Search results for "phage"

showing 10 items of 1573 documents

Characterization of the Microenvironment in Positive and Negative Sentinel Lymph Nodes from Melanoma Patients

2015

Melanomas are aggressive skin tumors characterized by high metastatic potential. Our previous results indicate that Natural Killer (NK) cells may control growth of melanoma. The main defect of blood NK cells was a decreased expression of activating NCR1/NKp46 receptor and a positive correlation of NKp46 expression with disease outcome in stage IV melanoma patients was found. In addition, in stage III melanoma patients, we identified a new subset of mature NK cells in macro-metastatic Lymph nodes (LN). In the present studies, we evaluated the numbers of NK cells infiltrating primary cutaneous melanoma and analyzed immune cell subsets in a series of sentinel lymph nodes (SLN). First, we show …

AdultMalePathologymedicine.medical_specialtyCD34lcsh:MedicineCD8-Positive T-LymphocytesBiologyTumor MicroenvironmentmedicineHumansCytotoxic T celllcsh:ScienceMelanomaAgedNeoplasm StagingAged 80 and overTumor microenvironmentMultidisciplinarySentinel Lymph Node BiopsyMacrophagesMelanomalcsh:REndothelial CellsMiddle Agedmedicine.diseaseAntigens Differentiation3. Good healthKiller Cells NaturalGranzyme BCutaneous melanomalcsh:QFemaleLymphCD8Research ArticlePLOS ONE
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Ultrastructural study on human lung in alveolitis versus pulmonary fibrosis

1993

Lung specimens of 21 patients with diffuse interstitial lung disease were examined. The present ultrastructural study outlines the topography and distribution of inflammatory changes in the interstitium, endothelium, and in pneumocytes and phagocytes. Alveolitis is characterized by marked regenerative activity of type II pneumocytes (cuboid metaplasia), intraluminal macrophage accumulation, endothelial swelling, multilamination of the endothelial basement membrane, pericapillary edema, and primarily by cellular infiltrates in the interstitial space. The most prominent feature of the interstitium in pulmonary fibrosis is the lack of immunoinflammatory cells. In some areas there is a marked a…

AdultMalePathologymedicine.medical_specialtyEndotheliumPulmonary FibrosisEpitheliumInterstitial spaceEdemaMacrophages AlveolarDrug DiscoveryPulmonary fibrosismedicineHumansLymphocytesLungGenetics (clinical)InflammationBasement membraneMetaplasiaLungbusiness.industryPneumonia PneumocystisType-II PneumocytesInterstitial lung diseaseGeneral MedicineMiddle Agedrespiratory systemmedicine.diseaseCapillariesmedicine.anatomical_structureImmunologyMolecular MedicineFemalemedicine.symptombusinessAlveolitis Extrinsic AllergicThe Clinical Investigator
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Image cytometric DNA analysis of mucosal biopsies in patients with primary achalasia.

2006

(P < 0.0001), and 9c- (P = 0.0001) exceeding rate with progredient DNA alterations in the respective order. CONCLUSION: The finding that DNA aneuploidy was identifi ed by image cytometry in esophageal specimens of patients with achalasia, which may be due to specifi c chromosomal alterations presenting as precancerous lesions in 27% of patients, leads us to conclude that image cytometry represents a valuable screening tool. Abstract AIM: To determine DNA aneuploidy in mucosal biopsies of achalasia patients for subsequent rapid diagnosis. METHODS: Biopsies from the middle third of the esophagus were obtained in 15 patients with achalasia. Immunohistochemical staining was carried out with mon…

AdultMalePathologymedicine.medical_specialtyEsophageal NeoplasmsBiopsyAchalasiaAneuploidyBiologydigestive systemBiopsyotorhinolaryngologic diseasesCarcinomamedicineHumansGenetic TestingFeulgen stainEsophagusAgedImage CytometryMucous Membranemedicine.diagnostic_testGastroenterologyDNAGeneral MedicineMiddle AgedAneuploidymedicine.diseaseImmunohistochemistrydigestive system diseasesEsophageal AchalasiaBasic ResearchKi-67 Antigenmedicine.anatomical_structureDysplasiaCarcinoma Squamous CellImage CytometryFemaleTumor Suppressor Protein p53Precancerous Conditions
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A macrophage-suppressing 40-kD protein in a case of pulmonary alveolar proteinosis.

