Search results for "physical exercise"

showing 10 items of 462 documents

Fructose 2,6-bisphosphate and glycolytic flux in skeletal muscle of swimming frog

1990

AbstractGlycolytic flux in skeletal muscle is controlled by 6-phosphofructokinase but how this is achieved is controversial. Brief exercise (swimming) in frogs caused a dramatic increase in the phosphofructokinase activator, fructose 2,6-bisphosphate, in working muscle. The kinetics of phosphofructokinase suggest that in resting muscle, the enzyme is inhibited by ATP plus citrate and that the increase in fructose 2,6-bisphosphate is part of the mechanism to activate phosphofructokinase when exercise begins. When exercise was sustained, fructose 2,6-bisphosphate in muscle was decreased as was the rate of lactate accumulation. Glycolytic flux and the content of fructose 2,6-bisphosphate appea…

medicine.medical_specialtyPhosphofructokinase-1Rana temporariaBiophysicsSkeletal musclePhysical exerciseMotor ActivityBiologyBiochemistrychemistry.chemical_compoundStructural BiologyInternal medicineFructosediphosphatesGeneticsmedicineAnimalsGlycolysisLactic AcidExerciseMolecular BiologySwimmingchemistry.chemical_classificationMusclesSkeletal muscleFructoseCell BiologyEnzyme ActivationKineticsFructose 26-bisphosphateEndocrinologymedicine.anatomical_structureEnzymeBiochemistrychemistryFructose 26-bisphosphateLactates6-PhosphofructokinaseAnuraHexosediphosphatesGlycolysisFlux (metabolism)PhosphofructokinaseFEBS Letters
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Sestrins: Novel antioxidant and AMPK-modulating functions regulated by exercise?

2013

Oxidative stress results from damage to tissues caused by free radicals and is increased by exercise. Peroxiredoxins (PRXs) maintain the cellular reducing environment by scavenging intracellular hydrogen peroxide. It has been recently noted that physical exercise has a positive effect on the PRX system, exerting a protective effect against oxidative stress-induced damage. However, other compounds, such as sestrins (SESNs), a stress-inducible protein family with antioxidant properties, should also be considered in the function of PRXs. SESNs are clearly involved in the regeneration process of PRXs and therefore may also be modulated by physical exercise. In addition, SESNs are clearly involv…

medicine.medical_specialtyPhysiologyClinical BiochemistryNeurodegenerationAMPKPhysical exerciseCell BiologyOxidative phosphorylationBiologymedicine.diseasemedicine.disease_causeCell biologyInsulin resistanceEndocrinologySarcopeniaInternal medicinemedicineSignal transductionOxidative stressJournal of Cellular Physiology
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Knee extension strength and walking speed in relation to quadriceps muscle composition and training in elderly women.

1994

Summary. Knee extension strength, walking speed, quadriceps muscle mass and composition of the muscle compartment were studied in 66 to 85-year-old female athletes and controls. Maximal voluntary knee extension force, force/body mass, extension torque, torque/body mass and walking speed were higher for the athletes than the controls. A muscle index indicating intramuscular fat and connective tissue measured using ultrasonography was lower for the athletes than the controls. There were no differences between the study groups in knee extension force related either to cross-sectional area (CSA) or lean tissue area (CSAL) of the quadriceps. Within the subgroups, there was no significant correla…

medicine.medical_specialtyPhysiologyPhysical exerciseIsometric exerciseWalkingKnee extensionIsometric ContractionmedicineHumansKneeMuscle SkeletalAgedUltrasonographyAged 80 and overPhysical Education and TrainingbiologyAthletesbusiness.industryBody WeightQuadriceps muscleGeneral Medicinebiology.organism_classificationBody HeightPreferred walking speedAdipose TissuePhysical therapyBody CompositionFemaleIntramuscular fatUltrasonographybusinessTomography X-Ray Computedhuman activitiesClinical physiology (Oxford, England)
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The metabolic syndrome in hypertension: European society of hypertension position statement.

