Search results for "polymorphism"

showing 10 items of 1968 documents

Variability in human hepatic MRP4 expression: influence of cholestasis and genotype

2007

The multidrug resistance protein 4 (MRP4) is an efflux transporter involved in the transport of endogenous substrates and xenobiotics. We measured MRP4 mRNA and protein expression in human livers and found a 38- and 45-fold variability, respectively. We sequenced 2 kb of the 5'-flanking region, all exons and intron/exon boundaries of the MRP4 gene in 95 patients and identified 74 genetic variants including 10 non-synonymous variations, seven of them being located in highly conserved regions. None of the detected polymorphisms was significantly associated with changes in the MRP4 mRNA or protein expression. Immunofluorescence microscopy indicated that none of the non-synonymous variations af…

AdultMaleGenotypeProtein ConformationBiologyPolymorphism Single NucleotideExonCholestasisTerminology as TopicGenotypeGenetic variationGeneticsmedicineHumansRNA MessengerGeneCellular localizationPharmacologyMessenger RNACholestasisPolymorphism GeneticReverse Transcriptase Polymerase Chain ReactionIntronGenetic VariationDNAmedicine.diseaseImmunohistochemistryMolecular biologyIntronsGene Expression RegulationHaplotypesLiverMicroscopy FluorescenceMolecular MedicineFemaleMultidrug Resistance-Associated ProteinsThe Pharmacogenomics Journal
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Increased frequency of the CTLA-4 49 A/G polymorphism in patients with acquired haemophilia A compared to healthy controls

2007

Acquired haemophilia (AH) is an autoimmune disorder characterized by autoantibodies against endogenous factor VIII (FVIII). Half of the patients present with an underlying disease known to cause the FVIII autoantibodies whereas in the other half the disease is of idiopathic nature. Recently, it has been shown that variants of the polymorphic cytotoxic T lymphocyte antigen-4 (CTLA-4) gene are associated with autoimmune diseases and also represent a risk factor for inhibitor formation in inherited haemophilia A. In the present study, we investigated whether CTLA-4 variants also play a role in the pathogenesis of AH. Therefore, we analyzed three single nucleotide polymorphisms (SNPs) of the CT…

AdultMaleGenotypeSingle-nucleotide polymorphismHemophilia AHaemophiliaPolymorphism Single NucleotideGene FrequencyAntigens CDGenotypemedicineHumansCTLA-4 AntigenGenetic Predisposition to DiseaseAlleleAllele frequencyGenetics (clinical)AgedAutoantibodiesAged 80 and overAutoimmune diseaseFactor VIIIbusiness.industryAutoantibodyHematologyGeneral MedicineMiddle Agedmedicine.diseaseAntigens DifferentiationCase-Control StudiesImmunologyFemaleGene polymorphismbusinessHaemophilia
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Precision Medicine in Graves’ Disease: CD40 Gene Variants Predict Clinical Response to an Anti-CD40 Monoclonal Antibody

2021

BackgroundCD40, a key co-stimulatory molecule expressed on antigen-presenting cells, is genetically associated with a number of autoimmune diseases including Graves’ disease (GD). Therefore, recent therapies targeting CD40 have been developed, including the anti-CD40 monoclonal antibody Iscalimab. In a recent pilot study, Iscalimab was shown to induce clinical remission in ~ 50% of GD patients, but the reason why only 50% of GD patients responded is not known. The aim of our study was to test the hypothesis that specific CD40 single nucleotide polymorphism (SNP) genotypes and haplotypes are associated with clinical response of GD patients to Iscalimab.MethodsWe extracted genomic DNA from th…

AdultMaleGenotypemedicine.drug_classprecision medicineEndocrinology Diabetes and MetabolismGraves' diseaseSingle-nucleotide polymorphismmedicine.disease_causeMonoclonal antibodyPolymorphism Single NucleotideDiseases of the endocrine glands. Clinical endocrinologyAutoimmunityYoung AdultEndocrinologyGenotypeCD40HumansMedicineSNPRNA MessengerCD40 AntigensgeneGeneAgedOriginal Researchbusiness.industryHaplotypeAntibodies MonoclonalMiddle AgedRC648-665medicine.diseaseGraves DiseasevariantImmunologyFemaleGraves’ diseasebusinessFrontiers in Endocrinology
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The -308G/A polymorphism of TNF-alpha influences immunological parameters in old subjects affected by infectious diseases.

