Search results for "profilin"

showing 10 items of 900 documents

Mantle-cell lymphoma genotypes identified with CGH to BAC microarrays define a leukemic subgroup of disease and predict patient outcome

2005

To identify recurrent genomic changes in mantle cell lymphoma (MCL), we used high-resolution comparative genomic hybridization (CGH) to bacterial artificial chromosome (BAC) microarrays in 68 patients and 9 MCL-derived cell lines. Array CGH defined an MCL genomic signature distinct from other B-cell lymphomas, including deletions of 1p21 and 11q22.3-ATM gene with coincident 10p12-BMI1 gene amplification and 10p14 deletion, along with a previously unidentified loss within 9q21-q22. Specific genomic alterations were associated with different subgroups of disease. Notably, 11 patients with leukemic MCL showed a different genomic profile than nodal cases, including 8p21.3 deletion at tumor necr…

MaleChromosomes Artificial BacterialGenotypeImmunologyLocus (genetics)Lymphoma Mantle-CellBiologyBiochemistryGene duplicationmedicineHumansAgedOligonucleotide Array Sequence AnalysisSequence DeletionAged 80 and overGeneticsLeukemiaGene Expression ProfilingGenomic signatureGenomicsCell BiologyHematologyMiddle Agedmedicine.diseaseLymphomaSurvival RateGene expression profilingTreatment OutcomeGenomic ProfileCancer researchFemaleMantle cell lymphomaComparative genomic hybridizationBlood
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Profiling movement behaviours in pre-school children: A self-organised map approach.

2019

Application of machine learning techniques has the potential to yield unseen insights into movement and permits visualisation of complex behaviours and tangible profiles. The aim of this study was to identify profiles of relative motor competence (MC) and movement behaviours in pre-school children using novel analytics. One-hundred and twenty-five children (4.3 ± 0.5y, 1.04 ± 0.05 m, 17.8 ± 3.2 kg, BMI: 16.2 ± 1.9 kg

MaleComputer scienceMovementPhysical activity030209 endocrinology & metabolismPhysical Therapy Sports Therapy and Rehabilitation030229 sport sciencesFitness TrackersMotor ActivityVisualizationBody Mass IndexMachine Learning03 medical and health sciences0302 clinical medicineCross-Sectional StudiesHuman–computer interactionChild PreschoolAccelerometryProfiling (information science)HumansOrthopedics and Sports MedicinePre schoolFemaleExerciseJournal of sports sciences
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Transcriptional profiling reveals functional links between RasGrf1 and Pttg1 in pancreatic beta cells

2014

This article is published under license to BioMed Central Ltd. This is an Open Access article distributed under the terms of the Creative Commons Attribution License .

MaleComputingMilieux_LEGALASPECTSOFCOMPUTINGTranscriptomeCytosolRas-GRF1Insulin-Secreting CellsGlucose homeostasisPromoter Regions GeneticOligonucleotide Array Sequence AnalysisMice KnockoutGeneticsCell biologySecurinERKPhenotypemedicine.anatomical_structureMitogen-Activated Protein KinasesBeta cellSignal transductionResearch ArticleSignal TransductionBiotechnologyCell signalingMedicina InvestigacióMedicinaPancreatic isletsBiologyGeneticsmedicineAnimalsCell LineagePttg1TranscriptomicsTranscription factorBinding Sitesras-GRF1Gene Expression ProfilingPancreatic isletsBeta cellsMolecular Sequence AnnotationGlucose Tolerance TestMice Inbred C57BLPàncrees MalaltiesGenetic LociData_GENERALTranscriptional factorsras ProteinsCalciumRasGrf1RasBMC Genomics
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Expression of somatic DNA repair genes in human testes

