Search results for "primer"

showing 10 items of 530 documents

Nuclear insulin receptor substrate 1 interacts with estrogen receptor alpha at ERE promoters.

2004

Insulin receptor substrate 1 (IRS-1) is a major signaling molecule activated by the insulin and insulin-like growth factor I receptors. Recent data obtained in different cell models suggested that in addition to its conventional role as a cytoplasmic signal transducer, IRS-1 has a function in the nuclear compartment. However, the role of nuclear IRS-1 in breast cancer has never been addressed. Here we report that in estrogen receptor alpha (ERalpha)-positive MCF-7 cells, (1) a fraction of IRS-1 was translocated to the nucleus upon 17-beta-estradiol (E2) treatment; (2) E2-dependent nuclear translocation of IRS-1 was blocked with the antiestrogen ICI 182,780; (3) nuclear IRS-1 colocalized and…

Cancer Researchmedicine.medical_specialtyTranscription Geneticmedicine.medical_treatmentBlotting WesternEstrogen receptorBiologyInsulin-like growth factorInternal medicineCell Line TumorGeneticsmedicineAnimalsHumansReceptorPromoter Regions GeneticMolecular BiologyNuclear receptor co-repressor 1DNA PrimersBase SequenceReverse Transcriptase Polymerase Chain ReactionEstrogen Receptor alphaPromoterAntiestrogenPhosphoproteinsPrecipitin TestsIRS1Cell biologyProtein TransportEndocrinologyNuclear receptorReceptors EstrogenInsulin Receptor Substrate ProteinsProtein BindingOncogene
researchProduct

The Synthetic Cannabinoid WIN 55,212-2 Sensitizes Hepatocellular Carcinoma Cells to Tumor Necrosis Factor-Related Apoptosis-Inducing Ligand (TRAIL)-I…

2010

In this article, we demonstrate that the synthetic cannabinoid R-(+)-(2,3-dihydro-5-methyl-3-[(4-morpholinyl)methyl]pyrol[1,2,3-de]-1,4-benzoxazin-6-yl)-(1-naphthalenyl) methanone mesylate (WIN 55,212-2) sensitizes human hepatocellular carcinoma (HCC) cells to apoptosis mediated by tumor necrosis-related apoptosis inducing ligand (TRAIL). The apoptotic mechanism induced by treatment with WIN/TRAIL combination involved the loss of the mitochondrial transmembrane potential and led to the activation of caspases. In HCC cells, WIN treatment induced the up-regulation of TRAIL death receptor DR5, an effect that seemed to be related to the increase in the level of p8 and CHOP, two factors implicat…

Carcinoma HepatocellularDNA ComplementaryMorpholinesApoptosisNaphthalenesCHOPMembrane PotentialsTNF-Related Apoptosis-Inducing LigandCell Line TumorSurvivinmedicineHumansWIN 55212-2Protein kinase BTranscription factorCaspaseDNA PrimersPharmacologybiologyCannabinoidsReverse Transcriptase Polymerase Chain ReactionLiver NeoplasmsGene AmplificationDNA NeoplasmFlow CytometryBenzoxazinesReceptors TNF-Related Apoptosis-Inducing LigandApoptosisMitochondrial MembranesImmunologybiology.proteinCancer researchMolecular MedicineTumor necrosis factor alphaTranscription Factor CHOPmedicine.drugMolecular Pharmacology
researchProduct

Expression of WISPs and of their novel alternative variants in human hepatocellular carcinoma cells

2005

WISPs (Wnt-induced secreted proteins) are members of the CCN (CTGF/Cyr61/Nov) family involved in fibrotic disorders and tumorigenesis. They have a typical structure composed of four conserved cysteine-rich modular domains, but variants of CCN members lacking one or more modules, generated by alternative splicing or gene mutations, have been described in various pathological conditions. WISP genes were first described as downstream targets of the Wnt signaling pathway, which is frequently altered in human hepatocellular carcinoma (HCC). In the present study, WISP mRNA expression was analyzed by RT-PCR in four human HCC cell lines (HepG2, HuH-6, HuH-7, HA22T/VGH). Our results show for the fir…

Carcinoma HepatocellularWISPHepatocellular carcinomaApoptosisGene mutationBiologymedicine.disease_causeGeneral Biochemistry Genetics and Molecular BiologyCCN Intercellular Signaling ProteinsWntalternative splicingHistory and Philosophy of ScienceCell Line TumorProto-Oncogene ProteinsCCN Intercellular Signaling ProteinsmedicineHumansRNA MessengerGeneDNA PrimersOncogene ProteinsGeneticsCCNModels GeneticReverse Transcriptase Polymerase Chain ReactionGeneral NeuroscienceLiver NeoplasmsAlternative splicingIntracellular Signaling Peptides and ProteinsWnt signaling pathwaydigestive system diseasesNeoplasm ProteinsInsulin-Like Growth Factor Binding ProteinsRepressor ProteinsCTGFCYR61Cancer researchIntercellular Signaling Peptides and ProteinsRNACarcinogenesisWISPWntTranscription Factors
researchProduct

