Search results for "reactive nitrogen specie"

showing 10 items of 67 documents

DNA strand breaks induced by nuclear hijacking of neuronal NOS as an anti-cancer effect of 2-methoxyestradiol

2015

2-Methoxyestradiol (2-ME) is a physiological metabolite of 17β-estradiol. At pharmacological concentrations, 2-ME inhibits colon, breast and lung cancer in tumor models. Here we investigated the effect of physiologically relevant concentrations of 2-ME in osteosarcoma cell model. We demonstrated that 2-ME increased nuclear localization of neuronal nitric oxide synthase, resulting in nitro-oxidative DNA damage. This in turn caused cell cycle arrest and apoptosis in osteosarcoma cells. We suggest that 2-ME is a naturally occurring hormone with potential anti-cancer properties.

Pathologymedicine.medical_specialtyneuronal nitric oxide synthaseCell cycle checkpoint2-methoxyestradiolDNA damageAntineoplastic AgentsApoptosisBone NeoplasmsNitric Oxide Synthase Type Imedicine.disease_causeNitric OxideNitric oxidechemistry.chemical_compoundReactive nitrogen specieCell Line TumormedicineHumans2-MethoxyestradiolReactive nitrogen speciesCytokinesisOsteosarcomaEstradiolbusiness.industryDNA BreaksIntracellular Signaling Peptides and ProteinsCancermedicine.diseaseReactive Nitrogen SpeciesG2 Phase Cell Cycle CheckpointsOxidative StressOncologychemistryApoptosis2-methoxyestradiol; Neuronal nitric oxide synthase; Nitric oxide; Osteosarcoma; Reactive nitrogen species; OncologyCancer researchM Phase Cell Cycle CheckpointsbusinessTumor Suppressor p53-Binding Protein 1Oxidative stressmedicine.drugResearch Paper
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Drugs modulating the biological effects of peroxynitrite and related nitrogen species

2006

The term “reactive nitrogen species” includes nitrogen monoxide, commonly called nitric oxide, and some other remarkable chemical entities (peroxynitrite, nitrosoperoxycarbonate, etc.) formed mostly from nitrogen monoxide itself in biological environments. Regardless of the specific mechanisms implicated in their effects, however, it is clear that an integrated pharmacological approach to peroxynitrite and related species is only just beginning to take shape. The array of affected chemical and pathological processes is extremely broad. One of the most conspicuous mechanisms observed thus far has been the scavenging of the peroxynitrite anion by molecules endowed with antioxidant activity. T…

PharmacologyAntioxidantmedicine.medical_treatmentOxidative phosphorylationLung injuryIn vitroNitric oxidechemistry.chemical_compoundchemistryBiochemistryIn vivoDrug DiscoverymedicineMolecular MedicineReactive nitrogen speciesPeroxynitriteMedicinal Research Reviews
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Oxidative stress and pulmonary inflammation: pharmacological intervention with antioxidants.

1999

Reactive oxygen and nitrogen species are generated by several inflammatory and structural cells of the airways. These oxidant species may have important effects on different lung cells as regulators of signal transduction, activators of key transcription factors, and modulators of gene expression and apoptosis. Thus, an increased oxidative stress accompanied by reduced endogenous antioxidant defences may have a role in the pathogenesis of a number of inflammatory pulmonary diseases including asthma. Although antioxidant drugs could play a useful role in the therapy of inflammatory lung diseases, their clinical impact is relatively modest at present. Rigorous clinical investigation with the …

Pharmacologychemistry.chemical_classificationReactive oxygen speciesAntioxidantLungmedicine.medical_treatmentPneumoniaBiologymedicine.disease_causeAntioxidantsPathogenesischemistry.chemical_compoundOxidative Stressmedicine.anatomical_structurechemistryApoptosisImmunologymedicineAnimalsHumansSignal transductionLungReactive nitrogen speciesOxidative stressPharmacological research
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What is the function of neuroglobin?

2009

SUMMARY For a long time, haemoglobin and myoglobin had been assumed to represent the only globin types of vertebrates. In 2000, however, we discovered a third globin type by mining the genome sequence data. Based on a preferential expression in the nervous system, this globin is referred to as neuroglobin. Despite nine years of research, its function is still uncertain and a number of hypotheses have been put forward. Neuroglobin enhances cell viability under hypoxia and under various types of oxidative stress in transgenic systems, but does not appear to be strongly upregulated in response to stress. A close phylogenetic relationship with invertebrate nerve globins and its positive correla…

PhysiologyTransgeneCellNeuroglobinNerve Tissue ProteinsAquatic ScienceMitochondrionBiologymedicine.disease_causeNervous Systemchemistry.chemical_compoundmedicineAnimalsGlobinHypoxiaMolecular BiologyEcology Evolution Behavior and SystematicsReactive nitrogen speciesNeuronsCytoglobinReactive Nitrogen SpeciesCell biologyGlobinsOxygenmedicine.anatomical_structurechemistryBiochemistryGene Expression RegulationInsect ScienceNeuroglobinVertebratesAnimal Science and ZoologyReactive Oxygen SpeciesOxidative stressThe Journal of experimental biology
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Oxidative and nitrosative stress in the diaphragm of patients with COPD.

