Search results for "polymorph"

showing 10 items of 2115 documents

Opposite effects of interleukin 10 common gene polymorphisms in cardiovascular diseases and in successful ageing: genetic background of male centenar…

2004

Many aspects of ageing involve inflammatory processes. We evaluated the association with longevity of alleles of IL10 and TNFa, known to have opposite functions in inflammatory reactions, IL-10 acting predominantly as an anti-inflammatory and TNF-a as a proinflammatory factor. The number of male centenarians homozygous for the –1082G genotype, suggested to be associated with high IL-10 production, was significantly increased in comparison with younger control subjects. No significant differences were observed between women and controls. The genotypic frequencies of the TNFa promoter SNPs 308G and 308A, suggested to be associated with low and high TNF-a production respectively, were not sign…

AdultMaleAgingmedicine.medical_specialtyOffspringmedia_common.quotation_subjectLongevityMyocardial InfarctionPhysiologyDiseaseBiologyInternal medicineEpidemiologyGeneticsmedicineHumansMyocardial infarctionAlleleAllelesGenetics (clinical)Agedmedia_commonAged 80 and overPolymorphism GeneticBase SequenceHaplotypeLongevityMiddle Agedmedicine.diseaseInterleukin-10EndocrinologyHaplotypesItalyCardiovascular DiseasesAgeingLetter to JMGJournal of Medical Genetics
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No evidence for a correlation of glutathione S-tranferase polymorphisms and chronic rhinosinusitis.

2011

OBJECTIVE: Cellular detoxification mechanisms are mandatory for cellular protection against oxidative stress and reactive oxygen species. One major group of antioxidative active enzymes involved in cellular detoxification are the Glutathione S-Transferases (GST). Multiple subtypes like GSTM1, GSTP1, and GSTT1 and variants of them are known, arising from allelic variations of the GST loci. Moreover, functional variants occur in high percentages and have been associated with diseases like bronchial asthma and bronchial hyperresponsiveness. The interplay of oxidative stress, detoxifying genes like GSTs and the genesis of respiratory tract illness is under contradictory debate. In this study, w…

AdultMaleAllergyCellular detoxificationComorbidityGSTP1Nasal Polypsotorhinolaryngologic diseasesmedicineGenetic predispositionHypersensitivityHumansNasal polypsGenetic Predisposition to DiseaseSinusitisAsthmaGlutathione TransferaseRhinitisPolymorphism Geneticbusiness.industryGeneral Medicinerespiratory systemmedicine.diseaseAsthmaNasal MucosaOxidative Stressmedicine.anatomical_structureOtorhinolaryngologyGlutathione S-Transferase piBronchial hyperresponsivenessImmunologyChronic DiseaseFemalebusinessRespiratory tractRhinology
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Angiotensin II AT1 receptor gene polymorphism and microalbuminuria in essential hypertension.

2001

The objective of this study was to analyze the relationship of polymorphisms of the angiotensin II AT1 receptor gene with microalbuminuria in a group of young adults with essential hypertension. Essential hypertensives, less than 50 years old, never previously treated with antihypertensive drugs, and in absence of diabetes mellitus were included. Office blood pressure (BP), 24-h ambulatory BP monitoring, urinary albumin excretion (UAE) measurements, and DNA analysis were performed. Polymorphisms of the angiotensin II AT1-receptor gene (A1166C and C573T) were studied by polymerase chain reaction and single-strand conformation polymorphism techniques. One hundred eighty-three patients, 49 (27…

AdultMaleAngiotensin receptormedicine.medical_specialtyAmbulatory blood pressureGenotypeGenetic LinkageEssential hypertensionReceptor Angiotensin Type 2Receptor Angiotensin Type 1Internal medicineInternal MedicineMedicineAlbuminuriaHumansAngiotensin II receptor type 1ProteinuriaPolymorphism GeneticReceptors Angiotensinbusiness.industryMiddle Agedmedicine.diseaseAngiotensin IIEndocrinologyHypertensionMicroalbuminuriaGene polymorphismmedicine.symptombusinessAmerican journal of hypertension
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Disease-associated polymorphisms in ERAP1 do not alter endoplasmic reticulum stress in patients with ankylosing spondylitis

2014

The mechanism by which human leukocyte antigen B27 (HLA-B27) contributes to ankylosing spondylitis (AS) remains unclear. Genetic studies demonstrate that association with and interaction between polymorphisms of endoplasmic reticulum aminopeptidase 1 (ERAP1) and HLA-B27 influence the risk of AS. It has been hypothesised that ERAP1-mediated HLA-B27 misfolding increases endoplasmic reticulum (ER) stress, driving an interleukin (IL) 23-dependent, pro-inflammatory immune response. We tested the hypothesis that AS-risk ERAP1 variants increase ER-stress and concomitant pro-inflammatory cytokine production in HLA-B27(+) but not HLA-B27(-) AS patients or controls. Forty-nine AS cases and 22 healthy…

