTLs), but on the other hand, activation of CD4(+) helper T lymphocytes (TH cells) promotes HIV replication. Mathematical modeling of these opposite selective forces revealed that selection at the intrapatient l…

Helper T lymphocyteQH301-705.5HIV AntigensEpitopes T-LymphocyteHIV InfectionsImmunodominanceBiologyVirus ReplicationGeneral Biochemistry Genetics and Molecular BiologyEpitopeEvolution Molecular03 medical and health sciencesImmune systemCytotoxic T cellHumansComputer SimulationAmino Acid SequenceBiology (General)BiologyConserved Sequence030304 developmental biologyImmune Evasion0303 health sciencesImmunity CellularGeneral Immunology and MicrobiologyModels Genetic030306 microbiologyGeneral NeuroscienceGenetic VariationViral LoadVirology3. Good healthEpitope mappingHIV AntigensViral replicationImmunologyHost-Pathogen InteractionsSynopsisHIV-1General Agricultural and Biological SciencesAlgorithms
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Hemocytes of Rhynchophorus ferrugineus (Olivier)(Coleoptera: Curculionidae) and their response to Saccharomyces cerevisiae and Bacillus thuringiensis

2011

Originally from tropical Asia, the Red Palm Weevil (RPW Rhynchophorus ferrugineus (Olivier) is the most dangerous and deadly pest of many palm trees, and there have been reports of its recent detection in France, Greece and Italy. At present, emphasis is on the development of integrated pest management based on biological control rather than on chemical insecticides, however the success of both systems is often insufficient. In this regard, RPW appears to be one pest that is very difficult to control. Thus inves- tigations into the natural defences of this curculionid are advisable. RPW hemocytes, the main immuno- competent cells in the insect, are described for the first time. We identifie…

HemocytesRed Palm Weevil Infection Insect immunity Hemolymph Biological controlBacillus thuringiensisBiological pest controlSettore BIO/05 - ZoologiaSaccharomyces cerevisiaeBiologyMicrobiologyPhagocytosisHemolymphBacillus thuringiensisBotanyHemolymphAnimalsPest Control BiologicalEcology Evolution Behavior and SystematicsLarvaWeevilfungibiology.organism_classificationBlood Cell CountBiopesticideRhynchophorusSettore AGR/11 - Entomologia Generale E ApplicataLarvaHost-Pathogen InteractionsWeevilsPEST analysis
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Hepatitis viruses: live and let die.

2007

Viral hepatitis is a diffuse inflammatory reaction of the liver caused by hepatotropic viruses. Among the hepatitis viruses, only hepatitis B virus and hepatitis C virus are able to persist in the host and cause chronic hepatitis. In the course of persistent infection, inflammation forms the pathogenetic basis of chronic hepatitis that can lead to nodular fibrosis, which can progress to cirrhosis and, eventually, hepatocellular carcinoma (HCC). Of the different antiviral defense systems employed by the host, apoptosis significantly contributes to the prevention of viral replication, dissemination, and persistence. Pathomorphologic studies have shown acidophilic bodies and hepatocyte dropout…

Hepatitis B virusHepatitis B virusHepatologyHepatitis C virusViral pathogenesisApoptosisHepacivirusHepatitis BBiologymedicine.diseasemedicine.disease_causeHepatitis BVirologyHepatitis CLiver diseaseViral replicationCytopathogenic Effect ViralLiverHepatocellular carcinomaImmunologymedicineHepatocytesAnimalsHumansViral hepatitisLiver international : official journal of the International Association for the Study of the Liver
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Hepatitis B subviral envelope particles use the COPII machinery for intracellular transport via selective exploitation of Sec24A and Sec23B

2020

Hepatitis B virus (HBV) is a leading cause of liver disease. Its success as a human pathogen is related to the immense production of subviral envelope particles (SVPs) contributing to viral persistence by interfering with immune functions. To explore cellular pathways involved in SVP formation and egress, we investigated host-pathogen interactions. Yeast-based proteomics revealed Sec24A, a component of the coat protein complex II (COPII), as an interaction partner of the HBV envelope S domain. To understand how HBV co-opts COPII as a proviral machinery, we studied roles of key Sec proteins in HBV-expressing liver cells. Silencing of Sar1, Sec23, and Sec24, which promote COPII assembly conco…