1987

Pulmonary alveolar proteinosis (PAP) is a rare disease of unknown etiology. Macrophage dysfunctions are claimed to be involved in the pathogenesis. We investigated phagocytosis and oxidative metabolism of alveolar macrophages in a case of pulmonary alveolar proteinosis. These cells phagocytize normally and phagocytizable stimulants cause a normal oxidative burst. In response to the membrane signals phorbolmyristate acetate and aggregated immunoglobulin, however, no stimulated turnover of the oxidative metabolism can be observed. A 40-kD protein found in the lavage fluid mediates this macrophage-inhibiting effect. This phenomenon may contribute to the frequent opportunistic infections seen i…

AdultMalePathologymedicine.medical_specialtyPhagocytosisOpportunistic InfectionsPulmonary Alveolar ProteinosisPathogenesisPhagocytosisDrug DiscoverymedicineMacrophageHumansMacrophage Migration-Inhibitory FactorsGenetics (clinical)Lungmedicine.diagnostic_testbiologyMacrophagesfood and beveragesProteinsGeneral MedicineMacrophage Activationmedicine.diseaseRespiratory burstMolecular WeightPulmonary AlveoliBronchoalveolar lavagemedicine.anatomical_structureImmunologyLuminescent Measurementsbiology.proteinMolecular MedicineAntibodyPulmonary alveolar proteinosisEnergy MetabolismBronchoalveolar Lavage FluidKlinische Wochenschrift
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Immune-mediated necrotizing myopathy is characterized by a specific Th1-M1 polarized immune profile.

2012

Immune-mediated necrotizing myopathy (IMNM) is considered one of the idiopathic inflammatory myopathies, comprising dermatomyositis, polymyositis, and inclusion body myositis. The heterogeneous group of necrotizing myopathies shows a varying amount of necrotic muscle fibers, myophagocytosis, and a sparse inflammatory infiltrate. The underlying immune response in necrotizing myopathy has not yet been addressed in detail. Affected muscle tissue, obtained from 16 patients with IMNM, was analyzed compared with eight non-IMNM (nIMNM) tissues. Inflammatory cells were characterized by IHC, and immune mediators were assessed by quantitative real-time PCR. We demonstrate that immune- and non–immune-…

AdultMalePathologymedicine.medical_specialtyT cellBiopsyCell CountBiologyCD8-Positive T-LymphocytesMajor histocompatibility complexReal-Time Polymerase Chain ReactionPolymyositisPathology and Forensic MedicineYoung AdultImmune systemSarcolemmamedicineHumansAgedAged 80 and overB-LymphocytesMyositisMacrophagesMusclesHistocompatibility Antigens Class IAutoantibodyImmunityComplement System ProteinsDermatomyositisMiddle AgedTh1 Cellsmedicine.diseaseCapillariesmedicine.anatomical_structureChild PreschoolImmunologybiology.proteinTumor necrosis factor alphaFemaleInclusion body myositisThe American journal of pathology
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Presence of estrogen-binding sites on macrophage-like synoviocytes and cd8+, cd29+, cd45ro+ t lymphocytes in normal and rheumatoid synovium

1993

Objective. To study the presence of estrogen-binding sites (EBS) in the synovial tissues of male and female patients with rheumatoid arthritis (RA) and in age and sex-matched healthy controls. Methods. Both type 1 (high affinity, low binding capacity) and type 2 (reduced affinity, higher binding capacity) EBS were investigated in both soluble and nuclear fractions of homogenized synovial tissue samples by a dextran-coated charcoal method. To determine what type of synovial cell was positive for EBS, cryosections of synovial tissues were immunostained with a specific monoclonal anti–estrogen receptor antibody (anti-ER MAb) using both immunofluorescence and immunoperoxidase techniques. Double…

AdultMalePathologymedicine.medical_specialtyT-LymphocytesT cellImmunologyImmunofluorescenceArthritis RheumatoidRheumatologyAntigenmedicineHumansImmunology and AllergyPharmacology (medical)Estrogen bindingAgedSex Characteristicsmedicine.diagnostic_testbusiness.industryMacrophagesSynovial MembraneMiddle AgedImmunohistochemistryMolecular biologymedicine.anatomical_structureReceptors EstrogenSynovial CellFemaleSynovial membranebusinessImmunostainingCD8Arthritis &amp; Rheumatism
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IL-10 and IL-10 receptor overexpression in oral giant cell lesions

2010

Objective: Central giant cell lesions (CGCL) and peripheral giant cell lesions (PGCL) occur in the jaws and contain osteoclast-like giant cells and mononuclear cells positive for the macrophage marker CD68. The participation of immune-inflammatory mechanisms has been proposed in the lesions development. As IL-10 is one of the most important anti-inflammatory cytokines and it is also an inhibitory cytokine to macrophage function and bone resorption, the purpose of the present study was to investigate its expression together with its receptor (IL-10R?) in CGCL and PGCL. Study Design: Six fragments of CGCL and seven fragments of PGCL were obtained by surgical excision. Frozen specimens were cu…

