Search results for "peroxiredoxins"

showing 10 items of 10 documents

Redox Proteomics of the Inflammatory Secretome Identifies a Common Set of Redoxins and Other Glutathionylated Proteins Released in Inflammation, Infl…

2015

Protein cysteines can form transient disulfides with glutathione (GSH), resulting in the production of glutathionylated proteins, and this process is regarded as a mechanism by which the redox state of the cell can regulate protein function. Most studies on redox regulation of immunity have focused on intracellular proteins. In this study we have used redox proteomics to identify those proteins released in glutathionylated form by macrophages stimulated with lipopolysaccharide (LPS) after pre-loading the cells with biotinylated GSH. Of the several proteins identified in the redox secretome, we have selected a number for validation. Proteomic analysis indicated that LPS stimulated the releas…

LipopolysaccharidesProteomicsglutaredoxins; glutathione; redox signalingBlotting Westernlcsh:MedicineDown-RegulationInflammationBiologyProteomicsmedicine.disease_causeAntioxidantsDexamethasoneCell LineMiceProfilinschemistry.chemical_compoundThioredoxinsInfluenza HumanmedicineExtracellularAnimalsHumansVimentinSulfhydryl Compoundsglutathionelcsh:Scienceredox signalingglutaredoxinsInflammationMultidisciplinarylcsh:RRProteinsPeroxiredoxinsGlutathioneCell biologyBlotOxidative StressRAW 264.7 CellschemistryQR180lcsh:QTumor necrosis factor alphamedicine.symptomPeroxiredoxinOxidation-ReductionOxidative stressResearch ArticlePLOS ONE
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Formation, TEM study and 3D reconstruction of the human erythrocyte peroxiredoxin-2 dodecahedral higher-order assembly.

2004

The production of a higher-order assembly of peroxiredoxin-2 (Prx-2) from human erythrocytes has been achieved during specimen preparation on holey carbon support films, in the presence of ammonium molybdate and polyethylene glycol. TEM study suggested that this assembly is a regular dodecahedron, containing 12 Prx-2 decamers (Mr 2.62 MDa, external diameter approximately 20 nm). This interpretation has been supported by production of a approximately 1.6 nm 3D reconstruction from the negative stain TEM data, with automated docking of the available X-ray data of the Prx-2 decamer. Comparison with other known protein dodecahedral and viral icosahedral structures indicates that this arrangement…

Models MolecularMaterials scienceErythrocytesIcosahedral symmetryMacromolecular SubstancesMacromolecular SubstancesGeneral Physics and AstronomyCell BiologyPeroxiredoxin 2Polyethylene glycolPeroxiredoxinsNegative stainDodecahedronCrystallographychemistry.chemical_compoundProtein structurechemistryMicroscopy Electron TransmissionPeroxidasesStructural BiologyImage Processing Computer-AssistedHumansGeneral Materials ScienceProtein Structure QuaternaryMacromoleculeMicron (Oxford, England : 1993)
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Saccharomyces cerevisiae Cytosolic Thioredoxins Control Glycolysis, Lipid Metabolism, and Protein Biosynthesis under Wine-Making Conditions.

2019

Thioredoxins are small proteins that regulate the cellular redox state, prevent oxidative damage, and play an active role in cell repair. Oxidative stress has proven to be of much relevance in biotechnological processes when the metabolism of Saccharomyces cerevisiae is mainly respiratory. During wine yeast starter production, active dry yeast cytosolic thioredoxin Trx2p is a key player in protecting metabolic enzymes from being oxidized by carbonylation. Less is known about the role of redox control during grape juice fermentation. A mutant strain that lacked both cytosolic thioredoxins, Trx1p and Trx2p, was tested for grape juice fermentation. Its growth and sugar consumption were greatly…

ProteomicsSaccharomyces cerevisiae ProteinsSaccharomyces cerevisiaethioredoxin-thioredoxin reductase systemsyeastsWineOxidative phosphorylationSaccharomyces cerevisiaeApplied Microbiology and Biotechnology03 medical and health sciencesCytosolThioredoxinsYeastsMetabolomicsVitis030304 developmental biology0303 health sciencesEcologybiology030306 microbiologyChemistryfood and beveragesMembrane ProteinsLipid metabolismMetabolismPeroxiredoxinsglycolysisbiology.organism_classificationLipid MetabolismmetabolomicsYeastYeast in winemakingOxidative StressBiochemistryProtein BiosynthesisFermentationFood MicrobiologyFermentationThioredoxinThioredoxin-thioredoxin reductase systemsGlycolysisOxidation-ReductionGene DeletionFood ScienceBiotechnology
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Structure, mechanism and regulation of peroxiredoxins.

