Search results for "reactive oxygen specie"

showing 10 items of 880 documents

Cytotoxicity of two naturally occurring flavonoids (dorsmanin F and poinsettifolin B) towards multi-factorial drug-resistant cancer cells.

2015

Abstract Introduction The expression of diverse resistance mechanisms in cancer cells is one of the major barriers to successful cancer chemotherapy. Methods In the present study, we assessed the cytotoxicity of two naturally occurring flavonoids dorsmanin F ( 1 , a flavanone) and poinsettifolin B ( 2 , a chalcone) against 9 drug-sensitive and multidrug-resistant (MDR) cancer cell lines. The resazurin reduction assay was used to evaluate the cytotoxicity of these compounds, whilst caspase-Glo assay was used to detect caspase activation. Cell cycle, mitochondrial membrane potential (MMP) and levels of reactive oxygen species (ROS) were all analysed via flow cytometry. Results Compounds 1 and…

ChalconePharmaceutical ScienceApoptosisPharmacologyBiologychemistry.chemical_compoundInhibitory Concentration 50ChalconesCell Line TumorDrug DiscoverymedicineCytotoxic T cellHumansDoxorubicinCytotoxicityPharmacologyFlavonoidsMembrane Potential MitochondrialMolecular StructureCell CycleCell cycleMolecular biologyAntineoplastic Agents PhytogenicDrug Resistance MultipleComplementary and alternative medicinechemistryApoptosisDrug Resistance NeoplasmCaspasesCancer cellMolecular MedicineReactive Oxygen SpeciesFlavanonemedicine.drugPhytomedicine : international journal of phytotherapy and phytopharmacology
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Cytotoxicity and modes of action of 4'-hydroxy-2',6'-dimethoxychalcone and other flavonoids toward drug-sensitive and multidrug-resistant cancer cell…

2014

Abstract Introduction Resistance of cancer to chemotherapy is a main cause in treatment failure. Naturally occurring chalcones possess a wide range of biological activities including anti-cancer effects. In this work, we evaluated the antiproliferative activity of three chalcones [4′-hydroxy-2′,6′-dimethoxychalcone ( 1 ), cardamomin ( 2 ), 2′,4′-dihydroxy-3′,6′-dimethoxychalcone ( 3 )], and four flavanones [( S )-(–)-pinostrobin ( 4 ), ( S )-(–)-onysilin ( 5 ) and alpinetin ( 6 )] toward nine cancer cell lines amongst which were multidrug resistant (MDR) types. Methods The resazurin reduction assay was used to detect the antiproliferative activity of the studied samples whilst flow cytometr…

ChalconePharmaceutical ScienceApoptosisPharmacologyBiologychemistry.chemical_compoundInhibitory Concentration 50ChalconesCell Line TumorDrug DiscoverymedicineHumansCytotoxicityPharmacologyFlavonoidsMembrane Potential MitochondrialMolecular StructurePlant ExtractsCancerHep G2 CellsCell cyclemedicine.diseaseMolecular biologyAntineoplastic Agents PhytogenicDrug Resistance MultipleMultiple drug resistanceLeukemiaComplementary and alternative medicinechemistryApoptosisCell cultureDrug Resistance NeoplasmMolecular MedicinePolygonumReactive Oxygen SpeciesPhytomedicine : international journal of phytotherapy and phytopharmacology
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Cytotoxicity of three naturally occurring flavonoid derived compounds (artocarpesin, cycloartocarpesin and isobavachalcone) towards multi-factorial d…

2015

Abstract Introduction Cancer remains an aggressive deadly disease, if drug resistance develops. This problem is aggravated by the fact that multiple rather than single mechanisms are involved in resistance and that multidrug resistance (MDR) phenomena cause inefficacy of many clinical established anticancer drugs. We are seeking for novel cytotoxic phytochemicals to combat drug-resistant tumour cells. Methods In the present study, we investigated the cytotoxicity of three naturally occurring flavonoids including two flavones artocarpesin (1) and cycloartocarpesin (2) and one chalcone, isobavachalcone (3) against 9 drug-sensitive and MDR cancer cell lines. The resazurin reduction assay was u…

ChalconePharmaceutical SciencePharmacologyBiologyFlavoneschemistry.chemical_compoundInhibitory Concentration 50ChalconesCell Line TumorDrug DiscoverymedicineCytotoxic T cellHumansCytotoxicityPharmacologychemistry.chemical_classificationMembrane Potential MitochondrialMolecular StructureCell CycleCell cyclemedicine.diseaseFlavonesAntineoplastic Agents PhytogenicDrug Resistance MultipleMultiple drug resistanceLeukemiaComplementary and alternative medicinechemistryDrug Resistance NeoplasmCaspasesCancer cellMolecular MedicineReactive Oxygen SpeciesPhytomedicine : international journal of phytotherapy and phytopharmacology
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Inhibitory effects of N-acetylcysteine on the functional responses of human eosinophils in vitro

