Search results for "phosphodiester"

showing 10 items of 176 documents

Treatment of experimental autoimmune encephalomyelitis with adenylate deaminase from Penicillium lanoso-viride.

2000

The effect of intramuscularly administered immunomodulator, adenylate deaminase (E.C. 3.5.4.6), from Penicillium lanoso-viride on the clinical score of acute experimental autoimmune encephalomyelitis (EAE), a T cell-mediated autoimmune disease, was examined by inoculation of guinea pigs with rabbit brain and spinal cord homogenate (encephalitogen) and complete Freund's adjuvant. Adenylate deaminase (ADA) was effective in delaying the onset of clinical disease. ADA inhibited the severity of EAE. There was a significant decrease in clinical signs. A decrease in the number of morbid and dead animals was observed. Of ADA treated animals, 50-80% developed no clinical manifestations of EAE. The o…

MaleEncephalomyelitis Autoimmune ExperimentalEncephalomyelitisImmunologyGuinea PigsCross Reactionsmedicine.disease_causeInjections IntramuscularAutoimmunityAMP DeaminaseMiceBlood serumAdjuvants Immunologicimmune system diseasesImmunology and AllergyMedicineAnimalsHypersensitivity DelayedComplement ActivationSkin TestsAutoimmune diseaseMice Inbred BALB Cbiologybusiness.industryMultiple sclerosisExperimental autoimmune encephalomyelitisPenicilliumBrainAMP deaminasemedicine.diseaseSpinal CordImmunologybiology.proteinFemaleImmunizationRabbitsAntibodybusiness2'3'-Cyclic-Nucleotide PhosphodiesterasesJournal of autoimmunity
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Mechanisms underlying the inhibitory effects induced by pituitary adenylate cyclase-activating peptide in mouse ileum

2005

Abstract The aim of this study was to investigate the signal transduction mechanisms underlying the inhibitory effect induced by pituitary adenylate cyclase activating peptide (PACAP-27) on the spontaneous contractile activity of longitudinal muscle of mouse ileum. Mechanical activity of ileal segments was recorded isometrically in vitro. PACAP-27 produced apamin-sensitive reduction of the amplitude of the spontaneous contractions. 9-(Tetrahydro-2-furanyl)-9H-purin-6-amine (SQ 22,536), adenylate cyclase inhibitor, or genistein and tyrphostin 25, tyrosine kinase inhibitors, had negligible effects on PACAP-27-induced inhibition. PACAP-27 effects were significantly inhibited by U-73122, phopho…

MaleIndolesPhosphodiesterase InhibitorsVasodilator AgentsMouse ileumStimulationSettore BIO/09 - FisiologiaMicechemistry.chemical_compoundInositolEnzyme InhibitorsEstrenesRyanodineRyanodine receptorProtein-Tyrosine KinasesTyrphostinsGenisteinPyrrolidinonesCell biologyPituitary adenylate cyclase-activating peptideNG-Nitroarginine Methyl EsterPituitary Adenylate Cyclase-Activating PolypeptideThapsigarginSignal transductionCyclopiazonic acidhormones hormone substitutes and hormone antagonistsMuscle ContractionBoron Compoundsendocrine systemmedicine.medical_specialtyThapsigarginMuscular inhibitionCalcium-Transporting ATPasesIn Vitro TechniquesInositol 145-triphosphateBiologyPACAP-27 (pituitary adenylate cyclase activating peptide)IleumPhospholipase CInternal medicinemedicineAnimalsPharmacologyDose-Response Relationship DrugPhospholipase CAdenineMuscle SmoothMice Inbred C57BLEndocrinologyApaminchemistryAdenylyl Cyclase InhibitorsCalciumNitric Oxide SynthaseEuropean Journal of Pharmacology
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Pentoxifylline Prevents Loss of PP2A Phosphatase Activity and Recruitment of Histone Acetyltransferases to Proinflammatory Genes in Acute Pancreatitis