2003

Peroxiredoxins (Prxs) are a ubiquitous family of antioxidant enzymes that also control cytokine-induced peroxide levels which mediate signal transduction in mammalian cells. Prxs can be regulated by changes to phosphorylation, redox and possibly oligomerization states. Prxs are divided into three classes: typical 2-Cys Prxs; atypical 2-Cys Prxs; and 1-Cys Prxs. All Prxs share the same basic catalytic mechanism, in which an active-site cysteine (the peroxidatic cysteine) is oxidized to a sulfenic acid by the peroxide substrate. The recycling of the sulfenic acid back to a thiol is what distinguishes the three enzyme classes. Using crystal structures, a detailed catalytic cycle has been deriv…

Peroxiredoxin-4Binding SitesChemistryProtein ConformationPeroxiredoxin IIIPeroxiredoxin 2PeroxiredoxinsBiochemistryCatalysischemistry.chemical_compoundSulfiredoxinCatalytic cycleBiochemistryPeroxidasesSulfenic acidPeroxiredoxinMolecular BiologyDimerizationOxidation-ReductionCysteineTrends in biochemical sciences
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Comparison of the decameric structure of peroxiredoxin-II by transmission electron microscopy and X-ray crystallography

2001

Abstract The decameric human erythrocyte protein torin is identical to the thiol-specific antioxidant protein-II (TSA-II), also termed peroxiredoxin-II (Prx-II). Single particle analysis from electron micrographs of Prx-II molecules homogeneously orientated across holes in the presence of a thin film of ammonium molybdate and trehalose has facilitated the production of a ≥20 A 3-D reconstruction by angular reconstitution that emphasises the D5 symmetry of the ring-like decamer. The X-ray structure for Prx-II was fitted into the transmission electron microscopic reconstruction by molecular replacement. The surface-rendered transmission electron microscopy (TEM) reconstruction correlates well…

Models MolecularMolybdenumErythrocytesSurface PropertiesCryo-electron microscopyChemistryResolution (electron density)BiophysicsTrehaloseSingle particle analysisPeroxiredoxinsCrystallography X-RayBiochemistryNegative stainMicroscopy ElectronCrystallographyPeroxidasesElectron tomographyStructural BiologyTransmission electron microscopyHumansEnergy filtered transmission electron microscopyOrthorhombic crystal systemMolecular BiologyBiochimica et Biophysica Acta (BBA) - Protein Structure and Molecular Enzymology
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Lack of a peroxiredoxin suppresses the lethality of cells devoid of electron donors by channelling electrons to oxidized ribonucleotide reductase

2017

The thioredoxin and glutaredoxin pathways are responsible of recycling several enzymes which undergo intramolecular disulfide bond formation as part of their catalytic cycles such as the peroxide scavengers peroxiredoxins or the enzyme ribonucleotide reductase (RNR). RNR, the rate-limiting enzyme of deoxyribonucleotide synthesis, is an essential enzyme relying on these electron flow cascades for recycling. RNR is tightly regulated in a cell cycle-dependent manner at different levels, but little is known about the participation of electron donors in such regulation. Here, we show that cytosolic thioredoxins Trx1 and Trx3 are the primary electron donors for RNR in fission yeast. Unexpectedly,…

0301 basic medicineCancer ResearchThioredoxin reductaseSynthesis PhaseYeast and Fungal ModelsBiochemistryElectron DonorsSchizosaccharomyces PombeThioredoxinsGlutaredoxinCell Cycle and Cell DivisionGenetics (clinical)Chemical ReactionsOxidesPeroxidesNucleic acidsChemistryRibonucleotide reductaseBiochemistryExperimental Organism SystemsCell ProcessesSchizosaccharomyces pombePhysical SciencesSynthesis phaseThioredoxinOxidation-ReductionResearch ArticleDNA Replicationlcsh:QH426-470DNA transcriptionElectron donorsBiologyDNA replicationResearch and Analysis MethodsCatalysisElectron Transport03 medical and health sciencesModel OrganismsSchizosaccharomycesRibonucleotide ReductasesOxidationGeneticsMolecular BiologyEcology Evolution Behavior and SystematicsGlutaredoxinsCell growthDNA replicationChemical CompoundsOrganismsFungiBiology and Life SciencesCell BiologyDNAPeroxiredoxinsbiology.organism_classificationYeastCell cycle and cell divisionCheckpoint Kinase 2lcsh:Genetics030104 developmental biologySchizosaccharomyces pombeGene expressionSchizosaccharomyces pombe ProteinsPeroxiredoxin
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The triterpenoid ursolic acid ameliorates stress in Caenorhabditis elegans by affecting the depression-associated genes skn-1 and prdx2.