2007

Background Oxidative stress appears to be relevant in the pathogenesis of inflammation in allergic diseases like bronchial asthma. Eosinophils are oxidant-sensitive cells considered as key effectors in allergic inflammation. Objective The aim of this work was to study the effects of the clinically used antioxidant N-acetyl-L-cysteine (NAC) on the functional responses of human-isolated eosinophils. Methods Human eosinophils were purified from the blood of healthy donors by a magnetic bead separation system. The effects of NAC were investigated on the generation of reactive oxygen species (chemiluminescence and flow cytometry), Ca2+ signal (fluorimetry), intracellular glutathione (GSH; flow c…

Chemokine CCL11EotaxinLuminescenceImmunologyhuman eosinophilsBiologyPharmacologymedicine.disease_causeTranslocation GeneticAllergic inflammationAcetylcysteinechemistry.chemical_compoundmedicineHumansImmunology and Allergychemistry.chemical_classificationreactive oxygen speciesEosinophil cationic proteinReactive oxygen speciesCell DeathEosinophil Cationic ProteinNADPH OxidasesFree Radical ScavengersGlutathioneEosinophilPhosphoproteinsGlutathioneN-acetylcysteineAcetylcysteineEosinophilsN-Formylmethionine Leucyl-Phenylalaninemedicine.anatomical_structurechemistryBiochemistryChemokines CCeosinophil cationic proteinCalciumReactive Oxygen SpeciesOxidative stressp47(phox)medicine.drugp67(phox)
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Toll-like receptor-2 is essential in murine defenses against Candida albicans infections

2004

In this work, we studied the role of toll-like receptor-2 (TLR2) in murine defenses against Candida albicans. TLR2-deficient mice experimentally infected intraperitoneally (i.p.) or intravenously (i.v.) in vivo had very significant impaired survival compared with that of control mice. In vitro production of TNF-alpha and macrophage inhibitory protein-2 (MIP-2) by macrophages from TLR2-/- mice in response to yeasts and hyphae of C. albicans were significantly lower (80% and 40%, respectively; P <0.05) than production by macrophages from wild-type mice. This impaired production of TNF-alpha and MIP-2 probably contributed to the 41% decreased recruitment of neutrophils to the peritoneal cavity…

Chemokinemedicine.medical_treatmentPhagocytosisChemokine CXCL2ImmunologyHyphaeCell CountReceptors Cell SurfaceMicrobiologyMicrobiologyMicePhagocytosisIn vivoCandida albicansmedicineAnimalsMacrophageCandida albicansCells CulturedMice KnockoutToll-like receptorMembrane GlycoproteinsbiologyTumor Necrosis Factor-alphaToll-Like ReceptorsCandidiasisFlow Cytometrybiology.organism_classificationImmunity InnateToll-Like Receptor 2Corpus albicansMice Inbred C57BLDisease Models AnimalInfectious DiseasesCytokineMacrophages Peritonealbiology.proteinChemokinesReactive Oxygen SpeciesMicrobes and Infection
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Lichen rehydration in heavy metal polluted environments: Pb modulates the oxidative response of both Ramalina farinacea thalli and its isolated micro…

2014

Lichens are adapted to desiccation/rehydration and accumulate heavy metals, which induce ROS especially from the photobiont photosynthetic pigments. Although their mechanisms of abiotic stress tolerance are still to be unravelled, they seem related to symbionts' reciprocal upregulation of antioxidant systems. With the aim to study the effect of Pb on oxidative status during rehydration, the kinetics of intracellular ROS, lipid peroxidation and chlorophyll autofluorescence of whole Ramalina farinacea thalli and its isolated microalgae (Trebouxia TR1 and T. TR9) was recorded. A genetic characterization of the microalgae present in the thalli used was also carried out in order to assess possib…

ChlorophyllTrebouxiaLichensSoil ScienceBiologyPhotosynthesisRamalina farinaceaLipid peroxidationchemistry.chemical_compoundAscomycotaChlorophytaBotanyFluorometrySymbiosisEcology Evolution Behavior and SystematicsEcologyAbiotic stressWaterbiology.organism_classificationThallusLeadchemistrySpainChlorophyllBotànicaEnvironmental PollutantsLipid PeroxidationReactive Oxygen SpeciesDesiccation
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High mobility group box 1 and markers of oxidative stress in human cord blood.

2019

Background Parturition induces considerable oxidative stress and many inflammatory mediators, such as high mobility group box 1 (HMGB1), are involved from the beginning of the pregnancy to birth. The aim of the present study was to evaluate serum cord blood concentration of diacron-reactive oxygen metabolites (d-ROM), biological antioxidant potential (BAP), and HMGB1 to investigate the perinatal oxidative status of neonates and correlation with mode of delivery, as well as the influence of labor. Methods The subjects consisted of 214 neonates delivered at University Hospital "G. Martino", Messina, in a 6 months period. Venous blood samples were collected from the umbilical cord after cord s…