2006

Meiosis is the key process for recombination and reduction of the diploid chromosome set to a haploid one. Many genes that have been found in yeast or mouse models to play a role in meiosis are also important for the repair of DNA damage in somatic cells. To study the DNA repair gene transcriptome during male germ cell development, we have developed a specialized cDNA microarray with 181 human genes which are involved in different somatic DNA repair pathways and/or cell cycle control and 45 control house-keeping genes. This DNA repair gene chip was used to quantify the mRNA expression levels in three human testes samples versus a fibroblast RNA pool. Two hundred twenty genes on the chip (in…

MaleDNA RepairDNA damageSomatic cellDNA repairBiologyBiochemistryTranscriptomeTestismedicineHumansMolecular BiologyGeneCells CulturedOligonucleotide Array Sequence AnalysisSkinReverse Transcriptase Polymerase Chain ReactionGene Expression ProfilingCell BiologyFibroblastsDNA repair protein XRCC4Molecular biologyMeiosismedicine.anatomical_structureGene Expression RegulationHuman genomeBiomarkersGerm cellJournal of Cellular Biochemistry
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Bone marrow cell transcripts from Fanconi anaemia patients revealin vivoalterations in mitochondrial, redox and DNA repair pathways

2013

Fanconi anaemia (FA) is a genetic cancer predisposition disorder associated with cytogenetic instability, bone marrow failure and a pleiotropic cellular phenotype, including low thresholds of responses to oxidative stress, cross-linking agents and selected cytokines. This study was aimed at defining the scope of abnormalities in gene expression using the publicly available FA Transcriptome Consortium (FTC) database (Gene Expression Omnibus, 2009 and publicly available as GSE16334). We evaluated the data set that included transcriptomal analyses on RNA obtained from low-density bone marrow cells (BMC) from 20 patients with FA and 11 healthy volunteers, by seeking to identify changes in expre…

MaleDNA Repairiron-chelating proteinsTranscriptome0302 clinical medicineFanconi anemiaGene expressioncytokineoxidative stressChildbioenergetic pathwayRegulation of gene expression0303 health sciencesHematologyGeneral Medicineheat-shock proteinMitochondria3. Good health030220 oncology & carcinogenesisFemaleFanconi anaemiaOxidation-ReductionSignal TransductionAdultiron-chelating proteinDNA repairDNA repairBone Marrow CellsBiologyProinflammatory cytokine03 medical and health sciencesmedicineHumanstranscriptsGene030304 developmental biologyoxidative streGene Expression Profilingheat-shock proteinsMolecular Sequence Annotationmedicine.diseaseMolecular biologycytokinesDNA repair Fanconi anaemia bioenergetic pathways cytokines heat-shock proteins iron-chelating proteins oxidative stress transcriptsGene expression profilingOxidative StressFanconi AnemiaCase-Control Studiesbioenergetic pathwaysTranscriptomeEuropean Journal of Haematology
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Defective nuclear localization of Hsp70 is associated with dyserythropoiesis and GATA-1 cleavage in myelodysplastic syndromes.

2012

Abstract Normal human erythroid cell maturation requests the transcription factor GATA-1 and a transient activation of caspase-3, with GATA-1 being protected from caspase-3–mediated cleavage by interaction with the chaperone heat shock protein 70 (Hsp70) in the nucleus. Erythroid cell dysplasia observed in early myelodysplastic syndromes (MDS) involves impairment of differentiation and excess of apoptosis with a burst of caspase activation. Analysis of gene expression in MDS erythroblasts obtained by ex vivo cultures demonstrates the down-regulation of a set of GATA-1 transcriptional target genes, including GYPA that encodes glycophorin A (GPA), and the up-regulation of members of the HSP70…