Hepatitis B virusImmunology610 MedizinVesicular Transport ProteinsBiologymedicine.disease_causeProteomicsEndoplasmic ReticulumMicrobiologyCell Line03 medical and health sciencesDownregulation and upregulationTranscription (biology)610 Medical sciencesVirologyddc:570medicineGene silencingHumansProtein IsoformsSecretionRNA Small InterferingCOPII030304 developmental biologyHepatitis B virus0303 health sciences030306 microbiologyEndoplasmic reticulumBiological TransportHepatitis Bdiseases infection microbe–cell interaction proteomics virusesCell biologyHost-Pathogen InteractionsHepatocytesCOP-Coated Vesicles
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Liver membrane autoantibodies in chronic active hepatitis

1987

Summary Target antigens relevant for immune reactions in inflammatory liver diseases should be expressed on the hepatocellular membrane. Using mechanically or enzymatically isolated rabbit hepatocytes, we evaluated the influence of cell integrity on the detection of membrane-expressed antigens by sera from patients with chronic hepatitis and by murine monoclonal antibodies. Our results provide evidence that target antigens of liver membrane autoantibodies (LMA) as well as liver kidney microsomal antibodies (LKM) are not expressed on the hepatocellular membrane of viable and intact isolated rabbit hepatocytes. However, LMA were detected in the sera of 56% of patients with autoimmune chronic …

HepatitisAutoimmune diseaseHepatologymedicine.drug_classAutoantibodyBiologymedicine.diseaseMonoclonal antibodyPathogenesismedicine.anatomical_structureAntigenHepatocyteImmunologymedicinebiology.proteinAntibodyJournal of Hepatology
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Expression of the 60 kDa heat shock protein in normal and inflamed liver.

1993

The 60 kDa heat shock proteins (HSP 60) have been well conserved throughout evolution and are highly immunogenic. Cross-reactivity between bacterial and mammalian HSP 60 is considered a likely mechanism in the pathogenesis of autoimmune diseases. T cell and B cell reactivity to HSP 60 is found in patients with rheumatoid or juvenile arthritis, and the expression of HSP 60 in the inflamed joint is found to be increased. In this study the presence of HSP 60 was demonstrated in normal and inflamed lives. HSP 60 was found to be predominantly expressed in hepatocytes and Kupffer cells, and mainly localized in mitochondria. Heat stress in the form of a 1 h incubation at 42 degrees C increased HSP…

HepatitisHepatologyT cellKupffer cellInflammationAutoimmune hepatitisChaperonin 60Biologymedicine.diseaseMolecular biologyHepatitisPathogenesismedicine.anatomical_structureLiverReference ValuesHeat shock proteinImmunologyChronic DiseasemedicineHumansmedicine.symptomB cellHeat-Shock ProteinsJournal of hepatology
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HSP10,HSP70 AND HSP90 IMMUNOHISTOCHEMICAL LEVELS CHANGE IN ULCERATIVE COLITIS AFTER THERAPY

2011

Ulcerative colitis (UC) is a form of inflammatory bowel disease (IBD) characterized by damage of large bowel mucosa and frequent extra-intestinal autoimmune comorbidities. The role played in IBD pathogenesis by molecular chaperones known to interact with components of the immune system involved in inflammation is unclear. We previously demonstrated that mucosal Hsp60 decreases in UC patients treated with conventional therapies (mesalazine, probiotics), suggesting that this chaperonin could be a reliable biomarker useful for monitoring response to treatment, and that it might play a role in pathogenesis. In the present work we investigated three other heat shock protein/molecular chaperones:…