AdultMalePathologymedicine.medical_specialtymedicine.medical_treatmentBiologyPeripheral blood mononuclear cellProinflammatory cytokineYoung AdultGranuloma Giant CellmedicineHumansMacrophageReceptors Interleukin-10ChildGeneral DentistryCD68Middle Agedmedicine.disease:CIENCIAS MÉDICAS [UNESCO]Interleukin-10Interleukin 10CytokineOtorhinolaryngologyGiant cellGranulomaUNESCO::CIENCIAS MÉDICASFemaleSurgeryMouth Diseases
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Macrophage phenotype in the subclinical gut inflammation of patients with ankylosing spondylitis

2014

OBJECTIVE: Long-term evolution of subclinical gut inflammation to overt Crohn's disease (CD) has been described in AS patients. The aim of this study was to evaluate macrophage polarization occurring in the inflamed gut of patients with AS. METHODS: Twenty-seven HLA-B27(+) AS patients, 20 CD patients and 17 normal controls were consecutively enrolled. Classic M1 (iNOS(+)IL-10(-)), resolution phase (iNOS(+)IL-10(+)), M2 and CD14(+) macrophages were characterized by immunohistochemistry and flow cytometry. Quantitative gene expression analysis of IFN-γ, IL-4, IL-5, IL-33 and STAT6 was performed by real time PCR. RESULTS: Classic M1 macrophages were expanded in CD and AS, where resolution phas…

AdultMalePathologymedicine.medical_specialtymedicine.medical_treatmentCD14BiopsyMacrophage-activating factorMacrophage polarizationInflammationReal-Time Polymerase Chain ReactionM2 macrophageYoung AdultRheumatologyIleumMedicineMacrophageHumansPharmacology (medical)Spondylitis AnkylosingAgedbusiness.industryMacrophagesresolution phase macrophagesDNAIleitisMiddle AgedFlow CytometryImmunohistochemistryInterleukin 10Settore MED/16 - Reumatologiaankylosing spondylitiCytokinePhenotypeGene Expression RegulationM1 macrophages M2 macrophages ankylosing spondylitis gut inflammation interleukin 33 resolution phase macrophagesImmunologyCytokinesFemalegut inflammationinterleukin 33medicine.symptombusinessCD163M1 macrophage
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Long-term survivors of esophageal cancer: Disease-specific quality of life, general health and complications

2009

Introduction The aim of our study was to assess the quality of life as well as secondary cancers/diseases and esophagectomy-related or unrelated interventions in the long-term course of surgery. Patients and Methods Out of 417 patients who underwent esophageal resection for cancer between September 1985 and November 2003, 85 were defined as long-term survivors (≥5 years). Fifty patients still alive in November 2008 complied with our inclusion criteria. The general (QLQ-C 30, version 3.0) as well as the esophagus specific quality of life (QLQ-OES 18) were analyzed with the help of the EORTC QLQ-questionnaires. Results The median observation interval since the operation was 100.1 (range 60–23…

AdultMalePediatricsmedicine.medical_specialtyEsophageal NeoplasmsHealth Statusmedicine.medical_treatmentSwallowingQuality of lifeSurveys and QuestionnairesCarcinomamedicineHumansSurvivorsEsophagusAgedbusiness.industryCarcinomaCancerGeneral MedicineMiddle AgedEsophageal cancermedicine.diseaseDysphagiahumanitiesEsophagectomymedicine.anatomical_structureOncologyEsophagectomyQuality of LifePhysical therapyFemaleSurgerymedicine.symptombusinessFollow-Up StudiesJournal of Surgical Oncology
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Identification of a peptide mimicking the binding pattern of an antiphospholipid antibody

2006

Our objective was to characterize monoclonal antiphospholipid antibodies (APL) and identify disease-associated antigens in patients with the antiphospholipid syndrome (APS). We used the monoclonal antibody HL-5B, derived from a patient with APS suffering from multiple ischemic events, to screen a 12-mer peptide phage display library (New England Biolabs, London, England). The identified phage clones were sequenced and the derived consensus peptide was synthesized. The peptide was used to perform competitive inhibition experiments for their ability to inhibit the binding of the monoclonal antibody and of serum antibodies to cardiolipin and phosphatidylserine. Additionally patients and contro…

AdultMalePhage displaymedicine.drug_classMolecular Sequence DataImmunologyEnzyme-Linked Immunosorbent AssayMonoclonal antibodyEpitopeAntigenAntibody SpecificityPeptide LibraryAntiphospholipid syndromemedicineHumansImmunology and AllergyAmino Acid SequencePeptide libraryPeptide sequenceAgedbiologyMolecular MimicryAntibodies MonoclonalHematologyMiddle AgedAntiphospholipid Syndromemedicine.diseaseVirologyMolecular biologyAntibodies Antiphospholipidbiology.proteinFemaleAntibodyPeptidesProtein BindingImmunobiology
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