MaleErythroblasts[SDV]Life Sciences [q-bio]Biochemistry0302 clinical medicineTranscription (biology)hemic and lymphatic diseasesGene expressionErythropoiesisGATA1 Transcription FactorCells CulturedCaspaseComputingMilieux_MISCELLANEOUSOligonucleotide Array Sequence AnalysisAged 80 and over0303 health sciencesbiologyCaspase 3Reverse Transcriptase Polymerase Chain ReactionCell DifferentiationU937 CellsHematologyMiddle Agedmedicine.anatomical_structure030220 oncology & carcinogenesisFemaleAdultGreen Fluorescent ProteinsImmunoblottingImmunology03 medical and health sciencesErythroid CellsmedicineHumansHSP70 Heat-Shock ProteinsTranscription factorAged030304 developmental biologyCell NucleusGene Expression ProfilingCell BiologyMolecular biologyCell nucleusMicroscopy FluorescenceApoptosisMyelodysplastic SyndromesChaperone (protein)Mutationbiology.proteinNuclear localization sequence
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Transcriptional profiling of rat hypothalamus response to 2,3,7,8-tetrachlorodibenzo-ρ-dioxin

2015

In some mammals, halogenated aromatic hydrocarbon (HAH) exposure causes wasting syndrome, defined as significant weight loss associated with lethal outcomes. The most potent HAH in causing wasting is 2,3,7,8-tetrachlorodibenzo-r-dioxin (TCDD), which exerts its toxic effects through the aryl hydrocarbon receptor (AHR). Since TCDD toxicity is thought to predominantly arise from dysregulation of AHR-transcribed genes, it was hypothesized that wasting syndrome is a result of to TCDD-induced dysregulation of genes involved in regulation of food-intake. As the hypothalamus is the central nervous systems' regulatory center for food-intake and energy balance. Therefore, mRNA abundances in hypothala…

MaleFOOD-INTAKETCDDPolychlorinated DibenzodioxinsTime FactorsTranscription GeneticMicroarrayTISSUE GROWTH-FACTORAHRAH GENE BATTERY413 Veterinary scienceToxicologyToxicogeneticsfeed restrictionTranscriptomeNAD(P)H Dehydrogenase (Quinone)RESISTANT RATheterocyclic compoundsMESSENGER-RNA EXPRESSIONhypothalamusWastingreproductive and urinary physiologyOligonucleotide Array Sequence Analysisbiologyta31413. Good healthPROBE LEVELHypothalamusToxicityENERGY-BALANCEmedicine.symptommicroarrayARYL-HYDROCARBON RECEPTORendocrine systemmedicine.medical_specialtyta3111Species SpecificityInternal medicineCytochrome P-450 CYP1A1medicineAnimalsRats Long-EvansRNA MessengerWasting SyndromeRats WistarWasting SyndromeGene Expression Profilingta1184Lethal doseAryl hydrocarbon receptorstomatognathic diseasesEndocrinologyINDUCED ANOREXIAGene Expression Regulationbiology.proteinToxicology
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Different impacts of cardiovascular risk factors on oxidative stress.

2011

The objective of the study was to evaluate oxidative stress (OS) status in subjects with different cardiovascular risk factors. With this in mind, we have studied three models of high cardiovascular risk: hypertension (HT) with and without metabolic syndrome, familial hypercholesterolemia (FH) and familial combined hyperlipidemia (FCH) with and without insulin resistance. Oxidative stress markers (oxidized/reduced glutathione ratio, 8-oxo-deoxyguanosine and malondialdehide) together with the activity of antioxidant enzyme triad (superoxide dismutase, catalase, glutathione peroxidase) and activation of both pro-oxidant enzyme (NAPDH oxidase components) and AGTR1 genes, as well as antioxidant…