2008

The metabolic syndrome considerably increases the risk of cardiovascular and renal events in hypertension. It has been associated with a wide range of classical and new cardiovascular risk factors as well as with early signs of subclinical cardiovascular and renal damage. Obesity and insulin resistance, beside a constellation of independent factors, which include molecules of hepatic, vascular, and immunologic origin with proinflammatory properties, have been implicated in the pathogenesis. The close relationships among the different components of the syndrome and their associated disturbances make it difficult to understand what the underlying causes and consequences are. At each of these …

medicine.medical_specialtyPhysiologySodium Chloride Symporter InhibitorsAdrenergic beta-AntagonistsPhysical exerciseAngiotensin-Converting Enzyme InhibitorsType 2 diabetesBioinformaticsInsulin resistanceWeight lossInternal medicineInternal MedicinemedicineHumansThiazideAntihypertensive AgentsMetabolic Syndromebusiness.industrymedicine.diseaseCalcium Channel BlockersObesityExercise TherapyEndocrinologyBlood pressureHypertensionmedicine.symptomMetabolic syndromeCardiology and Cardiovascular MedicinebusinessAngiotensin II Type 1 Receptor BlockersRisk Reduction Behaviormedicine.drugDiet TherapyJournal of hypertension
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Neuromuscular electrical stimulation: a new therapeutic option for chronic diseases based on contraction-induced myokine secretion

2019

Myokines are peptides known to modulate brain neuroplasticity, adipocyte metabolism, bone mineralization, endothelium repair and cell growth arrest in colon and breast cancer, among other processes. Repeated skeletal muscle contraction induces the production and secretion of myokines, which have a wide range of functions in different tissues and organs. This new role of skeletal muscle as a secretory organ means skeletal muscle contraction could be a key player in the prevention and/or management of chronic disease. However, some individuals are not capable of optimal physical exercise in terms of adequate duration, intensity or muscles involved, and therefore they may be virtually deprived…

medicine.medical_specialtyPhysiologyStimulationPhysical exerciseReviewskeletal muscle contractionTranscutaneous electrical nerve stimulationcytokines; electrotherapy; physical exercise; skeletal muscle contraction; transcutaneous electrical nerve stimulationlcsh:Physiologylaw.inventionMuscle hypertrophyParacrine signallingtranscutaneous electrical nerve stimulationlawphysical exerciseInternal medicinePhysiology (medical)MyokinemedicineAutocrine signallinglcsh:QP1-981business.industrySkeletal muscleDeporteEjercicio físicocytokinesElectroterapiaEndocrinologymedicine.anatomical_structureTerapiaelectrotherapyElectricidadbusiness
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Exercise causes blood glutathione oxidation in chronic obstructive pulmonary disease: prevention by O2 therapy

1996

Viña, José, Emilio Servera, Miguel Asensi, Juan Sastre, Federico V. Pallardó, José A. Ferrero, JoséGarcı́a-de-la-Asunción, Vicente Antón, and Julio Marı́n. Exercise causes blood glutathione oxidation in chronic obstructive pulmonary disease: prevention by O2therapy. J. Appl. Physiol. 81(5): 2199–2202, 1996.—The aim of the present study was to determine whether glutathione oxidation occurs in chronic obstructive pulmonary disease (COPD) patients who perform exercise and whether this could be prevented. Blood glutathione red-ox ratio [oxidized-to-reduced glutathione (GSSG/GSH)] was significantly increased when patients performed exercise for a short period of time until exhaustion. Their res…

medicine.medical_specialtyPhysiologybusiness.industryLactic acid bloodRespiratory diseasePulmonary diseasePhysical exerciseOxidation reductionGlutathioneHypoxia (medical)medicine.diseaseSurgerychemistry.chemical_compoundO2 therapychemistryPhysiology (medical)Internal medicinemedicineCardiologymedicine.symptombusinessJournal of Applied Physiology
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The effect of hormone replacement therapy and/or exercise on skeletal muscle attenuation in postmenopausal women: a yearlong intervention

2005

Hormone replacement therapy (HRT) has been reported to exert a positive effect on preserving muscle strength following the menopause, however, the mechanism of action remains unclear. We examined whether the mechanism involved preservation of muscle composition as determined by skeletal muscle attenuation. Eighty women aged 50-57 years were randomly assigned to either: HRT, exercise (Ex), HRT + exercise (ExHRT), and control (Co) for 1 year. The study was double-blinded with subjects receiving oestradiol and norethisterone acetate (Kliogest) or placebo. Exercise included progressive high-impact training for the lower limbs. Skeletal muscle attenuation in Hounsfield units (HU) was determined …

medicine.medical_specialtyPhysiologymedia_common.quotation_subjectAdipose tissuePhysical exerciseVertical jumpDouble-Blind MethodPhysiology (medical)Internal medicinemedicineHormone replacement therapy (male-to-female)Body SizeHumansMuscle SkeletalExerciseMenstrual cyclemedia_commonAnalysis of Variancebusiness.industryBody WeightEstrogen Replacement TherapySkeletal muscleGeneral MedicineMiddle Agedmedicine.diseaseNorethisterone acetatePostmenopauseMenopauseEndocrinologymedicine.anatomical_structureAdipose TissueFemaleTomography X-Ray Computedbusinessmedicine.drugClinical Physiology and Functional Imaging
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Combination of hormone replacement therapy and high physical activity is associated with differences in Achilles tendon size in monozygotic female tw…