2009

Mitogen-activated protein kinases (MAPKs) are considered major signal transducers early during the development of acute pancreatitis. Pentoxifylline is a phosphodiesterase inhibitor with marked anti-inflammatory properties through blockade of extracellular signal regulated kinase (ERK) phosphorylation and tumor necrosis factor alpha production. Our aim was to elucidate the mechanism of action of pentoxifylline as an anti-inflammatory agent in acute pancreatitis. Necrotizing pancreatitis induced by taurocholate in rats and taurocholate-treated AR42J acinar cells were studied. Phosphorylation of ERK and ERK kinase (MEK1/2), as well as PP2A, PP2B, and PP2C serine/threonine phosphatase activiti…

MaleMAPK/ERK pathwayChromatin ImmunoprecipitationPhosphodiesterase InhibitorsBlotting WesternPhosphataseAnti-Inflammatory AgentsPharmacologyBiologyCell LinePentoxifyllineProinflammatory cytokineCyclic AMPPhosphoprotein PhosphatasesmedicineAnimalsPentoxifyllineRats WistarExtracellular Signal-Regulated MAP KinasesHistone AcetyltransferasesInflammationPharmacologyReverse Transcriptase Polymerase Chain ReactionTumor Necrosis Factor-alphaProtein phosphatase 2medicine.diseaseCyclic Nucleotide Phosphodiesterases Type 2RatsEnzyme ActivationPancreatitisBiochemistryAcute DiseaseRNAMolecular MedicinePhosphorylationPancreatitisMitogen-Activated Protein KinasesChromatin immunoprecipitationmedicine.drugJournal of Pharmacology and Experimental Therapeutics
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Pharmacological activity of PF-904 in guinea pig in vivo, and on human bronchus and neutrophils in vitro.

1997

The effects of PF-904 (4-amino-1-ethyl-6-methylpyrazino[2,3-c][1,2,6]thiadiazine 2,2-dioxide), a pyrazinothiadiazine derivative, were examined in guinea-pig airways in vivo, in human isolated bronchus and human polymorphonuclear leukocytes. PF-904 (12.5-200 mg/kg, intraduodenal) reduced bronchoconstriction in response to histamine, arachidonic acid, platelet-activating factor (PAF) and methacholine. PF-904 (50-200 mg/kg) prevented PAF-induced airways hyperreactivity and inhibited antigen-induced bronchoconstriction, airway microvascular leakage and eosinophil lung accumulation, but antigen-induced airways hyperresponsiveness was not reduced. PF-904 (1 microM-1 mM) produced complete inhibiti…

MaleNeutrophilsPhosphodiesterase InhibitorsGuinea PigsBronchiPharmacologyIn Vitro TechniquesBronchial Provocation TestsCapillary Permeabilitychemistry.chemical_compoundIn vivoSuperoxidesmedicineAnimalsHumansAnti-Asthmatic AgentsPlatelet Activating FactorRolipramPharmacologyBronchusThiadiazinesAnti-Inflammatory Agents Non-SteroidalPhosphodiesteraseBiological activityrespiratory systemBronchodilator AgentsN-Formylmethionine Leucyl-Phenylalaninemedicine.anatomical_structurechemistryBiochemistryPyrazinesBronchoconstrictionMethacholinemedicine.symptomBronchial HyperreactivityHistaminemedicine.drugEuropean journal of pharmacology
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In rat pinealocytes the cyclic GMP response to NO is regulated by Ca2+ and protein kinase C

1995

There is ample evidence that beta-adrenergic stimulation of cyclic GMP formation is potentiated by alpha1-adrenergic mechanisms, the latter leading to elevation of intracellular Ca2+ concentration ([Ca2+]i) and protein kinase C (PKC) activation. Recent studies have shown that nitric oxide synthase (NOS) and nitric oxide (NO) are a component of the adrenoceptor-cyclic GMP signalling pathway. The aim of the present investigation was to study the roles of alpha1-adrenergic mechanisms, Ca2+ and PKC on NO-stimulated cyclic GMP formation. To this end suspension cultures of rat pinealocytes were treated with the NO donor sodium nitroprusside (SNP) in the presence of alpha1-adrenergic agonists, [Ca…