2021

Abstract Introduction Depression is one of the leading causes of death worldwide. Lower antioxidant concentrations and increased oxidative stress levels contribute to the development of depression. Effective and tolerable medications are urgently needed. Nrf2 and PRDX2 are promising targets in the treatment of oxidative stress and, therefore, promising for the development of novel antidepressants. Ursolic acid (UA), a natural triterpenoid found in various plants is known to exert neuroprotective and antioxidant effects. Skn-1 (which corresponds to human Nrf2) and prdx2 deficient mutants of the nematode Caenorhabditis elegans are suitable models to study the effect of UA on these targets. Ad…

Antioxidantmedicine.medical_treatmentPharmaceutical SciencePharmacologymedicine.disease_causeProtective AgentsNeuroprotectionAntioxidants03 medical and health scienceschemistry.chemical_compound0302 clinical medicineUrsolic acidStress PhysiologicalDrug DiscoveryAdaptogenmedicineAnimalsCaenorhabditis elegansCaenorhabditis elegans ProteinsCaenorhabditis elegans030304 developmental biologyPharmacologychemistry.chemical_classification0303 health sciencesReactive oxygen speciesbiologyDepressionPeroxiredoxinsbiology.organism_classificationAntidepressive AgentsTriterpenesDNA-Binding ProteinsMolecular Docking SimulationOxidative StressComplementary and alternative medicinechemistryGene Expression Regulation030220 oncology & carcinogenesisMutationMolecular MedicineReactive Oxygen SpeciesJugloneOxidative stressTranscription FactorsPhytomedicine : international journal of phytotherapy and phytopharmacology
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Wine yeast peroxiredoxin TSA1 plays a role in growth, stress response and trehalose metabolism in biomass propagation

2020

This article belongs to the Special Issue Wine Yeast 1.0.

Microbiology (medical)Protein moonlightingperoxiredoxinsThioredoxin reductaseSaccharomyces cerevisiaeMutantWineSaccharomyces cerevisiaeMicrobiology<i>Saccharomyces cerevisiae</i>03 medical and health scienceschemistry.chemical_compoundVirologyoxidative stressBiomasswinelcsh:QH301-705.5030304 developmental biologychemistry.chemical_classification0303 health sciencesTsa1biologybiomass030306 microbiologyChemistryPeroxiredoxinsbiology.organism_classificationTrehaloseYeast in winemakingEnzymeBiochemistrylcsh:Biology (General)Oxidative stressPeroxiredoxinMicroorganisms
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Characterization of NO-Induced Nitrosative Status in Human Placenta from Pregnant Women with Gestational Diabetes Mellitus

2017

Dysregulation of NO production is implicated in pregnancy-related diseases, including gestational diabetes mellitus (GDM). The role of NO and its placental targets in GDM pregnancies has yet to be determined. S-Nitrosylation is the NO-derived posttranslational protein modification that can modulate biological functions by forming NO-derived complexes with longer half-life, termed S-nitrosothiol (SNO). Our aim was to examine the presence of endogenous S-nitrosylated proteins in cysteine residues in relation to antioxidant defense, apoptosis, and cellular signal transduction in placental tissue from control (n=8) and GDM (n=8) pregnancies. S-Nitrosylation was measured using the biotin-switch …

0301 basic medicineAgingendocrine system diseasesPlacentaNitric Oxide Synthase Type IIExpressionApoptosisBiochemistryBody Mass Index0302 clinical medicineNitric-oxidePregnancyMitogen-Activated Protein Kinase 1Mitogen-Activated Protein Kinase 3biologyCaspase 3lcsh:CytologyNitrosylationP38General MedicineCatalaseCaspase 9TrophoblastsGestational diabetesmedicine.anatomical_structureCatalase030220 oncology & carcinogenesisFemaleResearch ArticleAdultmedicine.medical_specialtyArticle SubjectNitrosationNitric OxidePathophysiology03 medical and health sciencesErk1/2Internal medicinePlacentamedicineHumanslcsh:QH573-671Protein kinase BPregnancyFetusNitratesS-NitrosothiolsCesarean SectionCell BiologyPeroxiredoxinsmedicine.diseaseProtein s-nitrosylationDiabetes Gestational030104 developmental biologyEndocrinologyOxidative stressCase-Control Studiesbiology.proteinPeroxiredoxinProto-Oncogene Proteins c-aktOxidative Medicine and Cellular Longevity
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Changes in the expression of neurotransmitter receptors in Parkin and DJ-1 knockout mice – A quantitative multireceptor study

2015

Parkinson's disease (PD) is a well-characterized neurological disorder with regard to its neuropathological and symptomatic appearance. At the genetic level, mutations of particular genes, e.g. Parkin and DJ-1, were found in human hereditary PD with early onset. Neurotransmitter receptors constitute decisive elements in neural signal transduction. Furthermore, since they are often altered in neurological and psychiatric diseases, receptors have been successful targets for pharmacological agents. However, the consequences of PD-associated gene mutations on the expression of transmitter receptors are largely unknown. Therefore, we studied the expression of 16 different receptor binding sites …

MaleMice KnockoutOncogene ProteinsUbiquitin-Protein LigasesGeneral NeuroscienceProtein Deglycase DJ-1Glutamate receptorBrainKainate receptorPeroxiredoxinsAMPA receptorNeurotransmissionBiologyParkinReceptors NeurotransmitterMice Inbred C57BLParkinsonian DisordersNeurotransmitter receptorKnockout mouseAnimalsAutoradiographyReceptorNeuroscienceNeuroscience
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