2009

Estrogen concentration has been suggested to play a role in tendon abnormalities and injury. In physically active postmenopausal women, hormone replacement therapy (HRT) has been suggested to decrease tendon diameter. We hypothesized that HRT use and physical activity are associated with Achilles tendon size and tissue structure. The study applied cotwin analysis of fourteen 54- to 62-yr-old identical female twin pairs with current discordance for HRT use for an average of 7 yr. Achilles tendon thickness and cross-sectional areas were determined by ultrasonography, and tendon structural organization was analyzed from the images using linear discriminant analysis (LDA). Maximal voluntary and…

medicine.medical_specialtyPhysiologymedicine.drug_classEstroneHormone Replacement TherapyAnterior cruciate ligamentPhysical activityMonozygotic twinPhysical exerciseMotor ActivityAchilles Tendon03 medical and health sciences0302 clinical medicinePhysiology (medical)Internal medicineSex Hormone-Binding GlobulinMedicineHumansTestosteroneMuscle StrengthAgedUltrasonographyAchilles tendonbusiness.industryEstrogens030229 sport sciencesTwins MonozygoticMiddle AgedTendonmedicine.anatomical_structureEndocrinologyCholesterolTransgender hormone therapyEstrogenFemaleMenopausebusiness030217 neurology & neurosurgeryMuscle ContractionJournal of applied physiology (Bethesda, Md. : 1985)
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Effects of hormone replacement therapy and high-impact physical exercise on skeletal muscle in post-menopausal women: a randomized placebo-controlled…

2001

An age-related decline in muscle performance is a known risk factor for falling, fracture and disability. In women, a clear deterioration is observed from early menopause. The effect of hormone replacement therapy (HRT) in preserving muscle performance is, however, unclear. This trial examined the effects of a 12-month HRT and high-impact physical exercise regimen on skeletal muscle in women in early menopause. A total of 80 women aged 50–57 years were assigned randomly to one of four groups: exercise (Ex), HRT, exercise+HRT (ExHRT) and control (Co). The exercise groups participated in a high-impact training programme. The administration of HRT (oestradiol/noretisterone acetate) or placebo …

medicine.medical_specialtyPlacebo-controlled studyPhysical exercisePlacebolaw.inventionDouble-Blind MethodRandomized controlled triallawInternal medicineElectric ImpedancemedicineHumansExercise physiologyMuscle SkeletalExerciseAnalysis of VarianceEstradiolbusiness.industryEstrogen Replacement TherapySkeletal muscleGeneral MedicineMiddle Agedmedicine.diseaseCombined Modality TherapyBiomechanical PhenomenaPostmenopauseMenopausemedicine.anatomical_structureEndocrinologyTorqueAnesthesiaBody CompositionFemaleNorethindronemedicine.symptomTomography X-Ray ComputedbusinessMuscle contractionClinical Science
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Epidemiological, biological and clinical update on exercise-induced hemolysis

2019

Exercise-induced hemolysis can be conventionally defined as rupture and destruction of erythrocytes during physical exercise. The currently available epidemiologic information attests that a substantial degree of exercise-induced hemolysis is commonplace after short-, medium-, long- and ultra-long distance running, as reflected by significant decrease of serum or plasma haptoglobin and significant increase of plasma concentration (or overall blood content) of free hemoglobin. This paraphysiological intravascular hemolysis is typically mild (average variations of hemolysis biomarkers are usually comprised between 1.2- and 1.8-fold), almost self-limiting (completely resolving within 24-48 hou…

medicine.medical_specialtyPopulationPhysical exerciseReview Article030204 cardiovascular system & hematologyExercise-induced hemolysisHemolysis03 medical and health sciences0302 clinical medicinephysical exerciseEndurance trainingInternal medicinemedicineHemolysis; hemoglobin; red blood cells (RBC); sports physical exerciseeducationeducation.field_of_studyred blood cells (RBC)biologybusiness.industryHaptoglobinErythrocyte fragility030229 sport sciencesGeneral Medicinehemoglobinmedicine.diseaseHemolysisHypotonic ShockEndocrinologybiology.proteinsportsbusinessAnnals of Translational Medicine
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