MaleNitroprussidePhosphodiesterase InhibitorsNitric OxidePineal GlandPinealocyteNitric oxideRats Sprague-DawleyPhenylephrinechemistry.chemical_compoundCalmodulinmedicineAnimalsOuabainCyclic GMPProtein Kinase InhibitorsMolecular BiologyProtein Kinase CProtein kinase CbiologyKinaseGeneral NeuroscienceIsoproterenolPhosphodiesterasePhosphoric Monoester HydrolasesRatsNitric oxide synthaseCytosolBiochemistrychemistrybiology.proteinCalciumNeurology (clinical)Sodium nitroprussideDevelopmental Biologymedicine.drugBrain Research
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The nicardipine-isoprenaline interaction in human and guinea-pig isolated airways

1994

Summary— The effects of the dihydropyridine calcium antagonist nicardipine on the concentration-response curves of relaxant compounds acting through the adenylate-cyclase/cAMP system (isoprenaline, forskolin, adenosine and theophylline) or by the cGMP pathway (sodium nitroprusside) were studied on human isolated bronchus and guinea-pig isolated trachea. These effects were compared with those of nifedipine (a dihydropyridine derivative) and theophylline (a non-selective phosphodiesterase inhibitor). Nicardipine, in the range of 0.01 to 1 μM, significantly potentiated the relaxant effects of isoprenaline, forskolin, adenosine and theophylline, whereas the effects of sodium nitroprusside were …

MaleNitroprussidemedicine.medical_specialtyAdenosineNifedipineMuscle RelaxationGuinea PigsNicardipineBronchiPharmacologyNicardipinechemistry.chemical_compoundTheophyllineIsoprenalineInternal medicinemedicineAnimalsHumansPharmacology (medical)TheophyllinePhosphodiesterase inhibitorPharmacologyForskolinDose-Response Relationship DrugChemistryColforsinIsoproterenolDihydropyridinePhosphodiesteraseDrug SynergismTracheaEndocrinologySodium nitroprussidemedicine.drugFundamental & Clinical Pharmacology
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Ceramide inhibits Kv currents and contributes to TP-receptor-induced vasoconstriction in rat and human pulmonary arteries

2011

et al.

MalePatch-Clamp TechniquesPhysiologyReceptors ThromboxaneSpider Venoms030204 cardiovascular system & hematologyMuscle Smooth VascularMembrane Potentialschemistry.chemical_compound0302 clinical medicineHypoxic pulmonary vasoconstrictionVasoconstrictor AgentsProtein Kinase C0303 health sciencesAniline Compounds3. Good healthSphingomyelin Phosphodiesterasemedicine.anatomical_structurePotassium Channels Voltage-GatedCirculatory systemmedicine.symptomSphingomyelinSignal TransductionBlood vesselmedicine.medical_specialtyCeramidePhosphinesMyocytes Smooth MusclePulmonary ArteryBiologyCeramidesBenzylidene Compounds03 medical and health sciencesInternal medicinemedicineAnimalsHumansRats Wistar030304 developmental biologyCell BiologySphingolipidRatsHEK293 CellsEndocrinologychemistryVasoconstriction15-Hydroxy-11 alpha9 alpha-(epoxymethano)prosta-513-dienoic AcidVascular resistanceVascular ResistancePeptidesVasoconstrictionAmerican Journal of Physiology-Cell Physiology
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Effects of selective phosphodiesterase inhibitors on platelet-activating factor- and antigen-induced airway hyperreactivity, eosinophil accumulation,…

1996

There is currently interest in the potential use of selective inhibitors of cyclic nucleotide phosphodiesterases (PDE) in the treatment of asthma. In this study we examined the effects of three selective PDE inhibitors, milrinone (PDE III), rolipram (PDE IV) and zaprinast (PDE V), on the broncoconstriction produced by antigen and histamine, the airway hyperreactivity and microvascular leakage after aerosol exposure to platelet-activating factor (PAF) and antigen, and the antigen-induced eosinophil infiltration in guinea-pig lung. Inhaled rolipram (0.01-10 mg ml-1) inhibited dose dependently the bronchospasm produced by aerosol antigen (5 mg ml-1) an anaesthetised, ventilated guinea-pigs. Ro…