Cardenales y "nobleza eclesiástica" en Italia, entre ficción y biografía (siglos XVI-XVII)

2016

The literature on cardinals examined in this article (writings on conclaves, biographies, funeral orations and relations, as well as descriptions of funeral apparati) demonstrate an ostentatious prevalence of the “purple” on the “blood”, of the ecclesiastical nobility on the honour of the lineage, of the genealogy of the Church on that of the family trees. Such prevalence was of course rhetoric within a system that made the kinship relationships as its strength and had a noble social block behind itself. The exception of some cardinals or popes of humble birth, indeed, confirmed the rule of the patrician or aristocratic descent of the majority of them. In this regard, as Peter Burke (2001) …

Cardinals nobility literature Early modern ItalySettore M-STO/07 - Storia Del Cristianesimo E Delle ChieseCardenales nobleza literatura Italia de la primera edad modernaSettore M-STO/02 - Storia ModernaCardinali nobiltà letteratura Italia della prima età moderna
researchProduct

Complexity of IL-1β induced gene expression pattern in human articular chondrocytes

1997

The mRNA fingerprinting technique, differential display reverse transcription polymerase chain (DDRT-PCR), was used to detect changes in the overall pattern of gene expression in human articular knee chondrocytes-induced by interleukin-1 beta (IL-1 beta), the prototypical inducer of catabolic responses in degenerate joint diseases. One hundred different primer combinations generated approximately 10,000 different PCR fragments for IL-1 beta treated, as well as for untreated human chondrocytes, cultivated in alginate beads. This represented 53% of all expressed chondrocyte genes as based on statistical considerations. Side by side comparisons of differential display patterns originating from…

Cartilage ArticularDNA ComplementaryMolecular Sequence DataCell Culture TechniquesBiomedical EngineeringBiologyPolymerase Chain ReactionChondrocyteChondrocytesRheumatologyComplementary DNAGene expressionOsteoarthritismedicineHumansOrthopedics and Sports MedicineRNA MessengerGeneAgedDifferential displayDifferential displayIL-1Middle AgedBlotting NorthernMolecular biologyReverse transcriptaseReal-time polymerase chain reactionmedicine.anatomical_structureGene Expression RegulationFemalePrimer (molecular biology)Interleukin-1Osteoarthritis and Cartilage
researchProduct

Expression of M-cadherin protein in myogenic cells during prenatal mouse development and differentiation of embryonic stem cells in culture.

1994

Molecules regulating morphogenesis by cell-cell interactions are the cadherins, a class of calcium-dependent adhesion molecules. One of its members, M-cadherin, has been isolated from a myoblast cell line (Donalies et al. [1991] Proc. Natl. Acad. Sci. U.S.A. 88:8024—8028). In mouse development, expression of M-cadherin mRNA first appears at day 8.5 of gestation (E8.5) in somites and has been postulated to be down-regulated in developing muscle masses (Moore and Walsh [1993] Development 117:1409—1420). Affinity-purified polyclonal M-cadherin antibodies, detecting a protein of approximately 120 kDa, were used to study the cell expression pattern of M-cadherin protein. It was first visualized …

Cell Adhesion Molecules NeuronalRecombinant Fusion ProteinsMolecular Sequence DataMorphogenesisFluorescent Antibody TechniqueGestational AgeBiologyEmbryonic and Fetal DevelopmentMiceLamininPregnancyMyocyteAnimalsAmino Acid SequenceRNA MessengerMuscle SkeletalCells CulturedDNA PrimersMice Inbred BALB CBase SequenceCadherinCell adhesion moleculeStem CellsCell MembraneGene Expression Regulation DevelopmentalCadherinsEmbryonic stem cellMolecular biologyCell culturebiology.proteinDesminFemaleDevelopmental BiologyDevelopmental dynamics : an official publication of the American Association of Anatomists
researchProduct

Endoplasmic reticulum stress is involved in response of human laryngeal carcinoma cells to carboplatin but is absent in carboplatin resistant cells

2013

The major obstacle of successful tumor treatment with carboplatin (CBP) is the development of drug resistance. In the present study, we found that following treatment with CBP the amount of platinum which enters the human laryngeal carcinoma (HEp2)-derived CBP- resistant (7T) cells is reduced relative to the parental HEp2. As a consequence, the formation of reactive oxidative species (ROS) is reduced, the induction of endoplasmic reticulum (ER) stress is diminished, the amount of inter- and intrastrand cross-links is lower, and the induction of apoptosis is depressed. In HEp2 cells, ROS scavenger tempol, inhibitor of ER stress salubrinal, as well as gene silencing of ER stress marker CCAAT/…