2007

COPD is associated with an increased load on the diaphragm. Since chronic muscle loading results in changes in antioxidant capacity and formation of reactive oxygen and reactive nitrogen species, we hypothesized that COPD has a similar effect on the diaphragm, which is related to the severity of COPD. Catalase activity was determined spectrophotometrically. Levels of 4-hydroxy-2-nonenal (HNE)-protein adducts and 3-nitrotyrosine (NT) formation were measured using western blotting. Levels of malondialdehyde (MDA) were assessed by high-performance liquid chromatography. We found that catalase activity was approximately 89% higher in the diaphragm of severe COPD patients (FEV1 37+/-5% predicted…

Pulmonary and Respiratory Medicinemedicine.medical_specialtyrespiratory musclesNitrosationDiaphragmOxidative phosphorylationmedicine.disease_causeSeverity of Illness IndexNOLipid peroxidationPulmonary Disease Chronic Obstructivechemistry.chemical_compoundInternal medicineHumansCOPDMedicineReactive nitrogen speciesOriginal ResearchCOPDoxidantsbiologybusiness.industryHealth PolicyPublic Health Environmental and Occupational HealthGeneral MedicineMiddle Agedmedicine.diseaseMalondialdehydeDiaphragm (structural system)Surgeryrespiratory tract diseasesOxidative StressantioxidantsEndocrinologychemistryCatalasebiology.proteinbusinessOxidative stressInternational journal of chronic obstructive pulmonary disease
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Glycine-functionalized copper(ii) hydroxide nanoparticles with high intrinsic superoxide dismutase activity

2017

Superoxide dismutases (SOD) are a group of enzymes that catalyze the dismutation of superoxide (O2−) radicals into molecular oxygen (O2) and H2O2 as a first line of defense against oxidative stress. Here, we show that glycine-functionalized copper(II) hydroxide nanoparticles (Gly-Cu(OH)2 NPs) are functional SOD mimics, whereas bulk Cu(OH)2 is insoluble in water and catalytically inactive. In contrast, Gly-Cu(OH)2 NPs form water-dispersible mesocrystals with a SOD-like activity that is larger than that of their natural CuZn enzyme counterpart. Based on this finding, we devised an application where Gly-Cu(OH)2 NPs were incorporated into cigarette filters. Cigarette smoke contains high concent…

RadicalInorganic chemistryGlycine02 engineering and technology010402 general chemistrymedicine.disease_cause01 natural sciencesSuperoxide dismutasechemistry.chemical_compoundSmokeHydroxidesmedicineHumansGeneral Materials ScienceReactive nitrogen specieschemistry.chemical_classificationCopper(II) hydroxideReactive oxygen speciesbiologySuperoxide DismutaseSuperoxideHydrogen PeroxideTobacco Products021001 nanoscience & nanotechnologyCombinatorial chemistry0104 chemical scienceschemistryA549 Cellsbiology.proteinNanoparticlesHydroxideReactive Oxygen Species0210 nano-technologyCopperOxidative stressNanoscale
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Antineoplastic Drug-Induced Cardiotoxicity: A Redox Perspective

2018

Antineoplastic drugs can be associated with several side effects, including cardiovascular toxicity (CTX). Biochemical studies have identified multiple mechanisms of CTX. Chemoterapeutic agents can alter redox homeostasis by increasing the production of reactive oxygen species (ROS) and reactive nitrogen species RNS. Cellular sources of ROS/RNS are cardiomyocytes, endothelial cells, stromal and inflammatory cells in the heart. Mitochondria, peroxisomes and other subcellular components are central hubs that control redox homeostasis. Mitochondria are central targets for antineoplastic drug-induced CTX. Understanding the mechanisms of CTX is fundamental for effective cardioprotection, without…

Stromal cellPhysiologymedicine.medical_treatmentTyrosine kinase inhibitorChemotherapy; HER-2 inhibitors; Oxidative/nitrosative stress; Tyrosine kinase inhibitors; Vascular endothelial growth factorReviewOxidative phosphorylation030204 cardiovascular system & hematologyMitochondrionPharmacologyChemotherapy; HER-2 inhibitors; Oxidative/nitrosative stress; Tyrosine kinase inhibitors; Vascular endothelial growth factor; Physiology; Physiology (medical)chemotherapyHER-2 inhibitorlcsh:Physiology03 medical and health scienceschemistry.chemical_compound0302 clinical medicinePhysiology (medical)tyrosine kinase inhibitorsMedicinechemotherapy HER-2 inhibitors oxidative/nitrosative stress vascular endothelial growth factor tyrosine kinase inhibitorsReactive nitrogen specieschemistry.chemical_classificationCardioprotectionReactive oxygen speciesChemotherapyCardiotoxicitylcsh:QP1-981vascular endothelial growth factorbusiness.industryOxidative/nitrosative strechemistry030220 oncology & carcinogenesisbusinessHER-2 inhibitorsoxidative/nitrosative stress
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Indicaxanthin from