MalePhosphodiesterase InhibitorsGuinea PigsRespiratory SystemPharmacologychemistry.chemical_compoundmedicineAnimalsPlatelet Activating FactorRolipramPharmacologyPlatelet-activating factorDose-Response Relationship DrugChemistryPhosphodiesteraseGeneral Medicinerespiratory systemExtravasationAsthmaPyrrolidinonesEosinophilsImmunologyMilrinoneBronchoconstrictionmedicine.symptomZaprinastRolipramHistaminemedicine.drugHistamineNaunyn-Schmiedeberg's archives of pharmacology
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Effects of inhaled glaucine on pulmonary responses to antigen in sensitized guinea pigs.

2000

The alkaloid (S)-(+)-1,2,9,10-tetramethoxyaporphine (glaucine) is a phosphodiesterase 4 inhibitor with bronchodilator and anti-inflammatory activity in vitro. In this study, we examined the in vivo effects of glaucine on an animal model of asthma. In ovalbumin sensitized guinea pigs, inhaled glaucine (10 mg ml(-1), 3 min) inhibited the acute bronchoconstriction produced by aerosol antigen (antigen response was 256+/-42 and 95+/-14 cm H(2)O l(-1) s(-1) in control and glaucine-treated animals, respectively; P<0.05). Pretreatment with glaucine (10 mg ml(-1), 10 min inhalation, 30 min pre- and 3 h post-antigen exposure) markedly reduced airway hyperreactivity to histamine, eosinophil lung accum…

MalePulmonary CirculationAporphinesEosinophil Peroxidasemedicine.drug_classBronchoconstrictionGuinea PigsPharmacologyGuinea pigCapillary Permeabilitychemistry.chemical_compoundBronchodilatormedicineCyclic AMPAnimalsAntigensLungPharmacologybiologyInhalationDose-Response Relationship Drugbusiness.industryrespiratory systemEosinophilGlaucineCyclic Nucleotide Phosphodiesterases Type 4EosinophilsTracheamedicine.anatomical_structurechemistryPeroxidases3'5'-Cyclic-AMP PhosphodiesterasesImmunologybiology.proteinBronchoconstrictionVascular Resistancemedicine.symptomBronchial HyperreactivitybusinessEosinophil peroxidaseHistamineHistamineEuropean journal of pharmacology
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Phosphodiesterase 4 inhibition decreases MUC5AC expression induced by epidermal growth factor in human airway epithelial cells

2005

Background: A common pathological feature of chronic inflammatory airway diseases such as asthma and chronic obstructive pulmonary disease (COPD) is mucus hypersecretion. MUC5AC is the predominant mucin gene expressed in healthy airways and is increased in asthmatic and COPD patients. Recent clinical trials indicate that phosphodiesterase type 4 (PDE4) inhibitors may have therapeutic value for COPD and asthma. However, their direct effects on mucin expression have been scarcely investigated. Methods: MUC5AC mRNA and protein expression were examined in cultured human airway epithelial cells (A549) and in human isolated bronchial tissue stimulated with epidermal growth factor (EGF; 25 ng/ml).…

MalePulmonary and Respiratory Medicinemedicine.medical_specialtyBlotting WesternBronchiEnzyme-Linked Immunosorbent AssayRespiratory MucosaMucin 5ACp38 Mitogen-Activated Protein KinasesWestern blotEpidermal growth factorInternal medicineGene expressionCyclic AMPmedicineHumansRNA MessengerPhosphotyrosineCells CulturedRoflumilastRolipramAgedA549 cellEpidermal Growth Factormedicine.diagnostic_testReverse Transcriptase Polymerase Chain Reactionbusiness.industryCilomilastMucinMucinsEpithelial CellsMiddle Agedrespiratory systemMolecular biologyRecombinant ProteinsCyclic Nucleotide Phosphodiesterases Type 4respiratory tract diseasesEndocrinology3'5'-Cyclic-AMP PhosphodiesterasesAirway BiologyFemalebusinessmedicine.drugThorax
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