AdultMaleAnkylosing Spondylitismedicine.medical_treatmentImmunologyInflammationSingle-nucleotide polymorphismDiseaseBiologyERAP1AminopeptidasesPolymorphism Single NucleotideMinor Histocompatibility AntigensYoung AdultGene expressionGeneticsmedicineHumansSpondylitis AnkylosingERAP1 Ankylosing SpondylitisEndoplasmic Reticulum Chaperone BiPSpondylitisHLA-B27 AntigenGenetics (clinical)InflammationAnkylosing spondylitisEndoplasmic reticulumMiddle AgedEndoplasmic Reticulum Stressmedicine.diseaseCytokineImmunologyFemalemedicine.symptomGenes & Immunity
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Association of smoking but not HLA-DRB1*15:01, APOE or body mass index with brain atrophy in early multiple sclerosis

2019

Background: The course of multiple sclerosis (MS) shows substantial inter-individual variability. The underlying determinants of disease severity likely involve genetic and environmental factors. Objective: The aim of this study was to assess the impact of APOE and HLA polymorphisms as well as smoking and body mass index (BMI) in the very early MS course. Methods: Untreated patients ( n = 263) with a recent diagnosis of relapsing-remitting (RR) MS or clinically isolated syndrome underwent standardized magnetic resonance imaging (MRI). Genotyping was performed for single-nucleotide polymorphisms (SNPs) rs3135388 tagging the HLA-DRB1*15:01 haplotype and rs7412 (Ɛ2) and rs429358 (Ɛ4) in APOE. …

AdultMaleApolipoprotein EMultiple SclerosisAdolescentPolymorphism Single NucleotideBody Mass IndexYoung Adult03 medical and health sciencesApolipoproteins E0302 clinical medicineAtrophyMedizinische FakultätmedicineHumansSNPGenetic Predisposition to Disease030212 general & internal medicineddc:610Risk factorHLA-DRB1Agedbusiness.industryMultiple sclerosisSmokingNeurodegenerationBrainMiddle Agedmedicine.diseaseNeurologyImmunologyFemaleNeurology (clinical)AtrophybusinessBody mass index030217 neurology & neurosurgeryHLA-DRB1 Chains
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Saturated fat intake and alcohol consumption modulate the association between the APOE polymorphism and risk of future coronary heart disease: a nest…

2011

The association is still not clear between the common APOE polymorphism and coronary heart disease (CHD) risk, nor its modulation by diet. Thus, our aim was to study the association between the APOE genotypes and incident CHD and how dietary fat and alcohol consumption modify these effects. We performed a nested case-control study in the Spanish European Prospective Investigation into Cancer and Nutrition cohort. Healthy men and women (41,440, 30-69 years) were followed up over a 10-year period, with the incident CHD cases being identified. We analyzed 534 incident CHD cases and 1123 controls. APOE, dietary intake and plasma lipids were determined at baseline. The APOE polymorphism was sign…

AdultMaleApolipoprotein Emedicine.medical_specialtyAlcohol DrinkingGenotypeEndocrinology Diabetes and MetabolismSaturated fatClinical BiochemistryPopulationCoronary DiseaseBiochemistryGastroenterologyWhite PeopleApolipoproteins ERisk FactorsInternal medicineOdds RatiomedicineHumansLongitudinal StudieseducationMolecular BiologyAllelesAgededucation.field_of_studyPolymorphism GeneticNutrition and Dieteticsbusiness.industryFatty AcidsCase-control studyCholesterol LDLOdds ratioMiddle AgedDietEuropean Prospective Investigation into Cancer and NutritionEndocrinologySpainCase-Control StudiesMultivariate AnalysisNested case-control studySaturated fatty acidRegression AnalysisFemalebusinessThe Journal of Nutritional Biochemistry
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Associations of LPL and APOC3 gene polymorphisms on plasma lipids in a mediterranean population: Interaction with tobacco smoking and the APOE locus

2002

We conducted a cross-sectional study in a Spanish population (n = 1,029) to investigate associations between the LPL and APOC3 gene loci (LPL-HindIII, LPL-S447X, and APOC3-SstI) and plasma lipid levels and their interaction with APOE polymorphisms and smoking. Carriers of the H− or the X447 allele had higher levels of HDL cholesterol (HDL-C), and lower levels of TG, after adjustment for age, body mass index, alcohol, smoking, exercise, and education (P < 0.01). The APOC3 polymorphism presented additive effects to the LPL variants on TG and HDL-C levels in men, and on TG in women. The most and the least favorable haplotype combinations were H−/X447/S1 and H+/S447/S2, respectively. These comb…