2018

Oxidized low-density lipoproteins (oxLDL) play a pivotal role in the etiopathogenesis of atherosclerosis through the activation of inflammatory signaling events eventually leading to endothelial dysfunction and senescence. In the present work, we investigated the effects of indicaxanthin, a bioavailable, redox-modulating phytochemical from Opuntia ficus indica fruits, with anti-inflammatory activity, against oxLDL-induced endothelial dysfunction. Human umbilical vein cord cells (HUVEC) were stimulated with human oxLDL, and the effects of indicaxanthin were evaluated in a range between 5 and 20 μM, consistent with its plasma level after a fruit meal (7 μM). Pretreatment with indicaxanthin si…

Transcription GeneticCell SurvivalPyridinesNF-kappa BOpuntiaHydrogen PeroxideReactive Nitrogen SpeciesThiobarbituric Acid Reactive SubstancesBetaxanthinsUp-RegulationLipoproteins LDLHuman Umbilical Vein Endothelial CellsHumansRNA MessengerReactive Oxygen SpeciesCell Adhesion MoleculesOxidation-ReductionATP Binding Cassette Transporter 1Research ArticleOxidative medicine and cellular longevity
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Beclomethasone dipropionate and formoterol reduce oxidative/nitrosative stress generated by cigarette smoke extracts and IL-17A in human bronchial ep…

2013

Interleukin-17A (IL-17A), cigarette smoke and oxidative/nitrosative stress are involved in inflammatory airway diseases, and the mechanisms behind these processes are still poorly understood. We investigated whether recombinant human IL-17A (rhIL-17A), in combination with cigarette smoke extracts (CSE), increases the levels of inducibile nitric oxide synthase (iNOS), reactive oxygen species, nitrotyrosine (NT) and the activation of signal transducer and activator of transcription 1 (STAT-1) in normal human bronchial epithelial cells (16HBE). The effect of beclomethasone dipropionate (BDP), formoterol and their combination was also evaluated. We demonstrated that rhIL-17A or CSE alone increa…

Transcription GeneticNitric Oxide Synthase Type IIBronchiOxidative phosphorylationPharmacologyGene Expression Regulation EnzymologicCell Linechemistry.chemical_compoundFormoterol FumarateSmokeNitrilesmedicineButadienesGene silencingHumansGene SilencingPromoter Regions GeneticPharmacologychemistry.chemical_classificationReactive oxygen speciesbiologyNitrotyrosineInterleukin-17BeclomethasoneEpithelial CellsTobacco ProductsReactive Nitrogen SpeciesNitric oxide synthaseOxidative StressSTAT1 Transcription FactorchemistryEthanolaminesImmunologySTAT proteinbiology.proteinPhosphorylationFormoterolBiomarkersmedicine.drugEuropean journal of pharmacology
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Alpha-lipoic acid: molecular mechanisms and therapeutic potential in diabetes.

2015

International audience; Diabetes is a chronic metabolic disease with a high prevalence worldwide. Diabetes and insulin resistance are associated with the development of cardiovascular and nervous diseases. The development of these disorders reflects complex pathological processes in which the oxidative stress caused by reactive oxygen species (ROS) and reactive nitrogen species (RNS) plays a pivotal role. It is widely accepted that diabetes impairs endothelial nitric oxide synthase (eNOS) activity and increases the production of ROS, thus resulting in diminished NO bioavailability and increased oxidative stress. Alpha-lipoic acid (LA) possesses beneficial effects both in the prevention and …

antioxidantdiabètesAntioxidantPhysiologymedicine.medical_treatmentAlpha-Lipoic Acidmedicine.disease_causeAntioxidantschemistry.chemical_compound0302 clinical medicineprevention0303 health sciencesdiabetestreatmentThioctic AcidGeneral MedicineReactive Nitrogen Species[SDV.MHEP.CSC] Life Sciences [q-bio]/Human health and pathology/Cardiology and cardiovascular system3. Good healthMitochondriapréventionLipoic acid030220 oncology & carcinogenesismedicine.medical_specialtyBiologytraitement03 medical and health sciencesInsulin resistanceantioxydant[SDV.MHEP.CSC]Life Sciences [q-bio]/Human health and pathology/Cardiology and cardiovascular systemDihydrolipoic acidPhysiology (medical)Internal medicineDiabetes mellitusmedicineDiabetes Mellitusacide lipoïqueAnimalsHumansmétabolismeReactive nitrogen species030304 developmental biologyPharmacologylipoic acidmedicine.diseaseOxidative StressEndocrinologychemistryReactive Oxygen SpeciesmetabolismOxidative stressCanadian journal of physiology and pharmacology
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