Celllcsh:MedicineApoptosisCarboplatinSalubrinalapoptosis; carboplatin; drug resistance; endoplasmic reticulum stress; reactive oxidative species; tumor cellschemistry.chemical_compound0302 clinical medicineBlotting Southwesternlcsh:Science0303 health sciencesMultidisciplinaryThioureaGeologyEndoplasmic Reticulum Stress3. Good healthmedicine.anatomical_structure030220 oncology & carcinogenesisSignal transductionSignal TransductionResearch ArticleProgrammed cell deathCell SurvivalBlotting WesternBiologyReal-Time Polymerase Chain ReactionCyclic N-Oxides03 medical and health sciencesCell Line TumormedicineHumansGene SilencingLaryngeal NeoplasmsBiology030304 developmental biologyDNA PrimersPlatinumEndoplasmic reticulumlcsh:RCarcinomaMolecular biologychemistryCell cultureApoptosisCinnamatesDrug Resistance NeoplasmUnfolded protein responseCancer researchlcsh:QSpin LabelsReactive Oxygen Species
researchProduct

Cannabinoids mediate analgesia largely via peripheral type 1 cannabinoid receptors in nociceptors

2007

Although endocannabinoids constitute one of the first lines of defense against pain, the anatomical locus and the precise receptor mechanisms underlying cannabinergic modulation of pain are uncertain. Clinical exploitation of the system is severely hindered by the cognitive deficits, memory impairment, motor disturbances and psychotropic effects resulting from the central actions of cannabinoids. We deleted the type 1 cannabinoid receptor (CB1) specifically in nociceptive neurons localized in the peripheral nervous system of mice, preserving its expression in the CNS, and analyzed these genetically modified mice in preclinical models of inflammatory and neuropathic pain. The nociceptor-spec…

Central Nervous SystemCannabinoid receptorCannabinoid Receptor Modulatorsmedicine.medical_treatmentCentral nervous systemPharmacologyBiologyArticleMiceReceptor Cannabinoid CB1Ganglia SpinalCannabinoid Receptor ModulatorsPeripheral Nervous SystemmedicineAnimalsNeurons AfferentAllelesDNA PrimersMice KnockoutNerve Fibers UnmyelinatedCannabinoidsGeneral NeuroscienceNociceptorsPeripheral Nervous System DiseasesEndocannabinoid systemElectrophysiologyMice Inbred C57BLmedicine.anatomical_structurenervous systemPeripheral nervous systemNeuropathic painNociceptorlipids (amino acids peptides and proteins)CannabinoidAnalgesiaNeuroscience
researchProduct

Direct suppression of CNS autoimmune inflammation via the cannabinoid receptor CB1 on neurons and CB2 on autoreactive T cells.

2007

The cannabinoid system is immunomodulatory and has been targeted as a treatment for the central nervous system (CNS) autoimmune disease multiple sclerosis. Using an animal model of multiple sclerosis, experimental autoimmune encephalomyelitis (EAE), we investigated the role of the CB(1) and CB(2) cannabinoid receptors in regulating CNS autoimmunity. We found that CB(1) receptor expression by neurons, but not T cells, was required for cannabinoid-mediated EAE suppression. In contrast, CB(2) receptor expression by encephalitogenic T cells was critical for controlling inflammation associated with EAE. CB(2)-deficient T cells in the CNS during EAE exhibited reduced levels of apoptosis, a higher…

Central Nervous SystemCannabinoid receptorEncephalomyelitis Autoimmune Experimentalmedicine.medical_treatmentEncephalomyelitisT-LymphocytesInflammationApoptosisMice TransgenicBiologyGeneral Biochemistry Genetics and Molecular BiologyReceptor Cannabinoid CB2MiceReceptor Cannabinoid CB1medicineCannabinoid receptor type 2AnimalsCell ProliferationDNA PrimersAutoimmune diseaseNeuronsExperimental autoimmune encephalomyelitisGeneral Medicinemedicine.diseaseEndocannabinoid systemImmunohistochemistryImmunologyEncephalitislipids (amino acids peptides and proteins)Cannabinoidmedicine.symptomNature medicine
researchProduct

Abdominal-B and caudal inhibit the formation of specific neuroblasts in the Drosophila tail region

2013

The central nervous system of Drosophila melanogaster consists of fused segmental units (neuromeres), each generated by a characteristic number of neural stem cells (neuroblasts). In the embryo, thoracic and anterior abdominal neuromeres are almost equally sized and formed by repetitive sets of neuroblasts, whereas the terminal abdominal neuromeres are generated by significantly smaller populations of progenitor cells. Here we investigated the role of the Hox gene Abdominal-B in shaping the terminal neuromeres. We show that the regulatory isoform of Abdominal-B (Abd-B.r) not only confers abdominal fate to specific neuroblasts (e.g. NB6-4) and regulates programmed cell death of several proge…

Central Nervous SystemTailanimal structuresCNS developmentCellular differentiationParaHoxApoptosisBiologyTerminal neuromeresAbdominal-BHox genesNeural Stem CellsNeuroblastNeuroblastsImage Processing Computer-AssistedAnimalsDrosophila ProteinsHox geneMolecular BiologyIn Situ HybridizationDNA PrimersHomeodomain ProteinsfungiCell DifferentiationStem Cells and RegenerationNeuromereImmunohistochemistryMolecular biologyNeural stem cellSegmental patterningDrosophila melanogasterMicroscopy Fluorescencenervous systemembryonic structuresCaudalDrosophilaGanglion mother cellDrosophila ProteinTranscription FactorsDevelopmental BiologyDevelopment
researchProduct