2005

Abnormal increments of pro-inflammatory cytokines (IL-6 and TNF-alpha) characterize the outbreak of infectious diseases, which are the major cause of death in the elderly. A counterbalance to the inflammation is exerted by IL-10 with an inhibitory role on TNF-alpha production. As is well known, some cytokine gene polymorphisms influence the cytokine production, playing a role as susceptibility or resistance factors against immune-mediated and infectious disease. Genetic variations in the -308A/G locus for TNF-alpha seems to affect the clinical outcome of some infectious diseases. In fact, the -308A allele is associated with severe septic shock and death. On this basis, we have screened heal…

AdultMaleGenotypemedicine.medical_treatmentImmunologyCommunicable DiseasesGene FrequencyGenotypeGeneticsmedicineBronchopneumoniaHumansAlleleInterleukin 6Molecular BiologyAllele frequencyGenetics (clinical)AgedPolymorphism GeneticbiologyInterleukin-6Tumor Necrosis Factor-alphaOutbreakGeneral MedicineMiddle AgedBronchitis ChronicKiller Cells NaturalZincCytokineInfectious disease (medical specialty)Immunologybiology.proteinTumor necrosis factor alphaFemaleMetallothioneinInternational journal of immunogenetics
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Specific association of IL17A genetic variants with panuveitis.

2015

Background/aims A pathogenic role of Th17 cells in uveitis has become clear in recent years. Therefore, in the present study, we aimed to evaluate the possible influence of the IL 17A locus on susceptibility to non-anterior uveitis and its main clinical subgroups. Methods Five IL17A polymorphisms (rs4711998, rs8193036, rs3819024, rs2275913 and rs7747909), selected by tagging, were genotyped using TagMan assays in 353 Spanish patients with non-anterior uveitis and 1851 ethnically matched controls. Results The case/control analysis yielded a consistent association between two of the analysed genetic variants, rs8193036 and rs2275913, and the presence of panuveitis under a dominant model (p(FD…

AdultMaleGenotyping TechniquesImmunologyLocus (genetics)DiseaseReal-Time Polymerase Chain ReactionPolymorphism Single NucleotideWhite PeopleCellular and Molecular NeuroscienceGene FrequencyGenetic modelPanuveitismedicineGeneticsHumansGenetic Predisposition to DiseaseInflammationbusiness.industryPanuveitisInterleukin-17Middle Agedmedicine.diseaseSensory SystemsOphthalmologymedicine.anatomical_structureImmunologyIntermediate uveitisTh17 CellsFemaleIL17AChoroidbusinessUveitisThe British journal of ophthalmology
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A functional polymorphism in theIL-10 promoter influences the response after vaccination with HBsAg and hepatitis A

2005

The immune response to hepatitis B surface antigen (HBsAg) is mostly genetically determined. Interleukin 10 (IL-10) is a central immunoregulatory cytokine with important effects on B-cells. We have studied the influence of IL-10 promoter polymorphisms on the immune response to HBsAg and hepatitis A vaccination. We vaccinated 202 twin pairs in an open prospective study with a combined recombinant HBsAg/inactivated hepatitis A vaccine. IL-10 promoter polymorphisms were investigated in all individuals and their influence on anti-HBs, and anti-HAV responsiveness was studied. In the multiple regression analysis accounting for smoking, gender, body mass index and age, the ACC haplotype (-1082, -8…

AdultMaleHBsAgHepatitis A vaccineTwinsBiologyAntigenmedicineHumansProspective StudiesAllelePromoter Regions GeneticAntigens ViralHepatitisHepatitis A VaccinesHepatitis B Surface AntigensPolymorphism GeneticHepatologyHaplotypevirus diseasesHepatitis Amedicine.diseaseVirologydigestive system diseasesInterleukin-10VaccinationImmunologyFemaleHepatology
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Sequential transcriptome analysis of human liver cancer indicates late stage acquisition of malignant traits

2014

Background & Aims Human hepatocarcinogenesis is as a multi-step process starting from dysplastic lesions to early carcinomas (eHCC) that ultimately progress to HCC (pHCC). However, the sequential molecular alterations driving malignant transformation of the pre-neoplastic lesions are not clearly defined. This lack of information represents a major challenge in the clinical management of patients at risk. Methods We applied next-generation transcriptome sequencing to tumor-free surrounding liver (n=7), low- (n=4) and high-grade (n=9) dysplastic lesions, eHCC (n=5) and pHCC (n=3) from 8 HCC patients with hepatitis B infection. Integrative analyses of genetic and transcriptomic changes were pe…