MaleGPX1Antioxidantmedicine.medical_treatmentGlutathione reductaseHyperlipidemia Familial Combinedmedicine.disease_causelcsh:Chemistrychemistry.chemical_compoundRisk FactorsMalondialdehydeoxidative stressglutathione peroxidaselcsh:QH301-705.5Spectroscopychemistry.chemical_classificationbiologyfamilial hypercholesterolemiaChemistryGlutathione peroxidaseGeneral MedicineMiddle AgedCatalaseGlutathioneComputer Science ApplicationsGlutathione Reductase8-Hydroxy-2'-DeoxyguanosineCardiovascular DiseasesFemaleThioredoxinAdultmedicine.medical_specialtyhypertensionmRNACatalysisGlutathione SynthaseArticleInorganic ChemistrySuperoxide dismutaseHyperlipoproteinemia Type IIInternal medicinemedicineHumansPhysical and Theoretical ChemistryMolecular BiologySuperoxide DismutaseGene Expression ProfilingOrganic ChemistryDeoxyguanosineNADPH OxidasesGlutathionesuperoxide dismutasesPhosphoproteinscombined familial dyslipidemiaEndocrinologylcsh:Biology (General)lcsh:QD1-999biology.proteinOxidative stressBiomarkersInternational journal of molecular sciences
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Effects of different kinds of essentiality on sequence evolution of human testis proteins

2016

We asked if essentiality for either fertility or viability differentially affects sequence evolution of human testis proteins. Based on murine knockout data, we classified a set of 965 proteins expressed in human seminiferous tubules into three categories: proteins essential for prepubertal survival (“lethality proteins”), associated with male sub- or infertility (“male sub-/infertility proteins”), and nonessential proteins. In our testis protein dataset, lethality genes evolved significantly slower than nonessential and male sub-/infertility genes, which is in line with other authors’ findings. Using tissue specificity, connectivity in the protein-protein interaction (PPI) network, and mul…

MaleGene Expression ProfilingComputational BiologyProteinsMolecular Sequence AnnotationSeminiferous TubulesArticle570 Life sciencesEvolution MolecularMiceOrgan SpecificityProtein Interaction MappingTestisAnimalsHumansGene Regulatory NetworksProtein Interaction MapsSpermatogenesisTranscriptomeInfertility Male570 BiowissenschaftenScientific Reports
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Cross-species transcriptomic analysis elucidates constitutive aryl hydrocarbon receptor activity

2014

Background Research on the aryl hydrocarbon receptor (AHR) has largely focused on variations in toxic outcomes resulting from its activation by halogenated aromatic hydrocarbons. But the AHR also plays key roles in regulating pathways critical for development, and after decades of research the mechanisms underlying physiological regulation by the AHR remain poorly characterized. Previous studies identified several core genes that respond to xenobiotic AHR ligands across a broad range of species and tissues. However, only limited inferences have been made regarding its role in regulating constitutive gene activity, i.e. in the absence of exogenous ligands. To address this, we profiled transc…

MaleHEPATIC GENE-EXPRESSION413 Veterinary scienceMedical and Health SciencesTranscriptomeDIOXIN RECEPTORMice0302 clinical medicineTCDD-induced toxicityReceptorsTranscriptional regulationABNORMAL LIVER DEVELOPMENT2.1 Biological and endogenous factorsCluster AnalysisAetiologyReceptorAH RECEPTORIN-VIVOAryl hydrocarbon receptorGeneticsRegulation of gene expression0303 health sciencesBiological Sciencesrespiratory systemCore-gene batteryAryl HydrocarbonOrgan Specificity030220 oncology & carcinogenesisAHR endogenous ligands2378-TETRACHLORODIBENZO-P-DIOXIN TCDDSignal transductionResearch ArticleBiotechnologySignal TransductionProtein BindingBioinformatics1.1 Normal biological development and functioningeducationRAT-LIVERConstitutive gene expressionBiologyMICE LACKING03 medical and health sciencesSpecies SpecificityUnderpinning researchInformation and Computing SciencesGeneticsAnimals030304 developmental biologyAryl hydrocarbon receptor activityGene Expression ProfilingComputational BiologyAryl hydrocarbon receptorCELL-CYCLE CONTROLRatsrespiratory tract diseasesGene expression profilingReceptors Aryl HydrocarbonGene Expression RegulationSUBCHRONIC EXPOSUREbiology.proteinDigestive DiseasesTranscriptome
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