AdultMaleApolipoprotein Emedicine.medical_specialtyapolipoprotein C-IIIPopulationlipoprotein lipaseLocus (genetics)Deoxyribonuclease HindIIIQD415-436Biochemistrylipidschemistry.chemical_compoundApolipoproteins EEndocrinologyInternal medicineHumansMedicineAlleleApolipoproteins CDeoxyribonucleases Type II Site-SpecificeducationTriglyceridesGeneticseducation.field_of_studyLipoprotein lipasePolymorphism Geneticbusiness.industryCholesterolCholesterol HDLSmokingHaplotypeGenetic Variationnutritional and metabolic diseasesCell BiologyCross-Sectional StudiesEndocrinologychemistrySpainFemalelipids (amino acids peptides and proteins)gene-environmental interactionbusinessBody mass index
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Analysis of IL-6, IL-10 and IL-17 genetic polymorphisms as risk factors for sepsis development in burned patients.

2012

Abstract Infection risk, sepsis and mortality after severe burn are primarily determined by patient age, burn size, and depth. Whether genetic differences contribute to otherwise unexpected variability in outcomes is unknown. We sought to determine whether there was an association between IL-6, IL-10 and IL-17 polymorphisms with cytokine production and development of sepsis. We evaluated 71 patients with burns ≥15% TBSA and 109 healthy subjects. The genotypes of IL-6 (−174C/G), IL-10 (−819C/T and −1082A/G) and IL-17 (7488T/C) polymorphisms were identified applying polymerase chain reaction protocols. The cytokine levels in serum were determined with enzyme-linked immunoabsorbent assays. Our…

AdultMaleBurns Cytokine polymorphisms sepsis riskAdolescentGenotypemedicine.medical_treatmentCritical Care and Intensive Care MedicinePolymerase Chain Reactionlaw.inventionSepsisYoung AdultlawRisk FactorsSepsisGenotypeSettore MED/05 - Patologia ClinicaMedicineHumansInterleukin 6Polymerase chain reactionAgedAged 80 and overPolymorphism Geneticbiologybusiness.industryInterleukin-6Interleukin-17General MedicineMiddle Agedmedicine.diseaseGenotype frequencyInterleukin-10Interleukin 10CytokineImmunologyEmergency Medicinebiology.proteinSurgeryFemaleInterleukin 17businessBurnsBurns : journal of the International Society for Burn Injuries
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Duffy antigen receptor for chemokines (Darc) polymorphism regulates circulating concentrations of monocyte chemoattractant protein-1 and other inflam…

2010

AbstractTo identify the genetic basis of circulating concentrations of monocyte chemoattractant protein-1 (MCP-1), we conducted genome-wide association analyses for MCP-1 in 3 independent cohorts (n = 9598). The strongest association was for serum MCP-1 with a nonsynonymous polymorphism, rs12075 (Asp42Gly) in DARC, the gene for Duffy antigen receptor for chemokines, a known vascular reservoir of proinflammatory cytokines (minor allele frequency, 45.6%; P &lt; 1.0 * 10−323). This association was supported by family-based genetic linkage at a locus encompassing the DARC gene (genome-wide P = 8.0 * 10−13). Asp42Gly accounted for approximately 20% of the variability in serum MCP-1 concentration…

AdultMaleCCR2ChemokineErythrocytesImmunologyReceptors Cell SurfacePolymorphism Single NucleotideBiochemistryProinflammatory cytokineCohort StudiesmedicineHumansCytokine bindingReceptorInterleukin 6Chemokine CCL2biologyMonocyteCell BiologyHematologyMiddle AgedMolecular biologymedicine.anatomical_structureChromosomes Human Pair 1Genetic LociImmunologybiology.proteinFemaleInterleukin 18Inflammation MediatorsDuffy Blood-Group SystemGenome-Wide Association StudyBlood
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Association between platelet endothelial cellular adhesion molecule 1 (PECAM-1/CD31) polymorphisms and acute myocardial infarction: a study in patien…

2004

Summary Adhesion of circulating cells to the arterial surface is among the first detectable events in atherogenesis. Cellular adhesion molecules, expressed by the vascular endothelium and by circulating leucocytes, mediate cell recruitment and their transendothelial migration. Platelet endothelial cellular adhesion molecule 1 (PECAM-1/CD31), involved in this migration, has been associated with the developmental course of atherosclerosis. A few studies have investigated an association between coronary heart disease and single nucleotide polymorphisms (SNPs) located in functionally important domains of the PECAM-1/CD31 gene. In particular, Ser563Asn and Gly670Arg SNPs have been described as s…

AdultMaleCD31GenotypeCell adhesion moleculeImmunologyMyocardial InfarctionSingle-nucleotide polymorphismOdds ratioMiddle AgedBiologymedicine.diseasePlatelet Endothelial Cell Adhesion Molecule-1Gene FrequencyCase-Control StudiesImmunologyGenotypeGeneticsmedicineHumansGenetic Predisposition to DiseaseMyocardial infarctionCell adhesionSicilyAllele frequency
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