AdultMaleHepatocarcinogenesisCarcinoma HepatocellularCarcinogenesisBiologyBioinformaticsmedicine.disease_causePolymorphism Single NucleotideArticleMalignant transformationTranscriptomeCarcinomamedicineTumor MicroenvironmentHumansMolecular pathogenesisRNA NeoplasmGeneAgedTumor microenvironmentHepatologyGene Expression ProfilingLiver NeoplasmsWnt signaling pathwayRNA sequencingMiddle Agedmedicine.diseaseGene expression profilingCell Transformation NeoplasticMutationCancer researchDisease ProgressionFemaleCarcinogenesis
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Characterization of non-expressed C4 genes in a case of complete C4 deficiency: identification of a novel point mutation leading to a premature stop …

1998

The genetic basis of complete C4 deficiency in a patient with SLE was investigated. Previous studies have demonstrated that this patient has two different major histocompatibility complex (MHC) haplotypes that each contain a major deletion and a non-expressed C4 gene. In the present study, non-expression of the C4 genes was explained by the finding of two distinct C4 gene mutations. A previously described two base pair insertion in exon 29 of the C4 gene was detected in the paternal MHC haplotype [HLA-A2, B40, SC00, DR6]. The maternal haplotype [HLA-A30, B18, F1C00, DR3] carried a C4 gene with a one base pair deletion in exon 20 generating a premature stop codon. This mutation was neither f…

AdultMaleHeterozygoteImmunologyGene mutationBiologymedicine.disease_causePolymerase Chain ReactionCell LineMajor Histocompatibility ComplexExonmedicineImmunology and AllergyHumansLupus Erythematosus SystemicPoint MutationGenePolymorphism Single-Stranded ConformationalGeneticsMutationPoint mutationHaplotypeC4AComplement C4General MedicineExonsSequence Analysis DNAMolecular biologyIsotypePedigreeHaplotypesCodon TerminatorFemalePolymorphism Restriction Fragment LengthHuman immunology
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Perilipin polymorphism interacts with saturated fat and carbohydrates to modulate insulin resistance

2010

Macronutrient intakes and genetic variants have been shown to interact to alter the risk of insulin resistance, but replication of gene-nutrient interactions across independent populations may be difficult to achieve. Saturated fat and carbohydrate were previously shown to interact with genotype for insulin resistance for a variant of perilipin (PLIN), the major adipocyte-associated protein and a regulator of adipocyte metabolism. We investigated the same interaction for PLIN 11482G>A (rs894160) on insulin resistance in US men(n=462) and women(n=508) (mean ±SD, 49±16 years). In multivariate linear regression models, we found a significant (PA for HOMA-IR (homeostasis model assessment of ins…

AdultMaleHeterozygotePerilipin-1medicine.medical_specialtyAdolescentMinnesotaEndocrinology Diabetes and MetabolismSaturated fatmedicine.medical_treatmentMedicine (miscellaneous)BiologyPolymorphism Single NucleotideWhite PeopleArticleYoung Adultchemistry.chemical_compoundNutrigenomicsInsulin resistanceUtahAdipocyteInternal medicineDietary CarbohydratesmedicineHumansInsulinGenetic Association StudiesAgedAged 80 and overSex CharacteristicsNutrition and DieteticsInsulinMetabolismMiddle AgedCarbohydratePhosphoproteinsmedicine.diseaseDietary FatsEndocrinologychemistryPerilipinFemaleInsulin ResistanceCarrier ProteinsCardiology and Cardiovascular MedicineHomeostasisNutrition, Metabolism and Cardiovascular Diseases
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Obese Subjects Carrying the 11482G>A Polymorphism at the Perilipin Locus Are Resistant to Weight Loss after Dietary Energy Restriction

2005

Dietary treatment of obesity could be improved if predictive information about the individual's genetic response to diet was available. Adipose tissue has been the focus of efforts to identify candidate genes. Perilipin is a major protein found in adipocytes, and perilipin knockout mice are lean and resistant to diet-induced obesity.The objective of the study was to examine the association of several polymorphisms at the perilipin (PLIN) locus with obesity and weight reduction in response to a low-energy diet in obese patients.This study was a 1-yr randomized (depending on the PLIN genotype) trial with three follow-up evaluations.The study was conducted at a university research center.One h…

AdultMaleHeterozygotePerilipin-1medicine.medical_specialtyCandidate geneGuanineEndocrinology Diabetes and MetabolismClinical BiochemistryAdipose tissueBiologyBiochemistryEndocrinologyWeight lossInternal medicineWeight LossGenotypePrevalencemedicineHumansObesityAlleleAllelesAgedCaloric RestrictionPolymorphism GeneticAdenineBiochemistry (medical)Middle AgedPhosphoproteinsmedicine.diseaseObesityEndocrinologyDiabetes Mellitus Type 2PerilipinFemalemedicine.symptomCarrier ProteinsBody mass indexThe Journal of Clinical Endocrinology & Metabolism
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