HistologyBiophysicsDown-RegulationInflammationcomorbidity.Inflammatory bowel diseaseulcerative colitis heat shock proteins Hsp molecular chaperones inflammation comorbidity.Pathogenesischemistry.chemical_compoundMesalazineulcerative colitis heat shock proteins Hsp molecular chaperones inflammation comorbidityHeat shock proteinChaperonin 10MedicineHspHumansHSP70 Heat-Shock ProteinsHSP90 Heat-Shock ProteinsColitisMesalaminelcsh:QH301-705.5ulcerative colitisbusiness.industryBrief Reportmolecular chaperonesAnti-Inflammatory Agents Non-SteroidalCell Biologymedicine.diseaseUlcerative colitisImmunohistochemistrydigestive system diseaseschemistrylcsh:Biology (General)inflammationImmunologyheat shock proteinsBiomarker (medicine)Colitis Ulcerativemedicine.symptombusiness
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Extracellular vesicles in parasitic diseases

2014

Parasitic diseases affect billions of people and are considered a major public health issue. Close to 400 species are estimated to parasitize humans, of which around 90 are responsible for great clinical burden and mortality rates. Unfortunately, they are largely neglected as they are mainly endemic to poor regions. Of relevance to this review, there is accumulating evidence of the release of extracellular vesicles (EVs) in parasitic diseases, acting both in parasite parasite inter-communication as well as in parasite host interactions. EVs participate in the dissemination of the pathogen and play a role in the regulation of the host immune systems. Production of EVs from parasites or paras…

HistologyParàsitsProtozousReview ArticleexosomesparasitesDiagnostic toolsExosomesExtracellular vesiclesextracellular vesicles; microvesicles; exosomes; parasites; protozoa; helminthsprotozoaImmune systemHelminthsHelminthsParasitesProtozoalcsh:QH573-671HelmintsPathogenhelminthsPublic healthbiologyHost (biology)lcsh:CytologyCell BiologyExtracellular vesiclesbiology.organism_classificationSalut públicaParasitic diseasesMicrovesicles3. Good healthParasitologia mèdicaMalalties parasitàriesImmunologyProtozoaextracellular vesiclesmicrovesiclesMicrovesiclesJournal of Extracellular Vesicles
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Growth and Antagonism of <i>Trichoderma</i> spp. and Conifer Pathogen <i>Heterobasidion annosum</i&a…

2012

Variations in the radial growth rate of 24 isolates belonging to ten species of Trichoderma, three isolates of conifer pathogen Heterobasidion annosum s.s. and four isolates of H. parviporum were evaluated by incubation on a solid malt extract medium at a temperature of 4℃, 15℃ and 21℃. Trichoderma antagonism against Heterobasidion was investigated in dual culture in vitro. The slowest rate of growth was referable to all seven strains of Heterobasidion spp. All Heterobasidion spp. strains were overgrown by 63% of Trichoderma spp. strains after two weeks at 21℃ and by 33% of strains at 15℃. 21% of Trichoderma strains did not grow and only four strains belonging to T. koningii, T. viride and …

HorticulturebiologyTrichodermaHeterobasidion annosumGeneral Medicinebiology.organism_classificationAntagonismHeterobasidionTrichoderma sppPathogenIncubationIn vitroAdvances in Microbiology
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Looking for pathways related to COVID-19 phenotypes: Confirmation of pathogenic mechanisms by SARS-CoV-2 - Host interactome

2020

AbstractIn the last months, many studies have clearly described several mechanisms of SARS-CoV-2 infection at cell and tissue level. Host conditions and comorbidities were identified as risk factors for severe and fatal disease courses, but the mechanisms of interaction between host and SARS-CoV-2 determining the grade of COVID- 19 severity, are still unknown.We provide a network analysis on protein–protein interactions (PPI) between viral and host proteins to better identify host biological responses, induced by both whole proteome of SARS-CoV-2 and specific viral proteins. A host-virus interactome was inferred on published PPI, using an explorative algorithm (Random Walk with Restart) tri…

Host (biology)Viral proteinvirusesCellComputational biologyBiologymedicine.disease_causePhenotypeInteractomePathogenesismedicine.anatomical_structureProteomemedicineViral Accessory Proteins
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