CordPhysiology030204 cardiovascular system & hematologyHMGB1medicine.disease_causeUmbilical cordUmbilical veinAntioxidants03 medical and health sciences0302 clinical medicinePregnancy030225 pediatricsMedicineHumansHMGB1 ProteinPregnancyLabor Obstetricbiologybusiness.industryInfant NewbornCord blood high mobility group box 1 newborn oxidative stress pregnancy.cord blood high mobility group box 1 newborn oxidative stress pregnancyVenous bloodmedicine.diseaseDelivery ObstetricFetal BloodOxidative Stressmedicine.anatomical_structureItalyCord bloodPediatrics Perinatology and Child Healthbiology.proteinFemalebusinessReactive Oxygen SpeciesOxidative stressBiomarkers
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Peroxynitrite generated from constitutive nitric oxide synthase mediates the early biochemical injury in short-term cultured hepatocytes

2000

AbstractEarly loss of P450 in rat hepatocyte cultures appears directly related to nitric oxide (NO) overproduction. This study provides experimental evidence for the induction – shortly after isolation through the classical procedure – of strong oxidative stress that involves both oxygen-derived and NO-derived species. NO formation at this stage is due to the early activation of liver constitutive NO synthase (cNOS). Immunodetection of nitrated proteins provides direct evidence of endogenous peroxynitrite (PN) formation upon hepatocyte isolation. On the basis of the combined use of dihydrorhodamine 123 and NOS inhibitors, the analysis of the amount, time course and nature of the species inv…

CultureBiophysicsEndogenyNitric Oxidemedicine.disease_causeBiochemistryPeroxynitriteNitric oxideP450 contentchemistry.chemical_compoundStructural BiologyGeneticsmedicineAnimalsViability assayOverproductionMolecular BiologyCells CulturedNitratesHepatocyte isolationbiologyNitric oxide synthaseProteinsCell BiologyOxidantsRatsNitric oxide synthaseKineticsmedicine.anatomical_structureLiverchemistryBiochemistryOxidative stressHepatocytebiology.proteinReactive Oxygen SpeciesProtein nitrationPeroxynitriteOxidative stressFEBS Letters
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Miltirone Induces G2/M Cell Cycle Arrest and Apoptosis in CCRF-CEM Acute Lymphoblastic Leukemia Cells

2015

Miltirone (1) is a diterpene quinone extracted from a well-known Chinese traditional herb (Salvia miltiorrhiza). We investigated the cytotoxic effects of miltirone toward sensitive and multidrug-resistant acute lymphoblastic leukemia cell lines. Miltirone inhibited multidrug-resistant P-glycoprotein (P-gp)-overexpressing CEM/ADR5000 cells better than drug-sensitive CCRF-CEM wild-type cells, a phenomenon termed collateral sensitivity. Flow cytometric analyses revealed that miltirone induced G2/M arrest and apoptosis. Furthermore, miltirone stimulated reactive oxygen species (ROS) generation and mitochondrial membrane potential (MMP) disruption, which in turn induced DNA damage and activation…

Cyclin-Dependent Kinase Inhibitor p21ATP Binding Cassette Transporter Subfamily BDNA damagePoly ADP ribose polymeraseCellPharmaceutical ScienceApoptosisSalvia miltiorrhizaAnalytical ChemistryDrug DiscoverymedicineHumansCyclin B1CaspaseMembrane Potential MitochondrialPharmacologyCyclin-dependent kinase 1Molecular StructurebiologyOrganic ChemistryPhenanthrenesPrecursor Cell Lymphoblastic Leukemia-LymphomaMolecular biologyG2 Phase Cell Cycle Checkpointsmedicine.anatomical_structureComplementary and alternative medicineApoptosisCell cultureCaspasesbiology.proteinMolecular MedicineReactive Oxygen SpeciesJournal of Natural Products
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The role of reactive oxygen species and subsequent DNA-damage response in the emergence of resistance towards resveratrol in colon cancer models

2014

AbstractIn spite of the novel strategies to treat colon cancer, mortality rates associated with this disease remain consistently high. Tumour recurrence has been linked to the induction of resistance towards chemotherapy that involves cellular events that enable cancer cells to escape cell death. Treatment of colon cancer mainly implicates direct or indirect DNA-damaging agents and increased repair or tolerances towards subsequent lesions contribute to generate resistant populations. Resveratrol (RSV), a potent chemosensitising polyphenol, might share common properties with chemotherapeutic drugs through its indirect DNA-damaging effects reported in vitro. In this study, we investigated how…

Cyclin-Dependent Kinase Inhibitor p21SenescenceCancer ResearchProgrammed cell deathColonDNA damageColorectal cancerImmunologyApoptosisBiologyResveratrolS PhaseHistonesPolyploidyCellular and Molecular Neurosciencechemistry.chemical_compoundCell Line TumorStilbenesmedicineAnimalsHumansCHEK1Cyclin-Dependent Kinase Inhibitor p16Cell Biologymedicine.diseaseAntineoplastic Agents PhytogenicRatsGene Expression Regulation NeoplasticCheckpoint Kinase 2chemistryDrug Resistance NeoplasmResveratrolApoptosisCheckpoint Kinase 1Cancer cellImmunologyCancer researchOriginal ArticleTumor Suppressor Protein p53Reactive Oxygen SpeciesProtein KinasesDNA DamageSignal TransductionCell Death &amp; Disease
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